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American Journal of Transplantation, ISSN 1600-6135, 10/2013, Volume 13, Issue 10, pp. 2577 - 2589
Journal Article
Kidney International, ISSN 0085-2538, 11/2006, Volume 70, Issue 10, pp. 1717 - 1724
Dysregulated production of adipocytokines may be involved in the development of atherosclerotic cardiovascular disease in metabolic syndrome and chronic kidney... 
adipocytokine | adiponectin | angiotensin II type1 receptor blocker | NADPH oxidase | oxidative stress | Adipocytokine | Oxidative stress | Angiotensin II type1 receptor blocker | Adiponectin | 3T3-L1 ADIPOCYTES | METABOLIC SYNDROME | NECROSIS-FACTOR-ALPHA | DIABETIC-NEPHROPATHY | CARDIOVASCULAR MORBIDITY | INSULIN SENSITIVITY | angiotensin II type 1 receptor blocker | VASCULAR-DISEASE | ARTERIOSCLEROTIC OUTCOMES | NITRIC-OXIDE | UROLOGY & NEPHROLOGY | ESSENTIAL-HYPERTENSION | Kidney Diseases - physiopathology | Tumor Necrosis Factor-alpha - metabolism | Reactive Oxygen Species - metabolism | Tetrazoles - pharmacology | Oxidative Stress - physiology | Tumor Necrosis Factor-alpha - genetics | Metabolic Syndrome - metabolism | Serum Amyloid A Protein - genetics | RNA, Messenger - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Adipose Tissue - metabolism | Plasminogen Activator Inhibitor 1 - metabolism | Metabolic Syndrome - physiopathology | Adiponectin - metabolism | Female | Plasminogen Activator Inhibitor 1 - genetics | Serum Amyloid A Protein - metabolism | Atherosclerosis - physiopathology | Gene Expression Regulation - genetics | Kidney Diseases - prevention & control | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Obesity - physiopathology | Imidazoles - pharmacology | Obesity - metabolism | Gene Expression Regulation - drug effects | Animals | Signal Transduction - physiology | Mice | Oxidative Stress - drug effects | Adipose Tissue - drug effects | Angiotensin II - physiology | Atherosclerosis - prevention & control
Journal Article
Journal Article
Hypertension: Journal of the American Heart Association, ISSN 0194-911X, 05/2004, Volume 43, Issue 5, pp. 1003 - 1010
ABSTRACT—Angiotensin II has been shown to contribute to the pathogenesis of insulin resistance; however, the mechanism is not well understood. The present... 
Glucose | Angiotensin II | Insulin | Diabetes mellitus | CONVERTING ENZYME-INHIBITOR | CELLS | glucose | FRUCTOSE-FED RATS | MEDIATED GLUCOSE-UPTAKE | STIMULATION | insulin | ALPHA | diabetes mellitus | SIGNALING SYSTEMS | angiotensin II | RESISTANCE | PERIPHERAL VASCULAR DISEASE | DIFFERENTIATION | HYPERTENSION | Tetrazoles - pharmacology | Valsartan | Tumor Necrosis Factor-alpha - genetics | Male | Muscle, Skeletal - metabolism | Protein Transport - drug effects | Diabetes Mellitus, Type 2 - metabolism | Insulin - blood | Phosphoproteins - metabolism | Angiotensin II Type 1 Receptor Blockers | Protein Processing, Post-Translational - drug effects | Mice, Mutant Strains | Muscle, Skeletal - drug effects | Insulin Receptor Substrate Proteins | Muscle Proteins - metabolism | Superoxides - metabolism | Deoxyglucose - pharmacokinetics | Phosphorylation - drug effects | Monosaccharide Transport Proteins - metabolism | Disease Models, Animal | Blood Glucose - analysis | Glucose Transporter Type 4 | Mice, Inbred C57BL | Valine - analogs & derivatives | Insulin Resistance | Energy Intake - drug effects | Imidazoles - pharmacology | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Mice, Obese | Tetrazoles - therapeutic use | Valine - therapeutic use | Mice | Pyridines - pharmacology | Valine - pharmacology | Diabetes Mellitus, Type 2 - drug therapy | Tumor Necrosis Factor-alpha - biosynthesis | Crosses, Genetic
Journal Article
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 02/2016, Volume 6, Issue 1, p. 21359
Journal Article
Experimental and Molecular Pathology, ISSN 0014-4800, 02/2017, Volume 102, Issue 1, pp. 32 - 40
Scorpion stings are mainly associated with cardiovascular disturbances that may be the cause of death. In this study, the involvement of angiotensin II (Ang... 
Cardiovascular system | Inflammatory response | Angiotensin II | Scorpion venom | Angiotensin type-1 receptor | Angiotensin converting enzyme | MATRIX | ASSAY | RAT | SUPEROXIDE-PRODUCTION | MECHANISMS | PATHOLOGY | TROPONIN-I | TUMOR-NECROSIS-FACTOR | NAD(P)H OXIDASE | NITRIC-OXIDE | TNF-ALPHA | Inflammation - chemically induced | Eosinophils - metabolism | Captopril - pharmacology | Eosinophils - drug effects | Glutathione - metabolism | Creatine Kinase, MB Form - metabolism | Male | Aorta - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Valsartan - pharmacology | Inflammation - metabolism | Matrix Metalloproteinase 9 - metabolism | Myocardium - metabolism | Oxidation-Reduction - drug effects | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Eosinophils - pathology | Neutrophils - metabolism | Neutrophils - pathology | Angiotensin II - metabolism | Cytokines - metabolism | Matrix Metalloproteinase 2 - metabolism | Neutrophils - drug effects | Myocardium - pathology | Mice, Inbred Strains | Aorta - pathology | Catalase - metabolism | Scorpion Venoms | Animals | Receptor, Angiotensin, Type 1 - metabolism | Oxidative Stress - drug effects | Heart | Venom | Angiotensin | Inflammation | Cardiology | Cardiovascular agents | Creatine kinase | Creatine | Prevention | Interleukins | Nitric oxide | Analysis
Journal Article
Journal Article
Journal Article