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PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e64647
Globoid-cell Leukodystrophy (GLD; Krabbes disease) is a rapidly progressing inherited demyelinating disease caused by a deficiency of the lysosomal enzyme... 
TWITCHER MOUSE | ATHEROSCLEROTIC LESIONS | MURINE MODEL | BONE-MARROW-TRANSPLANTATION | MULTIDISCIPLINARY SCIENCES | SPINAL-CORD | GENE-THERAPY | MICE | EXPRESSION | AUTOIMMUNE ENCEPHALOMYELITIS | KRABBE-DISEASE | Inflammation - pathology | Oligodendroglia - metabolism | Cell Proliferation | Central Nervous System - metabolism | Lumbar Vertebrae - metabolism | Mice, Inbred C57BL | Leukodystrophy, Globoid Cell - pathology | Central Nervous System - pathology | Galactosylceramidase - deficiency | Chemokine CXCL1 - deficiency | Longevity | Receptors, Interleukin-8B - deficiency | Disease Progression | Oligodendroglia - pathology | Animals | Flow Cytometry | Bone Marrow - metabolism | Bone Marrow - pathology | Leukodystrophy, Globoid Cell - metabolism | Receptors, Interleukin-8B - metabolism | Lumbar Vertebrae - pathology | Chemokine CXCL1 - metabolism | Disease Models, Animal | Galactosylceramidase - metabolism | Cytokines | Physiological aspects | Development and progression | Genetic aspects | Research | Chemokine receptors | Globoid cell leukodystrophy | Neurosciences | Animal models | Spinal cord | Transplants & implants | Syngeneic grafts | Pathogenesis | Central nervous system | Cognitive ability | Bone marrow transplantation | Cytotoxicity | Nervous system | Transplantation | Macrophages | Immunology | Galactosylceramidase | Demyelination | Transgenic animals | Rodents | Bone marrow | Growth factors | Seizures | Enzymes | Internal medicine | Leukodystrophy | CXCR2 protein | Spasticity | Inflammation | Microglia | Medicine | Pathology | Catabolism | Mice | Bone | Gene therapy | Galactosylceramide | Cell migration | Chemokines
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