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Journal of Clinical Investigation, ISSN 0021-9738, 03/2017, Volume 127, Issue 3, pp. 1061 - 1074
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2017, Volume 127, Issue 1, pp. 293 - 305
Prader-Willi syndrome (PWS) is caused by a loss of paternally expressed genes in an imprinted region of chromosome 15q. Among the canonical PWS phenotypes are... 
MEDICINE, RESEARCH & EXPERIMENTAL | PLASMA GHRELIN | BODY-WEIGHT | HORMONE-RELEASING-HORMONE | HYPERPHAGIA | FOOD-INTAKE | PROPROTEIN CONVERTASE-1 | ONSET OBESITY | KNOCKOUT MICE | CIRCULATING GHRELIN LEVELS | CHILDREN | Diabetes Mellitus - pathology | Neurons - pathology | Proinsulin - metabolism | Diabetes Mellitus - genetics | Humans | Male | Obesity - genetics | RNA, Small Nucleolar - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Hypogonadism - metabolism | Prader-Willi Syndrome - genetics | Female | Neurons - metabolism | Induced Pluripotent Stem Cells - metabolism | Hypogonadism - pathology | Induced Pluripotent Stem Cells - pathology | Basic Helix-Loop-Helix Transcription Factors - genetics | Protein Precursors - genetics | Diabetes Mellitus - metabolism | Proinsulin - genetics | Prader-Willi Syndrome - metabolism | Growth Hormone-Releasing Hormone - genetics | Hyperphagia - metabolism | Proprotein Convertase 1 - deficiency | RNA, Small Nucleolar - genetics | Mice, Knockout | Obesity - metabolism | Obesity - pathology | Protein Precursors - metabolism | Hyperphagia - genetics | Hyperphagia - pathology | Hypogonadism - genetics | Growth Hormone-Releasing Hormone - metabolism | Animals | Prader-Willi Syndrome - pathology | Prader-Willi syndrome | Gene expression | Neurons | Analysis | Risk factors | Proteins | Enzymes | Plasma | Obesity | Rodents | Fibroblasts | Patients | Binding sites
Journal Article
FEBS Letters, ISSN 0014-5793, 05/2014, Volume 588, Issue 10, pp. 1935 - 1941
Androgen reduces fat mass, although the underlying mechanisms are unknown. Here, we examined the effect of testosterone on heat production and mitochondrial... 
Testosterone | Mitochondrial biogenesis | Skeletal muscle | Androgen receptor deficient mice | ANDROGEN RECEPTOR | ACTIVATED RECEPTOR-GAMMA | ADIPOCYTES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INSULIN SENSITIVITY | CELL BIOLOGY | OBESITY | BIOPHYSICS | MASS | MEN | LATE-ONSET | HYPOGONADISM | EXPRESSION | Myoglobin - metabolism | Troponin - genetics | Mitochondria, Muscle - metabolism | Myoglobin - genetics | Body Weight - drug effects | Male | Muscle, Skeletal - metabolism | Receptors, Androgen - deficiency | Cytochromes c - genetics | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Weight Loss - drug effects | Muscle, Skeletal - cytology | Testosterone - pharmacology | Nuclear Respiratory Factor 1 - genetics | Testosterone - metabolism | DNA, Mitochondrial - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Nuclear Respiratory Factor 1 - metabolism | Mitochondria, Muscle - genetics | Troponin - metabolism | Cell Line | DNA, Mitochondrial - metabolism | Cytochromes c - metabolism | Mice, Inbred C57BL | Transcription Factors - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Knockout | Transcription Factors - metabolism | Eating - drug effects | Animals | Receptors, Androgen - genetics | Androgens - pharmacology | Muscle Fibers, Skeletal - cytology | Mice | Androgens - metabolism | Blood Glucose - metabolism | Energy Metabolism - drug effects | Muscles | Biosynthesis | Body weight | Weight loss
Journal Article
American Journal of Physiology - Endocrinology And Metabolism, ISSN 0193-1849, 01/2011, Volume 300, Issue 1, pp. 202 - 210
Neurokinin B (NKB) and its cognate receptor neurokinin 3 (NK3R) play a critical role in reproduction. NKB and NK3R are coexpressed with dynorphin (Dyn) and... 
Estradiol | Hypothalamus | PHYSIOLOGY | GONADOTROPIN-SECRETION | ARCUATE NUCLEUS | MORPHOLOGIC EVIDENCE | MESSENGER-RNA | LUTEINIZING-HORMONE SECRETION | DYNORPHIN-A | ENDOCRINOLOGY & METABOLISM | MEDIAN-EMINENCE | GENE-EXPRESSION | NEURONS | HYPOGONADOTROPIC HYPOGONADISM | hypothalamus | estradiol | Rats, Wistar | Arcuate Nucleus of Hypothalamus - cytology | Peptide Fragments - pharmacology | Substance P - pharmacology | Neurokinin B - agonists | Neurokinin B - genetics | RNA, Messenger - metabolism | Kisspeptins | Arcuate Nucleus of Hypothalamus - drug effects | Receptors, Neurokinin-3 - metabolism | Female | Neurons - metabolism | Prosencephalon - metabolism | Neurons - drug effects | Substance P - analogs & derivatives | Receptors, Neurokinin-3 - genetics | Gonadotropin-Releasing Hormone - secretion | Estradiol - metabolism | Luteinizing Hormone - blood | Arcuate Nucleus of Hypothalamus - metabolism | Gene Expression Regulation | Proto-Oncogene Proteins c-fos - metabolism | Receptors, Neurokinin-3 - agonists | Rats | Organ Specificity | Neurokinin B - metabolism | Proteins - genetics | Feedback, Physiological | Animals | Proteins - metabolism | Signal Transduction - drug effects | Prosencephalon - cytology | Proto-Oncogene Proteins c-fos - genetics | Estrous Cycle - metabolism | Physiological aspects | Neurohormones | Genetic aspects | Research | Gonadotropin releasing hormone
Journal Article
European Journal of Endocrinology, ISSN 0804-4643, 06/2017, Volume 176, Issue 6, pp. R283 - R308
Journal Article
PLoS Biology, ISSN 1544-9173, 2014, Volume 12, Issue 9, p. e1001952
Characterization of the genetic defects causing gonadotropic deficiency has made a major contribution to elucidation of the fundamental role of Kisspeptins and... 
V-ATPASE | CATALYTIC SUBUNIT | GDP/GTP EXCHANGE PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | BIOLOGY | HYPOMYELINATING LEUKODYSTROPHY | POL III | MUTATIONS | GTPASE-ACTIVATING PROTEIN | HYPOGONADOTROPIC HYPOGONADISM | NEUROKININ B | PUBERTY | Hypothyroidism - pathology | Hypothyroidism - metabolism | Testis - metabolism | Humans | Nerve Tissue Proteins - deficiency | Diabetes Mellitus, Type 1 - metabolism | Male | Luteinizing Hormone - metabolism | Polyneuropathies - metabolism | Intellectual Disability - metabolism | Young Adult | Infertility, Male - genetics | Base Sequence | Neurons - metabolism | Hypoglycemia - genetics | Gonadotropin-Releasing Hormone - genetics | Diabetes Mellitus, Type 1 - pathology | Intellectual Disability - pathology | Hypothalamus - pathology | Hypoglycemia - metabolism | Infertility, Male - pathology | Testis - growth & development | Mice, Knockout | Hypothalamus - metabolism | Testis - pathology | Adaptor Proteins, Signal Transducing - deficiency | Adolescent | Gonadotropin-Releasing Hormone - metabolism | Polyneuropathies - genetics | Mice | Neurons - pathology | Sequence Deletion | Hypothalamus - growth & development | Pituitary Gland - metabolism | Molecular Sequence Data | Hypoglycemia - pathology | Intellectual Disability - genetics | Hypothyroidism - genetics | Follicle Stimulating Hormone - genetics | Sexual Maturation | Follicle Stimulating Hormone - metabolism | Luteinizing Hormone - genetics | Pituitary Gland - pathology | Diabetes Mellitus, Type 1 - genetics | Pituitary Gland - growth & development | Nerve Tissue Proteins - genetics | Syndrome | Haploinsufficiency | Homozygote | Animals | Polyneuropathies - pathology | Adaptor Proteins, Signal Transducing - genetics | Infertility, Male - metabolism | Life Sciences | Human health and pathology | Neurons and Cognition | Neurobiology | Pituitary gland | Neurons | Infertility | Rodents
Journal Article
Endocrine Reviews, ISSN 0163-769X, 06/2004, Volume 25, Issue 3, pp. 389 - 425
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 04/2012, Volume 97, Issue 4, pp. E694 - E699
Journal Article
Journal Article
Cell, ISSN 0092-8674, 2006, Volume 125, Issue 2, pp. 247 - 260
Journal Article
Journal Article