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Journal of Autoimmunity, ISSN 0896-8411, 2011, Volume 37, Issue 3, pp. 190 - 197
Abstract One way for intravenous Ig (IVIg) to affect responses of the B cells might be to operate through their TLR7 and TLR9. We confirm the ability of TLR... 
Allergy and Immunology | Intravenous immunoglobulin | B lymphocyte | TLR9 | Autoimmune diseases | SYSTEMIC-LUPUS-ERYTHEMATOSUS | ACTIVATION | KAPPA-B | IMMUNOLOGY | PROINFLAMMATORY CYTOKINE PRODUCTION | FC-GAMMA-RIIB | IN-VITRO | NEGATIVE REGULATION | TOLL-LIKE RECEPTORS | UP-REGULATION | BACTERIAL-DNA | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - immunology | Humans | Toll-Like Receptor 9 - genetics | Molecular Sequence Data | Sialic Acid Binding Ig-like Lectin 2 - immunology | Toll-Like Receptor 9 - agonists | Toll-Like Receptor 9 - immunology | Signal Transduction - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Sialic Acid Binding Ig-like Lectin 2 - metabolism | Toll-Like Receptor 7 - immunology | Myeloid Differentiation Factor 88 - immunology | Autoimmune Diseases - metabolism | Autoimmune Diseases - pathology | Autoimmune Diseases - drug therapy | B-Lymphocytes - metabolism | Interleukin-2 Receptor alpha Subunit - immunology | Toll-Like Receptor 7 - agonists | Toll-Like Receptor 7 - genetics | Amino Acid Sequence | B-Lymphocytes - cytology | Toll-Like Receptor 7 - metabolism | Autoimmune Diseases - immunology | Cells, Cultured | Reverse Transcriptase Polymerase Chain Reaction | Immunoglobulins, Intravenous - pharmacology | B-Lymphocytes - drug effects | B-Lymphocytes - immunology | Interleukin-2 Receptor alpha Subunit - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - metabolism | Lymphocyte Activation - drug effects | Interleukin-6 - biosynthesis | Interleukin-10 - biosynthesis | Oligodeoxyribonucleotides - pharmacology | Toll-Like Receptor 9 - metabolism | Myeloid Differentiation Factor 88 - metabolism | Employee recruitment | Enzymes | Phosphatases | Immunoglobulin G | Oligodeoxyribonucleotides | Interleukin-2 Receptor alpha Subunit | Signal Transduction | Interleukin-10 | Lymphocyte Activation | Toll-Like Receptor 9 | B-Lymphocytes | Myeloid Differentiation Factor 88 | Toll-Like Receptor 7 | Interleukin-6 | Life Sciences | Immunology | Autoimmune Diseases | Sialic Acid Binding Ig-like Lectin 2 | Protein Tyrosine Phosphatase, Non-Receptor Type 6 | Immunoglobulins, Intravenous
Journal Article
BMC Gastroenterology, ISSN 1471-230X, 12/2011, Volume 11, Issue 1, pp. 138 - 138
Background: Dysregulation of innate immune response by Toll-Like Receptors (TLRs) is a key feature in Ulcerative Colitis (UC). Most studies have focused on... 
MESSENGER-RNA EXPRESSION | POUCH MUCOSA | GASTROENTEROLOGY & HEPATOLOGY | TLR4 | INTESTINAL EPITHELIAL-CELLS | COLONIC-MUCOSA | BACTERIAL-DNA | BOWEL-DISEASE | Immunohistochemistry | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Toll-Like Receptor 2 - genetics | Toll-Like Receptor 5 - genetics | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Toll-Like Receptor 9 - genetics | Male | Gene Expression Profiling | RNA, Messenger - metabolism | Young Adult | Toll-Like Receptor 6 - genetics | Statistics, Nonparametric | Adult | Female | Toll-Like Receptor 3 - genetics | Toll-Like Receptors - metabolism | Interleukin-6 - metabolism | Severity of Illness Index | Toll-Like Receptor 6 - metabolism | Toll-Like Receptor 7 - genetics | Toll-Like Receptor 5 - metabolism | Toll-Like Receptor 7 - metabolism | Interleukin-6 - genetics | Colitis, Ulcerative - metabolism | Toll-Like Receptor 1 - metabolism | Toll-Like Receptor 4 - genetics | Colitis, Ulcerative - pathology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 3 - metabolism | Toll-Like Receptor 4 - metabolism | Toll-Like Receptor 1 - genetics | Biopsy | Toll-Like Receptors - genetics | Adolescent | Toll-Like Receptor 8 - genetics | Aged | Toll-Like Receptor 9 - metabolism | Toll-Like Receptor 8 - metabolism | Measurement | Toll-like receptors | Messenger RNA | Research | Patient outcomes | Ulcerative colitis | Inflammatory bowel disease | Studies | Proteins | Nutrition research | Gene expression | Manuscripts
Journal Article
Journal Article
FRONTIERS IN IMMUNOLOGY, ISSN 1664-3224, 08/2018, Volume 9, p. 1986
There is growing evidence that tumor necrosis factor (TNF) receptor-associated factors (TRAFs) bind to unconventional membrane-bound receptors in many cell... 
FACTOR FAMILY | NECROSIS-FACTOR RECEPTOR | MEMBERS | PROTEIN | RECOGNITION | TRAF2 | IMMUNOLOGY | IL-6 | autoimmunity | inflammation | STRUCTURAL BASIS | TRAF5 | T(H)17 | TRAF FAMILY | GENES | EXPRESSION | LYMPHOCYTES | Research | T cells | Cell differentiation | Tumor necrosis factor | Interleukin-6 | TH17
Journal Article
Experimental Eye Research, ISSN 0014-4835, 2010, Volume 91, Issue 3, pp. 462 - 471
Corneal epithelial injury induces release of endogenous metabolites that are cannabinoid receptor 1 (CB1) and transient receptor potential vanilloid 1 (TRPV1)... 
proinflammatory cytokine | cell proliferation | epidermal growth factor receptor (EGFR) | cell migration | transient receptor potential vanilloid 1 (TRPV1) | protein phosphorylation | cannabinoid receptor 1 (CB1) | transactivation | Cell proliferation | Transient receptor potential vanilloid 1 (TRPV1) | Epidermal growth factor receptor (EGFR) | Proinflammatory cytokine | Protein phosphorylation | Transactivation | Cannabinoid receptor 1 (CB1) | Cell migration | STIMULATION | EGFR ACTIVATION | MAP KINASE | WOUNDING SHEETS | ANANDAMIDE | PROLIFERATION | MECHANISMS | INFLAMMATION | NEURONS | OPHTHALMOLOGY | MOTILITY | Phosphorylation | Cell Proliferation | Capsaicin - pharmacology | Humans | Quinazolines | TRPV Cation Channels - metabolism | Receptor, Epidermal Growth Factor - metabolism | Piperidines - pharmacology | TRPV Cation Channels - antagonists & inhibitors | Epithelium, Corneal - drug effects | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Fluorescent Antibody Technique, Indirect | Pyrazoles - pharmacology | Enzyme-Linked Immunosorbent Assay | Mitogen-Activated Protein Kinase 9 - metabolism | Mitogen-Activated Protein Kinase 8 - metabolism | Cells, Cultured | Morpholines - pharmacology | Tyrphostins - pharmacology | Blotting, Western | Naphthalenes - pharmacology | Receptor, Cannabinoid, CB1 - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Benzoxazines - pharmacology | Epithelium, Corneal - metabolism | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Transcriptional Activation - physiology | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Cell Movement | Mitogen-Activated Protein Kinase 1 - metabolism | Tyrosine | Anticoagulants (Medicine) | Glycosaminoglycans | Epidermal growth factor | Metabolites
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2013, Volume 110, Issue 42, pp. 16975 - 16980
The activation of STAT3 by tyrosine phosphorylation, essential for normal development and for a normal inflammatory response to invading pathogens, is kept in... 
Hep G2 cells | Phosphorylation | Receptors | Cytokines | Secretion | Antibodies | Washing | Gene expression regulation | Endothelial cells | Cancer | MULTIDISCIPLINARY SCIENCES | EPIDERMAL-GROWTH-FACTOR | IN-VIVO | GENE-EXPRESSION | RESISTANCE | BLASTOCYST IMPLANTATION | LEUKEMIA INHIBITORY FACTOR | IDENTIFICATION | CANCER | T-CELLS | SIGNAL TRANSDUCER | Neoplasms - metabolism | Phosphorylation - physiology | Inflammation - pathology | Receptor, Epidermal Growth Factor - genetics | Cytokine Receptor gp130 - genetics | Humans | Oncostatin M - pharmacology | Antineoplastic Agents - metabolism | Janus Kinase 1 - metabolism | Inflammation - metabolism | Receptor, Epidermal Growth Factor - metabolism | Neoplasms - genetics | Janus Kinase 1 - genetics | Janus Kinase 2 - metabolism | Oncostatin M - metabolism | Antineoplastic Agents - pharmacology | Phosphorylation - drug effects | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Cytokine Receptor gp130 - metabolism | Interleukin-6 - genetics | Janus Kinase 2 - genetics | Gene Expression Regulation - physiology | Suppressor of Cytokine Signaling Proteins - genetics | Hep G2 Cells | Gene Expression Regulation - drug effects | Suppressor of Cytokine Signaling 3 Protein | Interleukin-6 - pharmacology | Inflammation - genetics | Interleukin-6 - biosynthesis | Suppressor of Cytokine Signaling Proteins - metabolism | Neoplasms - pathology | Protein Binding - physiology | Physiological aspects | Epidermal growth factor | Research | Interleukins | Health aspects | Binding sites (Biochemistry) | Biological Sciences
Journal Article
Journal Article