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Medicine, ISSN 0025-7974, 10/2018, Volume 97, Issue 40, p. e12507
Journal Article
Medicine (United States), ISSN 0025-7974, 12/2017, Volume 96, Issue 51, p. e9470
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 03/2017, Volume 28, Issue 3, pp. 903 - 914
Phosphate (Pi) homeostasis is regulated by renal, intestinal, and endocrine mechanisms through which Pi intake stimulates parathyroid hormone (PTH) and... 
VITAMIN-D | ACUTE RENAL ADAPTATION | IN-VITRO | SIGNALING AXIS | GROWTH-FACTOR 23 | UROLOGY & NEPHROLOGY | DIETARY PHOSPHATE | MOUSE KIDNEY | PTH SECRETION | OPOSSUM KIDNEY-CELLS | PROXIMAL TUBULE | parathyroid hormone | phosphate uptake | intestine | Basic Research | phosphate-sensing
Journal Article
The Journal of Pathology, ISSN 0022-3417, 03/2015, Volume 235, Issue 4, pp. 539 - 545
Journal Article
Clinical and Experimental Nephrology, ISSN 1342-1751, 10/2017, Volume 21, Issue 5, pp. 926 - 931
After kidney transplantation, fibroblast growth factor-23 (FGF-23) normally returns to baseline within 1 year whereas hyperparathyroidism persists in most... 
Hypercalcemia | Phosphaturia | Nephrology | Medicine & Public Health | Vitamin D | Renal transplantation | Urology | MORTALITY | SECONDARY HYPERPARATHYROIDISM | RENAL-TRANSPLANTATION | RISK-FACTOR | DISEASE | CALCIUM | GROWTH-FACTOR 23 | UROLOGY & NEPHROLOGY | PARATHYROID-HORMONE | NEPHROCALCINOSIS | PROGRESSION | Hyperparathyroidism - physiopathology | Hypophosphatemia - etiology | Hyperparathyroidism - diagnosis | Prospective Studies | Humans | Middle Aged | Male | Hypophosphatemia, Familial - diagnosis | Allografts | Time Factors | Phosphates - urine | Hyperparathyroidism - blood | Phosphates - blood | Adult | Female | Kidney - physiopathology | Hyperparathyroidism - etiology | Hypophosphatemia, Familial - etiology | Hypophosphatemia - blood | Glomerular Filtration Rate | Hypophosphatemia - diagnosis | Risk Factors | Graft Survival | Treatment Outcome | Renal Elimination | Hypophosphatemia - physiopathology | Hypophosphatemia, Familial - physiopathology | Hypophosphatemia, Familial - urine | Kidney Transplantation - adverse effects | Renal Dialysis | Phosphates | Kidneys | Albumin | Organ transplant recipients | Calcifediol | Transplantation | Censorship | Risk factors | Chronic kidney failure | Parathyroid hormone | Analysis | Physiological aspects | Fibroblast growth factors | Alfacalcidol | Hyperparathyroidism | Calcium (urinary) | Fibroblast growth factor | Excretion | Transplants & implants | Fibroblast growth factor 23 | Syngeneic grafts | Metabolism | Survival | Glomerular filtration rate | Calcium phosphates | Calcium (blood) | Phosphate | Cardiovascular diseases | Parathyroid | Kidney transplantation
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, p. e0159804
Contribution of salt wasting and volume depletion to the pathogenesis of hypercalciuria and hyperphosphaturia is poorly understood. Pendrin/NCC double KO... 
HYPERPROSTAGLANDIN-E-SYNDROME | COLLECTING DUCT | VOLUME DEPLETION | NA+-CL-COTRANSPORTER | MULTIDISCIPLINARY SCIENCES | PARATHYROID-HORMONE | KIDNEY | REPORTING ANIMAL RESEARCH | THICK ASCENDING LIMB | BARTTER-SYNDROME | DOUBLE KNOCKOUT | Hypophosphatemia, Familial - etiology | Calcium - metabolism | Hypercalciuria - etiology | Hypercalciuria - pathology | Hypophosphatemia, Familial - metabolism | Male | Nephrons - pathology | Solute Carrier Family 12, Member 1 - deficiency | Kidney Function Tests | Mice, Knockout | Dinoprostone - metabolism | Phenotype | Phosphates - metabolism | Animals | Hypophosphatemia, Familial - pathology | Diet | Hypercalciuria - metabolism | Anion Transport Proteins - deficiency | Mice | Nephrons - metabolism | Disease Models, Animal | Phosphates | Excretion | Calcium, Dietary | Analysis | Synthetic prostaglandins E | Physiological aspects | Research | Cytochrome | Salts | Calcium | Veterans | Pathogenesis | Prostaglandin E2 | Isotypes | Sodium phosphate | Hypercalciuria | Metabolites | Phosphate | Rodents | Inhibition | Prostaglandin E | Calcium (urinary) | Departments | Kidneys | Internal medicine | Ducts | Phosphate transporter | Cytochrome P450 | Reabsorption | Metabolism | Calcium phosphates | Medicine | Depletion | Sodium | Isoforms | Renal failure | Indomethacin | Mutation | Potassium | Transporter | Alkalosis
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 2005, Volume 16, Issue 6, pp. 1645 - 1653
X-linked hypophosphatemia (XLH), a disorder characterized by hypophosphatemia, impaired skeletal mineralization, and aberrant regulation of 1, 25(OH)(2)D(3),... 
Hypophosphatemia, Familial - etiology | Animals | Extracellular Matrix Proteins - physiology | Female | Hypophosphatemia, Familial - physiopathology | Male | Models, Animal | Mice | Phosphoproteins - physiology | Glycoproteins - physiology
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 1931-857X, 06/2010, Volume 298, Issue 6, pp. 1341 - 1350
Journal Article
Giornale italiano di nefrologia : organo ufficiale della Societa italiana di nefrologia, 01/2017, Volume 34, Issue 1
Residual renal function (RRF) and phosphaturia had not stimulated particular interest in studies regarding patients on hemodialysis. In the current year the... 
Hypophosphatemia, Familial - etiology | Kidney Failure, Chronic - physiopathology | Kidney Failure, Chronic - therapy | Humans | Kidney Failure, Chronic - complications | Female | Male | Renal Dialysis - statistics & numerical data | Aged | Hypophosphatemia, Familial - therapy
Journal Article
Kidney International, ISSN 0085-2538, 07/2011, Volume 80, Issue 1, pp. 13 - 15
Phosphate is a key constituent of several important molecules, and hyperphospatemia has been associated with increased cardiovascular mortality. The kidney... 
UROLOGY & NEPHROLOGY | SERUM | Hypophosphatemia, Familial - etiology | Protein-Serine-Threonine Kinases - deficiency | Female | Animals | Bone Density - physiology | Humans
Journal Article