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Circulation, ISSN 0009-7322, 2013, Volume 128, Issue 18, pp. 2016 - 2025
Journal Article
Circulation, ISSN 0009-7322, 11/2009, Volume 120, Issue 20, pp. 1951 - 1960
Background-The most important determinant of longevity in pulmonary arterial hypertension is right ventricular (RV) function, but in contrast to experimental... 
Heart failure | Angiogenesis | Microcirculation | Pressure | Pulmonary heart disease | pulmonary heart disease | OVERLOAD | heart failure | CARDIAC & CARDIOVASCULAR SYSTEMS | angiogenesis | pressure | microcirculation | PROGNOSTIC VALUE | MYOSIN HEAVY-CHAIN | ANTIOXIDANT RESPONSE ELEMENT | TRANSCRIPTION FACTOR NRF2 | VENTRICULAR HYPERTROPHY | CARDIAC-HYPERTROPHY | DISEASE | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | HYPERTENSION | HEMATOLOGY | Blood Pressure | Vascular Endothelial Growth Factor A - biosynthesis | Heart Failure - physiopathology | Hypertension, Pulmonary - physiopathology | Male | RNA, Messenger - biosynthesis | Hypertrophy, Right Ventricular - physiopathology | Pulmonary Artery - metabolism | Heme Oxygenase (Decyclizing) - biosynthesis | Hypertrophy, Right Ventricular - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - biosynthesis | Hypertrophy, Right Ventricular - metabolism | Disease Models, Animal | Gene Expression Regulation | Rats | Heart Failure - metabolism | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Heart Failure - pathology | Rats, Sprague-Dawley | Animals | Fibrosis | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Ventricular Function, Right | Apoptosis | Usage | Animal models in research | Heart ventricle, Left | Physiological aspects | Diagnosis | Pulmonary hypertension | Index Medicus | Abridged Index Medicus
Journal Article
Chest, ISSN 0012-3692, 2013, Volume 143, Issue 6, pp. 1699 - 1708
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2017, Volume 313, Issue 3, pp. H459 - H468
Arrhythmogenic right ventricular (RV) remodeling has been reported in response to regular training, but it remains unclear how exercise intensity affects the... 
Endurance exercise | Cardiac remodeling | Right ventricle | Training load | Fibrosis | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | endurance exercise | PULMONARY-HYPERTENSION | VOLUME OVERLOAD | training load | HEART-FAILURE | RATS | EUROPEAN ASSOCIATION | DEFORMATION | ATRIAL-FIBRILLATION | right ventricle | CARDIAC ADAPTATION | cardiac remodeling | PERIPHERAL VASCULAR DISEASE | fibrosis | ECHOCARDIOGRAPHY | AMERICAN SOCIETY | Adaptation, Physiological | Rats, Wistar | Ventricular Function, Left | Male | Hypertrophy, Right Ventricular - etiology | Echocardiography, Doppler, Color | Physical Endurance | Heart Ventricles - diagnostic imaging | Arrhythmias, Cardiac - physiopathology | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Time Factors | Hypertrophy, Right Ventricular - diagnostic imaging | Hypertrophy, Right Ventricular - pathology | Ventricular Dysfunction, Right - physiopathology | Heart Ventricles - pathology | Disease Models, Animal | Models, Cardiovascular | Myocardium - pathology | Arrhythmias, Cardiac - etiology | Cardiomegaly, Exercise-Induced | Animals | Heart Ventricles - physiopathology | Ventricular Dysfunction, Right - etiology | Running | Hemodynamics | Ventricular Dysfunction, Right - pathology | Ventricular Function, Right | Ventricular Dysfunction, Right - diagnostic imaging | Health aspects | Exercise | Heart ventricle, Right | Heart | Deformation | Volume | Experiments | Pressure | Index Medicus
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 11/2013, Volume 91, Issue 11, pp. 1315 - 1327
Journal Article
Journal of Cardiovascular Electrophysiology, ISSN 1045-3873, 01/2016, Volume 27, Issue 1, pp. 80 - 87
NICM VT Ablation AimTo determine whether ventricular tachycardia (VT) recurrences in arrhythmogenic RV cardiomyopathy (ARVC) and nonischemic cardiomyopathy... 
substrate map | arrhythmogenic right ventricular cardiomyopathy | catheter ablation | nonischemic cardiomyopathy | ventricular tachycardia | CARDIAC & CARDIOVASCULAR SYSTEMS | INTEGRATION | VENTRICULAR-TACHYCARDIA | SUBSTRATE | SCAR | OUTCOMES | Recurrence | Cicatrix - etiology | Ventricular Function, Left | Humans | Middle Aged | Male | Hypertrophy, Right Ventricular - etiology | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Time Factors | Cicatrix - physiopathology | Adult | Female | Retrospective Studies | Catheter Ablation - adverse effects | Heart Ventricles - pathology | Heart Conduction System - surgery | Heart Conduction System - pathology | Europe | Hypertrophy, Left Ventricular - etiology | Risk Factors | Tachycardia, Ventricular - etiology | Treatment Outcome | Disease Progression | Stroke Volume | Arrhythmogenic Right Ventricular Dysplasia - complications | Tachycardia, Ventricular - physiopathology | Tachycardia, Ventricular - surgery | Heart Conduction System - physiopathology | Heart Ventricles - physiopathology | Tachycardia, Ventricular - diagnosis | Heart Ventricles - surgery | Aged | Hypertrophy, Left Ventricular - physiopathology | Electrophysiologic Techniques, Cardiac | Ventricular Function, Right | Medical research | Ablation (Surgery) | Tachycardia | Heart beat | Analysis | Medicine, Experimental | Development and progression | Cardiomyopathy | Heart attacks | Maps | Fatalities | Ventricle | Remodeling | Ablation | Patients | Stretching | Substrates | Delay | Ejection | Index Medicus
Journal Article
CIRCULATION-CARDIOVASCULAR IMAGING, ISSN 1941-9651, 06/2017, Volume 10, Issue 6
Background-Right ventricular (RV) end-systolic dimensions provide information on both size and function. We investigated whether an internally scaled index of... 
SURVIVAL | heart failure | risk assessment | CARDIAC & CARDIOVASCULAR SYSTEMS | CARDIOLOGY | pulmonary hypertension | echocardiography | ventricular remodeling | FAILURE | RIGHT-VENTRICULAR DYSFUNCTION | right ventricular dysfunction | STRAIN | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | patient outcome assessment | Hypertrophy, Right Ventricular - therapy | Multivariate Analysis | Predictive Value of Tests | Prospective Studies | Humans | Middle Aged | Heart Failure - physiopathology | Hypertension, Pulmonary - diagnostic imaging | Hypertension, Pulmonary - physiopathology | Male | Hypertrophy, Right Ventricular - mortality | Case-Control Studies | Hypertension, Pulmonary - therapy | Heart Failure - therapy | Cardiac Catheterization | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Time Factors | Hypertrophy, Right Ventricular - diagnostic imaging | Adult | Female | Registries | Ventricular Dysfunction, Right - physiopathology | Heart Failure - diagnostic imaging | Ventricular Dysfunction, Right - therapy | Hypertension, Pulmonary - mortality | Reproducibility of Results | Echocardiography | Kaplan-Meier Estimate | Proportional Hazards Models | Heart Transplantation | Treatment Outcome | Hospitalization | California | Chi-Square Distribution | Stroke Volume | Ventricular Dysfunction, Right - mortality | Ventricular Function, Right | Ventricular Dysfunction, Right - diagnostic imaging | Index Medicus | strain | remodeling heart failure | right ventricular failure | pulmonary arterial hypertension | outcome
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 04/2018, Volume 314, Issue 3, pp. H627 - H642
Chronic thromboembolic pulmonary hypertension (CTEPH) develops in 4% of patients after pulmonary embolism and is accompanied by an impaired exercise tolerance,... 
Right ventricular remodeling | Chronic thromboembolic pulmonary hypertension | Animal model | Exercise | Vascular remodelling | EMBOLISM | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | right ventricular remodeling | exercise | vascular remodeling | AIR EMBOLIZATION | chronic thromboembolic pulmonary hypertension | TREADMILL EXERCISE | PIGLET MODEL | MORPHOMETRY | ARTERIAL-HYPERTENSION | NITRIC-OXIDE | RESISTANCE | PERIPHERAL VASCULAR DISEASE | RIGHT-VENTRICULAR RESERVE | animal model | ENDOTHELIAL GROWTH-FACTOR | Vascular Remodeling | Hypertension, Pulmonary - physiopathology | Male | Hypertrophy, Right Ventricular - etiology | Hypertrophy, Right Ventricular - physiopathology | Pulmonary Artery - metabolism | Ventricular Remodeling | Female | Ventricular Dysfunction, Right - physiopathology | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Physical Exertion | Disease Models, Animal | Pulmonary Embolism - complications | Endothelium, Vascular - physiopathology | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Disease Progression | Animals | Endothelium, Vascular - metabolism | Ventricular Dysfunction, Right - etiology | Chronic Disease | Hypertension, Pulmonary - etiology | Sus scrofa | Ventricular Function, Right | Pulmonary Embolism - metabolism | Pulmonary Embolism - physiopathology | Physiological aspects | Health aspects | Pulmonary hypertension | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 07/2016, Volume 311, Issue 1, pp. H85 - H95
Pulmonary arterial hypertension (PAH), the most serious chronic disorder of the pulmonary circulation, is characterized by pulmonary vasoconstriction and... 
Pulmonary artery banding | Right ventricle remodeling | Pulmonary hypertension | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | pulmonary hypertension | right ventricle remodeling | VOLUME | PULMONARY ARTERIAL CAPACITANCE | PROGNOSTIC VALUE | DIASTOLIC DYSFUNCTION | pulmonary artery banding | EJECTION FRACTION | CARDIAC-HYPERTROPHY | CALCIUM | PERIPHERAL VASCULAR DISEASE | ANIMAL-MODEL | HYPERTENSION | RIGHT-HEART-FAILURE | Rats, Wistar | Ventricular Function, Left | Hypertension, Pulmonary - physiopathology | Male | Hypertrophy, Right Ventricular - etiology | Pulmonary Artery - surgery | Hypertrophy, Right Ventricular - physiopathology | Ventricular Remodeling | Arterial Pressure | Time Factors | Hypertrophy, Right Ventricular - pathology | Ventricular Dysfunction, Right - physiopathology | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Calcium Signaling | Disease Models, Animal | Constriction | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Myocytes, Cardiac - pathology | Animals | Ventricular Dysfunction, Right - etiology | Fibrosis | Myocytes, Cardiac - metabolism | Ventricular Dysfunction, Right - pathology | Ventricular Function, Right | Adaptation, Psychological | Hypertension, Pulmonary - complications | Measurement | Physiological aspects | Mechanical properties | Models | Ventricular function | Heart ventricle, Right | Hypoxia | Pulmonary arteries | Medical prognosis | Index Medicus
Journal Article
Cardiovascular Research, ISSN 0008-6363, 08/2016, Volume 111, Issue 3, pp. 184 - 193
Pulmonary hypertension is a progressive disease with poor prognosis, characterized by pathological inward remodelling and loss of patency of the lung... 
Hypertension | Hypoxia | Pulmonary | Pulmonary heart disease | CELLS | LUNG | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | PULMONARY ARTERIAL-HYPERTENSION | RECEPTOR | BREAST-CANCER | HEART | GENE | EXPRESSION | MICRORNA | Receptor, IGF Type 1 - metabolism | Receptor, IGF Type 1 - antagonists & inhibitors | Ventricular Dysfunction, Right - genetics | Humans | Heart Failure - physiopathology | Hypertension, Pulmonary - physiopathology | Male | MicroRNAs - metabolism | Hypertension, Pulmonary - prevention & control | Hypoxia - metabolism | Heart Failure - prevention & control | Hypertrophy, Right Ventricular - physiopathology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Ventricular Dysfunction, Right - physiopathology | Lung - metabolism | Receptor, IGF Type 1 - deficiency | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Genetic Predisposition to Disease | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Heart Failure - genetics | Hypertension, Pulmonary - genetics | Heart Failure - metabolism | Hypertension, Pulmonary - metabolism | Hypoxia - complications | Imidazoles - pharmacology | Receptor, IGF Type 1 - genetics | Mice, Knockout | Ventricular Dysfunction, Right - prevention & control | Hypoxia - genetics | Phenotype | Animals | Heart Ventricles - physiopathology | Receptors, Somatomedin - metabolism | Heart Ventricles - metabolism | MicroRNAs - genetics | Pyridines - pharmacology | Ventricular Function, Right | Index Medicus
Journal Article
Journal of Thoracic and Cardiovascular Surgery, The, ISSN 0022-5223, 2012, Volume 143, Issue 6, pp. 1299 - 1304
Objectives In correction of tetralogy of Fallot (TOF), surgical strategies to minimize right ventricular outflow tract (RVOT) enlargement have recently been... 
Cardiothoracic Surgery | SURGERY | RESTRICTIVE PHYSIOLOGY | CAPACITY | CARDIAC & CARDIOVASCULAR SYSTEMS | DETERMINANTS | DIASTOLIC FUNCTION | PERFORMANCE | TRANSANNULAR PATCH | EXERCISE | CHILDREN | RESPIRATORY SYSTEM | PULMONARY REGURGITATION | MAGNETIC-RESONANCE | Multivariate Analysis | Pulmonary Valve Insufficiency - physiopathology | Humans | Pulmonary Valve Stenosis - surgery | Pulmonary Valve Stenosis - physiopathology | Male | Cardiac Surgical Procedures - adverse effects | Hypertrophy, Right Ventricular - etiology | Ventricular Outflow Obstruction - diagnosis | Ventricular Outflow Obstruction - etiology | Pulmonary Valve Insufficiency - etiology | Young Adult | Hypertrophy, Right Ventricular - physiopathology | Tetralogy of Fallot - diagnosis | Adult | Female | Ventricular Dysfunction, Right - physiopathology | Retrospective Studies | Child | Republic of Korea | Echocardiography | Risk Assessment | Risk Factors | Linear Models | Treatment Outcome | Pulmonary Valve Stenosis - complications | Chi-Square Distribution | Ventricular Dysfunction, Right - diagnosis | Ventricular Outflow Obstruction - prevention & control | Stroke Volume | Ventricular Dysfunction, Right - prevention & control | Magnetic Resonance Imaging | Ventricular Outflow Obstruction - physiopathology | Tetralogy of Fallot - surgery | Ventricular Dysfunction, Right - etiology | Adolescent | Hypertrophy, Right Ventricular - prevention & control | Pulmonary Valve Insufficiency - surgery | Ventricular Pressure | Tetralogy of Fallot - physiopathology | Ventricular Function, Right | Tetralogy of Fallot - complications | Tetralogy of Fallot | Valves | Care and treatment | Congenital heart disease | Cardiology | Personal video recorders | Index Medicus | Abridged Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 01/2009, Volume 104, Issue 2, pp. 236 - 244
Inflammatory cytokine interleukin (IL)-6 is elevated in the serum and lungs of patients with pulmonary artery hypertension (PAH). Several animal models of PAH... 
Pulmonary artery hypertension | Proliferation | Interleukin-6 | CARDIAC & CARDIOVASCULAR SYSTEMS | pulmonary artery hypertension | proliferation | UP-REGULATES EXPRESSION | interleukin-6 | VASCULAR STRUCTURE | ARTERIAL-HYPERTENSION | PLEXIFORM LESIONS | ENDOTHELIAL-CELLS | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | INFLAMMATORY CYTOKINES | HEMATOLOGY | NEOINTIMAL FORMATION | TRANSGENIC MICE | Blood Pressure | Up-Regulation | Cell Proliferation | Microtubule-Associated Proteins - metabolism | Arterioles - immunology | Hyperplasia | Repressor Proteins | Hypertension, Pulmonary - physiopathology | Muscle, Smooth, Vascular - immunology | Vascular Endothelial Growth Factor A - metabolism | Muscle, Smooth, Vascular - physiopathology | Hypertrophy, Right Ventricular - physiopathology | Time Factors | Hypertrophy, Right Ventricular - pathology | Interleukin-6 - metabolism | Proto-Oncogene Proteins - metabolism | Vascular Resistance | Inhibitor of Apoptosis Proteins | Interleukin-6 - genetics | Mice, Inbred C57BL | Hypertension, Pulmonary - immunology | Mice, Transgenic | Hypoxia - complications | Pulmonary Artery - physiopathology | Proto-Oncogene Proteins c-myc - metabolism | Endothelial Cells - immunology | Arterioles - pathology | Muscle, Smooth, Vascular - pathology | Animals | Hypertrophy, Right Ventricular - immunology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Ventricular Pressure | Proto-Oncogene Proteins c-bcl-2 | Endothelial Cells - pathology | Chronic Disease | Hypertension, Pulmonary - pathology | Transforming Growth Factor beta - metabolism | Pulmonary Artery - pathology | Ventricular Function, Right | Apoptosis | Mitogen-Activated Protein Kinases - metabolism | Pulmonary Artery - immunology | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 08/2013, Volume 305, Issue 3, pp. H354 - H364
Right ventricular (RV) failure due to chronically abnormal loading is a main determinant of outcome in pulmonary hypertension (PH) and congenital heart... 
PRESSURE-OVERLOAD | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | RAT | PULMONARY ARTERIAL-HYPERTENSION | contractility | pulmonary hypertension | VOLUME OVERLOAD | monocrotaline | FAILURE | pressure-volume analysis | PASSIVE STIFFNESS | EJECTION FRACTION | CONGENITAL HEART-DISEASE | CARDIAC-HYPERTROPHY | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | right ventricular failure | Adaptation, Physiological | Hypertension, Pulmonary - diagnosis | Rats, Wistar | Ventricular Dysfunction, Right - genetics | Monocrotaline | Heart Failure - physiopathology | Hypertension, Pulmonary - physiopathology | Male | Hypertrophy, Right Ventricular - etiology | Pulmonary Artery - surgery | Heart Ventricles - diagnostic imaging | Hypertension, Pulmonary - chemically induced | Hypertrophy, Right Ventricular - physiopathology | Time Factors | Ultrasonography | Ventricular Dysfunction, Right - physiopathology | Heart Failure - diagnosis | Heart Failure - etiology | Physical Exertion | Disease Models, Animal | Myocardial Contraction | Constriction | Arteriovenous Shunt, Surgical | Models, Cardiovascular | Gene Expression Regulation | Rats | Familial Primary Pulmonary Hypertension | Heart Failure - genetics | Hypertension, Pulmonary - genetics | Pulmonary Artery - physiopathology | Ventricular Dysfunction, Right - diagnosis | Stroke Volume | Animals | Heart Ventricles - physiopathology | Ventricular Dysfunction, Right - etiology | Ventricular Pressure | Hemodynamics | Hypertension, Pulmonary - etiology | Ventricular Function, Right | Physiological aspects | Congenital heart disease | Health aspects | Pulmonary hypertension | Heart ventricle, Right | Hypertension | Cardiovascular disease | Exercise | Pulmonary arteries | Rodents | Index Medicus
Journal Article