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1. Full Text Hypophosphatasia mimicking hypoxic–ischaemic encephalopathy: early recognition and management
by Picton, Aled and Nadar, Ruchi and Pelivan, Alexandra and Garikapati, Vidya and Saraff, Vrinda
Archives of disease in childhood, ISSN 0003-9888, 02/2021, Volume 106, Issue 2, pp. 189 - 191
Enzyme Replacement Therapy | Diagnosis, Differential | Hypoxia-Ischemia, Brain | Humans | Male | Hypophosphatasia - complications | Muscle Hypotonia - etiology | Hypophosphatasia - diagnosis | Respiratory Distress Syndrome, Newborn - etiology | Seizures - etiology | Hypophosphatasia - drug therapy | Infant, Newborn | Hypophosphatasia - diagnostic imaging | Ventilators | Alkaline phosphatase | Enzymes | Pediatrics | Funding | Phosphatase | Metabolism | Hypophosphatasia | Mimicry | Fractures | X rays | Mineralization | Encephalopathy | Hypoxia | Bones | Mutation | Diagnosis | Acidosis | Seizures | Hypothermia | Index Medicus | Abridged Index Medicus
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2. Full Text Neutrophil dynamics, plasticity and function in acute neurodegeneration following neonatal hypoxia–ischemia
by Mülling, Kerstin and Fischer, Alexa Josephine and Siakaeva, Elena and Richter, Mathis and Bordbari, Sharareh and Spyra, Ilona and Köster, Christian and Hermann, Dirk and Gunzer, Matthias and Felderhoff-Müser, Ursula and Bendix, Ivo and Jablonska, Jadwiga and Herz, Josephine
Brain, behavior, and immunity, ISSN 0889-1591, 02/2021, Volume 92, pp. 232 - 242
Brain ischemia | Ly6G | Neonatal hypoxia–ischemia | Neutrophils | Infants (Newborn) | Neonatology | Brain | Ischemia | Injuries | Index Medicus
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3. Full Text White matter injury in the neonatal hypoxic‐ischemic brain and potential therapies targeting microglia
by Shao, Rongjiao and Sun, Dawei and Hu, Yue and Cui, Derong
Journal of neuroscience research, ISSN 0360-4012, 04/2021, Volume 99, Issue 4, pp. 991 - 1008
hypoxia‐ischemia | perinatal brain injury | microglia | sexual dimorphism | white matter injury | therapeutic strategies | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neonates | Neuroprotection | Brain | Oxidative stress | Cognitive ability | Disabilities | Risk factors | Heterogeneity | Ischemia | Etiology | Oligodendrocytes | Paralysis | Damage | Injuries | Phenotypes | Stroke | Health risks | Risk analysis | Substantia alba | Morbidity | Microglia | Neurological diseases | Myelination | Excitotoxicity | Hypoxia | Brain injury | Hypothermia | Index Medicus
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4. Full Text A Review of Plant Extracts and Plant-Derived Natural Compounds in the Prevention/Treatment of Neonatal Hypoxic-Ischemic Brain Injury
by Mohsenpour, Hadi and Pesce, Mirko and Patruno, Antonia and Bahrami, Azam and Pour, Pardis Mohammadi and Farzaei, Mohammad Hosein
International journal of molecular sciences, ISSN 1661-6596, 01/2021, Volume 22, Issue 2, p. 833
Biochemistry & Molecular Biology | Physical Sciences | Chemistry | Life Sciences & Biomedicine | Chemistry, Multidisciplinary | Science & Technology | Neonates | Neuroprotection | Animal models | Traumatic brain injury | Infants | Literature reviews | Head injuries | Ischemia | Energy | Newborn babies | Encephalopathy | Tumor necrosis factor-TNF | Growth factors | Neurological disorders | Edema | Pathophysiology | Births | Inflammation | Plants | Metabolism | Investigations | Plant extracts | Injury prevention | Molecular modelling | Neurological complications | Brain damage | Hypoxia | Brain injury | Apoptosis | Hypothermia | Index Medicus | neonatal hypoxic-ischemic brain damage | newborns | neonates | hypoxic-ischemic encephalopathy | perinatal hypoxia-ischemia | birth asphyxia
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5. Full Text Brain-derived neurotrophic factor and its related enzymes and receptors play important roles after hypoxic-ischemic brain damage
by Xiong, Liu-Lin and Chen, Jie and Du, Ruo-Lan and Liu, Jia and Chen, Yan-Jun and Hawwas, Mohammed Al and Zhou, Xin-Fu and Wang, Ting-Hua and Yang, Si-Jin and Bai, Xue
Neural regeneration research, ISSN 1673-5374, 08/2021, Volume 16, Issue 8, pp. 1453 - 1459
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Brain damage | Development and progression | Models | Brain-derived neurotrophic factor | Health aspects | Patient outcomes | brain injury; brain-derived neurotrophic factor; enzyme; hypoxia-ischemia; receptors; recovery; repair
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6. Full Text L-Cysteine Provides Neuroprotection of Hypoxia-Ischemia Injury in Neonatal Mice via a PI3K/Akt-Dependent Mechanism
by Li, Tingting and Li, Jiangbing and Li, Tong and Zhao, Yijing and Ke, Hongfei and Wang, Shuanglian and Liu, Dexiang and Wang, Zhen
Drug design, development and therapy, ISSN 1177-8881, 2021, Volume 15, pp. 517 - 529
Neuroimaging | Neonates | Neuroprotection | Cysteine | Phosphorylation | Traumatic brain injury | Staining | Homology | AKT protein | Kinases | Neuroprotective agents | Phosphatase | Carotid arteries | Proteins | Hydrogen sulfide | Signal transduction | Cell activation | Ischemia | Chromosome 10 | Acute effects | Chromosomes | Tensin | Enzymes | Statistical analysis | Chromosome deletion | Exposure | Metabolism | 1-Phosphatidylinositol 3-kinase | International organizations | Signaling | Transmission electron microscopy | Microscopy | Brain damage | Hypoxia | Software | Laboratory animals | Immunofluorescence | Brain injury | PTEN protein | Apoptosis | Long-term effects | Index Medicus | h2s | hypoxia-ischemia | akt | neuroinflammation
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7. Full Text Pathophysiology of Cerebral Hyperperfusion in Term Neonates With Hypoxic-Ischemic Encephalopathy: A Systematic Review for Future Research
by Kleuskens, Dianne G and Gonçalves Costa, Filipe and Annink, Kim V and van den Hoogen, Agnes and Alderliesten, Thomas and Groenendaal, Floris and Benders, Manon J N and Dudink, Jeroen
Frontiers in pediatrics, ISSN 2296-2360, 2021, Volume 9, pp. 631258 - 631258
Infants (Newborn) | Encephalopathy | cerebral hyperperfusion | hyperemia | cerebral vasodilation | neonatal encephalopathy | perinatal hypoxia-ischemia | hypoxic-ischemic encephalopathy (HIE)
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8. Full Text Activation of MC1R with BMS-470539 attenuates neuroinflammation via cAMP/PKA/Nurr1 pathway after neonatal hypoxic-ischemic brain injury in rats
by Yu, Shufeng and Doycheva, Desislava Met and Gamdzyk, Marcin and Yang, Yijun and Lenahan, Cameron and Li, Gaigai and Li, Dujuan and Lian, Lifei and Tang, Jiping and Lu, Jun and Zhang, John H
Journal of neuroinflammation, ISSN 1742-2094, 01/2021, Volume 18, Issue 1, pp. 26 - 26
Neurosciences | Immunology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Medicine, Experimental | Brain | Medical research | Infants | Inflammation | Injuries | Protein kinase A | Neonates | Neuroprotection | Traumatic brain injury | Experiments | Carotid arteries | Interleukin 6 | Atrophy | Signal transduction | Ischemia | Encephalopathy | Rodents | Surgery | a-Melanocyte-stimulating hormone | Carotid artery | CRISPR | Cytokines | Cyclic AMP | Melanocortin | Tumor necrosis factor-α | IL-1β | Nuclear receptors | Microglia | Neurological diseases | Nurr1 protein | Animal behavior | Hypoxia | Laboratory animals | Immunofluorescence | Brain injury | Veins & arteries | Index Medicus | Nurr1 | Neuroinflammation | Melanocortin-1 receptor | Neonatal hypoxia-ischemia | BMS-470539 | Microglial polarization
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