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Acta neuropathologica, ISSN 1432-0533, 10/2007, Volume 114, Issue 6, pp. 619 - 631
Neuroimaging studies indicate reduced volumes of certain gray matter regions in survivors of prematurity with periventricular leukomalacia (PVL). We... 
Pathology | Perinatal hypoxia–ischemia | Basal ganglia | Medicine & Public Health | Neurodevelopmental disability | Brainstem | Thalamus | Perinatal panencephalopathy | White matter gliosis | Perinatal hypoxia-ischemia | Neurosciences | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neurons - pathology | Prevalence | Humans | Nerve Degeneration - physiopathology | Cerebellar Nuclei - growth & development | Infant | Male | Gliosis - physiopathology | Leukomalacia, Periventricular - pathology | Thalamus - pathology | Brain - growth & development | Gliosis - pathology | Cerebellar Nuclei - physiopathology | Leukomalacia, Periventricular - physiopathology | Gliosis - epidemiology | Premature Birth - physiopathology | Nerve Fibers, Myelinated - pathology | Female | Globus Pallidus - physiopathology | Brain Damage, Chronic - pathology | Globus Pallidus - pathology | Infant, Newborn | Leukomalacia, Periventricular - epidemiology | Globus Pallidus - growth & development | Comorbidity | Brain - physiopathology | Cerebellar Nuclei - pathology | Nerve Degeneration - epidemiology | Brain Damage, Chronic - physiopathology | Nerve Degeneration - pathology | Thalamus - growth & development | Thalamus - physiopathology | Brain - pathology | Brain Damage, Chronic - epidemiology | Index Medicus | Original Paper
Journal Article
Molecular brain, ISSN 1756-6606, 2015, Volume 8, Issue 1, pp. 11 - 11
Background: Our previous study found that suppression of TRPM7 reduced neuronal death in adult rat ischemic brain injury... 
Neuroprotection | Carvacrol | TRPM7 | Neonatal hypoxic-ischemic brain injury | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neuroprotective Agents - therapeutic use | Neurons - pathology | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Brain Infarction - pathology | Glucose | Hippocampus - drug effects | Hypoxia-Ischemia, Brain - pathology | Neuroprotection - drug effects | Monoterpenes - pharmacology | Brain Infarction - complications | Neuroprotective Agents - pharmacology | HEK293 Cells | Behavior, Animal - drug effects | Neurons - metabolism | Phosphorylation - drug effects | Neurons - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Monoterpenes - therapeutic use | Animals, Newborn | Oxygen | Brain - physiopathology | Hypoxia-Ischemia, Brain - drug therapy | bcl-2-Associated X Protein - metabolism | Rats | Hippocampus - pathology | Brain Infarction - physiopathology | Hypoxia-Ischemia, Brain - complications | Brain - drug effects | Animals | TRPM Cation Channels - antagonists & inhibitors | Brain - pathology | Mice | TRPM Cation Channels - metabolism | Hippocampus - physiopathology | Hypoxia-Ischemia, Brain - physiopathology | Ion Channel Gating - drug effects | Infants (Newborn) | Stroke (Disease) | Brain | Ischemia | Neurons | Research | Instrument industry | Injuries | Index Medicus
Journal Article
Journal Article
Nature neuroscience, ISSN 1097-6256, 08/2011, Volume 14, Issue 8, pp. 1009 - 1016
Permanent damage to white matter tracts, comprising axons and myelinating oligodendrocytes, is an important component of brain injuries of the newborn that cause cerebral palsy and cognitive... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Humans | Ki-67 Antigen - metabolism | Male | Brain Injuries - metabolism | Wnt Proteins - metabolism | Oligodendroglia - drug effects | Hypoxia-Ischemia, Brain - therapy | Infant, Newborn | Organ Culture Techniques | Disease Models, Animal | Animals, Newborn | Basic Helix-Loop-Helix Transcription Factors - genetics | Mice, Transgenic | Oligodendrocyte Transcription Factor 2 | Brain Injuries - therapy | Multiple Sclerosis - therapy | Myelin Proteins - genetics | beta Catenin - metabolism | Heterocyclic Compounds, 3-Ring - therapeutic use | Spinal Cord - physiology | Axin Protein | Mice | Myelin Sheath - ultrastructure | beta-Galactosidase - genetics | Cerebellum - ultrastructure | Myelin Proteins - metabolism | Corpus Callosum - metabolism | Spinal Cord - drug effects | Heterocyclic Compounds, 3-Ring - pharmacology | Myelin Proteins - therapeutic use | Cerebellum - drug effects | Myelin Sheath - drug effects | Hypoxia-Ischemia, Brain - pathology | Cerebral Cortex - cytology | Cytoskeletal Proteins - deficiency | Dose-Response Relationship, Drug | Oligodendroglia - physiology | Wnt Proteins - genetics | beta-Galactosidase - metabolism | Adult | Cytoskeletal Proteins - metabolism | Female | Demyelinating Diseases - chemically induced | Demyelinating Diseases - pathology | Neurons - drug effects | Cell Differentiation - physiology | Hypoxia-Ischemia, Brain - metabolism | Microscopy, Electron, Transmission | Myelin Sheath - pathology | Gene Expression Regulation - genetics | Cerebellum - metabolism | Cells, Cultured | Gene Expression Regulation - physiology | Corpus Callosum - drug effects | Nerve Tissue Proteins - genetics | beta Catenin - genetics | Multiple Sclerosis - complications | Nerve Tissue Proteins - metabolism | Animals | Cell Differentiation - drug effects | Multiple Sclerosis - pathology | Stem Cells - drug effects | Brain Injuries - etiology | Lysophosphatidylcholines - toxicity | Postmortem Changes | Infants (Newborn) | Brain | Care and treatment | Physiological aspects | Research | Binding proteins | Health aspects | Injuries | Index Medicus
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 02/2014, Volume 34, Issue 2, pp. 285 - 293
...–coupled receptor (GPR) 91. We postulate that succinate/GPR91 enhances post-HI vascularization and reduces infarct size in a model of newborn HI brain injury... 
animals newborn | succinic acid | GPR91 protein mouse | hypoxia-ischemia brain | intracellular signaling peptides and proteins | Peripheral Vascular Disease | Life Sciences & Biomedicine | Hematology | Cardiovascular System & Cardiology | Science & Technology | Receptors, G-Protein-Coupled - metabolism | Succinic Acid - administration & dosage | Astrocytes - pathology | Cerebral Cortex - pathology | Injections, Intraventricular | Neuroprotective Agents - metabolism | Hypoxia-Ischemia, Brain - etiology | Cerebral Infarction - physiopathology | Neuroprotective Agents - pharmacology | Time Factors | Neurons - metabolism | Cerebral Cortex - drug effects | Disease Models, Animal | Animals, Newborn | Endothelial Cells - metabolism | Mice, Knockout | Dinoprostone - metabolism | Signal Transduction - drug effects | Succinic Acid - metabolism | Cerebral Infarction - etiology | Mice | Receptors, G-Protein-Coupled - genetics | Endothelial Cells - pathology | Angiogenic Proteins - metabolism | Astrocytes - metabolism | Cerebral Infarction - drug therapy | Neovascularization, Physiologic - drug effects | Neurons - pathology | Cerebral Cortex - blood supply | Hypoxia-Ischemia, Brain - pathology | Receptors, G-Protein-Coupled - agonists | Cerebral Infarction - genetics | Hypoxia-Ischemia, Brain - genetics | Cerebral Cortex - metabolism | Cerebral Infarction - metabolism | Prostaglandin Antagonists - pharmacology | Neurons - drug effects | Neuroprotective Agents - administration & dosage | Hypoxia-Ischemia, Brain - metabolism | Receptors, Prostaglandin E, EP4 Subtype - drug effects | Cerebral Infarction - pathology | Receptors, Prostaglandin E, EP4 Subtype - metabolism | Astrocytes - drug effects | Cell Line | Tissue Culture Techniques | Mice, Inbred C57BL | Hypoxia-Ischemia, Brain - drug therapy | Cyclooxygenase Inhibitors - pharmacology | Animals | Succinic Acid - pharmacology | Endothelial Cells - drug effects | Hypoxia-Ischemia, Brain - physiopathology | Index Medicus
Journal Article
NeuroRehabilitation (Reading, Mass.), ISSN 1053-8135, 2010, Volume 26, Issue 1, pp. 5 - 13
Journal Article