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Gut, ISSN 0017-5749, 02/2006, Volume 55, Issue 2, pp. 182 - 190
Background and aim: Abdominal pain and discomfort are common symptoms in functional disorders and are attributed to visceral hypersensitivity. These symptoms... 
DEXAMETHASONE | IMMUNE-SYSTEM | EXPERIMENTAL COLITIS | POSTINFECTIVE GUT DYSFUNCTION | DISEASE | LACTOBACILLUS-PARACASEI | HYPERALGESIA | DECONTAMINATION | SUBSTANCE-P | GASTROENTEROLOGY & HEPATOLOGY | IRRITABLE-BOWEL-SYNDROME | Hyperalgesia - chemically induced | Abdominal Pain - prevention & control | Culture Media, Conditioned - pharmacology | Anti-Inflammatory Agents - therapeutic use | Female | Electromyography | Anti-Bacterial Agents - toxicity | Irritable Bowel Syndrome - prevention & control | Irritable Bowel Syndrome - chemically induced | Abdominal Pain - chemically induced | Irritable Bowel Syndrome - metabolism | Hyperalgesia - metabolism | Abdominal Pain - microbiology | Hyperalgesia - prevention & control | Abdominal Pain - metabolism | Hyperalgesia - microbiology | Physical Stimulation - methods | Irritable Bowel Syndrome - microbiology | Dexamethasone - therapeutic use | Animals | Intestines - microbiology | Probiotics - therapeutic use | Substance P - metabolism | Mice | Mice, Inbred BALB C | Peroxidase - metabolism | Complications and side effects | Probiotics | Antibiotics | Viscera | Irritable bowel syndrome | Physiological aspects | Development and progression | Research | Pathogens | Drinking water | Laboratories | Infections | Inflammation | Experiments | Studies | Inflammatory bowel disease | Rodents | Bacteria | probiotic therapy | inflammation | mice | gut flora | Neurogastroenterology | visceral hypersensitivity
Journal Article
Journal Article
Experimental Neurology, ISSN 0014-4886, 11/2016, Volume 285, Issue Pt A, pp. 41 - 50
Transient receptor potential melastatin 2 (TRPM2) is a thermosensitive, Ca2+-permeable cation channel. TRPM2 contributes to the pathogenesis of inflammatory... 
Inflammatory bowel disease | TRPM2 | Sensory nerve | Enteric nervous system | Visceral hypersensitivity | CA2+ INFLUX | NERVE-FIBERS | PRIMARY AFFERENT NEURONS | ROOT GANGLION NEURONS | ADP-RIBOSE | NEUROSCIENCES | NEUROPATHIC PAIN | EXPERIMENTAL COLITIS | HYDROGEN-PEROXIDE | PRIMARY SENSORY NEURONS | TRPV1 EXPRESSION | Fluorescein-5-isothiocyanate - pharmacokinetics | Evoked Potentials, Motor - physiology | Colitis - complications | Colon - innervation | Evoked Potentials, Motor - drug effects | Male | Ganglia, Spinal - cytology | Antigens, CD - metabolism | Dextrans - pharmacokinetics | Hypersensitivity - metabolism | Colitis - chemically induced | Receptors, Purinergic P2X - metabolism | Hypersensitivity - genetics | Electromyography | Gastrointestinal Tract - innervation | Disease Models, Animal | Calcium-Binding Proteins | Calbindin 2 - metabolism | Mice, Inbred C57BL | Stilbamidines - pharmacokinetics | Rats | Fluorescein-5-isothiocyanate - analogs & derivatives | Mice, Transgenic | Integrin alpha Chains - metabolism | Visceral Pain - metabolism | Rats, Sprague-Dawley | Gastrointestinal Tract - metabolism | Nerve Tissue Proteins - metabolism | Visceral Pain - genetics | Animals | Microfilament Proteins | TRPM Cation Channels - genetics | Substance P - metabolism | Mice | TRPM Cation Channels - metabolism | Trinitrobenzenesulfonic Acid - toxicity | Visceral Pain - etiology | Allergy | Gastrointestinal diseases | Analysis | Gastrointestinal system | Physiological aspects | Colitis | Allergic reaction | Index Medicus
Journal Article
Journal Article
Journal Article
Journal Article
Neurogastroenterology & Motility, ISSN 1350-1925, 09/2017, Volume 29, Issue 9, pp. e13081 - n/a
Background Abdominal pain is one of the major symptoms of inflammatory Bowel Disease (IBD). The inflammatory mediators released by colon inflammation are known... 
sympathetic activity | IBD abdominal pain | BDNF | visceral sensitivity | INFECTIONS | RISK | DISORDERS | MECHANISMS | PREVALENCE | CHILDHOOD | NEUROSCIENCES | CLINICAL NEUROLOGY | NARCOTIC USE | INFLAMMATORY-BOWEL-DISEASE | PAIN | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | Animals, Newborn | Inflammation - chemically induced | Abdominal Pain - etiology | Spinal Cord - metabolism | Hyperalgesia - metabolism | Epigenesis, Genetic | Gene Expression Regulation - physiology | Rats | Male | Abdominal Pain - metabolism | Visceral Pain - metabolism | Rats, Sprague-Dawley | Gene Expression Regulation - drug effects | Inflammation - metabolism | Animals | Brain-Derived Neurotrophic Factor - biosynthesis | Trinitrobenzenesulfonic Acid - toxicity | Visceral Pain - etiology | Tyrosine | Infants (Newborn) | Allergy | Medical research | Epigenetic inheritance | Cyclic adenylic acid | Allergic reaction | Epidemiology | Risk factors | Acetylene | Gastrointestinal diseases | Medicine, Experimental | Hydroxylases | Neonates | Spinal cord | Inflammatory bowel diseases | Transcription | Cerebrospinal fluid | Tyrosine 3-monooxygenase | Distension | Pain | Intestine | Opioids | Sensory neurons | Remission | Colon | Nonsteroidal anti-inflammatory drugs | Immune system | Hydroxylase | Hypersensitivity | Cyclic AMP | Sulfonic acid | Abdomen | Inflammatory bowel disease | Brain-derived neurotrophic factor | Epigenetics | Adrenergic receptors | Norepinephrine
Journal Article
Journal Article