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American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 07/2011, Volume 45, Issue 1, pp. 1 - 15
Idiopathic pulmonary fibrosis (IPF) is a disabling disease of the lung parenchyma, characterized by progressive accumulation of scar tissue and myofibroblast... 
Angiogenesis | Growth factors | Idiopathic pulmonary fibrosis | Pulmonary hypertension | Apoptosis | BRONCHOALVEOLAR LAVAGE FLUID | FACTOR-BETA | growth factors | pulmonary hypertension | angiogenesis | BIOCHEMISTRY & MOLECULAR BIOLOGY | apoptosis | PLASMINOGEN-ACTIVATOR INHIBITOR-1 | idiopathic pulmonary fibrosis | ENDOTHELIAL-CELL APOPTOSIS | SYSTEMIC-SCLEROSIS | PIGMENT-EPITHELIUM | CELL BIOLOGY | INTERSTITIAL LUNG-DISEASE | EPITHELIUM-DERIVED FACTOR | RECEPTOR TYROSINE KINASES | RESPIRATORY SYSTEM | ARTERIAL-HYPERTENSION | Hypertension, Pulmonary - diagnosis | Epithelial Cells - metabolism | Prognosis | Humans | Hypertension, Pulmonary - physiopathology | Hypertension, Pulmonary - therapy | Idiopathic Pulmonary Fibrosis - metabolism | Neovascularization, Pathologic - pathology | Pulmonary Artery - metabolism | Angiogenesis Inducing Agents - metabolism | Neovascularization, Pathologic - physiopathology | Idiopathic Pulmonary Fibrosis - complications | Fibroblasts - metabolism | Biomarkers - metabolism | Neovascularization, Pathologic - diagnosis | Vasoconstriction | Epithelial Cells - pathology | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Fibroblasts - pathology | Angiogenesis Inhibitors - metabolism | Animals | Idiopathic Pulmonary Fibrosis - diagnosis | Idiopathic Pulmonary Fibrosis - pathology | Idiopathic Pulmonary Fibrosis - physiopathology | Neovascularization, Pathologic - metabolism | Hypertension, Pulmonary - etiology | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 05/2014, Volume 370, Issue 22, pp. 2071 - 2082
Journal Article
Thorax, ISSN 0040-6376, 05/2013, Volume 68, Issue 5, pp. 436 - 441
Journal Article
Clinical Immunology, ISSN 1521-6616, 2010, Volume 137, Issue 1, pp. 89 - 101
Abstract Activated macrophages have been characterized as M1 and M2 according to their inflammatory response pattern. Here we analyzed the M2 marker expression... 
Allergy and Immunology | M2 phenotype | Th2 cytokines | Pulmonary fibrosis | IL-1RA | CCL22 | Sarcoidosis | JAK/STAT pathway | CCL18 | Alveolar macrophages | CCL17 | INTERLEUKIN-1 RECEPTOR ANTAGONIST | LUNG INJURY | CYTOKINE | IMMUNOLOGY | SYSTEMIC-SCLEROSIS | STAT3 ACTIVATION | IN-VITRO | GENE-EXPRESSION | TNF-ALPHA RELEASE | GROWTH-FACTOR | T-LYMPHOCYTES | Mannose-Binding Lectins - metabolism | Tumor Necrosis Factor-alpha - metabolism | Gene Expression - drug effects | Humans | Middle Aged | STAT Transcription Factors - metabolism | Interleukin 1 Receptor Antagonist Protein - genetics | Male | Monocytes - metabolism | Monocytes - immunology | Sarcoidosis, Pulmonary - diagnosis | Idiopathic Pulmonary Fibrosis - metabolism | Young Adult | Lectins, C-Type - metabolism | Signal Transduction - immunology | Interleukin-4 - pharmacology | Interleukin 1 Receptor Antagonist Protein - metabolism | Scleroderma, Systemic - complications | Aged, 80 and over | Adult | Female | Bronchoalveolar Lavage Fluid - cytology | Pulmonary Fibrosis - metabolism | Interleukin-8 - metabolism | Macrophages, Alveolar - immunology | Pulmonary Fibrosis - etiology | Sarcoidosis, Pulmonary - complications | Cytokines - metabolism | Macrophage Activation - immunology | Scleroderma, Systemic - metabolism | Pulmonary Fibrosis - immunology | Receptors, Cell Surface - metabolism | Idiopathic Pulmonary Fibrosis - immunology | Monocytes - drug effects | Signal Transduction - drug effects | Macrophages, Alveolar - drug effects | Sarcoidosis, Pulmonary - metabolism | Chemokines, CC - genetics | Aged | Macrophages, Alveolar - metabolism | Macrophage Activation - drug effects | Interleukin-10 - pharmacology | Chemokines, CC - metabolism | Respiratory tract diseases | Systemic scleroderma | Transforming growth factors | Macrophages | Scleroderma (Disease) | Alveoli
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2008, Volume 105, Issue 35, pp. 13051 - 13056
Idiopathic interstitial pneumonias (IIPs) have a progressive and often fatal course, and their enigmatic etiology has complicated approaches to effective... 
Telomeres | Pulmonary alveoli | Lungs | Interstitial lung diseases | Idiopathic pulmonary fibrosis | Liver | Fibrosis | Dyskeratosis congenita | Genetic mutation | Liver cirrhosis | Liver fibrosis | Interstitial lung disease | Telomerase | Aplastic anemia | RNA | MULTIDISCIPLINARY SCIENCES | DOMINANT DYSKERATOSIS-CONGENITA | REVERSE-TRANSCRIPTASE | LENGTH | IN-SITU HYBRIDIZATION | TERMINAL TRANSFERASE | liver fibrosis | dyskeratosis congenita | telomerase | GENE | interstitial lung disease | FAMILIES | aplastic anemia | MUTATIONS | APLASTIC-ANEMIA | Biomarkers - metabolism | Leukocytes, Mononuclear - metabolism | Epithelium - pathology | Pulmonary Alveoli - pathology | Pulmonary Fibrosis - complications | Fibrosis - complications | Humans | Risk Factors | In Situ Hybridization, Fluorescence | Tomography, X-Ray Computed | Pulmonary Fibrosis - genetics | Pulmonary Fibrosis - pathology | Case-Control Studies | RNA - genetics | Fibrosis - diagnostic imaging | Telomerase - genetics | Pulmonary Fibrosis - diagnostic imaging | Germ-Line Mutation | Family | Heterozygote | Telomere - metabolism | Fibrosis - pathology | Genetic aspects | Pulmonary fibrosis | Properties | Health aspects | Risk factors | Pneumonia | Epithelial cells | Lung | Lung diseases | Heredity | Leukocytes | Cirrhosis | Etiology | Mutation | telomerase reverse transcriptase | Alveoli | Age | Biological Sciences
Journal Article