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Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2009, Volume 106, Issue 32, pp. 13505 - 13510
Diabetes is associated with poor outcomes following acute vascular occlusive events. This results in part from a failure to form adequate compensatory... 
Diabetic angiopathies | Wound healing | Diabetes complications | Diabetes mellitus | Fibroblasts | Hypoxia | Mice | Diabetes | Type 2 diabetes mellitus | Transactivation | HIF-1 | Hyperglycemia | Ischemia | Deferoxamine | MOBILIZATION | STABILIZATION | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | VESSEL FORMATION | MELLITUS | ischemia | HYDROXYLASES | hyperglycemia | deferoxamine | METABOLISM | COMPLICATIONS | INDUCIBLE FACTOR-1 | ENDOTHELIAL GROWTH-FACTOR | Diabetes Mellitus - pathology | Reactive Oxygen Species - metabolism | Humans | Transcriptional Activation - drug effects | Hyperglycemia - complications | Vascular Endothelial Growth Factor A - metabolism | Neovascularization, Pathologic - complications | Neovascularization, Pathologic - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Protein Binding - drug effects | Hyperglycemia - pathology | Diabetes Mellitus, Experimental - complications | Diabetes Complications - pathology | Diabetes Mellitus, Experimental - metabolism | Wound Healing - drug effects | Disease Models, Animal | Deferoxamine - pharmacology | Pyruvaldehyde - pharmacology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Diabetes Complications - metabolism | Cells, Cultured | Diabetes Mellitus - metabolism | Glucose - pharmacology | Hypoxia - complications | Up-Regulation - drug effects | p300-CBP Transcription Factors - metabolism | Animals | Diabetes Mellitus, Experimental - pathology | Research | Neovascularization | Vascular endothelial growth factor | Biological Sciences
Journal Article
Liver international, ISSN 1478-3223, 2011, Volume 31, Issue 2, pp. 146 - 162
.... Therefore, knowledge about the pathophysiology of CLDs and its complications is of uttermost importance... 
chronic liver diseases | non‐alcoholic steatohepatitis | portal hypertension | angiogenesis | hepatocellular carcinoma | Angiogenesis | Chronic liver diseases | Hepatocellular carcinoma | Non-alcoholic steatohepatitis | Portal hypertension | ADVANCED HEPATOCELLULAR-CARCINOMA | HYPOXIA-INDUCIBLE FACTOR | PORTAL HYPERTENSIVE-RATS | ENDOTHELIAL-GROWTH-FACTOR | NONALCOHOLIC STEATOHEPATITIS | FACTOR RECEPTOR | PHASE-II TRIAL | HEPATIC STELLATE CELLS | TYROSINE KINASE INHIBITOR | HYPERDYNAMIC SPLANCHNIC CIRCULATION | GASTROENTEROLOGY & HEPATOLOGY | non-alcoholic steatohepatitis | Microvessels - physiopathology | Hypertension, Portal - physiopathology | Cadherins - metabolism | Hypertension, Portal - complications | Humans | Fatty Liver - complications | Liver Neoplasms - complications | Vascular Endothelial Growth Factor A - metabolism | Hypoxia-Inducible Factor 1 - metabolism | Integrins - metabolism | Neovascularization, Pathologic - etiology | Fibroblast Growth Factors - metabolism | Carcinoma, Hepatocellular - drug therapy | Receptors, Vascular Endothelial Growth Factor - metabolism | Carcinoma, Hepatocellular - complications | Liver Neoplasms - physiopathology | Non-alcoholic Fatty Liver Disease | Angiogenesis Inhibitors - therapeutic use | Neovascularization, Pathologic - physiopathology | Angiostatins - metabolism | Angiopoietins - metabolism | Carcinoma, Hepatocellular - physiopathology | Endostatins - metabolism | Liver Cirrhosis - complications | Fatty Liver - physiopathology | Liver Neoplasms - drug therapy | Disease Progression | Platelet-Derived Growth Factor - metabolism | Models, Biological | Signal Transduction - physiology | Liver Cirrhosis - physiopathology | Chronic Disease | Thrombospondins - metabolism | Hypertension | Complications and side effects | Liver cancer | Development and progression | Mortality
Journal Article
Journal Article