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Cellular Microbiology, ISSN 1462-5814, 06/2006, Volume 8, Issue 6, pp. 1047 - 1057
Summary Infection of epithelial cells by the intracellular pathogen, Chlamydia trachomatis, leads to activation of NF-κB and secretion of pro-inflammatory... 
GENITAL-TRACT INFECTION | DEVELOPMENTAL CYCLE | MOUSE PNEUMONITIS | IFN-GAMMA | TOLL-LIKE RECEPTORS | INNATE IMMUNE-RESPONSE | MICROBIOLOGY | EPITHELIAL-CELL LINES | BACTERIAL PEPTIDOGLYCAN | INFLAMMATORY RESPONSE | NF-KAPPA-B | CELL BIOLOGY | Toll-Like Receptor 2 - genetics | Fibroblasts - physiology | Receptor-Interacting Protein Serine-Threonine Kinases | Chlamydia trachomatis - chemistry | Humans | Receptors, Cell Surface - analysis | Epithelium - chemistry | RNA, Messenger - analysis | Male | Chlamydia Infections - pathology | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins - physiology | Epithelium - microbiology | Chlamydia Infections - physiopathology | DNA, Bacterial - analysis | Female | Chlamydia muridarum - physiology | Receptor-Interacting Protein Serine-Threonine Kinase 2 | Receptors, Cell Surface - physiology | Fibroblasts - microbiology | NF-kappa B - analysis | Epithelium - pathology | Protein-Serine-Threonine Kinases - physiology | RNA, Messenger - genetics | Toll-Like Receptor 4 - genetics | Chlamydia muridarum - chemistry | Gene Expression Regulation, Bacterial - physiology | Toll-Like Receptor 4 - physiology | Adaptor Proteins, Signal Transducing - physiology | Animals | DNA, Bacterial - genetics | Toll-Like Receptor 2 - physiology | Chlamydia muridarum - pathogenicity | Adaptor Proteins, Signal Transducing - genetics | Chlamydia trachomatis - pathogenicity | NF-kappa B - physiology | Mice, Inbred NOD | Epithelium - physiology | Mice | Nod1 Signaling Adaptor Protein | Vagina - microbiology | HeLa Cells | Chlamydia trachomatis - physiology | Chlamydia | Health aspects | Sexually transmitted diseases | Toll-Like Receptor 2 | Toll-Like Receptor 4 | Protein-Serine-Threonine Kinases | Receptors, Cell Surface | Innate immunity | Chlamydia trachomatis | DNA, Bacterial | Life Sciences | NF-kappa B | Immunology | Chlamydia Infections | Fibroblasts | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins | Vagina | Epithelium | Adaptor Proteins, Signal Transducing | Gene Expression Regulation, Bacterial | RNA, Messenger | Hela Cells | Chlamydia muridarum
Journal Article
Immunology, ISSN 0019-2805, 11/2013, Volume 140, Issue 3, pp. 352 - 361
Journal Article
Journal Article
Science, ISSN 0036-8075, 06/2016, Volume 352, Issue 6292, pp. aad1210 - aad1210
Journal Article
Journal Article
Neuropsychobiology, ISSN 0302-282X, 12/2011, Volume 65, Issue 1, pp. 12 - 19
Aims: There is evidence that psychological stress can modulate immune functions. It has been hypothesized that acute stressors can affect both immune balance... 
Original Paper | Acute stress | IFN-γ | Adrenergic receptor | IFN-γ receptor | Immunomodulation | Glucocorticoid receptor | beta-Adrenergic receptor | SOCIAL STRESS | PSYCHIATRY | INDUCED ANXIETY | NEUROSCIENCES | INFLAMMATORY CYTOKINE | PSYCHOLOGY | LEUKOCYTE DISTRIBUTION | MENTAL STRESS | RESPONSES | IN-VITRO | IFN-gamma | IFN-gamma receptor | MESSENGER-RNA | PSYCHOSOCIAL STRESS | ACUTE PSYCHOLOGICAL STRESS | T-Lymphocytes, Regulatory - metabolism | Catecholamines - metabolism | Humans | Middle Aged | Male | Receptors, Adrenergic, beta-2 - immunology | Receptors, Glucocorticoid - metabolism | Th2 Cells - immunology | Receptors, Catecholamine - immunology | Th1 Cells - immunology | Receptors, Catecholamine - metabolism | Stress, Psychological - immunology | T-Lymphocytes, Regulatory - immunology | Th1 Cells - metabolism | Interleukin-10 - metabolism | Adult | Female | Receptors, Cytokine - metabolism | Receptors, Catecholamine - genetics | Cytokines - genetics | Real-Time Polymerase Chain Reaction | Receptors, Cytokine - genetics | Receptors, Interferon - genetics | Transforming Growth Factor beta - immunology | Interferon-gamma | Gene Expression | Cytokines - metabolism | Receptors, Adrenergic, beta-2 - genetics | Receptors, Interleukin-4 - metabolism | Receptors, Glucocorticoid - immunology | Receptors, Interleukin-4 - immunology | Th2 Cells - metabolism | Receptors, Adrenergic, beta-2 - metabolism | Transforming Growth Factor beta - genetics | Receptors, Interferon - immunology | Interleukin-10 - genetics | Receptors, Interleukin-4 - genetics | Receptors, Glucocorticoid - genetics | Receptors, Cytokine - immunology | Interleukin-10 - immunology | Transforming Growth Factor beta - metabolism
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 06/2010, Volume 207, Issue 6, pp. 1283 - 1292
Human BDCA3(+) dendritic cells (DCs) were suggested to be homologous to mouse CD8 alpha(+) DCs. We demonstrate that human BDCA3(+) DCs are more efficient than... 
B-CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | NATURAL-KILLER-CELLS | C-TYPE LECTIN | IFN-GAMMA | IN-VIVO | SUBSETS | IMMUNOLOGY | CD8(+) T-CELLS | VIRAL-INFECTION | CUTTING EDGE | ANTIGEN
Journal Article
Journal Article
Journal Article
Nature Immunology, ISSN 1529-2908, 2013, Volume 14, Issue 5, pp. 480 - 488
NOD2 receptor and the cytosolic protein kinase RIPK2 regulate NF-kappaB and MAP kinase signaling during bacterial infections, but the role of this immune axis... 
LISTERIA-MONOCYTOGENES | IMMUNE-RESPONSE | 1918 INFLUENZA-VIRUS | DOUBLE-STRANDED-RNA | IFN-GAMMA PRODUCTION | DENDRITIC CELLS | IL-1-BETA PRODUCTION | INNATE | AUTOPHAGY | IMMUNOLOGY | CD8(+) T-CELLS | Immune response | Influenza | Physiological aspects | Causes of | Genetic aspects | Cellular signal transduction | Research | Protein kinases | mitophagy | autophagy | RIPK2 | NLR | inflammasome | IL-18 | Caspase-1
Journal Article