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Circulation research, ISSN 0009-7330, 06/2014, Volume 115, Issue 1, pp. 176 - 188
Forkhead box protein O1 (FOXO1) | G protein receptor kinase 2 (GRK2) | adrenergic receptors | glycolysis | pyruvate dehydrogenase complex | fluorodeoxyglucose | glutamine | Cardiac & Cardiovascular Systems | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Humans | Heart Failure - physiopathology | Hypertension, Pulmonary - physiopathology | Familial Primary Pulmonary Hypertension | Receptors, Adrenergic, beta - physiology | Neovascularization, Pathologic - etiology | Hypertrophy, Right Ventricular - physiopathology | Animals | Cyclic Nucleotide Phosphodiesterases, Type 5 - physiology | Fibrosis | Glycolysis | Heart Ventricles - anatomy & histology | Signal Transduction - physiology | Heart Ventricles - metabolism | Hypertrophy, Right Ventricular - complications | Heart Ventricles - pathology | Ventricular Function, Right | Index Medicus | microvascular ischemia | Congenital Heart Disease | cardiac magnetic resonance imaging (CMR) | adrenoreceptors | Right ventricular failure (RVF) | fluorodeoxyglucose positron emission tomography (FDG-PET) | aerobic glycolysis | glutaminolysis | pyruvate dehydrogenase (PDH)
Journal Article
Cardiovascular research, ISSN 0008-6363, 10/2017, Volume 113, Issue 12, pp. 1433 - 1440
Noncoding RNAs | MicroRNAs | Pulmonary hypertension | Right ventricular remodelling | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Ventricular Dysfunction, Right - genetics | Humans | Heart Failure - physiopathology | Hypertension, Pulmonary - physiopathology | MicroRNAs - metabolism | Hypertrophy, Right Ventricular - etiology | Arrhythmogenic Right Ventricular Dysplasia - physiopathology | Arrhythmogenic Right Ventricular Dysplasia - genetics | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling - genetics | Ventricular Dysfunction, Right - physiopathology | Heart Failure - etiology | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Hypertrophy, Right Ventricular - genetics | Ventricular Function, Right - genetics | Gene Expression Regulation | Heart Failure - genetics | Hypertension, Pulmonary - genetics | Heart Failure - metabolism | Genetic Markers | Disease Progression | Arrhythmogenic Right Ventricular Dysplasia - complications | Animals | Ventricular Dysfunction, Right - etiology | MicroRNAs - genetics | Hypertension, Pulmonary - complications | Index Medicus
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 12/2020, Volume 319, Issue 6, pp. H1459 - H1473
Mitochondria, Heart - metabolism | Mitochondria, Heart - pathology | Male | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Rats, Mutant Strains | Myocardium - metabolism | Estrogen Receptor alpha - metabolism | Female | Hypertrophy, Right Ventricular - pathology | Ventricular Dysfunction, Right - physiopathology | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Disease Models, Animal | Fibrillar Collagens - metabolism | Signal Transduction | Proto-Oncogene Proteins c-jun - genetics | Kallikreins - genetics | Myocardium - pathology | Rats, Sprague-Dawley | Ventricular Dysfunction, Right - prevention & control | Animals | Estrogen Receptor alpha - genetics | Proto-Oncogene Proteins c-jun - metabolism | Fibrosis | Sex Factors | Hypertrophy, Right Ventricular - prevention & control | Mutation | Kallikreins - metabolism | Ventricular Dysfunction, Right - pathology | Ventricular Function, Right | Heart | Matrix metalloproteinases | Estrogens | Collagens | Estrogen receptors | Overloading | Matrix metalloproteinase | Tissue inhibitor of metalloproteinase 1 | Gene sequencing | Receptors | Rodents | Metalloproteinase | Adaptation | Hypertension | Phenotypes | Peptidase | Pulmonary arteries | Waveforms | Kallikrein | Ribonucleic acid--RNA | Pulmonary artery | Gelatinase B | Peptidases | Collagen | Men | Ventricle | Females | Hypertrophy | Index Medicus | adverse remodeling | right ventricle | estrogen receptor-α | pressure overload
Journal Article
Cardiovascular research, ISSN 0008-6363, 08/2016, Volume 111, Issue 3, pp. 184 - 193
Hypertension | Hypoxia | Pulmonary | Pulmonary heart disease | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Receptor, IGF Type 1 - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Ventricular Dysfunction, Right - genetics | Humans | Heart Failure - physiopathology | Hypertension, Pulmonary - physiopathology | Male | MicroRNAs - metabolism | Hypertension, Pulmonary - prevention & control | Hypoxia - metabolism | Heart Failure - prevention & control | Hypertrophy, Right Ventricular - physiopathology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Ventricular Dysfunction, Right - physiopathology | Lung - metabolism | Receptor, IGF Type 1 - deficiency | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Genetic Predisposition to Disease | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Heart Failure - genetics | Hypertension, Pulmonary - genetics | Heart Failure - metabolism | Hypertension, Pulmonary - metabolism | Hypoxia - complications | Imidazoles - pharmacology | Receptor, IGF Type 1 - genetics | Mice, Knockout | Ventricular Dysfunction, Right - prevention & control | Hypoxia - genetics | Phenotype | Animals | Heart Ventricles - physiopathology | Receptors, Somatomedin - metabolism | Heart Ventricles - metabolism | MicroRNAs - genetics | Pyridines - pharmacology | Ventricular Function, Right | Index Medicus
Journal Article
Journal of molecular medicine (Berlin, Germany), ISSN 0946-2716, 1/2012, Volume 90, Issue 1, pp. 31 - 43
Human Genetics | QTc interval | Myocardial fibrosis | Biomedicine | Pulmonic stenosis | Monophasic action potential duration | Right heart failure | Internal Medicine | Molecular Medicine | Genetics & Heredity | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Cardiology. Vascular system | Heart | Biological and medical sciences | General aspects | Medical sciences | Heart failure, cardiogenic pulmonary edema, cardiac enlargement | Vasodilator Agents - therapeutic use | Glucose Transporter Type 1 - metabolism | Male | Hypertrophy, Right Ventricular - drug therapy | Ranolazine | Glycogen - metabolism | Acetanilides - therapeutic use | Trimetazidine - pharmacology | Ventricular Function, Right - drug effects | Hexokinase - genetics | Fatty Acids - metabolism | Hypertrophy, Right Ventricular - metabolism | Trimetazidine - therapeutic use | Acetanilides - pharmacology | Vasodilator Agents - pharmacology | Oxidation-Reduction | Enzyme Inhibitors - pharmacology | Rats | Piperazines - therapeutic use | Enzyme Inhibitors - therapeutic use | Piperazines - pharmacology | Rats, Sprague-Dawley | Animals | Myocytes, Cardiac - drug effects | Glucose Transporter Type 1 - genetics | Glycolysis | Myocytes, Cardiac - metabolism | Hexokinase - metabolism | Physiological aspects | Biological oxidation (Metabolism) | Research | Heart enlargement | Index Medicus
Journal Article
Naunyn-Schmiedeberg's archives of pharmacology, ISSN 0028-1298, 7/2016, Volume 389, Issue 7, pp. 757 - 767
Calcitonin gene-related peptide | Right ventricular remodeling | eIF3a | Neurosciences | Biomedicine | p27 | Cardiac fibroblasts | Pharmacology/Toxicology | Rutaecarpine | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Hypoxia - drug therapy | Apoptosis - drug effects | Transforming Growth Factor beta1 - metabolism | Hypertrophy, Right Ventricular - etiology | Hypoxia - metabolism | Dose-Response Relationship, Drug | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Hypertrophy, Right Ventricular - physiopathology | Myocardium - metabolism | Ventricular Dysfunction, Right - physiopathology | Ventricular Function, Right - drug effects | Calcitonin Gene-Related Peptide - metabolism | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Disease Models, Animal | Fibroblasts - metabolism | Cells, Cultured | Ventricular Pressure - drug effects | Myocardium - pathology | Eukaryotic Initiation Factor-3 - metabolism | Hypoxia - complications | Fibroblasts - pathology | Ventricular Dysfunction, Right - prevention & control | Animals | Signal Transduction - drug effects | Fibroblasts - drug effects | Indole Alkaloids - pharmacology | Ventricular Dysfunction, Right - etiology | Fibrosis | Hypertrophy, Right Ventricular - prevention & control | Hypoxia - physiopathology | Cell Proliferation - drug effects | Quinazolines - pharmacology | Ventricular Remodeling - drug effects | Heart | Bone morphogenetic proteins | Transforming growth factors | Collagen | Index Medicus
Journal Article
Journal of molecular medicine (Berlin, Germany), ISSN 0946-2716, 11/2013, Volume 91, Issue 11, pp. 1315 - 1327
Human Genetics | Angiogenesis | Hypoxia-inducible factor 1α | Biomedicine | Ischemia | Internal Medicine | Molecular Medicine | Right ventricular failure | Metabolism | Genetics & Heredity | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Reactive Oxygen Species - metabolism | Hypertension, Pulmonary - physiopathology | Rats | Glucose Transporter Type 1 - metabolism | Male | Hypertension, Pulmonary - metabolism | Mitochondria - metabolism | Mitochondria - pathology | Rats, Sprague-Dawley | Neovascularization, Pathologic - complications | Hypertrophy, Right Ventricular - physiopathology | Animals | Heart Ventricles - physiopathology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Glucose - metabolism | Neovascularization, Pathologic - physiopathology | Heart Ventricles - metabolism | Neovascularization, Pathologic - metabolism | Hypertrophy, Right Ventricular - complications | Hypertrophy, Right Ventricular - metabolism | Hypertension, Pulmonary - complications | Causes of | Research | Neovascularization | Heart enlargement | Pulmonary hypertension | Index Medicus
Journal Article
Cardiovascular research, ISSN 0008-6363, 03/2019, Volume 115, Issue 4, pp. 788 - 799
Remodelling | Animal models of human disease | Right ventricle | Pulmonary hypertension | Gene expression and regulation | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Adaptation, Physiological | Species Specificity | Ventricular Dysfunction, Right - genetics | Rats, Inbred F344 | Heart Failure - physiopathology | Transcriptome | Hypertension, Pulmonary - physiopathology | Male | Hypertrophy, Right Ventricular - etiology | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Ventricular Dysfunction, Right - physiopathology | Heart Failure - etiology | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Hypertrophy, Right Ventricular - genetics | Disease Models, Animal | Severity of Illness Index | Signal Transduction | Gene Expression Regulation | Heart Failure - genetics | Hypertension, Pulmonary - genetics | Heart Failure - metabolism | Hypertension, Pulmonary - metabolism | Rats, Sprague-Dawley | Animals | Ventricular Dysfunction, Right - etiology | Exercise Tolerance | Myocytes, Cardiac - metabolism | Neovascularization, Physiologic | Ventricular Function, Right | Hypertension, Pulmonary - complications | Index Medicus | Original
Journal Article
American journal of respiratory cell and molecular biology, ISSN 1044-1549, 03/2019, Volume 60, Issue 3, pp. 346 - 356
Cardiac fibrosis | Nitric oxide synthase | Right ventricle | Pulmonary hypertension | Respiratory System | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Mice, Inbred C57BL | Oxidative Stress - physiology | Male | Hypertension, Pulmonary - metabolism | Ventricular Remodeling - physiology | Fibrosis - metabolism | Pulmonary Artery - metabolism | Animals | Heart Ventricles - metabolism | Nitric Oxide - metabolism | Ventricular Function, Right - physiology | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Disease Models, Animal | Nitric Oxide Synthase Type II - metabolism | Heart | Oxidative stress | Transcription | Experiments | Vasodilation | Oxidants | Fibroblasts | Physiology | Genotypes | Adaptation | Heart failure | Echocardiography | Cytokines | Catheterization | Pulmonary arteries | Cardiomyocytes | Superoxide | Pulmonary artery | Nitric-oxide synthase | Banding | Collagen | Nitric oxide | Fibrosis | Ventricle | Veins & arteries
Journal Article