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Comparative Clinical Pathology, ISSN 1618-5641, 2/2019, Volume 28, Issue 1, pp. 89 - 95
Ischemic reperfusion is an optic neuropathy in which photoreceptor cells may die through an apoptotic process. Free radicals are involved in the apoptotic... 
Pathology | Transmission electron microscopy | Medicine & Public Health | Hematology | Rat | Oncology | Curcumin | Photoreceptor cell | Ischemic reperfusion | Nutrient deficiency | Free radicals | Optic neuropathy | Retina | Neuropathy | Antioxidants | Reperfusion | Ischemia | Diet | Rodents | Photoreceptors | Cell size | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e46479
Purpose: Ischemia and reperfusion injury (I/R) of neuronal structures and organs is associated with increased morbidity and mortality due to neuronal cell... 
REPERFUSION LUNG INJURY | OXIDATIVE STRESS | ACTIVATED PROTEIN-KINASE | MICROGLIA | PATHWAY | MULTIDISCIPLINARY SCIENCES | HEAT-SHOCK RESPONSE | GANGLION-CELLS | CARDIOPULMONARY BYPASS | INFLAMMATORY RESPONSE | MULLER CELLS | Retina - drug effects | Heme Oxygenase-1 - metabolism | Phosphorylation | Gene Expression - drug effects | Apoptosis - drug effects | Caspase 3 - metabolism | Reperfusion Injury - drug therapy | Male | NF-kappa B - metabolism | Retinitis - prevention & control | Neuroglia - drug effects | Retinal Ganglion Cells - pathology | Retinal Vessels - pathology | Heme Oxygenase-1 - genetics | Proto-Oncogene Proteins c-bcl-2 - metabolism | Protein Processing, Post-Translational - drug effects | Caspase 3 - genetics | Anti-Inflammatory Agents - administration & dosage | Female | Neuroprotective Agents - administration & dosage | bcl-2-Associated X Protein - genetics | Neuroglia - physiology | bcl-2-Associated X Protein - metabolism | Administration, Inhalation | Rats | Rats, Sprague-Dawley | Cell Movement - drug effects | Animals | Ischemia - drug therapy | Enzyme Activation | Carbon Monoxide - administration & dosage | Proto-Oncogene Proteins c-bcl-2 - genetics | Retina - pathology | Retinal Ganglion Cells - drug effects | Mitogen-Activated Protein Kinases - metabolism | Perioperative care | Ischemia | Neurons | Analysis | Carbon monoxide | Research | Health aspects | Risk factors | Apoptosis | Traumatic brain injury | Bax protein | Bcl-2 protein | Critical care | Retina | Kinases | Macrophages | Caspase-3 | Proteins | Cell activation | Reperfusion | Rodents | Inhibition | Injuries | Injury analysis | Enzymes | NF-κB protein | Stroke | Glial fibrillary acidic protein | Organs | Extracellular signal-regulated kinase | Caspase | MAP kinase | Inflammation | Morbidity | Inhalation | Latency | Time lag | Microglia | Medicine | Retinal ganglion cells | Brain research | Cell death | Immigration | Anesthesiology | Laboratory animals | Respiration | Cell migration
Journal Article
Journal Article
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 02/2009, Volume 108, Issue 3, pp. 741 - 754
δ‐Opioid receptor (DOR) is an oxygen‐sensitive protein whose function in the rat retina is unknown. We examined whether DOR is involved in hypoxic... 
extracellular signal‐regulated kinase | hypoxia‐inducible factor‐1α | δ‐opioid receptor | hypoxic preconditioning | intraocular pressure | oxidative stress | Extracellular signal-regulated kinase | Hypoxic preconditioning | Hypoxia-inducible factor-1α | Oxidative stress | δ-opioid receptor | Intraocular pressure | extracellular signal-regulated kinase | OPTIC-NERVE | AMACRINE CELLS | delta-opioid receptor | RABBIT RETINA | BIOCHEMISTRY & MOLECULAR BIOLOGY | MAMMALIAN RETINA | NEUROSCIENCES | INTERMITTENT HYPOXIA | INDUCED ISCHEMIA | NG108-15 CELLS | GANGLION-CELLS | IN-VIVO | hypoxia-inducible factor-1 alpha | Immunohistochemistry | Naltrexone - analogs & derivatives | Rats, Wistar | Antioxidants - metabolism | Oxidative Stress - physiology | Male | Dinoprost - metabolism | Retinal Vessels - physiology | Naltrexone - pharmacology | Lipid Peroxidation - drug effects | Superoxides - metabolism | Receptors, Opioid, delta - antagonists & inhibitors | Dinoprost - analogs & derivatives | Hypoxia-Inducible Factor 1, alpha Subunit - biosynthesis | Enkephalin, Leucine - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | RNA, Small Interfering - pharmacology | Intraocular Pressure - physiology | Ischemic Preconditioning | Rats | Spectrometry, Fluorescence | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Retina - physiology | Animals | Narcotic Antagonists - pharmacology | Hypoxia - physiopathology | Signal Transduction - physiology | Receptors, Opioid, delta - physiology
Journal Article
Documenta Ophthalmologica, ISSN 0012-4486, 2/2010, Volume 120, Issue 1, pp. 67 - 86
Journal Article
Journal Article