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1. Full Text Retinal neuroprotective effects of quercetin in streptozotocin-induced diabetic rats
by Kumar, Binit and Gupta, Suresh Kumar and Nag, Tapas Chandra and Srivastava, Sushma and Saxena, Rohit and Jha, Kumar Abhiram and Srinivasan, Bharthu Parthasarthy
Experimental Eye Research, ISSN 0014-4835, 08/2014, Volume 125, pp. 193 - 202
The aim of the present study was to evaluate the effects of Quercetin (Qctn), a plant based flavonol, on retinal oxidative stress, neuroinflammation and... 
quercetin | apoptosis | bioflavonoid | aquaporin-4 | neuroinflammation | GFAP | Neuroinflammation | Aquaporin-4 | Bioflavonoid | Quercetin | Apoptosis | OXIDATIVE STRESS | ACTIVATION | THERAPEUTIC IMPLICATIONS | FLAVONOIDS | DEATH | GLIAL-CELLS | OPHTHALMOLOGY | RETINOPATHY | COMPLICATIONS | MICE | MULLER CELLS | Retina - drug effects | Tumor Necrosis Factor-alpha - metabolism | Diabetic Retinopathy - drug therapy | Microglia - metabolism | Diabetes Mellitus, Experimental - drug therapy | Retina - metabolism | Rats, Wistar | Apoptosis - drug effects | Glutathione - metabolism | Antioxidants - metabolism | Caspase 3 - metabolism | Male | Neuroprotective Agents - pharmacology | Interleukin-1beta - metabolism | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Superoxide Dismutase - metabolism | Rats | Antioxidants - pharmacology | Diabetic Retinopathy - metabolism | Catalase - metabolism | Animals | Analysis of Variance | Quercetin - pharmacology | Aquaporin 4 - metabolism | Oxidative Stress - drug effects | Antioxidants | Enzymes | Flavonoids | Flavones | Streptozocin | Bioflavonoids | Aquaporins | Superoxide | Diabetes | Intermediate filament proteins | Restaurants
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2. Full Text Skin changes in streptozotocin-induced diabetic rats
by Andrade, Thiago Antônio Moretti and Masson-Meyers, Daniela Santos and Caetano, Guilherme Ferreira and Terra, Vânia Aparecida and Ovidio, Paula Payão and Jordão-Júnior, Alceu Afonso and Frade, Marco Andrey Cipriani
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2017, Volume 490, Issue 4, pp. 1154 - 1161
Diabetes can cause serious health complications, which can affect every organ of the body, including the skin. The molecular etiology has not yet been... 
Diabetic complication | Computer-assisted image processing | Insulin pathway | Inflammation | Diabetes mellitus | Skin homeostasis | OXIDATIVE STRESS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | NEUTROPHILS | TIME | MELLITUS | BIOPHYSICS | ONCOSTATIN-M | WOUNDS | GROWTH | COMPLICATIONS | DURATION | Animals | Rats, Wistar | Skin - metabolism | Streptozocin | Diabetes Mellitus, Experimental - pathology | Rats | Male | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Skin - pathology | Disease Models, Animal | Proteins | Antioxidants | Medical colleges | Skin diseases | Skin | Growth factors | Medical research | Medicine, Experimental | Diabetes
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3. Full Text microRNAs in early diabetic retinopathy in streptozotocin-induced diabetic rats
by Kovacs, Beatrix and Lumayag, Stephen and Cowan, Colleen and Xu, Shunbin
Investigative Ophthalmology and Visual Science, ISSN 0146-0404, 06/2011, Volume 52, Issue 7, pp. 4402 - 4409
PURPOSE. Diabetic retinopathy (DR) is one of the leading causes of blindness. However, the roles of microRNAs (miRNAs) in DR are still unknown. The aims of... 
ACTIVATION | ANGIOGENESIS | MECHANISM | PHOSPHORYLATION | ENDOTHELIAL-CELLS | OPHTHALMOLOGY | MEMBRANE-PROTEIN | FACTOR-KAPPA-B | RETINA | INHIBITOR | EXPRESSION | Cell Line | Retina - metabolism | Diabetes Mellitus, Experimental - genetics | Rats | Male | MicroRNAs - biosynthesis | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Diabetic Retinopathy - genetics | Animals | Diabetic Retinopathy - chemically induced | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Diabetes Mellitus, Experimental - chemically induced | MicroRNAs - genetics | Retina - pathology
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4. Full Text Intermedin protects against myocardial ischemia-reperfusion injury in diabetic rats
by Li, Hong and Bian, Yunfei and Zhang, Nana and Guo, Jia and Wang, Cheng and Lau, Wayne Bond and Xiao, Chuanshi
Cardiovascular Diabetology, ISSN 1475-2840, 06/2013, Volume 12, Issue 1, pp. 91 - 91
Background: Diabetic patients, through incompletely understood mechanisms, endure exacerbated ischemic heart injury compared to non-diabetic patients.... 
Ischemia-reperfusion | Inflammatory | Oxidative stress | Diabetes | Intermedin | Apoptosis | NADPH OXIDASE | ISCHEMIA/REPERFUSION INJURY | METABOLIC SYNDROME | CARDIAC & CARDIOVASCULAR SYSTEMS | NITRIC-OXIDE SYNTHASE | CARDIAC DYSFUNCTION | ACUTE CORONARY SYNDROME | INFARCT SIZE | SIGNALING PATHWAY | ENDOCRINOLOGY & METABOLISM | CARDIOVASCULAR-DISEASE | Diabetes Mellitus, Experimental - drug therapy | Diabetic Cardiomyopathies - metabolism | Apoptosis - drug effects | Streptozocin | Male | Adrenomedullin - pharmacology | Diabetic Cardiomyopathies - physiopathology | Myocardial Reperfusion Injury - pathology | Time Factors | Myocardial Infarction - pathology | Neuropeptides - pharmacology | Inflammation Mediators - metabolism | Diabetic Cardiomyopathies - prevention & control | Myocardial Infarction - physiopathology | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Diabetes Mellitus, Experimental - physiopathology | Cytokines - metabolism | Ventricular Function, Left - drug effects | Neuropeptides - metabolism | Adrenomedullin - metabolism | Cardiotonic Agents - pharmacology | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - physiopathology | Myocardial Reperfusion Injury - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Diabetes Mellitus, Experimental - pathology | Myocytes, Cardiac - metabolism | Diabetic Cardiomyopathies - pathology | Myocardial Infarction - prevention & control | Oxidative Stress - drug effects | Myocardial Reperfusion Injury - prevention & control | Proteins | Hyperglycemia | Heart attacks | Mortality | Nitric oxide | Rodents | Cardiovascular disease
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5. Full Text Berberine improves glucose metabolism in diabetic rats by inhibition of hepatic gluconeogenesis
by Xia, Xuan and Yan, Jinhua and Shen, Yunfeng and Tang, Kuanxiao and Yin, Jun and Zhang, Yanhua and Yang, Dongjie and Liang, Hua and Ye, Jianping and Weng, Jianping
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 2, p. e16556
Berberine (BBR) is a compound originally identified in a Chinese herbal medicine Huanglian (Coptis chinensis French). It improves glucose metabolism in type 2... 
ACTIVATED PROTEIN-KINASE | MECHANISM DISTINCT | INSULIN-RESISTANCE | MUSCLE ATROPHY | LIVER | BIOLOGY | CARBOHYDRATE | ELEMENT-BINDING PROTEIN | FOXO TRANSCRIPTION FACTORS | LIPID HOMEOSTASIS | EXPRESSION | Diabetes Mellitus, Experimental - drug therapy | Streptozocin | Gluconeogenesis - drug effects | Male | Diabetes Mellitus, Type 2 - metabolism | Insulin - blood | Diabetes Mellitus, Experimental - blood | Liver - drug effects | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Drug Evaluation, Preclinical | Hypoglycemic Agents - therapeutic use | Blood Glucose - analysis | Liver - metabolism | Rats | Down-Regulation - drug effects | Rats, Sprague-Dawley | Hypoglycemic Agents - pharmacology | Blood Glucose - drug effects | Berberine - pharmacology | Diabetes Mellitus, Type 2 - blood | Insulin - metabolism | Animals | Glucose - metabolism | Berberine - therapeutic use | Diabetes Mellitus, Type 2 - chemically induced | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Type 2 diabetes | Phosphatases | Blood sugar | Genes | DNA binding proteins | Muscle proteins | Fatty acids | Insulin | Glucose metabolism | Synthesis | Adenylic acid | Protein kinases | Adenosine triphosphate | Protein binding | Transcription factors | Peptides | Liver | Glucose | Blood | Proteins | Signal transduction | Mitochondria | Hyperglycemia | Forkhead protein | Inhibition | Drug dosages | Gluconeogenesis | Binding | Carbohydrates | Berberine | AMP | Metabolism | Fatty-acid synthase | Hepatocytes | Protein synthesis | Glucose-6-phosphatase | Endocrinology | Biomedical research | Kinases | High fat diet | Fatty liver | FOXO1 protein | Rodents | Statins | Fasting | Adenosine monophosphate | Diabetes mellitus | Oxygen consumption | Steatosis | Herbal medicine | Musculoskeletal system | Signaling | Protein kinase | Adenosine kinase | Coronary vessels | Insulin resistance | Diabetes | Respiration | Laboratory animals | ATP
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6. Full Text Dexmedetomidine enhances ropivacaine-induced sciatic nerve injury in diabetic rats
by Yu, Z.Y and Geng, J and Li, Z.Q and Li, M and Sun, Y.B and Wang, S.L and Wang, D.X and Masters, J and Guo, X.Y and Ma, D
British Journal of Anaesthesia, ISSN 0007-0912, 01/2019, Volume 122, Issue 1, pp. 141 - 149
Previous studies suggest that dexmedetomidine has a protective effect against local anaesthetic-induced nerve injury in regional nerve blocks. Whether this... 
local anaesthetics | diabetes mellitus | neurotoxicity | peripheral nerve injuries | PERINEURAL DEXMEDETOMIDINE | BLOCK | REGIONAL ANESTHESIA | BUPIVACAINE | NEUROPATHY | INFLAMMATION | LIDOCAINE | ANESTHESIOLOGY | DURATION | ANALGESIA | Sciatic Nerve - injuries | Diabetic Neuropathies - etiology | Dexmedetomidine - toxicity | Male | Nerve Block - methods | Anesthetics, Local - toxicity | Rats, Sprague-Dawley | Drug Synergism | Nerve Block - adverse effects | Peripheral Nerve Injuries - etiology | Animals | Peripheral Nerve Injuries - physiopathology | Peripheral Nerve Injuries - chemically induced | Adjuvants, Anesthesia - toxicity | Ropivacaine - toxicity | Inflammation Mediators - metabolism | Diabetes Mellitus, Experimental - complications | Neural Conduction - drug effects | Sciatic Nerve - drug effects | Cytokines - biosynthesis | Diabetes Mellitus, Experimental - physiopathology | Diabetic Neuropathies - physiopathology | Index Medicus
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7. Effect of Azadirachta indica   leaf extract on serum lipid profile changes in normal and streptozotocin induced diabetic rats
by Chattopadhyay, R. R and Bandyopadhyay, M
Effect of Azadirachta indica   leaf extract on serum lipid profile changes in normal and streptozotocin induced diabetic rats, 12/2005
Effects of Azadirachta indica   leaf extract on serum lipid profile changes in normal and streptozotocin - induced diabetic rats have been studied with a view... 
cardiovascular disease | Streptozotocin induced diabetes | Serum lipid profiles | Azadirachta indica
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8. Full Text Curcumin enhances recovery of pancreatic islets from cellular stress induced inflammation and apoptosis in diabetic rats
by Rashid, Kahkashan and Sil, Parames C
Toxicology and Applied Pharmacology, ISSN 0041-008X, 02/2015, Volume 282, Issue 3, pp. 297 - 310
The phytochemical, curcumin, has been reported to play many beneficial roles. However, under diabetic conditions, the detail mechanism of its beneficial action... 
Oxidative and endoplasmic reticulum (ER) stress | Inflammation | Curcumin | ER/mitochondrial dependent and independent apoptosis | Diabetes | Antioxidant | ACTIVATED PROTEIN-KINASE | D-SACCHARIC ACID-1,4-LACTONE | BETA-CELLS | GLUCOSE-TRANSPORT | SIGNALING PATHWAY | INDUCED OXIDATIVE STRESS | PROTECTIVE ROLE | IN-VIVO | PHARMACOLOGY & PHARMACY | TOXICOLOGY | STREPTOZOTOCIN | NF-KAPPA-B | Diabetes Mellitus, Experimental - drug therapy | Rats, Wistar | Apoptosis - drug effects | Male | NF-kappa B - metabolism | Insulin - blood | Glucose Transporter Type 2 - metabolism | Diabetes Mellitus, Experimental - blood | Anti-Inflammatory Agents - therapeutic use | Diabetes Mellitus, Experimental - metabolism | Heme Oxygenase (Decyclizing) - metabolism | Cytokines - blood | Hypoglycemic Agents - therapeutic use | Cell Survival - drug effects | Blood Glucose - analysis | Curcumin - therapeutic use | Cytokines - metabolism | Anti-Inflammatory Agents - pharmacology | Curcumin - pharmacology | Pancreas - drug effects | Pancreas - pathology | Antioxidants - pharmacology | Pancreas - metabolism | Hypoglycemic Agents - pharmacology | Antioxidants - therapeutic use | Animals | NF-E2-Related Factor 2 - metabolism | Oxidative Stress - drug effects | Nitric Oxide - metabolism | Nitric Oxide Synthase Type II - metabolism | Glucose metabolism | Pancreatic beta cells | Blood sugar | Apoptosis | Index Medicus | APOPTOSIS | CURCUMIN | MATERIALS RECOVERY | MITOCHONDRIA | RIBOSE | PANCREAS | RATS | 60 APPLIED LIFE SCIENCES | STRESSES | INSULIN | LYMPHOKINES | GLUCOSE | INFLAMMATION | DIABETES MELLITUS
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9. Full Text Effect of Conversion to CTLA4Ig on Tacrolimus-Induced Diabetic Rats
by Jin, Long and Lim, Sun Woo and Jin, Jian and Luo, Kang and Ko, Eun Jeong and Chung, Byung Ha and Lin, Hong Li and Yang, Chul Woo
Transplantation, ISSN 0041-1337, 04/2018, Volume 102, Issue 4, pp. e137 - e146
BACKGROUNDThe effect of conversion to cytotoxic T lymphocyte-associated protein 4 immunoglobulin (CTLA4Ig) treatment on tacrolimus (TAC)-induced renal... 
SURGERY | OXIDATIVE STRESS | PHASE-III | RENAL-TRANSPLANTATION | MELLITUS | IMMUNOLOGY | TRANSPLANTATION | BELATACEPT | KIDNEY-TRANSPLANTATION | PANCREATIC BETA-CELLS | COSTIMULATION | DYSFUNCTION | CYCLOSPORINE | Abatacept - pharmacology | Diabetes Mellitus - blood | Diabetes Mellitus - pathology | Tacrolimus - toxicity | Cell Line | Islets of Langerhans - drug effects | Islets of Langerhans - pathology | Macrophages - pathology | Apoptosis - drug effects | Male | Biomarkers - blood | Immunosuppressive Agents - toxicity | Insulin - blood | Rats, Sprague-Dawley | Blood Glucose - drug effects | Abatacept - toxicity | Macrophages - metabolism | Animals | Islets of Langerhans - metabolism | Diabetes Mellitus - chemically induced | Macrophages - drug effects | Oxidative Stress - drug effects | Blood Glucose - metabolism | Drug Substitution | Development and progression | Care and treatment | Immunoglobulins | Genetic aspects | Diabetes | Health aspects
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10. Full Text Astaxanthin inhibits expression of retinal oxidative stress and inflammatory mediators in streptozotocin-induced diabetic rats
by Yeh, Po-Ting and Huang, Hsin-Wei and Huang, Hsin-Wei and Yang, Chung-May and Yang, Chung-May and Yang, Wei-Shiung and Yang, Chang-Hao and Yang, Chang-Hao
PLoS ONE, ISSN 1932-6203, 01/2016, Volume 11, Issue 1, p. e0146438
Purpose We evaluated whether orally administered astaxanthin (AST) protects against oxidative damage in the ocular tissues of streptozotocin (STZ)-induced... 
BETA-CAROTENE | IN-VITRO | HUMAN-DISEASE | DB/DB MICE | VITREOUS FLUID | MULTIDISCIPLINARY SCIENCES | EXPERIMENTAL GALACTOSEMIA | HUMAN HEALTH | HEME OXYGENASE-1 | ENDOTOXIN-INDUCED UVEITIS | NF-KAPPA-B | Retina - drug effects | Gene Expression Regulation, Enzymologic - drug effects | Diabetes Mellitus, Experimental - drug therapy | Retina - metabolism | Body Weight | NF-kappa B - metabolism | Retinal Ganglion Cells - metabolism | Aqueous Humor - metabolism | Inflammation Mediators - metabolism | Female | Chemokine CCL2 - metabolism | Diabetes Mellitus, Experimental - metabolism | Electroretinography | Gene Expression | Protective Agents | RNA, Messenger - genetics | Rats | Blood Glucose | Antioxidants - pharmacology | Intercellular Adhesion Molecule-1 - metabolism | Animals | NF-kappa B - genetics | Xanthophylls - pharmacology | Oxidative Stress - drug effects | Lutein - pharmacology | Retina - pathology | Retinal Ganglion Cells - drug effects | Complications and side effects | Oxidative stress | Streptozocin | Astaxanthin | Dosage and administration | Research | Drug therapy | Immunohistochemistry | Neuroprotection | Diabetic retinopathy | Pathogenesis | Retina | Proteins | Antioxidants | Acrolein | Carotenoids | Animal tissues | Heme | Cell adhesion | Fractalkine | Drug dosages | Enzyme-linked immunosorbent assay | NF-κB protein | Damage assessment | Cytokines | Intercellular adhesion molecule 1 | Diabetes mellitus | Oral administration | Lutein | Heme oxygenase (decyclizing) | Inflammation | Metabolism | Gene expression | Medicine | Polymerase chain reaction | Oxygenase | Hospitals | Deoxyguanosine | Monocyte chemoattractant protein | Electrophoretic mobility | Nitrotyrosine | Diabetes | Monocyte chemoattractant protein 1
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11. Full Text Hyperglycemia abrogates ischemic postconditioning cardioprotection by impairing AdipoR1/Caveolin-3/STAT3 signaling in diabetic rats
by Li, Haobo and Yao, Weifeng and Liu, Zipeng and Xu, Aimin and Huang, Yu and Ma, Xin Liang and Irwin, Michael G and Xia, Zhengyuan
Diabetes, ISSN 0012-1797, 04/2016, Volume 65, Issue 4, pp. 942 - 955
Signal transducer and activator of transcription 3 (STAT3) activation is key for ischemic postconditioning (IPo) to attenuate myocardial ischemia-reperfusion... 
OXIDATIVE STRESS | ADIPONECTIN CARDIOPROTECTION | INSULIN-RESISTANCE | PROTEIN-KINASE | REPERFUSION INJURY | ENDOCRINOLOGY & METABOLISM | MYOCARDIAL-ISCHEMIA | N-ACETYLCYSTEINE | MICE | INDUCED CARDIAC-HYPERTROPHY | DYSFUNCTION | Myocardial Reperfusion Injury - etiology | Coronary Vessels - pathology | Mice, Inbred C57BL | Hyperglycemia - complications | Rats | Receptors, Adiponectin - metabolism | Male | Rats, Sprague-Dawley | Coronary Vessels - metabolism | Mice, Knockout | Hyperglycemia - metabolism | Myocardial Reperfusion Injury - metabolism | Animals | Myocardium - metabolism | Diabetes Mellitus, Experimental - pathology | Ischemic Postconditioning | Signal Transduction - physiology | Diabetes Mellitus, Experimental - complications | Mice | Diabetes Mellitus, Experimental - metabolism | Caveolin 3 - metabolism | Myocardial Reperfusion Injury - prevention & control | STAT3 Transcription Factor - metabolism | Care and treatment | Ischemia | STAT1 | Cardiotonic agents | Influence | Dosage and administration | Diabetes | Cardiac glycosides
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12. Full Text Resveratrol prevents memory deficits and the increase in acetylcholinesterase activity in streptozotocin-induced diabetic rats
by Schmatz, Roberta and Mazzanti, Cinthia Melazzo and Spanevello, Roselia and Stefanello, Naiara and Gutierres, Jessié and Corrêa, Maisa and da Rosa, Michelle Melgarejo and Rubin, Maribel A and Chitolina Schetinger, Maria Rosa and Morsch, Vera Maria
European Journal of Pharmacology, ISSN 0014-2999, 2009, Volume 610, Issue 1, pp. 42 - 48
The objective of the present study was to investigate the effect of the administration of resveratrol (RV) on memory and on acetylcholinesterase (AChE)... 
Diabetes | Rat | Acetylcholinesterase | Streptozotocin | Memory | Resveratrol | OXIDATIVE STRESS | ALZHEIMERS-DISEASE | MELLITUS | SYNAPTIC PLASTICITY | NEUROTOXICITY | HIPPOCAMPUS | TRANSMISSION | BRAIN ACETYLCHOLINESTERASE | ENZYME | PHARMACOLOGY & PHARMACY | CEREBRAL-BLOOD-FLOW | Memory - drug effects | Memory Disorders - enzymology | Blood Glucose - analysis | Diabetes Mellitus, Experimental - enzymology | Rats | Random Allocation | Stilbenes - pharmacology | Streptozocin - pharmacology | Diabetes Mellitus, Experimental - blood | Dose-Response Relationship, Drug | Memory Disorders - prevention & control | Animals | Cholinesterase Inhibitors - pharmacology | Behavior, Animal - drug effects | Avoidance Learning - drug effects | Acetylcholinesterase - metabolism | Memory Disorders - chemically induced | Index Medicus
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13. Full Text Luteolin limits infarct size and improves cardiac function after myocardium ischemia/reperfusion injury in diabetic rats
by Sun, Dongdong and Huang, Jie and Zhang, Zheng and Gao, Haokao and Li, Jiayi and Shen, Min and Cao, Feng and Wang, Haichang
PLoS ONE, ISSN 1932-6203, 03/2012, Volume 7, Issue 3, p. e33491
Background: The present study was to investigate the effects and mechanism of Luteolin on myocardial infarct size, cardiac function and cardiomyocyte apoptosis... 
SURVIVAL | INDUCED APOPTOSIS | CARDIOMYOCYTE | PHOSPHORYLATION | ANTIOXIDANT | MULTIDISCIPLINARY SCIENCES | ISCHEMIA | FLAVONOIDS | CONTRACTILE FUNCTION | INHIBITORY FACTOR | PROTECTS | L-Lactate Dehydrogenase - metabolism | Leukocytes - pathology | Apoptosis - drug effects | Myocardial Reperfusion Injury - complications | Male | Myocardial Infarction - diagnostic imaging | Arrhythmias, Cardiac - physiopathology | Arrhythmias, Cardiac - pathology | Myocardial Infarction - pathology | Ultrasonography | Myocardial Infarction - physiopathology | Diabetes Mellitus, Experimental - complications | Myocardial Reperfusion Injury - drug therapy | Diabetes Mellitus, Experimental - physiopathology | Arrhythmias, Cardiac - prevention & control | Cytokines - metabolism | Ventricular Function, Left - drug effects | Rats | Luteolin - therapeutic use | Luteolin - pharmacology | Rats, Sprague-Dawley | Myocardial Reperfusion Injury - physiopathology | Cell Movement - drug effects | Myocardial Reperfusion Injury - diagnostic imaging | Myocytes, Cardiac - pathology | Animals | Myocardial Infarction - drug therapy | Myocytes, Cardiac - drug effects | Leukocytes - drug effects |