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1. Full Text Kinetic differences in the induction of interferon stimulated genes by interferon‐α and interleukin 28B are altered by infection with hepatitis C virus
by Jilg, Nikolaus and Lin, Wenyu and Hong, Jian and Schaefer, Esperance A and Wolski, David and Meixong, James and Goto, Kaku and Brisac, Cynthia and Chusri, Pattranuch and Fusco, Dahlene N and Chevaliez, Stephane and Luther, Jay and Kumthip, Kattareeya and Urban, Thomas J and Peng, Lee F and Lauer, Georg M and Chung, Raymond T
Hepatology, ISSN 0270-9139, 04/2014, Volume 59, Issue 4, pp. 1250 - 1261
Several genome‐wide association studies (GWAS) have identified a genetic polymorphism associated with the gene locus for interleukin 28B (IL28B), a type III... 
IL28B | CLEARANCE | REPLICATION | RIBAVIRIN | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | EPIDEMIOLOGY | Interferon-alpha - pharmacology | Carcinoma, Hepatocellular - epidemiology | Phosphorylation | Hepatitis C - metabolism | Comorbidity | Humans | Hepatocytes - pathology | Transcriptome - drug effects | Hepatocytes - metabolism | Dose-Response Relationship, Drug | Liver Neoplasms - epidemiology | STAT1 Transcription Factor - metabolism | Time Factors | Hepatitis C - epidemiology | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Liver Neoplasms - pathology | Gene Expression Regulation, Neoplastic - drug effects | Hepatitis C - pathology | Interleukins - pharmacology | Hepatocytes - drug effects | Carcinoma, Hepatocellular - metabolism
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2. Full Text Sequence-specific potent induction of IFN-α by short interfering RNA in plasmacytoid dendritic cells through TLR7
by de Fougerolles, Antonin and Hartmann, Gunther and Akira, Shizuo and Ablasser, Andrea and Manoharan, Muthiah and Schlee, Martin and Uematsu, Satoshi and Hornung, Veit and Bourquin, Carole and Guenthner-Biller, Margit and Endres, Stefan and Noronha, Anne
Nature Medicine, ISSN 1078-8956, 03/2005, Volume 11, Issue 3, pp. 263 - 270
Short interfering RNA ( siRNA) is used in RNA interference technology to avoid non-target-related induction of type I interferon (IFN) typical for long... 
MEDICINE, RESEARCH & EXPERIMENTAL | DOUBLE-STRANDED-RNA | ACTIVATION | NUCLEOSIDE ANALOGS | RECOGNITION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CPG DNA | CD8 T-CELLS | CELL BIOLOGY | B-CELLS | TOLL-LIKE RECEPTOR-7 | MESSENGER-RNA | BACTERIAL-DNA | Cell Line | Interferon-alpha - biosynthesis | Dendritic Cells - immunology | Humans | Toll-Like Receptor 9 | RNA, Viral - physiology | Toll-Like Receptor 7 | RNA, Small Interfering - chemistry | Toll-Like Receptors | Membrane Glycoproteins - physiology | Animals | RNA, Small Interfering - immunology | Base Sequence | Mice | Receptors, Cell Surface - physiology | Liposomes | Dendritic Cells - metabolism | RNA, Small Interfering - metabolism
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3. Full Text Identification of CpG oligonucleotide sequences with high induction of IFN‐α/β in plasmacytoid dendritic cells
by Krug, Anne and Rothenfusser, Simon and Hornung, Veit and Jahrsdörfer, Bernd and Blackwell, Susan and Ballas, Zuhair K and Endres, Stefan and Krieg, Arthur M and Hartmann, Gunther
European Journal of Immunology, ISSN 0014-2980, 07/2001, Volume 31, Issue 7, pp. 2154 - 2163
The immature plasmacytoid dendritic cell (PDC) is identical with the principal type I IFN‐producing cell upon viral infection. Oligodeoxynucleotides which... 
CpG oligonucleotide | Dendritic cell | Type I interferon | IFN‐α/IFN‐β | Plasmacytoid dendritic cell | IFN-α/IFN-β | IFN-beta | KILLER-CELLS | plasmacytoid dendritic cell | IMMUNOLOGY | IFN-alpha | HUMAN MONOCYTES | type I interferon | ADAPTIVE IMMUNITY | IN-VITRO | GAMMA PRODUCTION | HUMAN BLOOD | SYNTHETIC OLIGONUCLEOTIDES | dendritic cell | T-CELLS | BACTERIAL-DNA | INNATE IMMUNITY | Stem Cells - immunology | Cytotoxicity Tests, Immunologic | Adjuvants, Immunologic - pharmacology | Antigens, CD - biosynthesis | Interferon-alpha - biosynthesis | Dendritic Cells - immunology | Humans | Membrane Glycoproteins - biosynthesis | Cells, Cultured | Oligodeoxyribonucleotides - chemistry | Interferon-beta - biosynthesis | CD4-Positive T-Lymphocytes - immunology | B7-2 Antigen | Interferon Type I - biosynthesis | B-Lymphocytes - immunology | Adjuvants, Immunologic - chemistry | Killer Cells, Natural - immunology | Stem Cells - drug effects | Dendritic Cells - drug effects | B7-1 Antigen - biosynthesis | Oligodeoxyribonucleotides - pharmacology | Tumor Necrosis Factor-alpha - biosynthesis | Interferon-gamma - biosynthesis | b-Interferon | a-Interferon | oligonucleotides
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4. Full Text An essential role for IFN‐β in the induction of IFN‐stimulated gene expression by LPS in macrophages
by Sheikh, Faruk and Dickensheets, Harold and Gamero, Ana M and Vogel, Stefanie N and Donnelly, Raymond P
Journal of Leukocyte Biology, ISSN 0741-5400, 10/2014, Volume 96, Issue 4, pp. 591 - 600
Interferon‐β mediates the activation of ISGF3 and induction of interferon‐stimulated genes, by lipopolysaccharide in macrophages. TLR agonists such as LPS and... 
ISGF3 | STAT | NF‐κB | TLR4 | endotoxin | NF-κB | Endotoxin | BACTERIAL LIPOPOLYSACCHARIDE | CYTOKINE SIGNALING-1 | ENDOTOXIN-SHOCK | NF-B | INTERFERON-BETA | IMMUNOLOGY | CUTTING EDGE | CELL BIOLOGY | TARGETED DISRUPTION | I INTERFERONS | RECEPTOR COMPLEX | HEMATOLOGY | LIPOPOLYSACCHARIDE-INDUCIBLE GENES | INNATE IMMUNITY | Lipopolysaccharides - immunology | Toll-Like Receptors - agonists | Mice, Knockout | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Macrophages - metabolism | STAT1 Transcription Factor - metabolism | Animals | Interferon-beta - metabolism | Interferon-beta - genetics | Mice | Receptor, Interferon alpha-beta - genetics | STAT2 Transcription Factor - metabolism | Toll-Like Receptors - metabolism | Macrophages - immunology | Receptor, Interferon alpha-beta - metabolism | Receptors, Signal Transduction, & Genes
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5. Full Text Interferon-α induction through Toll-like receptors involves a direct interaction of IRF7 with MyD88 and TRAF6
by Coban, Cevayir and Sato, Shintaro and Terai, Kenta and Takeuchi, Osamu and Kawai, Taro and Uematsu, Satoshi and Akira, Shizuo and Yamamoto, Masahiro and Hemmi, Hiroaki and Matsuda, Michiyuki and Inoue, Jun-ichiro and Ishii, Ken J
Nature Immunology, ISSN 1529-2908, 10/2004, Volume 5, Issue 10, pp. 1061 - 1068
Toll-like receptors (TLRs) are involved in the recognition of microbial pathogens. A subset of TLRs, TLR7, TLR8 and TLR9, induces antiviral responses by... 
TRANSCRIPTION FACTORS | DOMAIN-CONTAINING ADAPTER | DOUBLE-STRANDED-RNA | VIRUS-INFECTION | DENDRITIC CELLS | SIGNALING PATHWAY | TANK-BINDING KINASE-1 | IMMUNOLOGY | NF-KAPPA-B | IFN-REGULATORY FACTOR-3 | CUTTING EDGE | Proteins - physiology | DNA-Binding Proteins - physiology | Receptors, Immunologic - physiology | Interferon-alpha - biosynthesis | Humans | Mice, Inbred C57BL | Ubiquitin - metabolism | Toll-Like Receptor 9 | Myeloid Differentiation Factor 88 | Toll-Like Receptor 7 | Antigens, Differentiation - physiology | Toll-Like Receptor 8 | Toll-Like Receptors | Adaptor Proteins, Signal Transducing | Membrane Glycoproteins - physiology | TNF Receptor-Associated Factor 6 | Animals | Interferon Regulatory Factor-7 | Mice | Receptors, Cell Surface - physiology
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6. Full Text IFN-α inhibits telomerase in human CD8+ T cells by both hTERT downregulation and induction of p38 MAPK signaling
by Lanna, Alessio and Coutavas, Elias and Levati, Lauretta and Seidel, Judith and Rustin, Malcolm H. A and Henson, Sian M and Akbar, Arne N and Franzese, Ornella
Journal of Immunology, ISSN 0022-1767, 10/2013, Volume 191, Issue 7, pp. 3744 - 3752
The cytokine IFN-alpha is secreted during viral infections and has been shown to inhibit telomerase activity and accelerate T cell differentiation in vivo.... 
INTERFERON-ALPHA | IN-VITRO | ACTIVATION | MEMORY | AKT SER PHOSPHORYLATION | PERSISTENT LCMV INFECTION | NUCLEAR-PORE COMPLEX | REVERSE-TRANSCRIPTASE | CD4(+) | PROTEIN PHOSPHATASE 2A | IMMUNOLOGY | Interferon-alpha - pharmacology | Transcription, Genetic - drug effects | Humans | Cells, Cultured | NF-kappa B - metabolism | Down-Regulation - drug effects | Enzyme Activation - drug effects | E2F Transcription Factors - metabolism | CD28 Antigens - metabolism | Lymphocyte Activation - immunology | Signal Transduction - drug effects | Lymphocyte Activation - drug effects | Tumor Necrosis Factor Receptor Superfamily, Member 7 - metabolism | CD8-Positive T-Lymphocytes - drug effects | CD8-Positive T-Lymphocytes - metabolism | Telomerase - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Telomerase - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - metabolism
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7. Full Text Influenza A virus inhibits type I IFN signaling via NF-κB-dependent induction of SOCS-3 expression
by Pauli, Eva-K and Schmolke, Mirco and Wolff, Thorsten and Viemann, Dorothee and Roth, Johannes and Bode, Johannes G and Ludwig, Stephan
PLoS Pathogens, ISSN 1553-7366, 11/2008, Volume 4, Issue 11, p. e1000196
The type I interferon (IFN) system is a first line of defense against viral infections. Viruses have developed various mechanisms to counteract this response.... 
VIRAL NS1 PROTEIN | DOUBLE-STRANDED-RNA | ANTIVIRAL THERAPY | CYTOKINE SIGNALING-3 | CHRONIC HEPATITIS-C | PRE-MESSENGER-RNAS | MICROBIOLOGY | PRIMARY ENDOTHELIAL-CELLS | INTERFERON-ALPHA | IMMUNE-SYSTEM | VIROLOGY | GENE-EXPRESSION | PARASITOLOGY
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8. Full Text Type I interferon potentiates T‐cell receptor mediated induction of IL‐10‐producing CD4+ T cells
by Corre, Béatrice and Perrier, Julie and El Khouri, Margueritte and Cerboni, Silvia and Pellegrini, Sandra and Michel, Frédérique
European Journal of Immunology, ISSN 0014-2980, 10/2013, Volume 43, Issue 10, pp. 2730 - 2740
Type I interferons (IFNs) have the dual ability to promote the development of the immune response and exert an anti‐inflammatory activity. We analyzed the... 
T helper cells | IFN‐α/β | IL‐10 | CD4+ T cells | Tr1 cells | CD4 | T cells | IL-10 | IFN-α/β | GAMMA | REGULATORY FUNCTION | INTERLEUKIN-10 | IMMUNOLOGY | CUTTING EDGE | IFN | MULTIPLE-SCLEROSIS | DUAL ROLE | DIFFERENTIATION | TH1 | CD4(+) T cells | IFN-ALPHA | T-Lymphocyte Subsets - immunology | Leukocyte Common Antigens - metabolism | Receptors, Antigen, T-Cell - metabolism | Lymphocyte Activation | Humans | Cells, Cultured | CD28 Antigens - immunology | Interferon-gamma - metabolism | Interferon-alpha - immunology | T-Lymphocytes, Regulatory - immunology | Receptor Cross-Talk - immunology | Cell Differentiation - immunology | Signal Transduction - immunology | Feedback, Physiological | Forkhead Transcription Factors - metabolism | Inflammation Mediators - metabolism | Interleukin-10 - metabolism | Protein Binding | Receptors, Antigen, T-Cell - immunology | Biological response modifiers | Interferon | T cell receptors | Lymphocytes | Cooperation
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9. Full Text Rational design of new CpG oligonucleotides that combine B cell activation with high IFN‐α induction in plasmacytoid dendritic cells
by Hartmann, Gunther and Battiany, Julia and Poeck, Hendrik and Wagner, Moritz and Kerkmann, Miren and Lubenow, Norbert and Rothenfusser, Simon and Endres, Stefan
European Journal of Immunology, ISSN 0014-2980, 06/2003, Volume 33, Issue 6, pp. 1633 - 1641
Two different types of CpG motif‐containing oligonucleotides (CpG ODN) have been described: CpG‐A with high induction of IFN‐α in plasmacytoid dendritic cells;... 
B cell | Innate immunity | CpG oligodeoxynucleotide | Type I IFN | Plasmacytoid dendritic cell | Type I IFN | SYSTEMIC-LUPUS-ERYTHEMATOSUS | type IIFN | plasmacytoid dendritic cell | PROLIFERATION | IMMUNOLOGY | MATURATION | HUMAN MONOCYTES | BETA | INTERFERON | innate immunity | OLIGODEOXYNUCLEOTIDE | EXPRESSION | BACTERIAL-DNA | BLOOD | Adjuvants, Immunologic - pharmacology | Dose-Response Relationship, Immunologic | Antigens, CD - biosynthesis | Dendritic Cells - immunology | Humans | Membrane Glycoproteins - biosynthesis | Middle Aged | Oligodeoxyribonucleotides - chemistry | Toll-Like Receptor 9 | Male | Structure-Activity Relationship | Antigens, CD - genetics | B7-2 Antigen | Interferon-alpha - genetics | Base Sequence | Drug Design | Adult | Dendritic Cells - drug effects | Receptors, Cell Surface - physiology | Dendritic Cells - metabolism | DNA-Binding Proteins - physiology | CpG Islands - immunology | Oligodeoxyribonucleotides - immunology | Interferon-alpha - biosynthesis | Membrane Glycoproteins - genetics | Gene Expression Regulation - drug effects | B-Lymphocytes - drug effects | B-Lymphocytes - immunology | Lymphocyte Activation - drug effects | Aged | Oligodeoxyribonucleotides - pharmacology | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap
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10. Full Text Virulence of a Mycobacterium tuberculosis Clinical Isolate in Mice Is Determined by Failure to Induce Th1 Type Immunity and Is Associated with Induction of IFN-α/β
by Claudia Manca and Liana Tsenova and Amy Bergtold and Sherry Freeman and Michael Tovey and James M. Musser and Clifton E. Barry and Victoria H. Freedman and Gilla Kaplan
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2001, Volume 98, Issue 10, pp. 5752 - 5757
To understand how virulent mycobacteria subvert host immunity and establish disease, we examined the differential response of mice to infection with various... 
Spleen | Biological Sciences | Messenger RNA | Tuberculosis | Lungs | Mycobacterium tuberculosis | Cytokines | Pulmonary tuberculosis | Infections | Splenic tuberculosis | Lymph nodes | IN-VITRO | VIRUS-INFECTION | INTERFERON-GAMMA | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | INTERLEUKIN-12 | RECEPTOR GENE | HUMAN MONOCYTES | AFRICANS | CD8(+) T-CELLS | VIRAL-INFECTION | Interferon-alpha - biosynthesis | RNA, Messenger - genetics | Virulence | Interferon-beta - biosynthesis | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Th1 Cells - immunology | Mycobacterium tuberculosis - pathogenicity | Tuberculosis, Pulmonary - immunology | Animals | Interferon-alpha - genetics | Cell Division | Interferon-beta - genetics | Female | Lung - metabolism | Mice | b-Interferon | a-Interferon | tumor necrosis factor-^a
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11. Full Text Interferon induction by avian reovirus
by Lostalé-Seijo, Irene and Martínez-Costas, José and Benavente, Javier
Virology, ISSN 0042-6822, 2015, Volume 487, pp. 104 - 111
Abstract We have previously shown that the replication of avian reovirus (ARV) in chicken embryo fibroblasts (CEF) is more resistant to the antiviral action of... 
Infectious Disease | Avian reovirus | Interferon induction | PKR | Virus uncoating | Apoptosis | CELLS | RNA | MECHANISM | MATRIX PROTEIN | IMMUNE EVASION | VIROLOGY | GENE-EXPRESSION | SIGMA-A | INFECTION | VESICULAR STOMATITIS-VIRUS | CHICKEN INTERFERON | Cricetinae | Cell Proliferation | Interferon-beta - immunology | Vesicular stomatitis Indiana virus - growth & development | Interferon-alpha - biosynthesis | Vaccinia virus - growth & development | eIF-2 Kinase - biosynthesis | Cells, Cultured | Interferon-beta - biosynthesis | eIF-2 Kinase - immunology | Chick Embryo | Orthoreovirus, Avian - growth & development | Vaccinia virus - immunology | Interferon-alpha - immunology | Animals | Apoptosis - immunology | Caspases - metabolism | Virus Replication | Chickens | Orthoreovirus, Avian - immunology | Primary Cell Culture | Vesicular stomatitis Indiana virus - immunology | Interferon | Biological response modifiers | Gene expression | Health aspects
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12. Full Text Plasmacytoid dendritic cell‐derived IFN‐α promotes murine liver ischemia/reperfusion injury by induction of hepatocyte IRF‐1
by Castellaneta, Antonino and Yoshida, Osamu and Kimura, Shoko and Yokota, Shinichiro and Geller, David A and Murase, Noriko and Thomson, Angus W
Hepatology, ISSN 0270-9139, 07/2014, Volume 60, Issue 1, pp. 267 - 277
Plasmacytoid dendritic cells (pDC) constitute the body's principal source of type I interferon (IFN) and are comparatively abundant in the liver. Among various... 
ISCHEMIA-REPERFUSION INJURY | INTERFERON | TOLERANCE | ANTIMICROBIAL PEPTIDE | MECHANISMS | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | T-CELLS | REGULATORY FACTOR-I | TRANSPLANTATION | Tumor Necrosis Factor-alpha - metabolism | Dendritic Cells - immunology | Liver Diseases - pathology | Male | Hepatocytes - metabolism | Hepatocytes - cytology | Tumor Necrosis Factor-alpha - immunology | Interleukin-6 - metabolism | Dendritic Cells - metabolism | Disease Models, Animal | Liver Diseases - immunology | Interferon-alpha - metabolism | Reperfusion Injury - pathology | Interferon Regulatory Factor-1 - immunology | Mice, Inbred C57BL | Cells, Cultured | Hepatocytes - immunology | Interferon-alpha - immunology | Mice, Knockout | Animals | Apoptosis - immunology | Up-Regulation - immunology | Reperfusion Injury - immunology | Interleukin-6 - immunology | Mice | Interferon Regulatory Factor-1 - metabolism
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