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Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 299 - 15
The ubiquitously expressed RNA-binding proteins Roquin-1 and Roquin-2 are essential for appropriate immune cell function and postnatal survival of mice. Roquin... 
B-CELLS | CONSTITUTIVE-DECAY ELEMENT | AUTOIMMUNITY | MULTIDISCIPLINARY SCIENCES | TFH-CELL-DIFFERENTIATION | INDUCIBLE COSTIMULATOR | STEM-LOOP | MENTAL-RETARDATION PROTEIN | STRESS GRANULE | HELPER T-CELLS | DEPENDENT DECAY | RNA, Small Interfering - genetics | RNA-Binding Proteins - genetics | Receptors, OX40 - genetics | Humans | Receptors, OX40 - immunology | CD4-Positive T-Lymphocytes - immunology | Ubiquitin-Protein Ligases - immunology | HEK293 Cells | Inducible T-Cell Co-Stimulator Protein - genetics | Inducible T-Cell Co-Stimulator Protein - antagonists & inhibitors | Nuclear Proteins - genetics | Nucleic Acid Conformation | Binding Sites | Inducible T-Cell Co-Stimulator Protein - immunology | Inverted Repeat Sequences | RNA-Binding Proteins - antagonists & inhibitors | Amino Acid Sequence | CD4-Positive T-Lymphocytes - cytology | Mice, Inbred C57BL | RNA-Binding Proteins - immunology | Gene Expression Regulation | Repressor Proteins - genetics | Recombinant Proteins - genetics | Nuclear Proteins - immunology | RNA Stability | Sequence Homology, Amino Acid | Sequence Alignment | Animals | Recombinant Proteins - immunology | Nuclear Proteins - antagonists & inhibitors | Repressor Proteins - immunology | Protein Binding | Mice | HeLa Cells | Primary Cell Culture | Ubiquitin-Protein Ligases - genetics | RNA, Small Interfering - metabolism | Receptors, OX40 - antagonists & inhibitors | Post-transcription | Target recognition | Cell survival | RNA-mediated interference | mRNA turnover | Cofactors | Proteins | Gene silencing | RNA-binding protein | Ribonucleic acids | Tumor necrosis factor | ICOS protein | Decay | Index Medicus
Journal Article
Immunology, ISSN 0019-2805, 09/2017, Volume 152, Issue 1, pp. 138 - 149
The Toll‐like receptor ( TLR ) adaptor proteins myeloid differentiating factor 88 (MyD88) and Toll, interleukin‐1 receptor and resistance protein ( TIR )... 
T helper type 2 | Toll, interleukin‐1R and resistance protein (TIR) domain‐containing adaptor inducing interferon | Toll‐like receptor 4 | inducible co‐stimulator | Asthma | inducible co-stimulator | Toll, interleukin-1R and resistance protein (TIR) domain-containing adaptor inducing interferon-β | Toll-like receptor 4 | DUST MITE ALLERGEN | TH2 RESPONSES | DENDRITIC CELLS | HELPER-CELLS | IMMUNOLOGY | RECEPTOR 4 | MONOPHOSPHORYL-LIPID-A | interleukin-1R and resistance protein (TIR) domain-containing adaptor inducing interferon | IN-VIVO | REGULATORY T-CELLS | AIRWAY HYPERRESPONSIVENESS | Toll | NF-KAPPA-B | Chemotaxis, Leukocyte | Rhinitis, Allergic, Seasonal - physiopathology | Asthma - metabolism | Cell Proliferation | Asthma - prevention & control | Bronchial Hyperreactivity - immunology | Rhinitis, Allergic, Seasonal - metabolism | Adaptor Proteins, Vesicular Transport - genetics | Adoptive Transfer | Pollen - immunology | Adaptor Proteins, Vesicular Transport - metabolism | Lipopolysaccharides - immunology | Rhinitis, Allergic, Seasonal - immunology | CD4-Positive T-Lymphocytes - immunology | Bronchoconstriction | Time Factors | Bronchial Hyperreactivity - physiopathology | Asthma - immunology | Bronchial Hyperreactivity - metabolism | Female | Lung - metabolism | Inducible T-Cell Co-Stimulator Protein - immunology | Disease Models, Animal | Genetic Predisposition to Disease | Asthma - physiopathology | Signal Transduction | Lymphocyte Activation | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Myeloid Differentiation Factor 88 - genetics | Antigens, Plant - immunology | Lung - physiopathology | Toll-Like Receptor 4 - immunology | Adaptor Proteins, Vesicular Transport - immunology | Bronchial Hyperreactivity - prevention & control | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Rhinitis, Allergic, Seasonal - prevention & control | Phenotype | Animals | CD4-Positive T-Lymphocytes - transplantation | Betula - immunology | Mice, Inbred BALB C | Inducible T-Cell Co-Stimulator Protein - metabolism | Cysteine Endopeptidases - immunology | Myeloid Differentiation Factor 88 - metabolism | Drug Combinations | Lung - immunology | Development and progression | Interferon | Interleukins | Biological response modifiers | T cells | β-Interferon | Lung | Mucosa | Interleukin | Stimulation | Lymphocytes T | Adoptive transfer | Lymph nodes | Recruitment | Respiratory tract | Proteins | Signal transduction | Lymphocytes | Interleukin 1 | Toll-like receptors | Foxp3 protein | Respiratory tract diseases | Pollen | Inhibition | Adaptor proteins | Limbs | Lymphatic system | Data processing | T cell receptors | TLR4 protein | Adaptors | CD4 antigen | Immune systems | Drainage | Signaling | Lungs | TLR2 protein | ICOS protein | MyD88 protein | Lymph | Index Medicus | Original | Toll, interleukin‐1R and resistance protein (TIR) domain‐containing adaptor inducing interferon‐β
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2012, Volume 188, Issue 8, pp. 3734 - 3744
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 05/2017, Volume 214, Issue 5, pp. 1529 - 1546
Journal Article
Journal of Immunology, ISSN 0022-1767, 10/2017, Volume 199, Issue 7, pp. 2305 - 2315
Journal Article
Journal Article
European Journal of Immunology, ISSN 0014-2980, 02/2017, Volume 47, Issue 2, pp. 280 - 290
Control of B‐cell signal transduction is critical to prevent production of pathological autoantibodies. Tandem PH domain containing proteins (TAPPs)... 
Autoimmunity | Antibodies | Signal transduction | B cells | Cellular activation | AUTOIMMUNE INFLAMMATORY DISEASES | SYSTEMIC-LUPUS-ERYTHEMATOSUS | ACTIVATION | DOMAIN | HIGH-AFFINITY | IMMUNOLOGY | PHOSPHOINOSITIDE 3-KINASE | PI3 KINASE | SHIP | INOSITOL POLYPHOSPHATE 5-PHOSPHATASE | ANTIGEN RECEPTOR | Autoantibodies - metabolism | Lymphocyte Activation | Membrane Proteins - genetics | Germinal Center - immunology | Mice, Inbred C57BL | Cells, Cultured | B-Lymphocytes - physiology | Intracellular Signaling Peptides and Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Mice, Knockout | Phosphatidylinositol 3-Kinases - genetics | Animals | Inducible T-Cell Co-Stimulator Protein - genetics | Immunologic Memory | Membrane Proteins - metabolism | Mice | Antibodies, Antinuclear - metabolism | Inducible T-Cell Co-Stimulator Protein - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Phosphatases | Autoantibodies | Resveratrol | Cellular signal transduction | Phosphotransferases | Protein binding | AKT protein | Activation | Kinases | Proteins | Cell activation | Germinal centers | Clonal deletion | Ships | Rodents | Deletion | Anti-DNA antibodies | Deoxyribonucleic acid--DNA | Antigen-antibody complexes | Adaptor proteins | Immunological memory | Immunization | Kidneys | Cell survival | Adaptors | Lymphocytes B | ICOS protein | Transduction | Mutation | Autoimmune diseases | Index Medicus
Journal Article
Nature Immunology, ISSN 1529-2908, 07/2003, Volume 4, Issue 7, pp. 670 - 679
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 10/2006, Volume 116, Issue 10, pp. 2817 - 2826
T cell activation by APCs is positively and negatively regulated by members of the B7 family. We have identified a previously unknown function for B7... 
MEDICINE, RESEARCH & EXPERIMENTAL | DENDRITIC CELLS | IMMUNE-RESPONSES | TOLERANCE | RECEPTOR | SUPERFAMILY | CTLA-4 | COSTIMULATION | ICOS | LYMPHOCYTE-ACTIVATION | MEMBER | Immunoglobulins - physiology | Myocarditis - chemically induced | Gene Expression - drug effects | Humans | Molecular Sequence Data | Immunoglobulins - genetics | Interferon-gamma - metabolism | Receptors, Complement - metabolism | Lymphocyte Activation - immunology | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | Gene Expression - immunology | Receptors, Complement - genetics | Female | B7-1 Antigen - pharmacology | Autoimmune Diseases - metabolism | Myocarditis - immunology | Interleukin-2 - metabolism | Amino Acid Sequence | Cell Line | Immunoglobulins - pharmacology | B7-H1 Antigen | Liver - metabolism | Membrane Glycoproteins - pharmacology | Mice, Inbred C57BL | Programmed Cell Death 1 Ligand 2 Protein | Autoimmune Diseases - immunology | Peptides - pharmacology | Sequence Homology, Amino Acid | Macrophages - metabolism | Animals | Thioglycolates - pharmacology | Lymphocyte Activation - drug effects | Lipopolysaccharides - pharmacology | Myocarditis - metabolism | Interleukin-2 - pharmacology | Macrophages - drug effects | T-Lymphocytes - immunology | Cell Proliferation - drug effects | Liver - cytology | Mice | Mice, Inbred BALB C | Receptors, Complement - physiology | Autoimmune Diseases - chemically induced | Medicine, Experimental | Medical research | Protein research | Research | T cells | Index Medicus | Abridged Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 05/2017, Volume 114, Issue 19, pp. 4993 - 4998
Exhausted T cells in chronic infections and cancer have sustained expression of the inhibitory receptor programmed cell death 1 (PD-1). Therapies that block...