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Neurotoxicology and Teratology, ISSN 0892-0362, 2011, Volume 33, Issue 5, pp. 592 - 597
Journal Article
Biological Trace Element Research, ISSN 0163-4984, 2/2019, Volume 187, Issue 2, pp. 341 - 356
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2017, Volume 104, pp. 1 - 14
Journal Article
Journal of NeuroVirology, ISSN 1355-0284, 2/2014, Volume 20, Issue 1, pp. 39 - 53
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2015, Volume 10, Issue 6, p. e0129881
Background Little is known about the effects of induced pluripotent stem cell (iPSC) treatment on acute cerebral inflammation and injuries after intracerebral... 
EXPERIMENTAL INTRACEREBRAL HEMORRHAGE | FUNCTIONAL RECOVERY | THERAPY | MODELS | MULTIDISCIPLINARY SCIENCES | INJURY | RATS | NEURONS | MECHANISMS | ISCHEMIC-STROKE | BRAIN | Inflammation - pathology | Neurons - pathology | Brain - cytology | Induced Pluripotent Stem Cells - transplantation | Neurons - immunology | Male | Neurons - cytology | Inflammation - immunology | Cerebral Hemorrhage - immunology | Neurogenesis | Rats, Sprague-Dawley | Interleukins - analysis | Tumor Necrosis Factor-alpha - analysis | Inflammation - therapy | Animals | Interleukins - immunology | Tumor Necrosis Factor-alpha - immunology | Brain - pathology | Cerebral Hemorrhage - therapy | Induced Pluripotent Stem Cells - cytology | Brain - immunology | Cerebral Hemorrhage - pathology | Apoptosis | Stroke (Disease) | Cytokines | Analysis | Stem cells | Inflammation | Transplantation | Intermediate filament proteins | Health aspects | Brain | Neuroprotection | Neurosciences | Laboratories | Interleukin | Neurobiology | Recovery of function | Stem cell transplantation | Hemorrhage | Neuromodulation | Neuronal-glial interactions | Interleukin 6 | Proteins | Ischemia | Moisture content | Rodents | Animal tissues | Water content | Neostriatum | Fibroblasts | Injuries | Edema | Stroke | Cell survival | Glial fibrillary acidic protein | Rats | Leukocytes (neutrophilic) | Collagenase | Survival | Microglia | Medicine | Neurology | Hospitals | Collagen | Interleukin 10 | Clinical medicine | Pluripotency | Brain injury | Tumors | Inhibitory postsynaptic potentials
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 09/2006, Volume 98, Issue 5, pp. 1353 - 1368
Increased levels of interleukin‐6 (IL‐6) play a role in post‐ischemic cerebral inflammation. IL‐6 binding to its receptors induces phosphorylation of the... 
infarction | suppressor of cytokine signaling | transcription factor | inflammation | stroke | interluekin‐6 | Suppressor of cytokine signaling | Stroke | Infarction | Inflammation | Transcription factor | Interluekin-6 | RNA INTERFERENCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | INDUCED INFLAMMATION | NEUROSCIENCES | interluekin-6 | GENE | PATHWAY | IN-VIVO | RAT-BRAIN | GROWTH-FACTOR | EXPRESSION | SIGNAL TRANSDUCER | Neurons - pathology | Protein-Tyrosine Kinases - metabolism | RNA, Messenger - metabolism | Janus Kinase 2 | Protein-Tyrosine Kinases - genetics | Time Factors | Reverse Transcriptase Polymerase Chain Reaction - methods | Neurons - metabolism | Cell Death - drug effects | Neurons - drug effects | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Ischemic Attack, Transient - pathology | Proto-Oncogene Proteins - metabolism | Blotting, Western - methods | RNA, Small Interfering - pharmacology | Enzyme Inhibitors - pharmacology | Rats | Proto-Oncogene Proteins - genetics | In Situ Nick-End Labeling - methods | Tyrphostins - pharmacology | Enzyme Activation - drug effects | Immunohistochemistry - methods | Nerve Tissue Proteins - metabolism | Animals | Analysis of Variance | Cell Death - physiology | Neurologic Examination - methods | Reperfusion - methods | Enzyme Activation - physiology | Ischemia | Neurons | Enzymes | Brain damage | Inflammatory diseases
Journal Article
Hippocampus, ISSN 1050-9631, 05/2016, Volume 26, Issue 5, pp. 658 - 667
Sprouty (Spry) proteins play a key role as negative feedback inhibitors of the Ras/Raf/MAPK/ERK pathway downstream of various receptor tyrosine kinases. Among... 
neurodegeneration | gliosis | hippocampus | granule cell dispersion | Sprouty | ERK | Granule cell dispersion | Gliosis | Neurodegeneration | Hippocampus | MESSENGER-RNA EXPRESSION | REELIN | PHOSPHORYLATION | RECEPTOR | PROLIFERATION | ERK ACTIVATION | NEUROSCIENCES | RAT DENTATE GYRUS | EPILEPSY | DIFFERENTIATION | Neurons - pathology | Nerve Tissue Proteins - deficiency | Cell Survival - genetics | Gliosis - chemically induced | Brain Injuries - complications | Male | Intracellular Signaling Peptides and Proteins - metabolism | Hippocampus - drug effects | RNA, Messenger - metabolism | Seizures - chemically induced | Gliosis - pathology | Intracellular Signaling Peptides and Proteins - deficiency | Membrane Proteins - deficiency | Pentylenetetrazole - toxicity | Female | Neurons - metabolism | Kainic Acid - toxicity | Intracellular Signaling Peptides and Proteins - genetics | Disease Models, Animal | Cell Survival - drug effects | Gene Expression Regulation - genetics | Membrane Proteins - genetics | Brain Injuries - chemically induced | Mice, Transgenic | Hippocampus - pathology | Signal Transduction - genetics | Nerve Tissue Proteins - genetics | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Animals | Excitatory Amino Acid Agonists - toxicity | Analysis of Variance | Signal Transduction - drug effects | Mice | Brain Injuries - pathology | Neurons | Analysis
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 01/2004, Volume 24, Issue 3, pp. 671 - 678
Mechanisms of selective neuronal death in the hippocampus after global cerebral ischemia remain to be clarified. Here, we explored a possible role for matrix... 
Neuroprotection | Neuroinflammation | Stroke | Mouse |