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Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2007, Volume 293, Issue 4, pp. 2210 - 2218
...B and RhoA activation and upregulation of adhesion molecules ICAM-1 and VCAM-1, increased expression of monocyte chemoattractant protein, enhanced transendothelial migration of monocytes... 
Adhesion molecules | Inflammation | RhoA | Endothelial activation | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Inflammation - chemically induced | Leukocyte Rolling - drug effects | Tumor Necrosis Factor-alpha - metabolism | Receptor, Cannabinoid, CB2 - agonists | Humans | Male | Monocytes - metabolism | NF-kappa B - metabolism | rhoA GTP-Binding Protein - metabolism | Aorta - metabolism | RNA, Messenger - metabolism | Receptor, Cannabinoid, CB2 - genetics | Coronary Vessels - metabolism | Lipopolysaccharides | Dose-Response Relationship, Drug | Inflammation - metabolism | Anti-Inflammatory Agents - therapeutic use | Chemokine CCL2 - metabolism | Disease Models, Animal | Coronary Vessels - drug effects | Anti-Inflammatory Agents - pharmacology | Endothelial Cells - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Cells, Cultured | Cannabinoids - pharmacology | Monocytes - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Cannabinoids - therapeutic use | Intercellular Adhesion Molecule-1 - metabolism | Animals | Signal Transduction - drug effects | Inflammation - prevention & control | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - drug effects | Index Medicus | inflammation | adhesion molecules | endothelial activation
Journal Article
Nature biotechnology, ISSN 1546-1696, 10/2011, Volume 29, Issue 11, pp. 1005 - 1010
Journal Article
Immunity (Cambridge, Mass.), ISSN 1074-7613, 10/2017, Volume 47, Issue 4, pp. 621 - 634
.... The lipoproteins acquire features of damage-associated molecular patterns and trigger first an innate immune response, dominated by monocyte-macrophages, and then an adaptive immune response... 
Innate and adaptive inflammatory responses drive the progression of atherosclerosis | which is the root cause of most cardiovascular disease. Tabas and Lichtman review the roles and regulation of monocyte-macrophages and T cells during the various stages of atherosclerosis and discuss how this knowledge suggests new therapeutic approaches.atherosclerosis | innate immunity | inflammation | T cell | monocyte | macrophage | effector T cell | adaptive immunity | regulatory T cell | regulatory T cell | effector T cell | Life Sciences & Biomedicine | Immunology | Science & Technology | Immunity, Innate - immunology | Models, Immunological | Animals | Atherosclerosis - immunology | Humans | T-Lymphocytes - immunology | Monocytes - immunology | Adaptive Immunity - immunology | Lipoproteins - immunology | Macrophages - immunology | T cells | Macrophages | Atherosclerosis | Development and progression | Inflammation | Blood lipids | Apolipoproteins | Effector cells | Smooth muscle | Lymphocytes T | Retention | Cell activation | Lymphocytes | Hematopoiesis | Aging | Extracellular matrix | ABC transporters | Lipid metabolism | Thromboembolism | Immune system | Immune response | Neutrophils | Aging (artificial) | Phenotypic plasticity | Metabolism | Thrombosis | Cholesterol | Cells | Hemopoiesis | Damage patterns | Studies | Monocytes | Lipoproteins | Arteriosclerosis | Infarction | Diabetes | Plastic foam | Index Medicus
Journal Article