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Gastroenterology, ISSN 0016-5085, 2014, Volume 146, Issue 7, pp. 1763 - 1774
Background & Aims The NACHT, LRR, and pyrin domain–containing protein 3 (NLRP3) inflammasome induces inflammation in response to organ injury, but little is... 
Gastroenterology and Hepatology | Innate Immune Response | Immune Regulation | Pancreas | Mouse Model | ACTIVATION | NLRP3 INFLAMMASOME | GPR81 | AGONISTS | GENE | TLR9 | MICE | HEPATOTOXICITY | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | SEVERITY | Liver - pathology | Inflammasomes - metabolism | Receptors, G-Protein-Coupled - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Male | NF-kappa B - metabolism | Monocytes - immunology | Lipopolysaccharides | Liver - immunology | Liver - drug effects | RNA Interference | Interleukin-1beta - metabolism | Toll-Like Receptors - drug effects | Anti-Inflammatory Agents - administration & dosage | Toll-Like Receptors - metabolism | Cytoprotection | Disease Models, Animal | Galactosamine | Chemical and Drug Induced Liver Injury - prevention & control | Anti-Inflammatory Agents - pharmacology | Down-Regulation | Liver - metabolism | Injections, Intraperitoneal | Pancreas - pathology | Pancreas - metabolism | Pancreas - immunology | Toll-Like Receptor 4 - metabolism | Chemical and Drug Induced Liver Injury - immunology | Monocytes - drug effects | Macrophages - metabolism | Signal Transduction - drug effects | Chemical and Drug Induced Liver Injury - metabolism | Sodium Lactate - pharmacology | beta-Arrestins | Mice | Receptors, G-Protein-Coupled - genetics | RNA, Small Interfering - metabolism | Monocytes - metabolism | Pancreatitis - prevention & control | Arrestins - metabolism | Dose-Response Relationship, Drug | Pancreatitis - genetics | Transfection | Inflammasomes - drug effects | Sodium Lactate - administration & dosage | Pancreatitis - immunology | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - etiology | Macrophages - immunology | Cell Line | Immunity, Innate - drug effects | Toll-Like Receptor 4 - drug effects | Mice, Inbred C57BL | Pancreas - drug effects | Chemical and Drug Induced Liver Injury - genetics | Pancreatitis - chemically induced | Animals | Carrier Proteins - metabolism | beta-Arrestin 2 | Inflammasomes - immunology | Macrophages - drug effects | Pancreatitis - pathology | Pancreatitis - metabolism | Ceruletide | Lactates | Gastrointestinal diseases | Inflammation | Index Medicus | Abridged Index Medicus
Journal Article
Nature, ISSN 0028-0836, 04/2014, Volume 508, Issue 7494, pp. 123 - 7
Journal Article
Nature, ISSN 0028-0836, 09/2017, Volume 549, Issue 7671, pp. 282 - 286
The type 2 cytokines interleukin (IL)-4, IL-5, IL-9 and IL-13 have important roles in stimulating innate and adaptive immune responses that are required for... 
CRTH2 | CYTOKINES | IMMUNE INTERACTIONS | RECEPTORS | MULTIDISCIPLINARY SCIENCES | Inflammation - chemically induced | Inflammation - pathology | Eosinophils - drug effects | Pneumonia - pathology | Interleukin-13 - immunology | Male | Adoptive Transfer | Nippostrongylus - immunology | Receptors, Neurotransmitter - genetics | Eosinophils - immunology | Gastrointestinal Tract - immunology | Lymphocytes - immunology | Pneumonia - chemically induced | Neuropeptides - pharmacology | Interleukin-9 - metabolism | Pneumonia - immunology | Female | GTP-Binding Protein alpha Subunits, Gq-G11 - metabolism | Gastrointestinal Tract - innervation | Cytokines - immunology | Cytokines - metabolism | Immunity, Innate - drug effects | Cholinergic Neurons - metabolism | Interleukin-5 - metabolism | Neuropeptides - metabolism | Inflammation - immunology | Lymphocytes - cytology | Cholinergic Neurons - drug effects | Interleukin-13 - metabolism | Interleukin-5 - immunology | Receptors, Neurotransmitter - deficiency | Animals | Signal Transduction - drug effects | Lymphocytes - drug effects | Gastrointestinal Tract - cytology | Mice | Receptors, Neurotransmitter - metabolism | Eosinophils - cytology | Interleukin-9 - immunology | Lung | Genomics | Mucosa | Interleukin | Homeostasis | Nervous system | Neuropeptides | Activation | Small intestine | Antiinfectives and antibacterials | Proteins | Cell activation | Microorganisms | Enteric nervous system | Interleukin 5 | Immune system | Interleukin 9 | Lymphoid cells | Immune response | Cytokines | Secretion | Neurons | Hypersensitivity | Gastrointestinal tract | Inflammation | Interleukin 13 | Signaling | Ligands | In vitro methods and tests | Neuromedin | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2016, Volume 113, Issue 17, pp. 4777 - 4782
Journal Article
Nature, ISSN 0028-0836, 09/2017, Volume 549, Issue 7671, pp. 277 - 281
Group 2 innate lymphoid cells (ILC2s) regulate inflammation, tissue repair and metabolic homeostasis(1), and are activated by host-derived cytokines and... 
HOMEOSTASIS | IMMUNE INTERACTIONS | RAT | IMMUNOREACTIVITY | PROBE LEVEL | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | MICE | RECEPTOR RET | IDENTIFICATION | ORGANOGENESIS | Cytokines - metabolism | Immunity, Innate - drug effects | Calcium - metabolism | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Male | Neuropeptides - metabolism | Lymphocytes - cytology | Nippostrongylus - immunology | Animals | Lymphocytes - immunology | MAP Kinase Signaling System - drug effects | Strongylida Infections - parasitology | Lymphocytes - drug effects | Neuropeptides - pharmacology | Strongylida Infections - immunology | Female | Mice | Neurons - metabolism | Receptors, Neurotransmitter - metabolism | Neurons - drug effects | Calcineurin - metabolism | Cytokines - immunology | Physiological aspects | Physiological research | Lymphoid tissue | Research | Biological control systems | Neurons | Transcription factors | Calcium | Calcineurin | Mucosa | Homeostasis | Nervous system | Neuropeptides | Innate immunity | Activation | NF-AT protein | Kinases | Immunity | Calcium influx | Calcium signalling | Cell activation | Lymphocytes | Bone marrow | Lymphoid cells | Cytokines | Dendritic cells | Extracellular signal-regulated kinase | Inflammation | Metabolism | Ablation | Immune systems | Studies | Calcium (extracellular) | Neuromedin | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2009, Volume 106, Issue 49, pp. 20842 - 20846
Journal Article
The Plant Cell, ISSN 1040-4651, 12/2006, Volume 18, Issue 12, pp. 3721 - 3744
We show that oomycete-derived Nep1 (for necrosis and ethylene-inducing peptide1)-like proteins (NLPs) trigger a comprehensive immune response in Arabidopsis... 
Disease resistance | Pathogens | Cell death | Genes | Innate immunity | Toxins | Cell membranes | Plants | Animal cells | Plant cells | H+-ATPASE ACTIVITY | ARABIDOPSIS-THALIANA | SIGNALING PATHWAYS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PLANT SCIENCES | CELL BIOLOGY | PROGRAMMED CELL-DEATH | CAROTOVORA SUBSP CAROTOVORA | GENE-EXPRESSION | MULTIPLE DEFENSE RESPONSES | PORE-FORMING TOXINS | DISEASE RESISTANCE | HYPERSENSITIVE RESPONSE | Cell Death - radiation effects | Genes, Plant | Arabidopsis - immunology | Seedlings - microbiology | Phylogeny | Genetic Variation | Arabidopsis - radiation effects | Light | Protein Binding - drug effects | Gene Expression Regulation, Plant | Protein Binding - radiation effects | Plant Leaves - drug effects | Cell Death - drug effects | Germination - radiation effects | Plant Leaves - enzymology | Cell Membrane - drug effects | Germination - drug effects | Immunity, Innate - drug effects | Tobacco - drug effects | Plant Leaves - radiation effects | Seedlings - drug effects | Tobacco - radiation effects | Arabidopsis - genetics | Arabidopsis - microbiology | Immunity, Innate - radiation effects | Tobacco - microbiology | Fungal Proteins - pharmacology | Lipid Bilayers - metabolism | Seedlings - radiation effects | Cytoplasm - drug effects | Plant Leaves - microbiology | Fungal Proteins - metabolism | Arabidopsis thaliana | Immune response | Research | Peptides | Risk factors | Necrosis | Index Medicus
Journal Article
Journal Article
Cell Reports, ISSN 2211-1247, 09/2016, Volume 16, Issue 10, pp. 2576 - 2592
The mechanisms underlying Zika virus (ZIKV)-related microcephaly and other neurodevelopment defects remain poorly understood. Here, we describe the derivation... 
NEURAL PROGENITORS | LONG-TERM | HUMAN BRAIN | ADAPTER | CENTRAL-NERVOUS-SYSTEM | INFECTION | MICE | BINDING KINASE 1 | ORGANOIDS | INNATE IMMUNITY | CELL BIOLOGY | Neocortex - pathology | Neurons - pathology | Transcription, Genetic - drug effects | Brain - embryology | Neuroglia - ultrastructure | Neuroglia - pathology | Humans | Brain - virology | Centrosome - drug effects | Gene Expression Profiling | Microcephaly - virology | Neural Stem Cells - ultrastructure | Zika Virus Infection - virology | Neural Stem Cells - immunology | Neuroepithelial Cells - immunology | Neuroprotective Agents - pharmacology | Spinal Cord - pathology | Microcephaly - pathology | Neuroepithelial Cells - virology | Nucleosides - pharmacology | Fetus - virology | Cell Death - drug effects | Phosphorylation - drug effects | Neurons - drug effects | Protein-Serine-Threonine Kinases - metabolism | Zika Virus - pathogenicity | Proto-Oncogene Proteins - metabolism | Zika Virus - ultrastructure | Neurons - virology | Virus Replication - drug effects | Neuroepithelial Cells - ultrastructure | Immunity, Innate - drug effects | Zika Virus Infection - pathology | Neural Stem Cells - virology | Zika Virus - physiology | Zika Virus - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Protein-Tyrosine Kinases - metabolism | Neural Stem Cells - enzymology | Centrosome - metabolism | Mitosis - drug effects | Brain - pathology | Protein Kinase Inhibitors - pharmacology | Neuroglia - virology | Neuroepithelial Cells - drug effects | Index Medicus | Neurons/pathology | Zika Virus/pathogenicity | Mitochondria/metabolism | Virus Replication/drug effects | Microcephaly/pathology | Neurons/drug effects | Protein-Serine-Threonine Kinases/metabolism | Neural Stem Cells/immunology | Neuroglia/ultrastructure | Neuroepithelial Cells/drug effects |