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Genes and immunity, ISSN 1476-5470, 1999
Journal
Nature (London), ISSN 1476-4687, 2011, Volume 471, Issue 7336, pp. 119 - 123
Misrepair of DNA double-strand breaks produced by the V(D)J recombinase (the RAG1/RAG2 proteins) at immunoglobulin (Ig) and T cell receptor (Tcr) loci has been... 
THYMIC LYMPHOMA | REPAIR | LYMPHOCYTE DEVELOPMENT | B-CELL | ROLES | TRANSLOCATIONS | MULTIDISCIPLINARY SCIENCES | V(D)J RECOMBINATION | DOUBLE-STRAND BREAKS | GENE AMPLIFICATION | ATM-DEFICIENT MICE | Genomic Instability | Protein-Serine-Threonine Kinases - deficiency | Gene Rearrangement, T-Lymphocyte - genetics | Chromosomes, Mammalian - genetics | DNA-Binding Proteins - deficiency | DNA-Binding Proteins - metabolism | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - chemistry | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Recombination, Genetic - genetics | Lymphoma - pathology | Chromosome Deletion | Tumor Suppressor Proteins - metabolism | Kaplan-Meier Estimate | Protein-Serine-Threonine Kinases - genetics | Thymus Gland - cytology | Lymphoma - genetics | In Situ Hybridization, Fluorescence | Ataxia Telangiectasia Mutated Proteins | Genes, p53 - genetics | DNA-Binding Proteins - genetics | DNA-Binding Proteins - chemistry | Disease Progression | Animals | Genes, Immunoglobulin Heavy Chain - genetics | Mice | Receptors, Antigen, T-Cell - genetics | Translocation, Genetic - genetics | Causes of | Tumor suppressor genes | Lymphomas | Genetic aspects | Abnormalities | Software | Chromosomes | Deoxyribonucleic acid--DNA | Cell cycle | Apoptosis | Tumors | Thymus Gland | Translocation, Genetic | Protein-Serine-Threonine Kinases | Gene Rearrangement, T-Lymphocyte | Receptors, Antigen, T-Cell | Lymphoma | Chromosomes, Mammalian | Genes, Immunoglobulin Heavy Chain | Life Sciences | Genes, p53 | Immunology | Recombination, Genetic | Cell Cycle Proteins | Tumor Suppressor Proteins | DNA-Binding Proteins
Journal Article
PLoS genetics, ISSN 1553-7404, 2017, Volume 13, Issue 2, p. e1006609
Journal Article
PloS one, ISSN 1932-6203, 2009, Volume 4, Issue 12, pp. e8154 - e8154
Journal Article
Blood, ISSN 1528-0020, 2011, Volume 117, Issue 23, pp. 6287 - 6296
B-cell receptor (BCR) signaling is aberrantly activated in chronic lymphocytic leukemia (CLL). Bruton tyrosine kinase (BTK) is essential to BCR signaling and... 
B-CELLS | SURVIVAL | APOPTOSIS | ACTIVATION | FLUDARABINE PLUS CYCLOPHOSPHAMIDE | RITUXIMAB | LYMPHOMA | PROLIFERATION | INHIBITOR | HEMATOLOGY | EXPRESSION | T-Lymphocytes - enzymology | Apoptosis - drug effects | Humans | Tumor Necrosis Factor-alpha - genetics | Cell Survival - genetics | Apoptosis - genetics | Male | NF-kappa B - metabolism | Neoplasm Proteins - antagonists & inhibitors | Receptors, Antigen, B-Cell - metabolism | Mitogen-Activated Protein Kinase 1 - genetics | Neoplasm Proteins - genetics | Interleukin-6 - metabolism | Cell Survival - drug effects | Drug Screening Assays, Antitumor - methods | Interleukin-6 - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Gene Expression Regulation, Leukemic - drug effects | Pyrimidines - pharmacology | Leukemia, Lymphocytic, Chronic, B-Cell - enzymology | B-Cell Activating Factor - metabolism | CD40 Ligand - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Gene Expression Regulation, Enzymologic - genetics | Cell Line, Tumor | Receptors, Antigen, B-Cell - genetics | Mice | Leukemia, Lymphocytic, Chronic, B-Cell - drug therapy | Mitogen-Activated Protein Kinase 1 - metabolism | Protein-Tyrosine Kinases - antagonists & inhibitors | Tumor Necrosis Factor-alpha - metabolism | Gene Expression Regulation, Enzymologic - drug effects | Phosphatidylinositol 3-Kinases - metabolism | Gene Expression Regulation, Leukemic - genetics | Protein-Tyrosine Kinases - genetics | CD40 Ligand - metabolism | MAP Kinase Signaling System - genetics | Female | Interleukin-4 - genetics | Protein-Tyrosine Kinases - biosynthesis | Pyrazoles - pharmacology | B-Lymphocytes - enzymology | Neoplasm Proteins - biosynthesis | Interleukin-4 - metabolism | Phosphatidylinositol 3-Kinases - genetics | Animals | MAP Kinase Signaling System - drug effects | NF-kappa B - genetics | B-Cell Activating Factor - genetics | Cell Proliferation - drug effects | Lymphoid Neoplasia
Journal Article
PLoS genetics, ISSN 1553-7404, 2008, Volume 4, Issue 8, p. e1000166
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2005, Volume 174, Issue 12, pp. 7853 - 7858
textabstractT cells require both primary and costimulatory signals for optimal activation. The primary Ag-specific signal is delivered by engagement of the... 
Cytotoxicity, Immunologic/genetics | Adjuvants, Immunologic/genetics/metabolism/toxicity | Antigens, CD28/genetics/immunology/physiology | Comparative Study | Receptors, IgE/genetics/metabolism/physiology | Humans | Binding Sites, Antibody/genetics | HLA-A1 Antigen/genetics/immunology/metabolism | Immunoglobulin Variable Region/genetics/metabolism | T-Lymphocytes, Cytotoxic/immunology/metabolism | Transduction, Genetic/methods | Neoplasm Proteins/genetics/immunology/metabolism | Lymphocyte Activation/genetics | Immunoglobulin Fab Fragments/genetics/metabolism/toxicity | Research Support, Non-U.S. Gov't | Protein Structure, Tertiary/genetics | K562 Cells | Signal Transduction/genetics/immunology | Cell Line, Tumor | Receptors, Antigen, T-Cell/biosynthesis/genetics/metabolism | Epitopes, T-Lymphocyte/genetics/immunology/toxicity | Cytokines/biosynthesis | Melanoma/immunology/pathology | TUMOR-INFILTRATING LYMPHOCYTES | SIGNAL-TRANSDUCTION | METASTATIC MELANOMA | ACTIVATION | TCR | CANCER REGRESSION | CO-STIMULATION | PROLIFERATION | IMMUNOLOGY | CHIMERIC SINGLE-CHAIN | RECEPTOR GENES | HLA-A1 Antigen - genetics | Neoplasm Proteins - immunology | Immunoglobulin Variable Region - metabolism | Epitopes, T-Lymphocyte - genetics | Immunoglobulin Fab Fragments - toxicity | Neoplasm Proteins - metabolism | Receptors, Antigen, T-Cell - biosynthesis | Signal Transduction - immunology | Lymphocyte Activation - genetics | CD28 Antigens - genetics | Immunoglobulin Fab Fragments - metabolism | Adjuvants, Immunologic - metabolism | Adjuvants, Immunologic - genetics | Epitopes, T-Lymphocyte - immunology | Neoplasm Proteins - genetics | Transduction, Genetic - methods | HLA-A1 Antigen - metabolism | CD28 Antigens - physiology | T-Lymphocytes, Cytotoxic - immunology | Cytotoxicity, Immunologic - genetics | Melanoma-Specific Antigens | Receptors, IgE - physiology | Antigens, Neoplasm | Binding Sites, Antibody - genetics | Immunoglobulin Fab Fragments - genetics | Receptors, Antigen, T-Cell - metabolism | Adjuvants, Immunologic - toxicity | CD28 Antigens - immunology | Protein Structure, Tertiary - genetics | Signal Transduction - genetics | Melanoma - pathology | Receptors, IgE - genetics | Receptors, IgE - metabolism | T-Lymphocytes, Cytotoxic - metabolism | Melanoma - immunology | Epitopes, T-Lymphocyte - toxicity | Immunoglobulin Variable Region - genetics | HLA-A1 Antigen - immunology | Receptors, Antigen, T-Cell - genetics | Cytokines - biosynthesis
Journal Article
Journal Article
American journal of human genetics, ISSN 0002-9297, 2012, Volume 91, Issue 2, pp. 391 - 395
Journal Article
Blood, ISSN 1528-0020, 2017, Volume 129, Issue 6, pp. 759 - 770
Contrasting patterns of basal phosphorylation levels and α-BCR–induced signaling between CLL and MCL tumors. Direct association between BCR-induced signaling... 
SURFACE IGM | BCR | CHRONIC-LYMPHOCYTIC-LEUKEMIA | BRUTONS TYROSINE KINASE | HUMAN FOLLICULAR LYMPHOMA | GENE-EXPRESSION | KINASE INHIBITOR IBRUTINIB | MUTATIONS | TARGETING BTK | HEMATOLOGY | NF-KAPPA-B | Lymphoma, Follicular - metabolism | Protein-Tyrosine Kinases - metabolism | Humans | Lymphoma, Mantle-Cell - pathology | Leukemia, Lymphocytic, Chronic, B-Cell - genetics | Receptors, Antigen, B-Cell - metabolism | STAT1 Transcription Factor - metabolism | Lymphoma, Follicular - genetics | Mitogen-Activated Protein Kinase 1 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Single-Cell Analysis | Immunoglobulin M - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | STAT1 Transcription Factor - genetics | Leukemia, Lymphocytic, Chronic, B-Cell - pathology | Lymphoma, Large B-Cell, Diffuse - diagnosis | Mitogen-Activated Protein Kinase 3 - metabolism | Leukemia, Lymphocytic, Chronic, B-Cell - metabolism | Receptors, Antigen, B-Cell - genetics | CD79 Antigens - genetics | src-Family Kinases - genetics | Mitogen-Activated Protein Kinase 1 - metabolism | Phosphorylation | Gene Expression Regulation, Neoplastic | Lymphoma, Follicular - diagnosis | Lymphoma, Large B-Cell, Diffuse - metabolism | Phosphoproteins - metabolism | Proto-Oncogene Proteins c-akt - genetics | Lymphoma, Mantle-Cell - diagnosis | STAT5 Transcription Factor - genetics | STAT5 Transcription Factor - metabolism | Protein-Tyrosine Kinases - genetics | Flow Cytometry | Syk Kinase - genetics | Phospholipase C gamma - genetics | src-Family Kinases - metabolism | Lymphoma, Mantle-Cell - genetics | Lymphoma, Mantle-Cell - metabolism | Diagnosis, Differential | Phospholipase C gamma - metabolism | Lymphoma, Large B-Cell, Diffuse - pathology | Lymphoma, Follicular - pathology | p38 Mitogen-Activated Protein Kinases - genetics | Phosphoproteins - genetics | CD79 Antigens - metabolism | Syk Kinase - metabolism | Lymphoma, Large B-Cell, Diffuse - genetics | Leukemia, Lymphocytic, Chronic, B-Cell - diagnosis | Life Sciences | Human health and pathology | Hematology | Cancer | Lymphoid Neoplasia
Journal Article
11.