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Nature Medicine, ISSN 1078-8956, 2011, Volume 17, Issue 5, pp. 610 - 617
Journal Article
Science, ISSN 0036-8075, 04/2016, Volume 352, Issue 6284, pp. 453 - 459
Journal Article
NATURE STRUCTURAL & MOLECULAR BIOLOGY, ISSN 1545-9985, 05/2006, Volume 13, Issue 5, pp. 462 - 464
Journal Article
Genes and Immunity, ISSN 1466-4879, 09/2017, Volume 18, Issue 3, pp. 197 - 199
Agammaglobulinemia is a primary immunodeficiency disorder characterized by profoundly low or absent serum antibodies and low or absent circulating B cells. The... 
Immunoglobulin mu-Chains - genetics | Agammaglobulinemia - pathology | Heavy Chain Disease - genetics | Agammaglobulinemia - genetics | Gene Deletion | Humans | Female | Heterozygote | Infant | Codon, Nonsense | Genes, Recessive | Index Medicus
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2016, Volume 36, Issue 4, pp. 682 - 691
OBJECTIVE—Little is known about the role(s) B cells play in obesity-induced metabolic dysfunction. This study used a mouse with B-cell–specific deletion of Id3... 
adipose tissue | cytokines | B cells | inflammation | obesity | T15 IDIOTYPE | GREATER OMENTUM | CROWN-LIKE STRUCTURES | OXIDATION-SPECIFIC EPITOPES | NATURAL ANTIBODIES | MILKY SPOTS | STREPTOCOCCUS-PNEUMONIAE | ADIPOSE-TISSUE INFLAMMATION | APOPTOTIC CELLS | PERIPHERAL VASCULAR DISEASE | OXIDIZED PHOSPHOLIPIDS | HEMATOLOGY | B-Lymphocyte Subsets - transplantation | Obesity - immunology | Homeodomain Proteins - metabolism | Humans | Inhibitor of Differentiation Proteins - genetics | Male | Adoptive Transfer | Insulin - blood | Glucose Intolerance - immunology | Obesity - blood | Obesity - genetics | Glucose Intolerance - blood | Inflammation - blood | Glucose Intolerance - prevention & control | Time Factors | Inflammation Mediators - metabolism | B-Lymphocyte Subsets - immunology | Disease Models, Animal | Intra-Abdominal Fat - immunology | Glucose Intolerance - genetics | Cytokines - metabolism | Immunoglobulin mu-Chains - immunology | Obesity - complications | Tissue Culture Techniques | Mice, Inbred C57BL | Cells, Cultured | Insulin Resistance | Immunoglobulin mu-Chains - metabolism | Genotype | Inflammation - immunology | Inhibitor of Differentiation Proteins - metabolism | Biomarkers - blood | Intra-Abdominal Fat - metabolism | Homeodomain Proteins - genetics | Mice, Knockout | Immunoglobulin mu-Chains - genetics | Phenotype | Animals | B-Lymphocyte Subsets - metabolism | Inflammation - genetics | Inflammation - prevention & control | Blood Glucose - metabolism | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e90383 - e90383
Journal Article
Blood, ISSN 0006-4971, 03/2013, Volume 121, Issue 13, pp. 2512 - 2521
Mechanisms of spontaneous tumor regression have been difficult to characterize in a systematic manner due to their rare occurrence and the lack of model... 
T-Lymphocytes - physiology | Molecular Sequence Data | Apoptosis - genetics | DNA-Binding Proteins - metabolism | T-Lymphocytes - metabolism | Cell Transformation, Neoplastic - genetics | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Killer Cells, Natural - immunology | Neoplasm Regression, Spontaneous - immunology | Protein-Serine-Threonine Kinases - metabolism | Genes, myc - physiology | Amino Acid Sequence | Neoplasm Regression, Spontaneous - genetics | DNA-Binding Proteins - physiology | Tumor Suppressor Proteins - metabolism | Protein-Serine-Threonine Kinases - physiology | Cell Cycle Proteins - metabolism | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Leukemia, B-Cell - pathology | Mice, Transgenic | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | Mice, SCID | Cell Transformation, Neoplastic - metabolism | Killer Cells, Natural - physiology | Immunoglobulin mu-Chains - genetics | Tumor Suppressor Proteins - physiology | Animals | Apoptosis - immunology | Leukemia, B-Cell - genetics | T-Lymphocytes - immunology | Enhancer Elements, Genetic - genetics | Mice | Killer Cells, Natural - metabolism | Cell Transformation, Neoplastic - pathology | Leukemia, B-Cell - immunology | Cell Cycle Proteins - physiology | Index Medicus | Abridged Index Medicus | Lymphoid Neoplasia
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 11/2018, Volume 506, Issue 1, pp. 20 - 26
Visceral leishmaniasis, the most severe form of leishmaniasis, is caused by and . Immunity to infection has been shown to depend on the development of Th1... 
AID and μs double-deficient mice | Circulating immunoglobulins | Interferon-γ | Leishmania donovani | NADPH oxidase | Interferon-gamma | CELLS | BIOPHYSICS | BIOCHEMISTRY & MOLECULAR BIOLOGY | NITRIC-OXIDE | DIFFERENTIATION | VISCERAL LEISHMANIASIS | AID and mu s double-deficient mice | Cytidine Deaminase - immunology | Luciferases - metabolism | Reactive Oxygen Species - metabolism | B-Lymphocytes - parasitology | NADPH Oxidases - immunology | Leishmaniasis, Visceral - immunology | Th1 Cells - immunology | Luciferases - genetics | Reactive Oxygen Species - immunology | NADPH Oxidases - genetics | Female | Leishmaniasis, Visceral - parasitology | Interferon-gamma - genetics | Disease Resistance - genetics | Genes, Reporter | Immunization, Passive - methods | Leishmania donovani - immunology | Immunoglobulin mu-Chains - immunology | Signal Transduction | Cytidine Deaminase - genetics | Immune Sera - administration & dosage | Parasite Load | Gene Expression Regulation | Leishmania donovani - pathogenicity | Mice, Knockout | Immunoglobulin mu-Chains - genetics | Animals | B-Lymphocytes - immunology | Interferon-gamma - immunology | Leishmaniasis, Visceral - genetics | Cytidine Deaminase - deficiency | Th1 Cells - parasitology | Mice | Enzyme Activation | Immunoglobulin mu-Chains - blood | Oxidases | Medical colleges | Immunoglobulins | Leishmaniasis | B cells | Biological response modifiers | Interferon gamma | Index Medicus
Journal Article