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Journal Article
Journal Article
Journal Article
CELLULAR PHYSIOLOGY AND BIOCHEMISTRY, ISSN 1015-8987, 2005, Volume 16, Issue 1-3, pp. 9 - 14
Triggers of Jurkat T cell apoptosis include sphingosine and ceramide. Sphingosine and ceramide further inhibit capacitative Ca2+ entry (I-CRAC), an effect... 
FAS-INDUCED APOPTOSIS | DEATH RECEPTORS | PHYSIOLOGY | mitochondria | ACTIVATED CALCIUM CURRENT | APOPTOTIC VOLUME DECREASE | apoptosis | BH3 DOMAIN | CELL BIOLOGY | CYTOCHROME-C | INHIBITION | CHANNEL | Jurkat cells | capacitative Ca2+ channel I-CRAC | HUMAN ACID CERAMIDASE | I-CRAC
Journal Article
Journal of General Physiology, ISSN 0022-1295, 04/1998, Volume 111, Issue 4, pp. 521 - 537
We used whole-cell recording to characterize ion permeation, rectification, and block of monovalent current through calcium release-activated calcium (CRAC)... 
Inward rectification | CRAC channel | Calcium channel | I(CRAC) | Ion selectivity | calcium channel | ion selectivity | PHYSIOLOGY | INTRACELLULAR CA2+ STORES | FROG-MUSCLE | DEPLETION | CRAC CHANNELS | FUNCTIONAL EXPRESSION | MOLLUSK NEURONS | inward rectification | CONCENTRATION-DEPENDENCE | RAT MAST-CELLS | SELECTIVITY | I-CRAC | PERMEATION | Ion-permeable membranes | Calcium channels | Research | T cells
Journal Article
Journal of General Physiology, ISSN 0022-1295, 05/1996, Volume 107, Issue 5, pp. 597 - 610
The depletion of intracellular Ca2+ stores triggers the opening of Ca2+ release-activated Ca2+ (CRAC) channels in the plasma membrane of T lymphocytes. We have... 
capacitative calcium entry | Ca21 channel | patch clamp | thapsigargin | I(CRAC) | SYSTEM | PHYSIOLOGY | ENTRY | MAST-CELLS | DEPLETION | CA-2 | I-CRAC | Physiological aspects | Calcium channels | T cells | Calcium ions
Journal Article
Pflügers Archiv - European Journal of Physiology, ISSN 0031-6768, 4/2004, Volume 448, Issue 1, pp. 93 - 104
Journal Article
Journal of General Physiology, ISSN 0022-1295, 06/1997, Volume 109, Issue 6, pp. 703 - 715
Ca2+ currents activated by depletion of Ca2+ stores in Xenopus oocytes were studied with a two-electrode voltage clamp. Buffering of cytosolic Ca2+ with EGTA... 
Thapsigargin | MeBAPTA | I(SOC) | I(CRAC) | Calcium influx | CALPHOSTIN-C | CHLORIDE CURRENT | PROTEIN-KINASE-C | PHYSIOLOGY | ENTRY | calcium influx | INTRACELLULAR CA2+ STORES | MAST-CELLS | INFLUX | INHIBITION | K UM PHYSIOLOGY | I-SOC | thapsigargin | I-CRAC | CHANNEL BLOCKERS | LYMPHOCYTES | Physiological aspects | Research | Calcium | Oocytes
Journal Article
SCIENCE, ISSN 0036-8075, 10/2010, Volume 330, Issue 6000, pp. 105 - 109
Calcium signals, pivotal in controlling cell function, can be generated by calcium entry channels activated by plasma membrane depolarization or depletion of... 
OPERATED CA2+ ENTRY | TRPC CHANNELS | ACTIVATION | INFLUX | MULTIDISCIPLINARY SCIENCES | SIGNALS | SMOOTH-MUSCLE-CELLS | DOMAINS | I-CRAC
Journal Article
Journal Article
CELL BIOCHEMISTRY AND BIOPHYSICS, ISSN 1085-9195, 2004, Volume 41, Issue 3, pp. 367 - 380
The cytoplasmic Ca(2+)concentration ([Ca2+](cyt)) in resting cells is an equilibrium between several influx and efflux mechanisms. Here we address the question... 
2-AMINOETHOXYDIPHENYL BORATE | Ca2+ influx | ACTIVATED CALCIUM CURRENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | capacitative Ca2+ entry | CURRENT I-CRAC | Ca2+ homoeostasis | PLASMA-MEMBRANE | CELL BIOLOGY | BIOPHYSICS | STORES | INOSITOL TRISPHOSPHATE RECEPTOR | ACINAR-CELLS | CHANNELS | I-CRAC | T-LYMPHOCYTES
Journal Article
Nature Cell Biology, ISSN 1465-7392, 2009, Volume 11, Issue 6, pp. 669 - 677
Calcium influx through plasma membrane store-operated Ca2+ (SOC) channels is triggered when the endoplasmic reticulum (ER) Ca2+ store is depleted - a... 
CALCIUM SENSOR STIM1 | TRPC CHANNELS | CA2+-SELECTIVE ARC CHANNELS | STROMAL INTERACTION MOLECULE-1 | SMOOTH-MUSCLE-CELLS | ENDOPLASMIC-RETICULUM | NEOINTIMA FORMATION | ACTIVATES CRAC CHANNELS | CURRENT I-CRAC | PLASMA MEMBRANE JUNCTIONS | CELL BIOLOGY
Journal Article