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Journal Article
The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 09/2017, Volume 140, Issue 3, pp. 809 - 821.e3
Hypertension is considered an immunologic disorder. However, the role of the IL-17 family in genetic hypertension in the spontaneously hypertensive rat (SHR)... 
Hypertension | CD161 | IL-17 | spontaneously hypertensive rat | digoxin | T cells | retinoic acid receptor–related orphan receptor γt | TH17 | Toll-like receptor | gene expression | innate immune system | T | 17 | INTERLEUKIN 17 | ACTIVATION | ENDOTHELIAL DYSFUNCTION | ORGAN DAMAGE | IMMUNOLOGY | NICOTINIC ACETYLCHOLINE-RECEPTORS | RESPONSES | ALLERGY | INFLAMMATION | T(H)17 | retinoic acid receptor-related orphan receptor gamma t | DIFFERENTIATION | TH17 CELLS | LYMPHOCYTES | Animals, Newborn | Rats, Inbred SHR | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - antagonists & inhibitors | Poly I-C - pharmacology | Cells, Cultured | Interleukin-17 - immunology | Interleukin-17 - physiology | Interleukin-17 - genetics | Rats, Inbred WKY | Male | Hypertension - immunology | Spleen - cytology | Toll-Like Receptor 3 - agonists | NK Cell Lectin-Like Receptor Subfamily B - immunology | Digoxin - pharmacology | Hypertension - physiopathology | Vasodilation | Aging - immunology | Animals | Aorta, Thoracic - physiology | Blood Pressure - drug effects | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | RNA - metabolism | Flow cytometry | Transcription factors | Digoxin | Disease | Helper cells | Nervous system | Lymphocytes T | Birth | Kinases | Signal transduction | Splenocytes | Lymphocytes | Rodents | Measurement methods | Blood pressure | Immune system | Spleen | Immune response | Cytokines | Rats | Pharmacology | Inflammation | T cell receptors | Interleukin 17 | Endothelium | Immune systems | Acids | Prostate | Surface markers | Retinoic acid | Nicotine | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2010, Volume 107, Issue 33, pp. 14751 - 14756
In several murine models of autoimmune arthritis, Th17 cells are the dominant initiators of inflammation. In human arthritis the majority of IL-17–secreting... 
T lymphocytes | Synovial fluid | Phenotypes | Cytokines | Juvenile rheumatoid arthritis | Cultured cells | Arthritis | Joint diseases | Joints | T cell antigen receptors | Juvenile | CD161 | RORC2 | RHEUMATOID-ARTHRITIS | GAMMA | MULTIDISCIPLINARY SCIENCES | JUVENILE IDIOPATHIC ARTHRITIS | CD4(+) T-CELLS | INDUCTION | MEMORY CELLS | IL-17 | juvenile | INFLAMED JOINTS | T-H-17 CELLS | LINEAGE | Receptors, CCR6 - immunology | Humans | Interleukin-17 - immunology | Molecular Sequence Data | T-Box Domain Proteins - immunology | Interferon-gamma - metabolism | Th1 Cells - immunology | Th1 Cells - metabolism | Flow Cytometry | T-Lymphocytes, Helper-Inducer - immunology | Base Sequence | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Receptors, CCR6 - metabolism | Interferon-gamma - genetics | Child | Cell Lineage - genetics | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Transforming Growth Factor beta - immunology | Amino Acid Sequence | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Gene Expression | T-Lymphocytes, Helper-Inducer - metabolism | NK Cell Lectin-Like Receptor Subfamily B - metabolism | Interleukin-17 - genetics | Interleukin-12 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Arthritis, Juvenile - genetics | NK Cell Lectin-Like Receptor Subfamily B - immunology | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Arthritis, Juvenile - metabolism | Interleukin-17 - metabolism | NK Cell Lectin-Like Receptor Subfamily B - genetics | Arthritis, Juvenile - immunology | Interferon-gamma - immunology | Interleukin-12 - immunology | Receptors, CCR6 - genetics | Transforming Growth Factor beta - metabolism | Cell Lineage - immunology | Autoimmunity | Research | Adaptation (Physiology) | Genotype & phenotype | Rodents | T cell receptors | Gene expression | Inflammatory diseases | Children & youth | Index Medicus | Animal models | Transcription factors | Helper cells | Interleukin 12 | Interleukin 23 | Inflammation | Lymphocytes T | Transforming growth factor- beta | double prime T-cell receptor | CCR6 protein | Interleukin 17 | Plasticity | Children | Biological Sciences
Journal Article
The Journal of Immunology, ISSN 0022-1767, 10/2007, Volume 179, Issue 8, pp. 5462 - 5473
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2008, Volume 181, Issue 4, pp. 2277 - 2284
Journal Article
Cancer Research, ISSN 0008-5472, 02/2012, Volume 72, Issue 3, pp. 581 - 591
The immunosuppressive microenvironment in tumors hampers the induction of antitumor immunity by vaccines or immunotherapies. Toll-like receptor (TLR) ligands... 
RESPONSES | TGF-BETA | DENDRITIC CELLS | ONCOLOGY | INDUCTION | BORDETELLA-PERTUSSIS | SUPPRESSION | PHOSPHOINOSITIDE 3-KINASE | CANCER | INNATE IMMUNITY | TH17 CELLS | Immunotherapy - methods | Carcinoma, Lewis Lung - therapy | Carcinoma, Lewis Lung - immunology | Interleukin-17 - immunology | Phosphatidylinositol 3-Kinases - metabolism | Interferon-gamma - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Thiazolidinediones - administration & dosage | Toll-Like Receptor 5 - agonists | Enzyme Inhibitors - administration & dosage | Neoplasms, Experimental - pathology | Melanoma, Experimental - immunology | T-Lymphocytes - metabolism | Colonic Neoplasms - immunology | T-Lymphocytes - drug effects | Neoplasms, Experimental - immunology | Flagellin - administration & dosage | Colonic Neoplasms - therapy | Toll-Like Receptor 5 - metabolism | Cancer Vaccines - administration & dosage | Melanoma, Experimental - therapy | Mice, Inbred C57BL | Neoplasms, Experimental - therapy | Interleukin-17 - genetics | Melanoma, Experimental - pathology | Combined Modality Therapy | Drug Resistance, Neoplasm - immunology | Blotting, Western | Mice, Knockout | Cancer Vaccines - immunology | Interleukin-17 - metabolism | Animals | Tumor Burden - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Triazines - administration & dosage | Colonic Neoplasms - pathology | Interferon-gamma - immunology | Carcinoma, Lewis Lung - pathology | Cell Line, Tumor | T-Lymphocytes - immunology | Mice | Tumor Burden - immunology | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2011, Volume 186, Issue 6, pp. 3745 - 3752
A major neurotransmitter dopamine transmits signals via five different seven-transmembrane G protein-coupled receptors termed D1-D5. Several studies have shown... 
ENDOGENOUS CATECHOLAMINES | INTERLEUKIN-17 RECEPTOR | CD4+AND CD8+T CELLS | SYNOVIAL ENDOTHELIAL-CELLS | DENDRITIC CELLS | AUTOIMMUNE INFLAMMATION | TH2 CYTOKINES | PERIPHERAL-BLOOD LYMPHOCYTES | TNF-ALPHA | IMMUNOLOGY | TH17 CELLS | Humans | Synovial Membrane - immunology | Synovial Membrane - pathology | Male | CD4-Positive T-Lymphocytes - pathology | CD4-Positive T-Lymphocytes - immunology | Synovitis - therapy | Th17 Cells - metabolism | Interleukin-6 - physiology | Synovitis - immunology | Disease Models, Animal | Th17 Cells - pathology | Receptors, Dopamine D1 - antagonists & inhibitors | Synovial Membrane - transplantation | Interleukin-17 - secretion | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Dopamine - pharmacology | Benzazepines - pharmacology | Interleukin-17 - biosynthesis | Random Allocation | Mice, SCID | Transplantation Chimera | Arthritis, Rheumatoid - pathology | Synovitis - pathology | Arthritis, Rheumatoid - therapy | Cartilage, Articular - pathology | Cell Differentiation - immunology | Animals | Receptors, Dopamine D1 - physiology | Resting Phase, Cell Cycle - immunology | Th17 Cells - immunology | Benzazepines - administration & dosage | Mice | Arthritis, Rheumatoid - immunology | Cartilage, Articular - drug effects | Cartilage, Articular - immunology | Index Medicus | Abridged Index Medicus
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 05/2014, Volume 66, Issue 5, pp. 1185 - 1194
Journal Article