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Pediatrics, ISSN 0031-4005, 06/2016, Volume 137, Issue 6, pp. e20160191 - e20160191
Journal Article
Neuropharmacology, ISSN 0028-3908, 12/2015, Volume 99, pp. 38 - 50
Previous studies have demonstrated that the early suppression of HIF-1α after hypoxia–ischemia (HI) injury provides neuroprotection. Vitexin (5, 7,... 
Hypoxia-inducible factor | Hypoxia–ischemia | Vitexin | Neonatal rat | Hypoxia-ischemia | Capillary Permeability - physiology | Neurons - pathology | Blood-Brain Barrier - physiopathology | Capillary Permeability - drug effects | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Vascular Endothelial Growth Factor A - metabolism | Hypoxia-Ischemia, Brain - pathology | Apigenin - chemistry | Neuroprotective Agents - pharmacology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Neurons - physiology | Atrophy - drug therapy | Cell Death - drug effects | Drug Evaluation, Preclinical | Neurons - drug effects | Disease Models, Animal | Animals, Newborn | Atrophy - physiopathology | Neuroprotective Agents - chemistry | Maze Learning - physiology | Brain - physiopathology | Brain Edema - pathology | Hypoxia-Ischemia, Brain - drug therapy | Treatment Outcome | Random Allocation | Maze Learning - drug effects | Blood-Brain Barrier - drug effects | Rats, Sprague-Dawley | Brain - drug effects | Blood-Brain Barrier - pathology | Animals | Brain Edema - physiopathology | Cell Death - physiology | Brain - pathology | Brain Edema - drug therapy | Apigenin - pharmacology | Hypoxia-Ischemia, Brain - physiopathology | Medicinal plants | Immunohistochemistry | Brain | Ischemia | Immunoglobulin G | Physiological aspects | Permeability | Vascular endothelial growth factor | Injuries
Journal Article
Pediatrics, ISSN 0031-4005, 08/2009, Volume 124, Issue 2, pp. e218 - e226
OBJECTIVE: The purpose of this study was to evaluate the efficacy and safety of erythropoietin in neonatal hypoxic-ischemic encephalopathy (HIE), by using a... 
Neonates | Asphyxia | Erythropoietin | Hypoxic-ischemic encephalopathy | NEURONAL APOPTOSIS | BRAIN-INJURY | FETAL SHEEP | neonates | CEREBROSPINAL-FLUID | asphyxia | STROKE | BIRTH-WEIGHT INFANTS | NEONATAL ENCEPHALOPATHY | PHARMACOKINETICS | hypoxic-ischemic encephalopathy | PEDIATRICS | CEREBRAL-ISCHEMIA | DOSE RECOMBINANT ERYTHROPOIETIN | erythropoietin | Brain Damage, Chronic - diagnosis | Prospective Studies | Follow-Up Studies | Humans | Developmental Disabilities - prevention & control | Erythropoietin - adverse effects | Infant | Male | Recombinant Proteins | Asphyxia Neonatorum - drug therapy | Dose-Response Relationship, Drug | Intensive Care Units, Neonatal | Asphyxia Neonatorum - diagnosis | China | Injections, Subcutaneous | Female | Developmental Disabilities - diagnosis | Psychomotor Disorders - prevention & control | Infant, Newborn | Disability Evaluation | Erythropoietin - administration & dosage | Drug Administration Schedule | Hypoxia-Ischemia, Brain - drug therapy | Hypoxia-Ischemia, Brain - diagnosis | Psychomotor Disorders - diagnosis | Neurologic Examination - drug effects | Infusions, Intravenous | Brain Damage, Chronic - prevention & control | Babies | Pediatrics | Developmental psychology | Clinical trials | Glycoproteins | Drug therapy | Drug dosages | Clinical outcomes | Effectiveness studies | Developmental Disabilities | Hypoxia-Ischemia | Brain | Neonatal | MEDICIN OCH HÄLSOVETENSKAP | administration & dosage | Asphyxia Neonatorum | MEDICAL AND HEALTH SCIENCES | Recombinant | Drug | Intensive Care Units | drug therapy | Intravenous | Subcutaneous | Infusions | diagnosis | drug effects | prevention & control | Chronic | Injections | Newborn | Neurologic Examination | adverse effects | Dose-Response Relationship | Brain Damage | Psychomotor Disorders
Journal Article
Lancet Neurology, The, ISSN 1474-4422, 2015, Volume 14, Issue 5, pp. 469 - 477
Journal Article
Frontiers in Neuroendocrinology, ISSN 0091-3022, 2008, Volume 30, Issue 1, pp. 65 - 91
Abstract DHEA and DHEAS are steroids synthesized in human adrenals, but their function is unclear. In addition to adrenal synthesis, evidence also indicates... 
Endocrinology & Metabolism | DHEA | Neuroprotection | Dehydroepiandrosterone | DHEAS | Schizophrenia | Depression | Cortisol | Neurogenesis | Apoptosis | Dementia | POSTTRAUMATIC-STRESS-DISORDER | LINKED-IMMUNOSORBENT-ASSAY | TREATED SCHIZOPHRENIA-PATIENTS | NECROSIS-FACTOR-ALPHA | DWELLING OLDER MEN | NEUROSCIENCES | ACTIVATED-RECEPTOR-ALPHA | NEUROACTIVE STEROIDS | ENDOCRINOLOGY & METABOLISM | CENTRAL-NERVOUS-SYSTEM | CORONARY-ARTERY-DISEASE | METHYL-D-ASPARTATE | Dehydroepiandrosterone - pharmacology | Dementia - drug therapy | Humans | Middle Aged | Dehydroepiandrosterone Sulfate - pharmacology | Hypoxia-Ischemia, Brain - drug therapy | Dehydroepiandrosterone - therapeutic use | Male | Dehydroepiandrosterone - physiology | Antioxidants - pharmacology | Brain - drug effects | Brain - metabolism | Depression - drug therapy | Animals | Neuroprotective Agents - pharmacology | Stress Disorders, Post-Traumatic - drug therapy | Aging | Adult | Female | Aged | Schizophrenia - drug therapy | Hydrogen peroxide | Peptides | Streptozocin | Depression, Mental | Leukemia | ACTH | Oxides | Nerve growth factor | Sodium nitroferricyanide | Hormones | Sulfates | Post-traumatic stress disorder | Health aspects | Protein kinases | Glutathione | Protein binding | Epinephrine | Corticosteroids | Endothelium | Methyl aspartate | Androgens | Nitric oxide | GABA | Acetylcholine | Hydroxylases | Mitogens | Literature Review | Psychiatric Disorders | Neuroendocrinology | Glucocorticoids | cortisol | neuroprotection | neurogenesis | apoptosis | dementia | depression | schizophrenia
Journal Article
Neurology, ISSN 0028-3878, 2014, Volume 82, Issue 16, pp. 1425 - 1433
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 12/2014, Volume 34, Issue 49, pp. 16467 - 16481
Journal Article
Journal of Comparative Neurology, ISSN 0021-9967, 07/2009, Volume 515, Issue 1, pp. 116 - 124
Mesencephalic astrocyte‐derived neurotrophic factor (MANF), also known as arginine‐rich, mutated in early stage of tumors (ARMET), is a secreted protein that... 
stroke | armet | ischemia | Ischemia | Armet | Stroke | MIDDLE CEREBRAL-ARTERY | ARGININE-RICH PROTEIN | DOPAMINERGIC-NEURONS | TIME WINDOW | NEUROSCIENCES | UNFOLDED PROTEIN RESPONSE | ZOOLOGY | BONE MORPHOGENETIC PROTEIN-7 | OSTEOGENIC PROTEIN-1 | EXPRESSION | OCCLUSION | Necrosis - drug therapy | Motor Activity - physiology | Nerve Tissue Proteins - therapeutic use | Recovery of Function - drug effects | Apoptosis - drug effects | Humans | Nerve Degeneration - physiopathology | Brain Infarction - metabolism | Motor Activity - drug effects | Male | Cytoprotection - physiology | Nerve Degeneration - metabolism | Brain - metabolism | Tetrazolium Salts | Necrosis - physiopathology | Cytoprotection - drug effects | Recovery of Function - physiology | Behavior, Animal - drug effects | Brain Infarction - drug therapy | Hypoxia-Ischemia, Brain - metabolism | Disease Models, Animal | Brain - physiopathology | Hypoxia-Ischemia, Brain - drug therapy | Rats | Behavior, Animal - physiology | Necrosis - metabolism | Treatment Outcome | Brain Infarction - physiopathology | Recombinant Proteins - pharmacology | Rats, Sprague-Dawley | Brain - drug effects | Animals | Nerve Growth Factors | Staining and Labeling | Indicators and Reagents | Apoptosis - physiology | Nerve Degeneration - drug therapy | Hypoxia-Ischemia, Brain - physiopathology
Journal Article