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Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 03/2012, Volume 73, Issue 6, pp. 1116 - 1126
The precise connectivity of inputs and outputs is critical for cerebral cortex function; however, the cellular mechanisms that establish these connections are... 
SYNAPTIC SPECIFICITY | ADULT BRAIN | SIGNALING PATHWAY | IN-VIVO | BOC | GROWTH | MECHANISMS | AXON-GUIDANCE | MEMBRANE-PROTEINS | NEUROSCIENCES | CEREBRAL-CORTEX | Silver Staining - methods | RNA, Small Interfering - genetics | Furans - metabolism | Electric Stimulation | gamma-Aminobutyric Acid - metabolism | Channelrhodopsins | Hedgehog Proteins - metabolism | Dendritic Spines - metabolism | Functional Laterality - genetics | Nerve Net - cytology | Synaptophysin - genetics | Neurons - ultrastructure | Neurons - metabolism | Repressor Proteins - metabolism | Gene Expression Regulation, Developmental - physiology | Synaptophysin - metabolism | Animals, Newborn | Fluorobenzenes - metabolism | Tumor Suppressor Proteins - metabolism | Matrix Attachment Region Binding Proteins - metabolism | Dendritic Spines - physiology | Mice, Transgenic | Synapses - ultrastructure | Electroporation - methods | Mutation - genetics | Corpus Callosum - cytology | Patch-Clamp Techniques | Immunoglobulin G - genetics | Cerebral Cortex - growth & development | Luminescent Proteins - genetics | Corpus Callosum - growth & development | Mice | Immunoglobulin G - metabolism | RNA, Small Interfering - metabolism | Receptors, Cell Surface - genetics | Membrane Potentials - genetics | Age Factors | Gene Expression Regulation, Developmental - genetics | Phosphopyruvate Hydratase - metabolism | Cerebral Cortex - cytology | DNA-Binding Proteins - metabolism | Synapses - metabolism | Hedgehog Proteins - genetics | Pyramidal Tracts - physiology | Stilbamidines - metabolism | Receptors, Cell Surface - metabolism | Nuclear Proteins - metabolism | Transcription Factors - metabolism | Animals | Nerve Net - metabolism | In Vitro Techniques | Luminescent Proteins - metabolism | Stem cell research | Neurosciences | Nervous system diseases | Neurons | Stem cells | Collateralized debt obligations | Physiological aspects | Cells | Proteins | Transcription factors | Software | Sucrose | Synaptogenesis | Circuits | Hedgehog protein | Neural networks | Pyramidal cells | Cortex | Synapses
Journal Article
Molecular and cellular biology, ISSN 0270-7306, 2003, Volume 23, Issue 4, pp. 1428 - 1440
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2007, Volume 133, Issue 2, pp. 559 - 573
Background & Aims: Recent studies have revealed that murine intestinal mucosa contains several kinds of lineage markers (lin)– c-kit+ immune precursor cells.... 
Gastroenterology and Hepatology | INFLAMMATORY-BOWEL-DISEASE | GUT CRYPTOPATCHES | NK CELLS | DENDRITIC CELLS | PEYERS-PATCHES | ALPHA-BETA | LYMPH-NODES | DELTA-T-CELLS | GASTROENTEROLOGY & HEPATOLOGY | LYMPHOCYTES-T | FETAL THYMUS | Membrane Glycoproteins - metabolism | Humans | Colitis, Ulcerative - genetics | Crohn Disease - metabolism | Hematopoietic Stem Cells - pathology | Lymphotoxin-alpha - metabolism | Interferon-gamma - metabolism | RNA, Messenger - metabolism | Colitis, Ulcerative - immunology | Antigens, CD - metabolism | Immunoglobulins - metabolism | Lymphotoxin-alpha - genetics | Time Factors | Intestinal Mucosa - immunology | Inhibitor of Differentiation Protein 2 - genetics | Cell Differentiation | Gene Expression | Colitis, Ulcerative - metabolism | Colitis, Ulcerative - pathology | Inhibitor of Differentiation Protein 2 - metabolism | Cell Lineage | Adult Stem Cells - metabolism | Interleukin-2 Receptor alpha Subunit - metabolism | Crohn Disease - pathology | ADP-ribosyl Cyclase 1 - metabolism | Killer Cells, Natural - metabolism | Intestinal Mucosa - pathology | Crohn Disease - genetics | Intestinal Mucosa - metabolism | Antigens, CD34 - metabolism | Killer Cells, Natural - pathology | Proto-Oncogene Proteins c-kit - metabolism | Adult Stem Cells - immunology | DNA-Binding Proteins - metabolism | Hematopoietic Stem Cells - immunology | Trans-Activators - genetics | Killer Cells, Natural - immunology | Adult Stem Cells - pathology | Sialic Acid Binding Ig-like Lectin 3 | Proto-Oncogene Proteins - metabolism | CD56 Antigen - metabolism | Receptors, Interleukin-7 - metabolism | Cells, Cultured | Immunophenotyping | Proto-Oncogene Proteins - genetics | Hematopoietic Stem Cells - metabolism | Integrin alpha Chains - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Crohn Disease - immunology | Transcription Factors - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | Trans-Activators - metabolism
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2011, Volume 141, Issue 2, pp. 621 - 632
Background & Aims Epithelial cells that cover the intestinal mucosal surface maintain immune homeostasis and tolerance in the gastrointestinal tract. However,... 
Gastroenterology and Hepatology | IgA | Inflammatory Bowel Disease | T Cell | Immune Response | CELLS | COMPLEX | ACTIVATION | POLYMERIC IMMUNOGLOBULIN RECEPTOR | INTESTINAL EPITHELIUM | TRANSPORT | INFLAMMATORY-BOWEL-DISEASE | ADAPTER | HOST-DEFENSE | INFECTION | GASTROENTEROLOGY & HEPATOLOGY | Tumor Necrosis Factor-alpha - metabolism | Intestinal Mucosa - metabolism | Adaptor Protein Complex mu Subunits - metabolism | Epithelial Cells - metabolism | Adaptor Protein Complex beta Subunits - deficiency | Humans | Cell Membrane Permeability | Crohn Disease - metabolism | Cell Membrane - physiology | Ribonucleases - metabolism | Adaptor Protein Complex 1 - metabolism | Th17 Cells - metabolism | Intestinal Mucosa - immunology | alpha-Defensins - metabolism | Adaptor Protein Complex beta Subunits - immunology | Cell Membrane - metabolism | beta-Defensins - metabolism | Colon | Receptors, Cytokine - metabolism | Colitis - immunology | Adaptor Protein Complex 1 - deficiency | Lipocalins - metabolism | Adaptor Protein Complex 1 - immunology | Immunoglobulin A - metabolism | Signal Transduction | Acute-Phase Proteins - metabolism | Down-Regulation | Oncogene Proteins - metabolism | Homeostasis - immunology | Epithelial Cells - pathology | S100 Proteins - metabolism | Muramidase - metabolism | Intestinal Mucosa - microbiology | Mice, Knockout | Interleukin-17 - metabolism | Adaptor Protein Complex beta Subunits - metabolism | Animals | Colitis - microbiology | Proteins - metabolism | Ribonuclease, Pancreatic - metabolism | Lipocalin-2 | Cathelicidins - metabolism | Epithelial Cells - immunology | Receptors, Cytokine - immunology | Mice | Intestinal Mucosa - pathology | Medical colleges | Fc receptors | Gastrointestinal diseases | Homeostasis | Biological response modifiers | Immunoglobulin A | T cells
Journal Article
Journal of hepatology, ISSN 0168-8278, 2011, Volume 54, Issue 4, pp. 795 - 809
The unfolded protein response (UPR) is activated upon the accumulation of misfolded proteins in the endoplasmic reticulum (ER) that are sensed by the binding... 
Gastroenterology and Hepatology | Hepatosteatosis | Endoplasmic reticulum stress | Unfolded protein response | Liver injury | MESSENGER-RNA TRANSLATION | UNFOLDED-PROTEIN RESPONSE | TRANSCRIPTION FACTOR XBP-1 | NECROSIS-FACTOR-ALPHA | ISCHEMIA-REPERFUSION INJURY | PROGRAMMED CELL-DEATH | GLUCOSE-REGULATED PROTEINS | HEPATITIS-C VIRUS | HUMAN HEPATOCELLULAR-CARCINOMA | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | Hepatitis C, Chronic - metabolism | Fatty Liver - metabolism | Cholestasis - metabolism | eIF-2 Kinase - metabolism | Humans | Liver Diseases - pathology | Stress, Physiological | Endoplasmic Reticulum - metabolism | Hyperhomocysteinemia - metabolism | Hepatocytes - pathology | Hepatocytes - metabolism | Unfolded Protein Response | Liver Diseases, Alcoholic - metabolism | Activating Transcription Factor 6 - metabolism | Inflammation - metabolism | Animals | Models, Biological | Liver Neoplasms - metabolism | Non-alcoholic Fatty Liver Disease | Protein Conformation | Liver Diseases - metabolism | Reperfusion Injury - metabolism | Apoptosis | Carcinoma, Hepatocellular - metabolism | Inositol | Immunoglobulins | Stress (Physiology) | Ribonuclease | Glucose | Genetic translation | Dextrose | Messenger RNA | Proteases | Proteolysis | Analysis | Hepatitis C | Adenylic acid | Hepatitis C virus | Health aspects | Protein binding | Tumors
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 12/2015, Volume 35, Issue 12, pp. 2605 - 2616
OBJECTIVE—Kawasaki disease (KD) is the most common cause of acute vasculitis and acquired cardiac disease among US children. We have previously shown that both... 
dendritic cells | interleukin-1 | mucocutaneous lymph node syndrome | MyD88 | Endothelial cells | protein | endothelial cells | ELEMENT-BINDING PROTEIN-2 | ACTIVATION | RECEPTOR | CORONARY-ARTERY ANEURYSMS | NLRP3 INFLAMMASOME | POLYMORPHISM | ACUTE-PHASE | INTRAVENOUS IMMUNOGLOBULIN | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ASSOCIATION | Lactobacillus casei | Mucocutaneous Lymph Node Syndrome - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Stromal Cells - pathology | Caspase 1 - metabolism | Interleukin-1alpha - metabolism | Aortitis - chemically induced | Aorta - metabolism | Coronary Vessels - metabolism | Cell Wall | DNA-Binding Proteins - metabolism | Receptors, Interleukin-1 Type I - metabolism | Interleukin-1beta - metabolism | Bone Marrow Transplantation | Coronary Artery Disease - pathology | Aortitis - pathology | Dendritic Cells - metabolism | Disease Models, Animal | Mucocutaneous Lymph Node Syndrome - pathology | Coronary Vessels - pathology | Signal Transduction | Coronary Artery Disease - chemically induced | Endothelial Cells - metabolism | Mucocutaneous Lymph Node Syndrome - chemically induced | Coronary Artery Disease - metabolism | Mice, Inbred C57BL | Stromal Cells - metabolism | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Aortitis - genetics | DNA-Binding Proteins - genetics | Transplantation Chimera | Mice, Knockout | Aorta - pathology | Mucocutaneous Lymph Node Syndrome - genetics | Aortitis - metabolism | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Coronary Artery Disease - genetics | Receptors, Interleukin-1 Type I - genetics | Myeloid Differentiation Factor 88 - metabolism | Bone Marrow Cells - metabolism | coronary artery vasculitis | Kawasaki disease | IL-1
Journal Article
Journal of allergy and clinical immunology, ISSN 0091-6749, 2013, Volume 131, Issue 2, pp. 501 - 511.e1
Background Sphingosine-1-phosphate (S1P), which is produced by 2 sphingosine kinase (SphK) isoenzymes, SphK1 and SphK2, has been implicated in IgE-mediated... 
Allergy and Immunology | airway hyperresponsiveness | asthma | nuclear factor κB | mast cells | sphingosine kinase | Sphingosine-1-phosphate | IMMUNITY | FC-EPSILON-RI | nuclear factor kappa B | FACTOR-KAPPA-B | IMMUNOLOGY | MULTIPLE FEATURES | REGULATES MUCIN PRODUCTION | RESPONSES | CYTOKINE PRODUCTION | ALLERGY | GENE-EXPRESSION | MICE | Tumor Necrosis Factor-alpha - metabolism | Lysophospholipids - metabolism | Asthma - metabolism | Bronchial Hyperreactivity - drug therapy | Humans | Goblet Cells - drug effects | NF-kappa B - metabolism | Amino Alcohols - pharmacology | Interferon-gamma - metabolism | Interleukins - metabolism | Inflammation - metabolism | Inflammation - drug therapy | Methacholine Chloride - pharmacology | Mast Cells - metabolism | Bronchial Hyperreactivity - metabolism | Female | Chemokine CCL2 - metabolism | Hyperplasia - drug therapy | Lung - metabolism | Sphingosine - metabolism | Hyperplasia - metabolism | Asthma - enzymology | Immunoglobulin E - metabolism | Mice, Inbred C57BL | Cells, Cultured | Ovalbumin - pharmacology | Asthma - drug therapy | Goblet Cells - metabolism | Mast Cells - drug effects | Asthma - chemically induced | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Bronchial Hyperreactivity - enzymology | Phosphotransferases (Alcohol Group Acceptor) - antagonists & inhibitors | Bronchial Hyperreactivity - pathology | Sphingosine - analogs & derivatives | Animals | Lung - drug effects | Mice | Bronchoalveolar Lavage Fluid - chemistry | Phosphates | Medical colleges | Isoenzymes | Analysis | Sphingosine | Inflammation | Asthma | Studies | Cytokines | Lungs | Rodents | Kinases | Laboratory animals | Allergies
Journal Article
PloS one, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e35905
Hematopoietic progenitor CD133(+)/c-kit(+) cells have been shown to be involved in myocardial healing following myocardial infarction (MI). Previously we... 
PROGENITOR CELLS | CD31(+) CELLS | ISCHEMIA/REPERFUSION INJURY | ACTIVATION | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | TISSUE | APELIN | TOPCARE-AMI | EXPRESSION | Up-Regulation | Capillaries - pathology | Receptors, Notch - metabolism | Cardiomegaly - pathology | Angiopoietin-1 - metabolism | Antigens, CD - metabolism | Peptides - metabolism | Serrate-Jagged Proteins | Bone Marrow - metabolism | Myocardium - metabolism | Endomyocardial Fibrosis - complications | Myocardial Infarction - physiopathology | Capillaries - metabolism | Apelin | Jagged-1 Protein | Capillaries - physiopathology | Signal Transduction | Cardiomegaly - physiopathology | Myocardial Infarction - metabolism | Receptors, CXCR4 - metabolism | Chemokine CXCL12 - metabolism | Hematopoietic Stem Cells - cytology | Endomyocardial Fibrosis - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Cardiomegaly - metabolism | Cell Movement | Actins - metabolism | Glycoproteins - metabolism | Diabetes Mellitus, Type 2 - metabolism | Proto-Oncogene Proteins c-kit - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Myocardial Infarction - pathology | Endomyocardial Fibrosis - pathology | Membrane Proteins - metabolism | Heart Function Tests | Diabetes Mellitus, Type 2 - complications | Calcium-Binding Proteins - metabolism | Myocardium - pathology | Adipokines | AC133 Antigen | Cardiomegaly - complications | Myocardial Infarction - complications | Animals | Diabetes Mellitus, Type 2 - physiopathology | Bone Marrow - pathology | Receptor, Notch3 | Endomyocardial Fibrosis - physiopathology | Apoptosis | Advertising executives | Vascular endothelial growth factor | Adenoviruses | Heart attack | Myocardial infarction | Heart | Diabetic retinopathy | Heart attacks | Angiopoietin | Recovery of function | Smooth muscle | Cardiovascular disease | Recruitment | Angiogenesis | Toxicology | Cell growth | Ischemia | Data recovery | Rodents | Bone marrow | Repair | Heart diseases | Immunoglobulins | Diabetes mellitus | Coronary artery | Muscles | Pharmacology | c-Kit protein | Hemopoiesis | Studies | Ostomy | Coronary vessels | Fibrosis | Stem cells | Myocardium | Infarction | Diabetes | Laboratory animals
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2015, Volume 149, Issue 6, pp. 1564 - 1574.e3
Background & Aims Many patients with inflammatory bowel disease (IBD) fail to respond to anti–tumor necrosis factor (TNF) agents such as infliximab and... 
Gastroenterology and Hepatology | Therapy | Inflammation | EFFECTOR FUNCTIONS | CROHNS-DISEASE | VIVO | ULCERATIVE-COLITIS | MATRIX METALLOPROTEINASES | IN-VITRO | HINGE | MONOCLONAL-ANTIBODIES | INFLIXIMAB | ETANERCEPT | GASTROENTEROLOGY & HEPATOLOGY | Tumor Necrosis Factor-alpha - metabolism | Intestinal Mucosa - metabolism | Epitopes - metabolism | Humans | Inflammatory Bowel Diseases - immunology | Middle Aged | Crohn Disease - metabolism | Etanercept - metabolism | Colon - immunology | Male | Colitis, Ulcerative - immunology | Inflammatory Bowel Diseases - metabolism | Case-Control Studies | Intestinal Mucosa - drug effects | Immunoblotting - methods | Infliximab - metabolism | Proteolysis - drug effects | Intestinal Mucosa - immunology | Female | Colitis, Ulcerative - drug therapy | Biological Factors - metabolism | Inflammatory Bowel Diseases - drug therapy | Colon - pathology | Colitis, Ulcerative - metabolism | Antibodies, Monoclonal, Humanized - metabolism | Matrix Metalloproteinase 3 - metabolism | Matrix Metalloproteinase 12 - metabolism | Adalimumab - metabolism | Crohn Disease - immunology | Colon - metabolism | Biopsy | Crohn Disease - drug therapy | Biological Factors - pharmacology | Immunoglobulin G - metabolism | Medical colleges | Tumor necrosis factor | Gastrointestinal diseases | Tumors | Necrosis
Journal Article