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Journal Article
Science Translational Medicine, ISSN 1946-6234, 08/2018, Volume 10, Issue 455, p. eaaq0693
It is anticipated that bioactive fragments of the extracellular matrix (matrikines) can influence the development and progression of chronic diseases. The... 
NEUTROPHILIC INFLAMMATION | MEDICINE, RESEARCH & EXPERIMENTAL | LUNG | OBSTRUCTIVE PULMONARY-DISEASE | SEVERE ASTHMA | INDUCED SPUTUM | CHEMOATTRACTANT | LEUKOTRIENE A HYDROLASE | B-4 | PROLINE-GLYCINE-PROLINE | INHIBITOR | CELL BIOLOGY | Index Medicus
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 2, pp. 462 - 473
Background Steroid-resistant asthma is a major clinical problem that is linked to activation of innate immune cells. Levels of IFN-γ and LPS are often... 
Allergy and Immunology | pulmonary macrophages | MicroRNA | innate immunity | steroid-resistant airway hyperresponsiveness | protein phosphatase 2A | EMERGING ROLES | N-TERMINAL KINASE | INFLAMMATORY SUBTYPES | IMMUNOLOGY | CANCER-THERAPY | NEUTROPHILIC ASTHMA | INTERFERON-GAMMA | ALLERGY | DISEASE | GENE-EXPRESSION | GLUCOCORTICOID-RECEPTOR PHOSPHORYLATION | Oligonucleotides - genetics | Egg Hypersensitivity - drug therapy | Macrophages, Alveolar - pathology | MicroRNAs - antagonists & inhibitors | Bronchial Hyperreactivity - immunology | Eosinophils - drug effects | Bronchial Hyperreactivity - drug therapy | Humans | Protein Phosphatase 2 - immunology | Bronchial Hyperreactivity - genetics | Eosinophils - immunology | Luciferases - genetics | Egg Hypersensitivity - immunology | Dexamethasone - pharmacology | Egg Hypersensitivity - etiology | Luciferases - immunology | Asthma - immunology | Eosinophils - pathology | Genes, Reporter | Macrophages, Alveolar - immunology | Disease Models, Animal | Egg Hypersensitivity - genetics | Neutrophils - pathology | Bronchial Hyperreactivity - chemically induced | Ovalbumin | Signal Transduction | Neutrophils - drug effects | Gene Expression Regulation | Neutrophils - immunology | Protein Phosphatase 2 - genetics | Receptors, Glucocorticoid - immunology | MicroRNAs - immunology | Asthma - drug therapy | Asthma - chemically induced | Asthma - genetics | Oligonucleotides - metabolism | Animals | Macrophages, Alveolar - drug effects | Lipopolysaccharides - pharmacology | Receptors, Glucocorticoid - genetics | Glucocorticoids - pharmacology | Mice | Mice, Inbred BALB C | MicroRNAs - genetics | Primary Cell Culture | Interferon-gamma - pharmacology | Medical colleges | Phosphatases | Corticosteroids | Analysis | Pharmacy | Drugstores | Genetic translation | Asthma | Proteins | Phosphorylation | Disease | Pathogenesis | Rodents | MicroRNAs | Inflammation | Gene expression | Phosphatase | Index Medicus | Abridged Index Medicus
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 09/2015, Volume 309, Issue 7, pp. L736 - L746
Ozone causes airway hyperresponsiveness (AHR) and pulmonary inflammation. Rho kinase (ROCK) is a key regulator of smooth muscle cell contraction and... 
Osteopontin | Hyaluronan | Inflammation | Bronchoalveolar lavage | Fasudil | RHO-ASSOCIATED KINASE | CA2+ SENSITIZATION | PHYSIOLOGY | SERINE-THREONINE KINASE | fasudil | hyaluronan | osteopontin | INDUCED INFLAMMATION | FORMING PROTEIN-KINASE | HUMAN ENDOTHELIAL-CELLS | inflammation | RESPIRATORY SYSTEM | COILED-COIL | bronchoalveolar lavage | SMOOTH-MUSCLE CONTRACTION | T-CELLS | MYOSIN PHOSPHATASE | Osteopontin - genetics | Tumor Necrosis Factor-alpha - metabolism | Gene Expression Regulation, Enzymologic - drug effects | Pneumonia - physiopathology | Muscle Contraction - genetics | Humans | Tumor Necrosis Factor-alpha - genetics | Pneumonia - pathology | Osteopontin - metabolism | Bronchial Hyperreactivity - genetics | Dose-Response Relationship, Drug | Mice, Mutant Strains | Pneumonia - chemically induced | Bronchial Hyperreactivity - physiopathology | Bronchial Hyperreactivity - metabolism | rho-Associated Kinases - biosynthesis | Oxidants, Photochemical - adverse effects | Oxidants, Photochemical - pharmacology | Pneumonia - genetics | Bronchial Hyperreactivity - chemically induced | Ozone - adverse effects | Interleukin-17 - genetics | rho-Associated Kinases - genetics | Muscle, Smooth - metabolism | Interleukin-17 - metabolism | Bronchial Hyperreactivity - pathology | Animals | Ozone - pharmacology | Muscle Contraction - drug effects | Muscle, Smooth - physiopathology | Mice | Pneumonia - metabolism | Muscle, Smooth - pathology | Physiological aspects | Air pollution | Ozone | Health aspects | Airway (Medicine) | Smooth muscle | Physiology | Kinases | Rodents | Cell adhesion & migration | Index Medicus
Journal Article
Respiratory Research, ISSN 1465-9921, 10/2013, Volume 14, Issue 1, pp. 105 - 105
Background: Asthma is a chronic disease that is characterized by airway hyperresponsiveness and airway remodeling. The underlying mechanisms that mediate the... 
Tyrosine kinase | Airway remodeling | Airway smooth muscle | Airway hyperresponsiveness | ACTIVATION | PHOSPHORYLATION | ALPHA | ACTIN DYNAMICS | IL-13 | VIMENTIN | RESPIRATORY SYSTEM | ASTHMA | REQUIRES | SMOOTH-MUSCLE-CELLS | Asthma - metabolism | Humans | Myocytes, Smooth Muscle - pathology | Male | Proto-Oncogene Proteins c-abl - physiology | Bronchi - drug effects | Bronchial Hyperreactivity - physiopathology | Ovalbumin - adverse effects | Bronchi - metabolism | Bronchial Hyperreactivity - metabolism | Female | Benzamides - pharmacology | Chemokine CCL2 - metabolism | Airway Remodeling - physiology | Bronchi - pathology | Disease Models, Animal | Bronchial Hyperreactivity - chemically induced | Proto-Oncogene Proteins c-abl - deficiency | Proto-Oncogene Proteins c-abl - genetics | Asthma - physiopathology | Mice, Inbred C57BL | Cells, Cultured | Myocytes, Smooth Muscle - physiology | Pyrimidines - pharmacology | Interleukin-13 - metabolism | Imatinib Mesylate | Piperazines - pharmacology | Asthma - chemically induced | Mice, Knockout | Animals | Mice | Protein Kinase Inhibitors - pharmacology | In Vitro Techniques | Proliferating Cell Nuclear Antigen - metabolism | Tyrosine | Medical research | Analysis | Albumin | Medicine, Experimental | Gene expression | Asthma | Cell culture | Cell growth | Airway management | Pathogenesis | Rodents | Smooth muscle | Muscular system | Chemokines | Index Medicus | Research
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 11/2018, Volume 315, Issue 5, pp. L787 - L798
Journal Article
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 10/2017, Volume 174, Issue 19, pp. 3346 - 3358
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 08/2015, Volume 309, Issue 3, pp. L205 - L210
Journal Article