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Oncogene, ISSN 0950-9232, 11/2012, Volume 31, Issue 44, pp. 4718 - 4724
The transcription of ERCC1 and other nucleotide excision repair (NER) genes is strongly influenced by c-jun. C-jun is transcriptionally regulated by Gli... 
XRCC1 | cisplatin | Gli1 | ovarian cancer | XPD | ERCC1 | HEDGEHOG PATHWAY | OVARIAN-CARCINOMA CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MESSENGER-RNA LEVELS | FLUOROURACIL CHEMOTHERAPY | NUCLEOTIDE EXCISION-REPAIR | CELL BIOLOGY | LUNG-CANCER | CANCER-PATIENTS | ANTICANCER CHEMOTHERAPY | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | RESISTANCE | Oncogene Proteins - genetics | Endonucleases - genetics | DNA Adducts - drug effects | Humans | Drug Resistance, Neoplasm | Endonucleases - metabolism | DNA-Binding Proteins - metabolism | Ovarian Neoplasms - genetics | RNA Interference | Trans-Activators - genetics | Female | Antineoplastic Agents - pharmacology | Ovarian Neoplasms - metabolism | Phosphorylation - drug effects | DNA Repair - drug effects | Xeroderma Pigmentosum Group D Protein - genetics | Proto-Oncogene Proteins c-jun - genetics | Gene Silencing | Oncogene Proteins - metabolism | Cisplatin - pharmacology | DNA-Binding Proteins - genetics | Proto-Oncogene Proteins c-jun - metabolism | Xeroderma Pigmentosum Group D Protein - metabolism | Cell Line, Tumor | Trans-Activators - metabolism | Zinc Finger Protein GLI1 | X-ray Repair Cross Complementing Protein 1 | Genetic aspects | Research | Genetic transcription | Drug therapy | DNA repair | Ovarian cancer | Oncology | Pharmacology | Platinum | Phosphorylation | Transcription factors | XRCC1 protein | Transcription | c-Jun protein | Nucleotide excision repair | Tumor cell lines | Base excision repair | Cisplatin | Absorbance | Gli protein | Spectrometry | Tubulin | Hedgehog protein | XPD protein | ERCC1 protein | Glyceraldehyde-3-phosphate dehydrogenase | Adducts
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2012, Volume 122, Issue 12, pp. 4555 - 4568
Increased mucus production is a common cause of morbidity and mortality in inflammatory airway diseases, including asthma, chronic obstructive pulmonary... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATED PROTEIN-KINASE | BRONCHIAL EPITHELIAL-CELLS | MUCIN | P38-DELTA | MCLCA3 | P38 MAPK | INTERLEUKIN-13 | MEMBRANE-PROTEIN | VIRAL-INFECTION | RATIONAL DESIGN | Respiratory System - pathology | Interleukin-13 - physiology | Epithelial Cells - drug effects | Humans | Crystallography, X-Ray | Chloride Channels - genetics | Gene Knockdown Techniques | MAP Kinase Signaling System | Epithelial Cells - secretion | Pyrazoles - chemistry | Mitogen-Activated Protein Kinase 13 - antagonists & inhibitors | RNA Interference | Drug Design | Mitogen-Activated Protein Kinase 13 - chemistry | Mitogen-Activated Protein Kinase 13 - metabolism | Mucins - metabolism | Mitogen-Activated Protein Kinase 13 - genetics | Pulmonary Disease, Chronic Obstructive - metabolism | Binding Sites | Pyrazoles - pharmacology | Naphthalenes - chemistry | Cells, Cultured | Gene Expression Regulation | Models, Molecular | Mucus - secretion | Respiratory System - secretion | Naphthalenes - pharmacology | Chloride Channels - metabolism | Secretory Pathway - drug effects | Hydrogen Bonding | Protein Binding | Kinetics | Chloride Channels - physiology | Mucins - genetics | Respiratory mucosa | Interleukins | Genetic aspects | Research | Gene expression | Properties | Airway (Medicine) | Mitogen-activated protein kinases
Journal Article
Neuroscience and Biobehavioral Reviews, ISSN 0149-7634, 06/2010, Volume 34, Issue 7, pp. 1047 - 1054
Journal Article
Human Brain Mapping, ISSN 1065-9471, 10/2018, Volume 39, Issue 10, pp. 4065 - 4082
Journal Article
Trends in Cognitive Sciences, ISSN 1364-6613, 2008, Volume 12, Issue 11, pp. 418 - 424
Journal Article
Cell Cycle, ISSN 1538-4101, 07/2012, Volume 11, Issue 13, pp. 2507 - 2517
Targeting Chk1 protein kinase can enhance the antitumor effects of radio- and chemotherapy. Recent evidence disclosed a role of Chk1 in unperturbed cell... 
targeted therapy | WEE1 | Chk1 | in vivo antitumor activity | synthetic lethality | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | In vivo antitumor activity | Wee1 | Synthetic lethality | Targeted therapy | CHECKPOINT KINASE 1 | PHOSPHORYLATION | PROTEIN-KINASE | DNA-DAMAGE | G CHECKPOINT | CANCER-THERAPY | CELL BIOLOGY | GENOME INTEGRITY | CELL-CYCLE | ATR | Protein Kinases - metabolism | Protein Kinases - genetics | Pyrazoles - therapeutic use | Apoptosis - drug effects | DNA Replication - drug effects | Protein-Tyrosine Kinases - metabolism | Humans | Protein Kinases - chemistry | Transplantation, Heterologous | Cell Cycle Proteins - antagonists & inhibitors | Protein-Tyrosine Kinases - genetics | RNA Interference | Cell Cycle Proteins - genetics | Female | Drug Therapy, Combination | Nuclear Proteins - genetics | Ovarian Neoplasms - drug therapy | Pyrazoles - pharmacology | Benzodiazepinones - pharmacology | Cell Cycle Proteins - metabolism | Nuclear Proteins - metabolism | Pyrimidines - pharmacology | Drug Synergism | Animals | Mitosis - drug effects | Mice, Nude | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Checkpoint Kinase 1 | Mice | Protein Kinase Inhibitors - pharmacology | Benzodiazepinones - therapeutic use | Protein-Tyrosine Kinases - antagonists & inhibitors | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Journal Article
International Journal of Cancer, ISSN 0020-7136, 02/2013, Volume 132, Issue 3, pp. E149 - E157
Inhibition of centromere‐associated protein‐E (CENP‐E) has demonstrated preclinical anti‐tumor activity in a number of tumor types including neuroblastoma. A... 
CENP‐E | neuroblastoma | MEK | Ras | GSK923295 | ERK | CENP-E | MAP KINASE | RECEPTOR | ONCOLOGY | PATHWAY | THERAPEUTIC-EFFICACY | ALK KINASE | MUTATIONS | MITOTIC CHECKPOINT | SIGNAL-REGULATED KINASE | Lung Neoplasms - drug therapy | Colonic Neoplasms - genetics | Apoptosis - drug effects | Colonic Neoplasms - drug therapy | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Drug Resistance, Neoplasm | Biomarkers, Tumor | Chromosomal Proteins, Non-Histone - antagonists & inhibitors | Pancreatic Neoplasms - drug therapy | RNA Interference | Dacarbazine - pharmacology | Dacarbazine - analogs & derivatives | Antineoplastic Agents - pharmacology | Benzamides - pharmacology | Sarcosine - analogs & derivatives | Neuroblastoma - pathology | Sarcosine - pharmacology | Camptothecin - analogs & derivatives | Lung Neoplasms - genetics | M Phase Cell Cycle Checkpoints - drug effects | Mitogen-Activated Protein Kinase 3 - genetics | Pancreatic Neoplasms - genetics | Drug Synergism | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | MAP Kinase Signaling System - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Neuroblastoma - drug therapy | Cell Line, Tumor | Cell Proliferation - drug effects | Neuroblastoma - metabolism | RNA, Small Interfering | Camptothecin - pharmacology | Drug Screening Assays, Antitumor | Prevention | Colon cancer | Lung cancer | Oncology, Experimental | Genes | Research | Drug therapy, Combination | Protein kinases | Cancer | Proteins | Chemotherapy | Kinases | Index Medicus | Apoptosis
Journal Article
1995, ISBN 9780122089305, 443
Life scientists have long been familiar with the notion of interference and inhibition in biological systems 3/4 most notably in the neuron. Now these concepts... 
Interference (Perception) | Cognition | Inhibition | Biology, life sciences
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