X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (14979) 14979
Publication (1925) 1925
Book Review (115) 115
Book Chapter (78) 78
Newsletter (23) 23
Book / eBook (19) 19
Dissertation (17) 17
Conference Proceeding (14) 14
Data Set (2) 2
Magazine Article (2) 2
Paper (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
male (8299) 8299
animals (8295) 8295
index medicus (7506) 7506
humans (7497) 7497
hypertrophy (6356) 6356
cardiac & cardiovascular systems (5076) 5076
female (4839) 4839
rats (4388) 4388
myocardium - pathology (3864) 3864
heart (3309) 3309
middle aged (3012) 3012
hypertension (2873) 2873
mice (2697) 2697
peripheral vascular disease (2534) 2534
echocardiography (2530) 2530
heart failure (2424) 2424
adult (2413) 2413
heart-failure (2336) 2336
left-ventricular hypertrophy (2302) 2302
fibrosis (2108) 2108
cardiac-hypertrophy (2098) 2098
myocardium - metabolism (2069) 2069
aged (2001) 2001
disease models, animal (1998) 1998
hypertrophy, left ventricular - pathology (1847) 1847
heart ventricles - pathology (1816) 1816
physiology (1792) 1792
cell biology (1669) 1669
cardiomyopathy (1666) 1666
expression (1600) 1600
cardiomegaly - pathology (1543) 1543
rats, sprague-dawley (1524) 1524
myocytes, cardiac - pathology (1447) 1447
hypertrophy, left ventricular - physiopathology (1422) 1422
blood pressure (1374) 1374
risk factors (1362) 1362
myocytes, cardiac - metabolism (1314) 1314
cardiac hypertrophy (1279) 1279
heart diseases (1275) 1275
cardiology (1256) 1256
hypertension - physiopathology (1220) 1220
apoptosis (1210) 1210
disease (1169) 1169
gene expression (1156) 1156
pharmacology & pharmacy (1151) 1151
research (1129) 1129
rodents (1115) 1115
analysis (1109) 1109
article (1105) 1105
physiological aspects (1100) 1100
rats, wistar (1096) 1096
cells, cultured (1069) 1069
oxidative stress (1069) 1069
mice, inbred c57bl (1058) 1058
time factors (1051) 1051
hypertrophy, left ventricular - etiology (1049) 1049
cardiovascular (1037) 1037
signal transduction (1035) 1035
left ventricular hypertrophy (1019) 1019
myocardial-infarction (1015) 1015
blood-pressure (991) 991
heart enlargement (965) 965
hypertension - complications (961) 961
hypertension - pathology (955) 955
cardiomegaly - metabolism (949) 949
proteins (946) 946
cardiomyocytes (934) 934
gene-expression (930) 930
electrocardiography (907) 907
medicine, research & experimental (905) 905
medicine (889) 889
activation (885) 885
cardiovascular system (867) 867
dysfunction (854) 854
ventricular remodeling (833) 833
mortality (822) 822
biochemistry & molecular biology (818) 818
heart failure - physiopathology (810) 810
cardiomegaly - physiopathology (802) 802
heart failure - pathology (792) 792
blood pressure - drug effects (791) 791
mice, knockout (774) 774
organ size (772) 772
health aspects (765) 765
hypertrophy, left ventricular - diagnostic imaging (765) 765
myocytes, cardiac - drug effects (764) 764
multidisciplinary sciences (750) 750
cardiovascular disease (746) 746
ventricle (742) 742
pathology (741) 741
cardiovascular diseases (740) 740
magnetic resonance imaging (739) 739
research article (736) 736
myocardium (734) 734
adolescent (728) 728
hemodynamics (722) 722
pressure-overload (722) 722
failure (687) 687
heart - physiopathology (675) 675
heart ventricles - physiopathology (673) 673
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (14903) 14903
Russian (62) 62
Spanish (23) 23
German (21) 21
French (16) 16
Chinese (13) 13
Japanese (10) 10
Italian (9) 9
Portuguese (6) 6
Turkish (4) 4
Lithuanian (3) 3
Hungarian (2) 2
Serbian (1) 1
Slovak (1) 1
Ukrainian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Circulation, ISSN 0009-7322, 2013, Volume 128, Issue 18, pp. 2016 - 2025
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 10/2006, Volume 114, Issue 17, pp. 1799 - 1806
Background - Arrhythmogenic right ventricular cardiomyopathy (ARVC) is an inherited disorder that causes sudden death and right ventricular heart failure in... 
Conduction | Exercise | Arrhythmia | Cardiomyopathy | Desmosomes | Adherens junctions | Death, sudden | ARRHYTHMIAS | CARDIAC & CARDIOVASCULAR SYSTEMS | NAXOS-DISEASE | DYSPLASIA | exercise | RECEPTOR | death, sudden | adherens junctions | DELETION | arrhythmia | conduction | PLAKOPHILIN-2 MUTATIONS | EMBRYONIC HEART | WOOLLY HAIR | PERIPHERAL VASCULAR DISEASE | desmosomes | cardiomyopathy | HEMATOLOGY | EXPRESSION | DYSPLASIA/CARDIOMYOPATHY | Arrhythmogenic Right Ventricular Dysplasia - etiology | Ventricular Dysfunction, Right - genetics | Hypertrophy, Right Ventricular - etiology | Arrhythmogenic Right Ventricular Dysplasia - physiopathology | Arrhythmogenic Right Ventricular Dysplasia - genetics | Desmosomes - pathology | gamma Catenin - deficiency | Myocardium - metabolism | Electrocardiography | Tachycardia, Ventricular - genetics | Hypertrophy, Right Ventricular - pathology | Swimming | Arrhythmogenic Right Ventricular Dysplasia - pathology | Hypertrophy, Right Ventricular - genetics | Disease Models, Animal | Myocardium - ultrastructure | Physical Conditioning, Animal - adverse effects | Myocardial Contraction | Ventricular Premature Complexes - etiology | Genetic Predisposition to Disease | Models, Cardiovascular | Gene Expression Regulation | Tachycardia, Ventricular - etiology | gamma Catenin - genetics | Stress, Physiological - physiopathology | Arrhythmogenic Right Ventricular Dysplasia - epidemiology | Mice, Knockout | Phenotype | Animals | Aging - physiology | Ventricular Dysfunction, Right - etiology | Glucose - metabolism | Heterozygote | Mice | Ventricular Premature Complexes - genetics
Journal Article
Circulation, ISSN 0009-7322, 11/2009, Volume 120, Issue 20, pp. 1951 - 1960
Background-The most important determinant of longevity in pulmonary arterial hypertension is right ventricular (RV) function, but in contrast to experimental... 
Heart failure | Angiogenesis | Microcirculation | Pressure | Pulmonary heart disease | pulmonary heart disease | OVERLOAD | heart failure | CARDIAC & CARDIOVASCULAR SYSTEMS | angiogenesis | pressure | microcirculation | PROGNOSTIC VALUE | MYOSIN HEAVY-CHAIN | ANTIOXIDANT RESPONSE ELEMENT | TRANSCRIPTION FACTOR NRF2 | VENTRICULAR HYPERTROPHY | CARDIAC-HYPERTROPHY | DISEASE | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | HYPERTENSION | HEMATOLOGY | Blood Pressure | Vascular Endothelial Growth Factor A - biosynthesis | Heart Failure - physiopathology | Hypertension, Pulmonary - physiopathology | Male | RNA, Messenger - biosynthesis | Hypertrophy, Right Ventricular - physiopathology | Pulmonary Artery - metabolism | Heme Oxygenase (Decyclizing) - biosynthesis | Hypertrophy, Right Ventricular - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - biosynthesis | Hypertrophy, Right Ventricular - metabolism | Disease Models, Animal | Gene Expression Regulation | Rats | Heart Failure - metabolism | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Heart Failure - pathology | Rats, Sprague-Dawley | Animals | Fibrosis | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Ventricular Function, Right | Apoptosis | Usage | Animal models in research | Heart ventricle, Left | Physiological aspects | Diagnosis | Pulmonary hypertension
Journal Article
Journal of Thoracic and Cardiovascular Surgery, The, ISSN 0022-5223, 2017, Volume 154, Issue 6, pp. 2070 - 2079
Abstract Objectives Mechanisms of right ventricular (RV) adaptation to chronic pressure overload remain not well understood. We hypothesized that a lower... 
Cardiothoracic Surgery | fibrosis | adaptive remodeling | right ventricle | pulmonary hypertension | angiogenesis | SURVIVAL | SURGERY | CARDIAC & CARDIOVASCULAR SYSTEMS | PULMONARY ARTERIAL-HYPERTENSION | QUANTIFICATION | EISENMENGER-SYNDROME | HEART-FAILURE | GUIDELINES | FICK METHOD | TRANSITION | RESPIRATORY SYSTEM | CARDIAC-HYPERTROPHY | Adaptation, Physiological | Capillaries - pathology | Neovascularization, Pathologic | Hypertension, Pulmonary - physiopathology | Male | Hypertrophy, Right Ventricular - etiology | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Time Factors | Hypertrophy, Right Ventricular - pathology | Ventricular Dysfunction, Right - physiopathology | Capillaries - metabolism | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Heart Ventricles - pathology | Disease Models, Animal | Animals, Newborn | Capillaries - physiopathology | Animals | Heart Ventricles - physiopathology | Ventricular Dysfunction, Right - etiology | Fibrosis | Heart Ventricles - metabolism | Hemodynamics | Angiogenic Proteins - metabolism | Chronic Disease | Sus scrofa | Ventricular Dysfunction, Right - pathology | Ventricular Function, Right | Hypertension, Pulmonary - complications | Endothelial growth factors | Specific gravity | Analysis | Pulmonary hypertension
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2017, Volume 313, Issue 3, pp. H459 - H468
Arrhythmogenic right ventricular (RV) remodeling has been reported in response to regular training, but it remains unclear how exercise intensity affects the... 
Endurance exercise | Cardiac remodeling | Right ventricle | Training load | Fibrosis | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | endurance exercise | PULMONARY-HYPERTENSION | VOLUME OVERLOAD | training load | HEART-FAILURE | RATS | EUROPEAN ASSOCIATION | DEFORMATION | ATRIAL-FIBRILLATION | right ventricle | CARDIAC ADAPTATION | cardiac remodeling | PERIPHERAL VASCULAR DISEASE | fibrosis | ECHOCARDIOGRAPHY | AMERICAN SOCIETY | Adaptation, Physiological | Rats, Wistar | Ventricular Function, Left | Male | Hypertrophy, Right Ventricular - etiology | Echocardiography, Doppler, Color | Physical Endurance | Heart Ventricles - diagnostic imaging | Arrhythmias, Cardiac - physiopathology | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Time Factors | Hypertrophy, Right Ventricular - diagnostic imaging | Hypertrophy, Right Ventricular - pathology | Ventricular Dysfunction, Right - physiopathology | Heart Ventricles - pathology | Disease Models, Animal | Models, Cardiovascular | Myocardium - pathology | Arrhythmias, Cardiac - etiology | Cardiomegaly, Exercise-Induced | Animals | Heart Ventricles - physiopathology | Ventricular Dysfunction, Right - etiology | Running | Hemodynamics | Ventricular Dysfunction, Right - pathology | Ventricular Function, Right | Ventricular Dysfunction, Right - diagnostic imaging | Health aspects | Exercise | Heart ventricle, Right
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 11/2009, Volume 297, Issue 5, pp. 1606 - 1616
Pulmonary arterial hypertension (PAH) is a chronic lung disease that leads to right ventricular (RV) hypertrophy (RVH), remodeling, and failure. We tested... 
PROGENITOR CELLS | heart failure | remodeling | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | hypertrophy | PYRROLIZIDINE ALKALOIDS | monocrotaline | VASCULAR-DISEASE | MARROW-DERIVED CELLS | ARTERIAL-HYPERTENSION | PERIPHERAL VASCULAR DISEASE | GENE-TRANSFER | STROMAL CELLS | pulmonary arterial hypertension | NEOINTIMAL FORMATION | ENDOTHELIAL GROWTH-FACTOR | TENASCIN-C | Cell Proliferation | Rats, Wistar | Monocrotaline | Cardiac Output | Hypertension, Pulmonary - physiopathology | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Hypertension, Pulmonary - chemically induced | Recovery of Function | Transplantation, Homologous | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Time Factors | Myocardium - metabolism | Inflammation Mediators - metabolism | Female | Hypertrophy, Right Ventricular - pathology | Ventricular Dysfunction, Right - physiopathology | Cell Differentiation | Lung - metabolism | Ventricular Dysfunction, Right - chemically induced | Extracellular Matrix Proteins - metabolism | Disease Models, Animal | Myocardial Contraction | Heart Rate | Lung - pathology | Pulmonary Alveoli - pathology | Extracellular Matrix Proteins - genetics | Cells, Cultured | Hypertension, Pulmonary - surgery | Mesenchymal Stromal Cells - metabolism | Rats | Myocardium - pathology | Lung - physiopathology | Stroke Volume | Ventricular Dysfunction, Right - prevention & control | Arterioles - pathology | Animals | Hypertrophy, Right Ventricular - chemically induced | Hypertrophy, Right Ventricular - prevention & control | Ventricular Pressure | Lung - blood supply | Hypertension, Pulmonary - pathology | Mesenchymal Stem Cell Transplantation | Pulmonary Artery - pathology | Ventricular Dysfunction, Right - pathology | Ventricular Function, Right | Physiology, Pathological | Care and treatment | Stem cells | Development and progression | Transplantation | Research | Properties | Pulmonary hypertension | Methods | Heart ventricle, Right | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 12/2012, Volume 126, Issue 24, pp. 2859 - 2869
Background-The cause and consequences of impaired adrenergic signaling in right ventricular failure/hypertrophy (RVH) are poorly understood. We hypothesized... 
Hypertension | β-adrenergic receptor kinase | Right ventricular hypertrophy | β1-adrenoreceptor | Pulmonary | SU5416 | Dopamine receptors | RECEPTOR KINASE | SYSTEM | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | dopamine receptors | beta-adrenergic receptor kinase | hypertension, pulmonary | PERIPHERAL VASCULAR DISEASE | right ventricular hypertrophy | beta 1-adrenoreceptor | CONGESTIVE-HEART-FAILURE | Oligonucleotide Array Sequence Analysis | Humans | Myocardial Contraction - physiology | Myocardial Contraction - drug effects | Male | G-Protein-Coupled Receptor Kinase 2 - metabolism | Hypertrophy, Right Ventricular - drug therapy | Receptors, Dopamine D1 - metabolism | Female | Hypertrophy, Right Ventricular - pathology | Hypertension, Pulmonary - drug therapy | Receptors, Adrenergic, beta - metabolism | Hypertrophy, Right Ventricular - metabolism | Xanthenes - pharmacology | Myocytes, Cardiac - cytology | Cells, Cultured | Dopamine - pharmacology | Rats | Hypertension, Pulmonary - metabolism | Cardiotonic Agents - pharmacology | Down-Regulation - drug effects | Rats, Sprague-Dawley | Dobutamine - pharmacology | Down-Regulation - physiology | Animals | Myocytes, Cardiac - drug effects | Receptors, Adrenergic, beta - genetics | Signal Transduction - drug effects | Signal Transduction - physiology | Hypertension, Pulmonary - pathology | Receptors, Dopamine D1 - genetics | Care and treatment | Heart enlargement | Physiological aspects | Dosage and administration | Research | Adrenergic beta blockers | Pulmonary hypertension
Journal Article
Circulation Research, ISSN 0009-7330, 01/2009, Volume 104, Issue 2, pp. 236 - 244
Inflammatory cytokine interleukin (IL)-6 is elevated in the serum and lungs of patients with pulmonary artery hypertension (PAH). Several animal models of PAH... 
Pulmonary artery hypertension | Proliferation | Interleukin-6 | CARDIAC & CARDIOVASCULAR SYSTEMS | pulmonary artery hypertension | proliferation | UP-REGULATES EXPRESSION | interleukin-6 | VASCULAR STRUCTURE | ARTERIAL-HYPERTENSION | PLEXIFORM LESIONS | ENDOTHELIAL-CELLS | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | INFLAMMATORY CYTOKINES | HEMATOLOGY | NEOINTIMAL FORMATION | TRANSGENIC MICE | Blood Pressure | Up-Regulation | Cell Proliferation | Microtubule-Associated Proteins - metabolism | Arterioles - immunology | Hyperplasia | Repressor Proteins | Hypertension, Pulmonary - physiopathology | Muscle, Smooth, Vascular - immunology | Vascular Endothelial Growth Factor A - metabolism | Muscle, Smooth, Vascular - physiopathology | Hypertrophy, Right Ventricular - physiopathology | Time Factors | Hypertrophy, Right Ventricular - pathology | Interleukin-6 - metabolism | Proto-Oncogene Proteins - metabolism | Vascular Resistance | Inhibitor of Apoptosis Proteins | Interleukin-6 - genetics | Mice, Inbred C57BL | Hypertension, Pulmonary - immunology | Mice, Transgenic | Hypoxia - complications | Pulmonary Artery - physiopathology | Proto-Oncogene Proteins c-myc - metabolism | Endothelial Cells - immunology | Arterioles - pathology | Muscle, Smooth, Vascular - pathology | Animals | Hypertrophy, Right Ventricular - immunology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Ventricular Pressure | Proto-Oncogene Proteins c-bcl-2 | Endothelial Cells - pathology | Chronic Disease | Hypertension, Pulmonary - pathology | Transforming Growth Factor beta - metabolism | Pulmonary Artery - pathology | Ventricular Function, Right | Apoptosis | Mitogen-Activated Protein Kinases - metabolism | Pulmonary Artery - immunology
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 02/2014, Volume 189, Issue 3, pp. 314 - 324
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 07/2016, Volume 311, Issue 1, pp. H85 - H95
Pulmonary arterial hypertension (PAH), the most serious chronic disorder of the pulmonary circulation, is characterized by pulmonary vasoconstriction and... 
Pulmonary artery banding | Right ventricle remodeling | Pulmonary hypertension | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | pulmonary hypertension | right ventricle remodeling | VOLUME | PULMONARY ARTERIAL CAPACITANCE | PROGNOSTIC VALUE | DIASTOLIC DYSFUNCTION | pulmonary artery banding | EJECTION FRACTION | CARDIAC-HYPERTROPHY | CALCIUM | PERIPHERAL VASCULAR DISEASE | ANIMAL-MODEL | HYPERTENSION | RIGHT-HEART-FAILURE | Rats, Wistar | Ventricular Function, Left | Hypertension, Pulmonary - physiopathology | Male | Hypertrophy, Right Ventricular - etiology | Pulmonary Artery - surgery | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Arterial Pressure | Time Factors | Hypertrophy, Right Ventricular - pathology | Ventricular Dysfunction, Right - physiopathology | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Calcium Signaling | Disease Models, Animal | Constriction | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Myocytes, Cardiac - pathology | Animals | Ventricular Dysfunction, Right - etiology | Fibrosis | Myocytes, Cardiac - metabolism | Ventricular Dysfunction, Right - pathology | Ventricular Function, Right | Adaptation, Psychological | Hypertension, Pulmonary - complications | Measurement | Physiological aspects | Mechanical properties | Models | Ventricular function | Heart ventricle, Right
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 1/2010, Volume 88, Issue 1, pp. 47 - 60
Journal Article
Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 12/2013, Volume 8, Issue 12, pp. 2106 - 2116
Journal Article