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PLoS Biology, ISSN 1544-9173, 11/2004, Volume 2, Issue 11, pp. 1862 - 1879
Journal Article
2014, Advances in experimental medicine and biology, ISBN 1493903977, Volume 809, xxi, 188
Book
Journal of Biological Chemistry, ISSN 0021-9258, 04/2010, Volume 285, Issue 17, pp. 13107 - 13120
Journal Article
EMBO reports, ISSN 1469-221X, 07/2017, Volume 18, Issue 7, pp. 1100 - 1107
Journal Article
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 01/2018, Volume 24, Issue 1, pp. 95 - 105
Metastasis to the brain from breast cancer remains a significant clinical challenge, and may be targeted with CAR-based immunotherapy. CAR design optimization... 
Receptor, ErbB-2 - genetics | Breast Neoplasms - immunology | Humans | Tumor Necrosis Factor Receptor Superfamily, Member 9 - genetics | Recombinant Fusion Proteins - metabolism | Breast Neoplasms - metabolism | Brain Neoplasms - secondary | Lymphocyte Activation - genetics | Lymphocyte Activation - immunology | Brain Neoplasms - immunology | CD28 Antigens - genetics | T-Lymphocytes - metabolism | Inflammation Mediators - metabolism | Female | Receptors, Chimeric Antigen - metabolism | Disease Models, Animal | Immunotherapy, Adoptive - methods | Cytokines - metabolism | Receptors, Antigen, T-Cell - metabolism | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Brain Neoplasms - therapy | Receptors, Chimeric Antigen - genetics | Recombinant Fusion Proteins - genetics | T-Lymphocytes - immunology | Cytotoxicity, Immunologic | Cell proliferation | Brain | Animal models | Intravenous administration | Brain tumors | Xenotransplantation | Medical services | Intracellular signalling | Lymphocytes T | Metastasis | CD28 antigen | Optimization | Metastases | Design optimization | Anticancer properties | Design | Lymphocytes | Immunotherapy | Safety engineering | Xenografts | Meninges | Antigens | Phenotypes | Medical treatment | Breast cancer | T cell receptors | Automobiles | ErbB-2 protein | Experimental design | Exhaustion | Breast | Antitumor activity | Solid tumors | Cancer | Tumors
Journal Article
Apoptosis, ISSN 1360-8185, 9/2007, Volume 12, Issue 9, pp. 1543 - 1568
Apoptosis has been accepted as a fundamental component in the pathogenesis of cancer, in addition to other human diseases including neurodegeneration, coronary... 
Biochemistry, general | TNFR | TRAF | Oncology | NFkB | Virology | Cell Biology | Biomedicine | Cancer Research | IAPs | Caspases | Smac | Knock out mice | Apoptosis | Cancer | NEURONAL APOPTOSIS | X-LINKED INHIBITOR | SERINE-PROTEASE OMI/HTRA2 | MITOCHONDRIA-DERIVED ACTIVATOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | knock out mice | INDUCED PROXIMITY MODEL | apoptosis | CELL BIOLOGY | CELL LUNG-CANCER | SPINAL MUSCULAR-ATROPHY | IN-VIVO | cancer | CYTOCHROME-C RELEASE | caspases | CASPASE ACTIVATION | Caspases - physiology | Proto-Oncogene Proteins c-bcl-2 - physiology | Apoptosis - drug effects | Inhibitor of Apoptosis Proteins - genetics | Humans | Neoplasm Proteins - physiology | Serine Endopeptidases - physiology | Neoplasm Proteins - antagonists & inhibitors | Inhibitor of Apoptosis Proteins - physiology | X-Linked Inhibitor of Apoptosis Protein - antagonists & inhibitors | Neoplasms - genetics | Inhibitor of Apoptosis Proteins - antagonists & inhibitors | Oligonucleotides, Antisense - therapeutic use | Intracellular Signaling Peptides and Proteins - therapeutic use | Mitochondrial Proteins - therapeutic use | Peptide Termination Factors - physiology | Mitochondrial Proteins - physiology | Neoplasm Proteins - therapeutic use | Microtubule-Associated Proteins - antagonists & inhibitors | Caspase Inhibitors | High-Temperature Requirement A Serine Peptidase 2 | Neoplasms - drug therapy | Animals | Protein Kinases - physiology | Checkpoint Kinase 1 | Enzyme Activation | Genetic Vectors | Intracellular Signaling Peptides and Proteins - physiology | Proteins | Cancer therapies
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e66987
Non-steroidal anti-inflammatory drugs such as diclofenac exhibit potent anticancer effects. Up to now these effects were mainly attributed to its classical... 
MONOCARBOXYLATE TRANSPORTERS | COLON-CANCER CELLS | SALICYLIC-ACID | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | INDUCED APOPTOSIS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | GROWTH | C-MYC | MALIGNANT-MELANOMA | INDUCE APOPTOSIS | Melanoma - metabolism | Neoplasms - metabolism | Diclofenac - pharmacology | Humans | Lactic Acid - metabolism | Leukemia - metabolism | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Gene Expression Regulation - drug effects | Animals | Neoplasms - genetics | Melanoma - genetics | Intracellular Space - metabolism | Cell Line, Tumor | Glucose - metabolism | Carcinoma - genetics | Biological Transport - drug effects | Cell Proliferation - drug effects | Proto-Oncogene Proteins c-myc - genetics | Carcinoma - metabolism | Metabolic Networks and Pathways - drug effects | Leukemia - genetics | Glucose metabolism | Aspirin | Anti-inflammatory drugs | Leukemia | Genes | Melanoma | Glucose | Gene expression | Dextrose | Cell proliferation | Drugs | c-Myc protein | Myc protein | Kinases | Cancer therapies | Accumulation | Diclofenac | Anticancer properties | Cell growth | Psychiatrists | Nonsteroidal anti-inflammatory drugs | Glucose transporter | Efflux | Antiinflammatory agents | Secretion | Tumor cells | Lactate dehydrogenase | Inflammation | Metabolism | L-Lactate dehydrogenase | Inhibitors | Glycolysis | Lactic acid | Cyclooxygenase-2 | Intracellular | Transporter
Journal Article
Journal Article