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European Journal of Immunology, ISSN 0014-2980, 2013, Volume 43, Issue 2, pp. 416 - 426
IL-1 drives Th responses, particularly Th17, in host defense. Sharing the same co-receptor, the IL-1 family member IL-36 exhibits properties similar to those... 
Th responses | Pattern recognition | IL‐36 receptor antagonist | IL‐36 | IL-36 | IL-36 receptor antagonist | GENERALIZED PUSTULAR PSORIASIS | IMMUNOLOGY | IMMUNE RECOGNITION | FUNGAL-INFECTIONS | BETA-GLUCAN RECEPTOR | INVASIVE ASPERGILLOSIS | CANDIDA-ALBICANS | IL-18 | IN-VIVO | CRITICALLY-ILL PATIENTS | T-CELLS | Forkhead Transcription Factors - immunology | Toll-Like Receptor 2 - genetics | Humans | Interleukin-17 - immunology | Lectins, C-Type - immunology | Macrophage-1 Antigen - immunology | Interferon-gamma - metabolism | Lectins, C-Type - genetics | Th1 Cells - immunology | Receptors, Interleukin - metabolism | Th1 Cells - metabolism | Lectins, C-Type - metabolism | Signal Transduction - immunology | Th17 Cells - metabolism | Forkhead Transcription Factors - metabolism | Aspergillosis - metabolism | Macrophage-1 Antigen - metabolism | Interferon-gamma - genetics | Spores, Fungal - immunology | Receptors, Interleukin - immunology | RNA, Messenger - genetics | Aspergillosis - genetics | Interleukin-17 - genetics | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Forkhead Transcription Factors - genetics | Receptors, Interleukin - genetics | Toll-Like Receptor 4 - metabolism | Hyphae - immunology | Interleukin-17 - metabolism | Macrophage-1 Antigen - genetics | Aspergillus fumigatus - immunology | Interferon-gamma - immunology | Aspergillosis - microbiology | Th17 Cells - immunology | Toll-Like Receptor 2 - immunology | Aspergillosis - immunology | RNA | Fungi | Composting
Journal Article
Journal Article
OncoImmunology, ISSN 2162-4011, 06/2017, Volume 6, Issue 6, p. e1322238
We have previously reported that direct injection of dendritic cells (DC) engineered to express the Type-1 transactivator Tbet (i.e., DC.Tbet) into murine... 
tbet | Dendritic cells | interleukin (IL)-36γ | tertiary lymphoid organ | tumor | immunotherapy
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 05/2016, Volume 184, Issue 2, pp. 159 - 173
Journal Article
The FEBS Journal, ISSN 1742-464X, 06/2017, Volume 284, Issue 11, pp. 1712 - 1725
Journal Article
Experimental Dermatology, ISSN 0906-6705, 03/2019, Volume 28, Issue 3, pp. 233 - 239
Skin injury can trigger formation of new lesions in psoriasis (Koebner phenomenon). The mechanisms through which injury exacerbates psoriasis are unclear.... 
dsRNA | IL‐17A | keratinocytes | psoriasis | IL‐36γ | IL-36γ | IL-17A | Tumor necrosis factor receptors | Wound healing | Psoriasis | Double-stranded RNA | Keratinocytes | MAP kinase | mRNA | siRNA | Poly (I:C) | Inflammation | TLR3 protein | Signal transduction | Tumor necrosis factor | Toll-like receptors | CCL20 protein | Skin
Journal Article
Australasian Journal of Dermatology, ISSN 0004-8380, 02/2020, Volume 61, Issue 1, pp. e39 - e45
Background Members of the interleukin (IL)‐36 family, IL‐36α, IL‐36β and IL‐36γ, are potent chemoattractive cytokines for neutrophils and eosinophils. IL‐36... 
eosinophilic pustular folliculitis | IL‐36 receptor antagonist | folliculitis | IL‐36α | IL‐36β | IL‐36γ | DERMATITIS | IL-36 alpha | IL-36 beta | IL-36 gamma | IL-36 receptor antagonist | ACNE PATHOGENESIS | SKIN | PSORIASIS | DERMATOLOGY | Cytokines | Leukocytes (eosinophilic) | Epidermis | Leukocytes (neutrophilic) | Inflammation | Folliculitis | Epithelium | Follicles
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Journal Article
Journal Article
Journal Article
Journal Article