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Cellular Signalling, ISSN 0898-6568, 08/2019, Volume 60, pp. 39 - 56
Interferon-induced transmembrane proteins IFITM1 and IFITM3 (IFITM1/3) play a role in both RNA viral restriction and in human cancer progression. Using... 
IFITM1 | Interferon | SILAC mass spectrometry | MHC Class I molecule | CAS9 gene editing | Cervical cancer | CELLS | SAMPLE PREPARATION | INDUCED TRANSMEMBRANE PROTEIN-1 | CELL BIOLOGY | BREAST-CANCER | OVEREXPRESSION | RESISTANCE | INTERFERONS | EXPRESSION | CARCINOMA | PROTEOMICS
Journal Article
Pathology - Research and Practice, ISSN 0344-0338, 07/2019, Volume 215, Issue 7, p. 152444
We evaluated the relationship between interferon-induced transmembrane protein 1 (IFITM1) expression, epithelial–mesenchymal transition (EMT) signature and... 
Interferon-induced transmembrane protein 1 | Microvessel | Epithelial–mesenchymal transition | Prognosis | Lung adenocarcinoma | Density | REVISION | 8TH EDITION | CLASSIFICATION | MONOCLONAL-ANTIBODY | PROLIFERATION | PATHOLOGY | INDUCED TRANSMEMBRANE PROTEIN-1 | Epithelial-mesenchymal transition | BREAST-CANCER CELLS | TNM STAGE GROUPINGS | AGGREGATION
Journal Article
Cancer Letters, ISSN 0304-3835, 2015, Volume 368, Issue 1, pp. 135 - 143
Highlights • Elevated IFITM1 expression is important in the acquisition of an aggressive phenotype and poor prognostic in CRC. • IFITM1 expression is an... 
Hematology, Oncology and Palliative Medicine | IFITM1 | Metastasis | CAV1 | Colorectal cancer | ACTIVATION | PROLIFERATION | BETA-CATENIN | INDUCED TRANSMEMBRANE PROTEIN-1 | FAMILY | ONCOLOGY | GASTRIC-CANCER | SQUAMOUS-CELL CARCINOMA | EXPRESSION SUPPRESSES GROWTH | TUMORIGENESIS | CAVEOLIN-1 | Multivariate Analysis | Colorectal Neoplasms - genetics | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Gene Knockdown Techniques | Transfection | RNA Interference | Time Factors | Antigens, Differentiation - metabolism | Female | Colorectal Neoplasms - metabolism | Caco-2 Cells | Colorectal Neoplasms - mortality | Signal Transduction | Neoplasm Invasiveness | HCT116 Cells | Proportional Hazards Models | Caveolin 1 - genetics | Caveolin 1 - metabolism | HT29 Cells | Antigens, Differentiation - genetics | Colorectal Neoplasms - pathology | Cell Movement | Medical colleges | Oncology, Experimental | Interferon | Research | Biological response modifiers | Cancer | Caveolin-1 | Cell culture | Genes | Colorectal carcinoma | Colleges & universities | Experiments | Lymph nodes | Metastases | Confidence intervals | Proteins | Inhibition | Therapeutic applications | Caveolin | Mortality | Cyclin-dependent kinases | Tumorigenicity | Patients | Pathology | Molecular modelling | Overexpression | Medical prognosis | Cell migration
Journal Article
Cancer Letters, ISSN 0304-3835, 2017, Volume 399, pp. 29 - 43
Abstract Interferon induced transmembrane protein 1 (IFITM1) belongs to a family of interferon stimulated genes (ISGs) that is associated with tumor... 
Hematology, Oncology and Palliative Medicine | Interferon stimulated genes | Aromatase inhibitor-resistance | JAK/STAT signaling | Mouse mammary intraductal model | p21 | CELLS | MECHANISM | STIMULATED GENES | PHOSPHORYLATION | INDUCED TRANSMEMBRANE PROTEIN-1 | ENDOCRINE RESISTANCE | INTERFERON-ALPHA | ONCOLOGY | PIM-1 KINASE | COLORECTAL-CANCER | GENE-EXPRESSION | Aromatase Inhibitors - pharmacology | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Drug Resistance, Neoplasm | Janus Kinases - metabolism | Breast Neoplasms - enzymology | STAT1 Transcription Factor - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Transfection | MCF-7 Cells | RNA Interference | Aged, 80 and over | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Adult | Antigens, Differentiation - metabolism | Female | Transcription, Genetic | Retrospective Studies | Antineoplastic Agents, Hormonal - pharmacology | Neoplasm Invasiveness | Down-Regulation | Neoplasm Recurrence, Local | Mice, SCID | Breast Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Cell Movement - drug effects | Animals | Breast Neoplasms - genetics | Signal Transduction - drug effects | Breast Neoplasms - pathology | Mice, Inbred NOD | Aged | Antigens, Differentiation - genetics | Cell Proliferation - drug effects | Enzyme Activation | Neoplasm Staging | Medical colleges | Cell death | Analysis | Estrogen | Physiological aspects | Development and progression | Breast cancer | Interferon | Biological response modifiers | Chromatin | GTP-binding protein | Animal models | Transcription | DNA damage | Estrogens | Estrogen receptors | Amino acids | Metastasis | Kinases | Cancer therapies | Proteins | Genotype & phenotype | Rodents | Aromatase | Cell cycle | Localization | Deoxyribonucleic acid--DNA | Endocrine therapy | Cyclin-dependent kinase inhibitor p21 | Chemotherapy | DNA microarrays | Antibiotics | Medical prognosis | Breast | Position (location) | Tumors | Cancer | Apoptosis | STAT signaling | mouse mammary intraductal model | JAK | interferon stimulated genes
Journal Article
Oncology Reports, ISSN 1021-335X, 12/2014, Volume 32, Issue 6, pp. 2648 - 2656
Interferon-induced transmembrane protein 3 (IFITM3) has been recently identified as a potential molecular marker. IFITM3 has been reported to be upregulated in... 
epithelial-tomesenchymal transition | matrix metalloproteinase | gastric cancer | Wnt/β-catenin signaling pathway | interferon-induced transmembrane protein 3 | Interferon-induced transmembrane protein 3 | Matrix metalloproteinase | Gastric cancer | Epithelial-to-mesenchymal transition | GERM-CELL FATE | METASTASIS | SPECIFICATION | IDENTIFICATION | Wnt/beta-catenin signaling pathway | INDUCED TRANSMEMBRANE PROTEIN-1 | epithelial-to-mesenchymal transition | MATRIX METALLOPROTEINASES | GENE | ONCOLOGY | EXPRESSION | CARCINOMA | PROGRESSION | Stomach Neoplasms - genetics | RNA-Binding Proteins - genetics | Cell Proliferation - genetics | Matrix Metalloproteinase 9 - biosynthesis | Membrane Proteins - genetics | Humans | Gene Expression Regulation, Neoplastic | Stomach Neoplasms - pathology | Epithelial-Mesenchymal Transition - genetics | Gene Knockdown Techniques | RNA-Binding Proteins - biosynthesis | Membrane Proteins - biosynthesis | Wnt Signaling Pathway - genetics | Cell Line, Tumor | Biomarkers, Tumor - genetics | Biomarkers, Tumor - biosynthesis | Matrix Metalloproteinase 2 - biosynthesis | Physiological aspects | Genetic aspects | Research | Gene expression | Stomach cancer | Membrane proteins | Cell culture | Pathogenesis | Genes | Metastasis | Cell adhesion & migration | Studies | Proteins | Cell growth | Rodents | Cell cycle | Tumorigenesis | Interferon
Journal Article
Cancer Science, ISSN 1347-9032, 10/2018, Volume 109, Issue 10, pp. 3115 - 3128
This research aimed to analyze the effect of IFITM1 on the radioresistance of oral neoplasm. Using a multi‐group heat map from GSE9716 analysis of the GEO... 
radiotherapy | IFITM1 | oral neoplasm | oral cancer | radioresistance | PROSTATE | CELLS | MARKER | PHENOTYPE | RADIATION | INDUCED TRANSMEMBRANE PROTEIN-1 | BREAST | INVASION | THERAPY | ONCOLOGY | COLORECTAL-CANCER | Phosphorylation | Apoptosis - radiation effects | Tissue Array Analysis | Humans | Gene Expression Regulation, Neoplastic | STAT Transcription Factors - metabolism | Apoptosis - genetics | Male | Mouth - cytology | Gene Knockdown Techniques | Antigens, Differentiation - metabolism | Mouth Neoplasms - radiotherapy | Mouth Neoplasms - genetics | Cell Proliferation - genetics | Down-Regulation | Radiation Tolerance - genetics | STAT Transcription Factors - genetics | Animals | Mice, Nude | Cell Line, Tumor | Mouth - pathology | Antigens, Differentiation - genetics | Mice | Mice, Inbred BALB C | Histones - metabolism | Dose-Response Relationship, Radiation | Cell Proliferation - radiation effects | RNA, Small Interfering - metabolism | Immunohistochemistry | DNA microarrays | Radiotherapy | Analysis | Mouth cancer | Radiation | Cell proliferation | Flow cytometry | GTP-binding protein | Oral cancer | DNA damage | Radioresistance | Cancer therapies | Stomach cancer | Ovarian cancer | Cell adhesion & migration | Proteins | Cell growth | Databases | Cell cycle | Head and neck | Conflicts of interest | Stat1 protein | Cell survival | Caspase | siRNA | Radiation therapy | Gene expression | Polymerase chain reaction | Cyclin-dependent kinase inhibitor p21 | Chemotherapy | Medical prognosis | Cell lines | Biomarkers | Fatalities | Head & neck cancer | Immunofluorescence | Apoptosis | Tumors | Original
Journal Article
Pathology & Oncology Research, ISSN 1219-4956, 1/2019, Volume 25, Issue 1, pp. 157 - 167
Journal Article
Journal Article