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Cancer Research, ISSN 0008-5472, 04/2006, Volume 66, Issue 8, pp. 4273 - 4278
Journal Article
Journal Article
Current Molecular Medicine, ISSN 1566-5240, 2014, Volume 14, Issue 2, pp. 246 - 253
The caspases are a family of ubiquitously expressed cysteine proteases best known for their roles in programmed cell death. However, caspases play a number of... 
Calpain | Phosphorylation | Caspase 8 | SH2 domain | Nonapoptotic | Cell migration | CANCER-CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | X-LINKED INHIBITOR | TYROSINE PHOSPHORYLATION | OVARIAN-CANCER | caspase 8 | SIGNALING PATHWAY | cell migration | TRAIL-INDUCED APOPTOSIS | GROWTH-FACTOR | RAB PROTEINS | phosphorylation | NF-KAPPA-B | nonapoptotic | NEUROBLASTOMA METASTASIS
Journal Article
Cold Spring Harbor Perspectives in Biology, ISSN 1943-0264, 09/2017, Volume 9, Issue 9, p. a022046
Journal Article
by Kim, S and Lee, J and You, D and Jeong, Y and Jeon, M and Yu, J and Kim, SW and Nam, SJ and Lee, JE
CELLULAR PHYSIOLOGY AND BIOCHEMISTRY, ISSN 1015-8987, 2018, Volume 45, Issue 2, pp. 795 - 807
Background/Aims: Transforming growth factor-beta proteins (TGF-beta s) are multifunctional growth factors and powerful modulators of the epithelial-mesenchymal... 
Berberine | PHYSIOLOGY | TGF-BETA | TPA | TGF-beta 1 | Metastasis | CELL BIOLOGY | TO-MESENCHYMAL TRANSITION | GROWTH-FACTOR-BETA | INHIBITION | MMP-2 | SIGNALING PATHWAY | INDUCED MMP-9 | MMP-9 | EXPRESSION | MDA-MB-231
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2012, Volume 7, Issue 2, p. e31714
Background: Protein kinase C (PKC) epsilon, a key signaling transducer implicated in mitogenesis, survival, and cancer progression, is overexpressed in human... 
BREAST-CANCER | INVASION | CANCER CELLS | EPITHELIAL-CELLS | INDUCED APOPTOSIS | BIOLOGY | PROTEOLYTIC-ENZYMES | GROWTH-FACTOR | NUCLEOTIDE-EXCHANGE FACTORS | RHO-GTPASES | MURINE MAMMARY CELLS | Carcinoma, Non-Small-Cell Lung - pathology | Signal Transduction | Humans | Lung Neoplasms - metabolism | Carcinoma, Non-Small-Cell Lung - metabolism | Gene Expression Regulation, Neoplastic | Extracellular Matrix - metabolism | rac GTP-Binding Proteins - metabolism | Lung Neoplasms - pathology | Male | Guanosine Triphosphate - metabolism | Disease Progression | Neoplasm Metastasis | Animals | RNA Interference | Mice, Nude | Protein Kinase C-epsilon - metabolism | Cell Line, Tumor | Mice | Carcinoma - metabolism | Enzyme Activation | Carcinoma - pathology | Cell Movement | Epidermal growth factor | Protease inhibitors | Proteases | Analysis | Development and progression | Metastasis | B cells | Lung cancer, Non-small cell | Protein kinases | Protein kinase C | Phosphorylation | Motility | Lung cancer | Activation | Kinases | Metastases | Cell adhesion & migration | Proteins | Cell activation | Cell cycle | Extracellular matrix | Growth factors | Cell survival | Small cell lung carcinoma | Secretion | Invasiveness | Therapeutic applications | RNA-mediated interference | Non-small cell lung carcinoma | Rac1 protein | Pharmacology | Proteinase inhibitors | Lung carcinoma | Signaling | Lungs | Acetic acid | Phorbol 12-myristate 13-acetate | Cell migration | Cancer | Guanosinetriphosphatase | Apoptosis
Journal Article
Oncogene, ISSN 0950-9232, 10/2009, Volume 28, Issue 42, pp. 3714 - 3722
Journal Article
Oncogene, ISSN 0950-9232, 02/2012, Volume 31, Issue 5, pp. 595 - 610
Semaphorins are implicated in glioma progression, although little is known about the underlying mechanisms. We have reported plexin-B3 expression in human... 
glial differentiation | fascin | glioblastoma | invasion | GTPases | INDUCED ANGIOGENESIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PANCREATIC-CANCER | AXON GUIDANCE | CELL BIOLOGY | R-RAS | COMPLEMENTARY-DNA | INVASIVE GROWTH | ONCOLOGY | HUMAN ASTROCYTOMA-CELLS | FIBRILLARY ACIDIC PROTEIN | GENETICS & HEREDITY | GENE-EXPRESSION | TUMOR-GROWTH | Phosphorylation | Immunoprecipitation | Humans | Focal Adhesions | Immunoblotting | Glial Fibrillary Acidic Protein - metabolism | Glioma - metabolism | Glioma - genetics | Astrocytoma - pathology | RNA Interference | Protein Kinase C - metabolism | Glioma - pathology | HEK293 Cells | Membrane Proteins - metabolism | Microfilament Proteins - metabolism | Microfilament Proteins - genetics | Astrocytoma - genetics | Astrocytoma - metabolism | Actin Cytoskeleton - metabolism | Membrane Proteins - genetics | Neural Cell Adhesion Molecules - genetics | Nerve Tissue Proteins - genetics | Protein Transport | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Two-Hybrid System Techniques | Carrier Proteins - metabolism | Fluorescent Antibody Technique | Neural Cell Adhesion Molecules - metabolism | Cell Line, Tumor | Protein Binding | rac1 GTP-Binding Protein - metabolism | Cell Movement | rac1 GTP-Binding Protein - genetics | Cellular proteins | Motility | Semaphorins | Gliomas | Development and progression | Cell differentiation | Health aspects | Guanosine triphosphatase | Cells | Proteins | Cytoskeleton | Amino acids | Gene expression | Protein kinase C | semaphorins | Invasiveness | Brain tumors | Glial fibrillary acidic protein | Cytology | Rac1 protein | Fibers | Glioma | Actin | Glioma cells | Differentiation | Protein transport | Cell body | Astrocytoma | Cell migration
Journal Article
Gastroenterology, ISSN 0016-5085, 2012, Volume 142, Issue 4, pp. 928 - 937.e3
Background & Aims Myofibroblasts are the primary cell type involved in physiologic wound healing and its pathologic counterpart, fibrosis. Cellular fibronectin... 
Gastroenterology and Hepatology | Cirrhosis | Extracellular Matrix | Animal Model | Liver Disease | RAT-LIVER | ALTERNATIVELY SPLICED EIIIB | LIVER FIBROSIS | GROWTH-FACTOR-BETA | TRANSFORMING GROWTH-FACTOR-BETA-1 | ENDOTHELIAL-CELLS | EXTRACELLULAR-MATRIX | SUBSTRATE STIFFNESS | GASTROENTEROLOGY & HEPATOLOGY | PORTAL FIBROBLASTS | ISOFORM DISTRIBUTION | Hydrogels | Hepatic Stellate Cells - metabolism | Male | Myofibroblasts - metabolism | Liver Cirrhosis, Experimental - prevention & control | Ligation | Fibronectins - deficiency | Liver Cirrhosis, Biliary - etiology | Liver Cirrhosis, Experimental - pathology | Cell Transdifferentiation | Female | Cell Culture Techniques | Hepatic Stellate Cells - pathology | Fibroblasts - metabolism | Myofibroblasts - pathology | Mice, Inbred C57BL | Cells, Cultured | Rats | Integrin alpha Chains - metabolism | Liver Cirrhosis, Biliary - prevention & control | Bile Ducts - surgery | Liver Cirrhosis, Biliary - pathology | Fibroblasts - pathology | Rats, Sprague-Dawley | Fibronectins - metabolism | Mice, Knockout | Integrin beta1 - metabolism | Thioacetamide | Animals | Pseudopodia - metabolism | Liver Cirrhosis, Experimental - chemically induced | Protein Isoforms | Fibronectins - genetics | Mice | Liver Cirrhosis, Biliary - metabolism | Liver Cirrhosis, Experimental - metabolism | Cell Movement | Fibronectins | Medical colleges | Platelet-derived growth factor | Liver diseases | Actin | Analysis | Muscle proteins | Transforming growth factors | liver disease | animal model | extracellular matrix | cirrhosis
Journal Article