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Journal of allergy and clinical immunology, ISSN 0091-6749, 2012, Volume 129, Issue 2, pp. 294 - 305
Mucocutaneous candidiasis and dermatophyte infections occur either in isolation or alongside other symptoms in patients with various primary immunodeficiency... 
Allergy and Immunology | Chronic mucocutaneous candidiasis | IL-17 receptor | caspase recruitment domain–containing protein 9 | signal transducer and activator of transcription 3 | neutrophil | dedicator of cytokinesis 8 | TH17 cell | anti–IL-17 receptor antibody | autoimmune polyendocrinopathy–candidiasis–ectodermal dystrophy | hyper-IgE syndromes | signal transducer and activator of transcription 1 | dectin | IL-17 | primary immunodeficiency | anti–IL-17 antibody | 17 cell | anti-IL-17 antibody | caspase recruitment domain-containing protein 9 | autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy | anti-IL-17 receptor antibody | BETA-GLUCAN RECOGNITION | IMMUNOLOGY | ADAPTIVE IMMUNITY | DECTIN-2 RECOGNITION | COMBINED IMMUNODEFICIENCY | ALLERGY | ectodermal dystrophy | T-CELLS | PATTERN-RECOGNITION RECEPTOR | HYPER-IGE SYNDROME | CANDIDA-ALBICANS | T(H)17 cell | HOST-DEFENSE | autoimmune polyendocrinopathy-candidiasis | TH17 CELLS | Genetic Predisposition to Disease | Animals | Humans | Dermatomycoses - genetics | Dermatomycoses - immunology | Immunologic Deficiency Syndromes - genetics | Immunologic Deficiency Syndromes - immunology | Tyrosine | Autoimmunity | Peptides | Dendritic cells | Genes | Leukemia | Immunodeficiency | Antifungal agents | Immunoglobulin E | T cells | Diseases | Oligomers | Lectins | Lymphomas | Health aspects | Cytokines | Physicians | Mortality | Vitamin deficiency | Fungal infections | Defects | Fungi | Signal transduction | Chemotherapy | Genetic counseling | Acquired immune deficiency syndrome--AIDS | Antibiotics | Human immunodeficiency virus--HIV | Meningitis | Accreditation | Skin | Mutation | Diabetes | Immune system | Candidiasis | Transcription factors | Helper cells | Interleukin 22 | Lymphocytes T | Gene polymorphism | Immunity | Cell activation | Antimicrobial peptides | Interleukin 1 | Stat1 protein | Job's syndrome | Spleen | Autoantibodies | Stat3 protein | Caspase | Genetic diversity | Leukocytes (neutrophilic) | Interleukin 17 | Orphan receptors | Interleukin 17 receptors | Antigen-presenting cells | Bcl-10 protein | Degranulation | Autoimmune diseases | Phagocytosis
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2014, Volume 111, Issue 2, pp. 787 - 792
Journal Article
International Immunology, ISSN 0953-8178, 2012, Volume 24, Issue 3, pp. 137 - 146
Journal Article
Journal of neuroinflammation, ISSN 1742-2094, 2016, Volume 13, Issue 1, p. 223
Background: CCR5 is a CC chemokine receptor involved in the migration of effector leukocytes including macrophages, NK, and T cells into inflamed tissues.... 
CD4 | Tregs | IL-17 | Neuroinflammation | CCR5 | Foxp3 | Th17 | Japanese encephalitis | MIGRATION | NILE-VIRUS ENCEPHALITIS | PROINFLAMMATORY IL-17(+) | IMMUNOLOGY | NEUROSCIENCES | CCR5-DEFICIENT MICE | CD4(+)Foxp3(+) Tregs | FLAVIVIRUS INFECTIONS | CD154 EXPRESSION | NEURONAL DEATH | HOST-DEFENSE | IL-17(+)CD4(+) Th17 | CHEMOKINE RECEPTORS | WEST-NILE | Spleen - virology | Central Nervous System - metabolism | Virus Replication - genetics | Central Nervous System - pathology | Encephalitis, Japanese - genetics | Receptors, CCR5 - genetics | Male | Killer Cells, Natural - pathology | Cell Movement - physiology | Receptors, CCR5 - metabolism | Th17 Cells - metabolism | Forkhead Transcription Factors - metabolism | Female | Spleen - pathology | Cytokines - genetics | Central Nervous System - virology | Disease Models, Animal | Cytokines - metabolism | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Gene Expression Regulation - physiology | Interleukin-17 - genetics | Mice, Transgenic | Myeloid Cells - virology | Forkhead Transcription Factors - genetics | Disease Progression | Interleukin-17 - metabolism | Animals | Encephalitis, Japanese - pathology | Spleen - metabolism | Myeloid Cells - metabolism | Mice | Killer Cells, Natural - metabolism | Myeloid Cells - pathology | Complications and side effects | Care and treatment | Mortality | Analysis | Research | Gene expression | Risk factors
Journal Article
The Journal of allergy and clinical immunology, ISSN 0091-6749, 1/2019, Volume 143, Issue 1, pp. 229 - 244.e9
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 12/2013, Volume 188, Issue 11, pp. 1294 - 1302
Journal Article
European journal of immunology, ISSN 0014-2980, 2016, Volume 46, Issue 2, pp. 440 - 445
Mesenchymal stem cells are known to exert immunomodulatory effects in inflammatory diseases. Immuneregulatory cells lead to progressive joint destruction in... 
Cartilage Chondrogenesis ⋅ Chondrogenic progenitor cells ⋅ Rheumatoid arthritis ⋅ Interleukin 17 (IL‐17) | Interleukin 17 (IL-17) | Rheumatoid arthritis | Cartilage Chondrogenesis | Chondrogenic progenitor cells | ACTIVATION | Cartilage Chondrogenesis Chondrogenic progenitor cells Rheumatoid arthritis Interleukin 17 (IL-17) | ARTICULAR-CARTILAGE | DISEASE | TISSUE | IL-17 | IMMUNOLOGY | NF-KAPPA-B | EXPRESSION | Stem Cells - immunology | Chondrogenesis - drug effects | Immunotherapy - methods | Cytokines - metabolism | Cartilage - pathology | Humans | Middle Aged | Cells, Cultured | Interleukin-17 - immunology | Male | Matrix Metalloproteinase 3 - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Arthritis, Rheumatoid - therapy | Adalimumab - administration & dosage | Chondrogenesis - immunology | Antibodies, Monoclonal - administration & dosage | Antibodies, Blocking - therapeutic use | Stem Cells - drug effects | Female | Aged | Receptors, Interleukin-17 - metabolism | Arthritis, Rheumatoid - immunology | Core Binding Factor Alpha 1 Subunit - genetics | Interleukins | Rheumatoid factor | Arthritis | Proteins | Tumor necrosis factor-TNF | Rodents | Mesenchyme | Destruction | Interleukin | Antibodies | mRNA | Inflammatory diseases | Interleukin 6 | Cartilage | Receptors | Pharmaceutical industry | Chondrogenesis | Repair | Players | Cbfa-1 protein | Cytokines | Immunomodulation | Patients | Interleukin 17 | Diseases | Tumor necrosis factor | Stem cells | Interleukin 10 | Cells (biology) | Monoclonal antibodies | Tumors | Short Communication
Journal Article
Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 2014, Volume 133, Issue 2, pp. 388 - e5
Background The 3-gene signature of periostin, chloride channel accessory 1 (CLCA1), and Serpin β2 (SERPINB2) in airway epithelial brushings is used to classify... 
phenotypes | inflammation | IL-17 | IL-4 | eosinophils | eotaxin | IL-5 | cytokines | sputum | IL-13 | mast cells | Asthma
Journal Article
Journal of Dermatological Treatment: Safety of secukinumab in moderate-severe psoriasis and psoriatic arthritis: evidences from real-life experiences, ISSN 0954-6634, 12/2018, Volume 29, Issue 2, pp. 1 - 2
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2010, Volume 107, Issue 45, pp. 19402 - 19407
Journal Article
The Journal of infectious diseases, ISSN 1537-6613, 2019, Volume 221, Issue 9, pp. 1554 - 1563
Candida albicans, a ubiquitous commensal fungus that colonizes human mucosal tissues and skin, can become pathogenic, clinically manifesting most commonly as... 
Candida albicans | IMMUNITY | INFECTIOUS DISEASES | ACTIVATION | MICROBIOLOGY | IMMUNOLOGY | vulvovaginal candidiasis | fungal immunity | IL-17 FAMILY | IL-17 | HOST-DEFENSE | cytokines | CHEMOKINE | INFECTION | ALBICANS | vaginitis | Th17 | T-CELLS | TH17 CELLS | HORMONES
Journal Article