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Acta biomaterialia, ISSN 1742-7061, 2017, Volume 63, pp. 96 - 109
[Display omitted] Anticancer immune responses depend on efficient presentation of tumor antigens and co-stimulatory signals provided by antigen-presenting... 
Tumor microenvironment | Immunomodulation | APCs polarization | Nanotherapy | Colorectal cancer | POLY(GAMMA-GLUTAMIC ACID) | ACTIVATION | MATERIALS SCIENCE, BIOMATERIALS | GAMMA-GLUTAMIC ACID | DENDRITIC CELLS | ENGINEERING, BIOMEDICAL | CHITOSAN | CANCER-IMMUNOTHERAPY | TUMOR-ASSOCIATED MACROPHAGES | ANTITUMOR IMMUNITY | POLARIZATION | T-CELLS | Inflammation - pathology | Humans | Polyglutamic Acid - administration & dosage | Nanoparticles - adverse effects | Polyglutamic Acid - analogs & derivatives | Antigen-Presenting Cells - pathology | T-Lymphocytes - drug effects | Dendritic Cells - drug effects | Cell Polarity - drug effects | Chitosan - adverse effects | Dendritic Cells - metabolism | Cell Survival - drug effects | Neoplasm Invasiveness | Antigen-Presenting Cells - drug effects | Endocytosis - drug effects | Particle Size | Cell Movement - drug effects | Macrophages - metabolism | Phenotype | T-Lymphocytes - cytology | Cell Differentiation - drug effects | Colonic Neoplasms - pathology | Macrophages - drug effects | Cell Proliferation - drug effects | Interleukin-10 - pharmacology | Polyglutamic Acid - adverse effects | Glutamate | Antigens | Genetic aspects | Dendritic cells | Development and progression | Amino acids | Disease susceptibility | T cells | Macrophages | Prevention | Nanoparticles | Tumor antigens | Cancer
Journal Article
Scientific reports, ISSN 2045-2322, 01/2016, Volume 6, Issue 1, pp. 19200 - 19200
Although the heavy metals cadmium (Cd) and lead (Pb) are known environmental health concerns, their long-term impacts on gut ecology and susceptibility to... 
INFLAMMATORY-BOWEL-DISEASE | METALLOTHIONEIN | PROTECTIVE ROLE | MULTIDISCIPLINARY SCIENCES | ULCERATIVE-COLITIS | IN-VIVO | CHRONIC INGESTION | GENE-EXPRESSION | RATS | INDUCTION | METALLOHORMONE CADMIUM | Immunomodulation - drug effects | Dextran Sulfate - adverse effects | Colitis - pathology | Salmonella typhimurium | Intestinal Mucosa - drug effects | Cadmium - adverse effects | Lead - adverse effects | Trinitrobenzenesulfonic Acid - adverse effects | Time Factors | Immune System - immunology | Female | Metals, Heavy - administration & dosage | Disease Models, Animal | Metals, Heavy - adverse effects | Disease Susceptibility | Salmonella Infections - complications | Administration, Oral | Immune System - cytology | Colitis - etiology | Animals | Immune System - drug effects | Salmonella Infections - microbiology | Mice | Lead - administration & dosage | Cadmium - administration & dosage | Intestinal Mucosa - pathology | Salmonella | Cell culture | Cadmium | Drinking water | Inflammatory bowel diseases | Animal models | Digestive system | Sulfonic acid | Heavy metals | Dextran | Inflammatory bowel disease | Immunosuppression | Sodium | Intestine | Sodium sulfate | Rodents | Colon | Sulfate | Colitis | Autoimmune diseases | Digestive tract | Index Medicus | Dextran Sulfate/adverse effects | Disease Models | Trinitrobenzenesulfonic Acid/adverse effects | Metals | Heavy/adverse effects | Toxicology and food chain | Colitis/pathology | Immune System/immunology | Intestinal Mucosa/pathology | Intestinal Mucosa/drug effects | Life Sciences | Toxicology | Immunomodulation/drug effects | Salmonella Infections/microbiology | Animal | Colitis/etiology | Cadmium/administration & dosage
Journal Article
International journal of nanomedicine, ISSN 1178-2013, 2018, Volume 13, pp. 5799 - 5810
...) by stimulation with GO/RAW 264.7-conditioned culture medium were accessed. We also further investi-gated the possible mechanisms underlying the osteo-and angio-immunomodulatory effects of GO. Results... 
graphene oxide | angiogenesis | RAW264.7 cells | osteogenesis | bone marrow mesenchymal stem cells | HUVECs | osteo-immunomodulatory | SIGNALING PATHWAYS | MACROPHAGES | PHENOTYPE | NANOSCIENCE & NANOTECHNOLOGY | INDUCTION | MESENCHYMAL STEM-CELLS | AGENT | REPAIR | BONE REGENERATION | PHARMACOLOGY & PHARMACY | BIOMATERIALS | SCAFFOLDS | Immunomodulation - drug effects | Inflammation - pathology | Neovascularization, Physiologic - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Cell Survival - genetics | Culture Media, Conditioned - pharmacology | RNA, Messenger - metabolism | Cell Differentiation - genetics | Human Umbilical Vein Endothelial Cells - cytology | Mesenchymal Stem Cells - cytology | Mesenchymal Stem Cells - drug effects | Graphite - pharmacology | Mesenchymal Stem Cells - metabolism | Cell Survival - drug effects | Macrophages - ultrastructure | Human Umbilical Vein Endothelial Cells - drug effects | Neovascularization, Physiologic - genetics | Endocytosis - drug effects | Osteogenesis - drug effects | RNA, Messenger - genetics | Macrophages - cytology | Gene Expression Regulation - drug effects | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Inflammation - genetics | Macrophages - drug effects | RAW 264.7 Cells | Cell Proliferation - drug effects | Mice | Immunologic Factors - pharmacology | Biological products | Tissue engineering | Graphene | Analysis | Stem cells | Graphite | Research | Environmental engineering | Cytokines | Laboratories | Gene expression | Angiogenesis | Biomedical materials | Cell growth | Microscopy | Spectrum analysis | Bone marrow | Tumor necrosis factor-TNF | Vascular endothelial growth factor | Graphene oxide
Journal Article
International journal of molecular sciences, ISSN 1422-0067, 2017, Volume 18, Issue 7, p. 1414
.... Mechanisms of anticancer effects of HDAC inhibitors are not uniform; they may be different and depend on the cancer type, HDAC inhibitors, doses, etc... 
Histone deacetylase inhibitors | Anti-angiogenic effect | Cell cycle arrest | Drug combinations | Histone deacetylases | Autophagy | Apoptosis | Cancer | HDAC INHIBITORS | autophagy | SUBEROYLANILIDE HYDROXAMIC ACID | HUMAN LEUKEMIA-CELLS | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR-SUPPRESSOR GENES | apoptosis | VALPROIC ACID | cell cycle arrest | anti-angiogenic effect | CHEMISTRY, MULTIDISCIPLINARY | drug combinations | CELL LUNG-CANCER | EPITHELIAL-MESENCHYMAL TRANSITION | PHASE-II TRIAL | LONG NONCODING RNA | histone deacetylases | cancer | histone deacetylase inhibitors | Immunomodulation - drug effects | Apoptosis - drug effects | Humans | Angiogenesis Inhibitors - pharmacology | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Antineoplastic Agents - therapeutic use | Clinical Trials as Topic | Autophagy - drug effects | Acetylation - drug effects | Animals | Signal Transduction - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Cell Cycle Checkpoints - drug effects | Angiogenesis Inhibitors - therapeutic use | Histone Deacetylase Inhibitors - pharmacology | Antineoplastic Agents - pharmacology | Epigenesis, Genetic - drug effects | Histone Deacetylase Inhibitors - therapeutic use | Gene Expression Regulation, Neoplastic - drug effects | Drug Evaluation, Preclinical
Journal Article
Biomaterials, ISSN 0142-9612, 2015, Volume 37, pp. 367 - 382
Journal Article
PloS one, ISSN 1932-6203, 2009, Volume 4, Issue 12, pp. e8202 - e8202
... beneficial activities mediated by transrepression but avoid adverse effects of GR action requiring transactivation... 
APOPTOSIS | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | DNA-BINDING | REPRESSION | INFLAMMATION | TRANSACTIVATION | IN-VIVO | BIOLOGY | CENTRAL-NERVOUS-SYSTEM | METHYLPREDNISOLONE | T-CELLS | Organ Specificity - drug effects | Apoptosis - drug effects | Transcriptional Activation - drug effects | Myelin Sheath - drug effects | Receptors, Glucocorticoid - metabolism | Acetates | Myelin Sheath - metabolism | Dose-Response Relationship, Drug | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | Tyramine - analogs & derivatives | Disease Models, Animal | Fibroblasts - metabolism | Solvents | Guinea Pigs | Quaternary Ammonium Compounds - adverse effects | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Mice, Inbred C57BL | Quaternary Ammonium Compounds - therapeutic use | Aziridines - pharmacology | Mitochondria - metabolism | Interleukin-17 - biosynthesis | Mitochondria - drug effects | Cell Adhesion Molecules - metabolism | Animals | Embryo, Mammalian - cytology | Fibroblasts - drug effects | Multiple Sclerosis - pathology | Ligands | Mice | Synephrine - pharmacology | Multiple Sclerosis - drug therapy | Quaternary Ammonium Compounds - pharmacology | Complications and side effects | Multiple sclerosis | Corticosteroids | Nuclear magnetic resonance spectroscopy | Models | T cells | Apoptosis | Steroids | Nuclear magnetic resonance--NMR | Bcl-2 protein | Glucocorticoids | Neuropathology | Helper cells | Nervous system | Lymphocytes T | Alkylation | Medical schools | LFA-1 antigen | Magnetic resonance spectroscopy | Cell activation | Immunology | Blood-brain barrier | Lymphocytes | CD44 antigen | Rodents | Fibroblasts | Drug dosages | Deoxyribonucleic acid--DNA | Repressing | Spectroscopy | Myelin | Caspase | Inflammation | Metabolism | Gene expression | Experimental allergic encephalomyelitis | Interleukin 17 | Intermediates | Side effects | Brain research | NMR spectroscopy | Deoxyribonucleic acid | Nuclear magnetic resonance | NMR | DNA
Journal Article
by Feng, Q and Xu, M and Yu, Y. Y and Hou, Y and Mi, X and Sun, Y. X and Ma, S and Zuo, X. Y and Shao, L. L and Hou, M and Zhang, X. H and Peng, J
Journal of thrombosis and haemostasis, ISSN 1538-7933, 2017, Volume 15, Issue 9, pp. 1845 - 1858
Essentials M1/M2 imbalance is involved in many autoimmune diseases, and could be restored. The expressions and functions of M1 and M2 were investigated in an... 
dexamethasone | immunomodulation | macrophage | all‐trans‐retinoic acid | immune thrombocytopenia | all-trans-retinoic acid | PURPURA ITP | PHAGOCYTOSIS | DENDRITIC CELLS | MEDIATED CYTOTOXICITY | PLATELET | REGULATORY T-CELLS | PERIPHERAL VASCULAR DISEASE | DIFFERENTIATION | TYPE-1 | HEMATOLOGY | POLARIZATION | EXPRESSION | Purpura, Thrombocytopenic, Idiopathic - metabolism | T-Lymphocytes, Regulatory - metabolism | Spleen - immunology | Coculture Techniques | Humans | Middle Aged | Dexamethasone - adverse effects | Male | Spleen - drug effects | Case-Control Studies | Purpura, Thrombocytopenic, Idiopathic - drug therapy | T-Lymphocytes, Regulatory - immunology | T-Lymphocytes, Helper-Inducer - drug effects | Young Adult | Purpura, Thrombocytopenic, Idiopathic - immunology | T-Lymphocytes, Helper-Inducer - immunology | CD8-Positive T-Lymphocytes - metabolism | Adult | Female | Macrophages - immunology | Biomarkers - metabolism | Phagocytosis - drug effects | T-Lymphocytes, Helper-Inducer - metabolism | Cytokines - metabolism | Cells, Cultured | Treatment Outcome | Dexamethasone - therapeutic use | Macrophages - metabolism | Phenotype | T-Lymphocytes, Regulatory - drug effects | Tretinoin - therapeutic use | Spleen - metabolism | Lymphocyte Activation - drug effects | CD8-Positive T-Lymphocytes - drug effects | Adolescent | Tretinoin - adverse effects | Macrophages - drug effects | Aged | Cell Proliferation - drug effects | Immunologic Factors - adverse effects | CD8-Positive T-Lymphocytes - immunology | Macrophage Activation - drug effects | Immunologic Factors - therapeutic use | Thrombocytopenia | Dexamethasone | Dosage and administration | T cells | Macrophages | Tretinoin | Steroids | Cell proliferation | Cell culture | Flow cytometry | Polarization | Pathogenesis | CD8 antigen | Lymphocytes T | Immunosuppressive agents | Enzyme-linked immunosorbent assay | trans-Retinoic acid | Spleen | Antigens | Cytokines | Immunoregulation | Rupture | Inflammation | T cell receptors | Immunological tolerance | Environmental effects | CD4 antigen | Monocytes | Antigen-presenting cells | Autoimmune diseases | Immunofluorescence | Retinoic acid
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 3, p. e59119
Tuberculosis is an ongoing threat to global health, especially with the emergence of multi drug-resistant (MDR... 
IMMUNITY | CELLS | HUMAN NEUTROPHIL PEPTIDE-1 | BACTERIAL-INFECTIONS | KINETICS | MULTIDISCIPLINARY SCIENCES | PULMONARY TUBERCULOSIS | BETA-DEFENSINS | ANTIMICROBIAL PEPTIDES | INDUCTION | Antitubercular Agents - chemical synthesis | Immunomodulation - drug effects | Lung - microbiology | Tuberculosis, Pulmonary - microbiology | Humans | Mycobacterium tuberculosis - immunology | Pneumonia - pathology | Tuberculosis, Pulmonary - drug therapy | Mycobacterium tuberculosis - drug effects | Bacterial Load - drug effects | Tuberculosis, Pulmonary - complications | Microbial Sensitivity Tests | Cytokines - immunology | Antimicrobial Cationic Peptides - chemical synthesis | Cell Line | Lung - pathology | Immunity, Innate - drug effects | Antimicrobial Cationic Peptides - pharmacology | Antitubercular Agents - pharmacology | Pneumonia - microbiology | Tuberculosis, Pulmonary - pathology | Animals | Pneumonia - drug therapy | Lung - drug effects | Pneumonia - complications | Mice | Drug Resistance, Multiple, Bacterial - drug effects | Cytokines - biosynthesis | Tuberculosis | Peptides | Analysis | Immunotherapy | Models | Drug resistance | Health aspects | Animal models | Antimicrobial activity | Disease | Cytotoxicity | Innate immunity | Infections | Antiinfectives and antibacterials | Immunity | Immunology | Rodents | Occupational health | Public health | Medical research | Wound healing | Bacterial infections | Nutrition | Immunomodulation | Health risks | Antimicrobial agents | Inflammation | Pathology | Lungs | Minimum inhibitory concentration | Chemokines
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 4, p. e62241
Parenteral use of drugs; such as opiates exert immunomodulatory effects and serve as a cofactor in the progression of HIV-1 infection, thereby potentiating HIV related neurotoxicity ultimately leading to progression of NeuroAIDS... 
LONG-TERM-MEMORY | EXPOSURE ALTERS | MULTIDISCIPLINARY SCIENCES | ELEMENT-BINDING PROTEIN | CENTRAL-NERVOUS-SYSTEM | RAT HIPPOCAMPUS | HIV-ASSOCIATED DEMENTIA | SUBSTANTIA-NIGRA | SPINE DENSITY | FACTOR PREVENTS | CELL-DEATH | Blood-Brain Barrier - cytology | Blood Vessels - metabolism | Cell Count | Humans | Capillary Permeability - drug effects | Drug Carriers - chemistry | Brain - blood supply | Brain-Derived Neurotrophic Factor - pharmacology | Neuroprotective Agents - pharmacology | Dendritic Spines - drug effects | Morphine - antagonists & inhibitors | Astrocytes - cytology | Astrocytes - drug effects | Brain - cytology | Morphine - pharmacology | Endothelial Cells - metabolism | Blood-Brain Barrier - drug effects | Blood Vessels - cytology | Blood-Brain Barrier - metabolism | Magnetite Nanoparticles - chemistry | Endothelial Cells - cytology | Models, Biological | Blood Vessels - drug effects | Primary Cell Culture | Astrocytes - metabolism | Endothelial Cells - drug effects | Drugs | Brain | Drug delivery systems | Neurons | Morphine | Research | Health aspects | Apoptosis | Vehicles | Drug abuse | Neuroprotection | Density measurement | Spine | Memory | Opiates | Drug delivery | Infections | Synaptic density | Kinases | Density | Nanoparticles | Proteins | Dendritic spines | Neurotoxicity | Immunology | Blood-brain barrier | Neurodegeneration | Rodents | Human immunodeficiency virus--HIV | Physiology | Degeneration | Enzymes | Drug addiction | Narcotics | Immunomodulation | Pharmacology | Cyclic AMP response element-binding protein | Medicine | Brain-derived neurotrophic factor | Morphology | Cocaine | Dendritic branching | Dementia | HIV | Human immunodeficiency virus
Journal Article