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Clinical & Experimental Immunology, ISSN 0009-9104, 05/2013, Volume 172, Issue 2, pp. 333 - 348
Journal Article
Toxicology, ISSN 0300-483X, 2014, Volume 326, pp. 9 - 17
Abstract Galangin can suppress hepatocellular carcinoma (HCC) cell proliferation. In this study, we demonstrated that galangin induced autophagy by activating... 
Emergency | TGF-β receptor | Galangin | Smad | Autophagy | Autophagy-related gene | CANCER-CELLS | HEPATOCELLULAR-CARCINOMA CELLS | APOPTOSIS | BECLIN 1 | ENDOPLASMIC-RETICULUM STRESS | TGF-beta receptor | PROTEIN CONJUGATION SYSTEM | TUMOR-SUPPRESSOR | PHARMACOLOGY & PHARMACY | TOXICOLOGY | INHIBITS PROLIFERATION | UP-REGULATION | Apoptosis - drug effects | Humans | Autophagy - drug effects | Dose-Response Relationship, Drug | Transfection | RNA Interference | Carcinoma, Hepatocellular - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Liver Neoplasms - pathology | Antineoplastic Agents - pharmacology | Flavonoids - pharmacology | Protein-Serine-Threonine Kinases - metabolism | Pyrazoles - pharmacology | Liver Neoplasms - genetics | Smad Proteins - genetics | Apoptosis Regulatory Proteins - metabolism | Hep G2 Cells | Pyrroles - pharmacology | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Proliferation - drug effects | Smad Proteins - metabolism | Transforming Growth Factor beta - metabolism | Carcinoma, Hepatocellular - metabolism | Index Medicus | Receptors | Toxicology | Pathways | Genes | Blocking | Activation | Inhibition | Growth factors
Journal Article
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 12/2018, Volume 119, Issue 12, pp. 10131 - 10142
Journal Article
Peptides, ISSN 0196-9781, 04/2012, Volume 34, Issue 2, pp. 380 - 388
► Angiotensin-(1–7) [Ang-(1–7)] inhibited DNA, protein, and collagen synthesis in cardiac fibroblasts. ► Ang-(1–7) increased dual specificity phosphatase DUSP1... 
Cardiac fibrosis | Cyclooxygenase | Angiotensin-(1–7) | Prostaglandin E synthase | Dual specificity phosphatase | Mitogen-activated protein kinases | Angiotensin-(1-7) | CONVERTING ENZYME 2 | FIBROSIS | MYOCARDIAL-INFARCTION | MYOCYTES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INHIBITS GROWTH | HEART-FAILURE | RATS | HYPERTROPHY | ACE2 | PHARMACOLOGY & PHARMACY | ENDOTHELIN-1 GENE-EXPRESSION | Heart Diseases - metabolism | Endothelin-1 - pharmacology | Peptide Fragments - pharmacology | Fibrosis - metabolism | Cyclooxygenase 2 - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Heart Diseases - complications | DNA - biosynthesis | Angiotensin I - pharmacology | Collagen - biosynthesis | Phosphorylation - drug effects | Animals, Newborn | Prostaglandin-Endoperoxide Synthases - genetics | Dual Specificity Phosphatase 1 - genetics | Angiotensin II - pharmacology | Fibrosis - physiopathology | Mitogen-Activated Protein Kinase 3 - genetics | Heart Diseases - physiopathology | Fibrosis - complications | Cells, Cultured | Collagen - antagonists & inhibitors | Rats | DNA - antagonists & inhibitors | Myocardium - cytology | Gene Expression Regulation - drug effects | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Prostaglandin-Endoperoxide Synthases - metabolism | Fibroblasts - drug effects | Cyclooxygenase 2 - metabolism | Dual Specificity Phosphatase 1 - metabolism | Cell Proliferation - drug effects | Fibroblasts - cytology | Mitogen-Activated Protein Kinase 1 - metabolism | Prostaglandins | Phosphatases | Peptides | Collagen | Prostaglandins E | Angiotensin | Phosphotransferases | Activated | Synthesis | Deoxyribonucleic acid | Fibrosis | Fibroblasts | Hormones | Kinases | dual specificity phosphatase | angiotensin-(1-7) | cyclooxygenase | prostaglandin E synthase | cardiac fibrosis | mitogen-activated protein kinases
Journal Article
Molecular BioSystems, ISSN 1742-206X, 2/2013, Volume 9, Issue 3, pp. 47 - 411
Journal Article
Cancer and Metastasis Reviews, ISSN 0167-7659, 9/2010, Volume 29, Issue 3, pp. 405 - 434
Almost 25 centuries ago, Hippocrates, the father of medicine, proclaimed “Let food be thy medicine and medicine be thy food.” Exploring the association between... 
NF-κB | Biomedicine | Therapeutics | Oncology | Cancer Research | Inflammation | Tumorigenesis | Biomedicine general | Nutraceuticals | INHIBITS MATRIX METALLOPROTEINASE-2 | UROKINASE-PLASMINOGEN-ACTIVATOR | ANTIAPOPTOTIC GENE-PRODUCTS | INDUCED UP-REGULATION | BREAST-CANCER CELLS | NF-kappa B | ONCOLOGY | RECEPTOR-INTERACTING PROTEIN | ALPHA KINASE ACTIVATION | TRAIL-INDUCED APOPTOSIS | NF-KAPPA-B | HUMAN MULTIPLE-MYELOMA | Cell Proliferation | Neoplasm Invasiveness | Humans | Colorectal Neoplasms - prevention & control | Cell Adhesion | Inflammation - metabolism | Neoplasm Metastasis | Animals | Neovascularization, Pathologic - prevention & control | Apoptosis - physiology | Dietary Supplements | Cell Survival - physiology | Colorectal Neoplasms - metabolism | Cell Movement | Legumes | Catechin | Oncology, Experimental | Development and progression | Metastasis | Research | Mimosaceae | Prevention | Beans | Functional foods | Resveratrol | Physiological aspects | Cancer | Isoflavones | Cell proliferation | Transformation | Nuts | Vegetables | Capsaicin | Genistein | zerumbone | Fruits | Metastases | Angiogenesis | Signal transduction | Paclitaxel | sanguinarine | Curcumin | flavopiridol | Food | Sulforafan | Cell survival | Tumor cells | Caffeic acid | Diets | nutraceuticals | Quercetin | Spices | Apoptosis | Tumors | Index Medicus
Journal Article