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1. Full Text Drosophila insulin release is triggered by adipose Stunted ligand to brain Methuselah receptor
by Delanoue, Renald and Meschi, Eleonora and Agrawal, Neha and Mauri, Alessandra and Tsatskis, Yonit and McNeill, Helen and Léopold, Pierre
Science, ISSN 0036-8075, 09/2016, Volume 353, Issue 6307, pp. 1553 - 1556
Animals adapt their growth rate and body size to available nutrients by a general modulation of insulin-insulin-like growth factor signaling. In Drosophila,... 
LIPID TRANSFER | BETA-CHAIN | CELLS | ATP SYNTHASE | PROTEIN-COUPLED RECEPTOR | EXTENDED LIFE-SPAN | MULTIDISCIPLINARY SCIENCES | BINDING-PROTEIN | NUTRITION | SECRETION | STRESS RESISTANCE | Receptors, G-Protein-Coupled - metabolism | TOR Serine-Threonine Kinases - metabolism | Incretins - metabolism | Pupa - metabolism | Positive Transcriptional Elongation Factor B - metabolism | Drosophila Proteins - metabolism | Receptors, Gastrointestinal Hormone - metabolism | Drosophila melanogaster - genetics | Brain - metabolism | Drosophila melanogaster - metabolism | Pupa - genetics | Adipose Tissue - metabolism | Inhibitor of Apoptosis Proteins - metabolism | Membrane Proteins - metabolism | Fasting - metabolism | Pupa - growth & development | Fat Body - metabolism | Organ Culture Techniques | Food | Eating | Membrane Proteins - genetics | Hemolymph - metabolism | Insulin - metabolism | Animals | Receptors, Gastrointestinal Hormone - genetics | Drosophila melanogaster - growth & development | Ligands | Receptors, G-Protein-Coupled - genetics | Drosophila Proteins - genetics | Physiological aspects | Pancreatic beta cells | Research | Insulin | Drosophila | Signal transduction | Amino acids | Insulin-like growth factors | Cytokines | Brain | Receptors | Peptides | Organs | Nutrients | Sun
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2. Full Text Impaired enteroendocrine development in intestinal-specific Islet1 mouse mutants causes impaired glucose homeostasis
by Terry, Natalie A and Terry, Natalie A and Walp, Erik R and Lee, Randall A and Kaestner, Klaus H and May, Catherine Lee
American journal of physiology. Gastrointestinal and liver physiology, ISSN 0193-1857, 11/2014, Volume 307, Issue 10, pp. G979 - G991
Enteroendocrine cells secrete over a dozen different hormones responsible for coordinating digestion, absorption, metabolism, and gut motility. Loss of... 
Islet1 | incretins | enteroendocrine | glucose regulation | PHYSIOLOGY | ENDOCRINE CELL-DEVELOPMENT | GASTROINTESTINAL-TRACT | FATE SPECIFICATION | ENTEROINSULAR AXIS | CONGENITAL MALABSORPTIVE DIARRHEA | CHROMOGRANIN-A | INCRETIN HORMONES | HOMEOBOX GENE ISL-1 | GLUCAGON-LIKE PEPTIDE-1 | RECEPTOR KNOCKOUT MICE | GASTROENTEROLOGY & HEPATOLOGY | Dietary Fats - metabolism | Intestinal Mucosa - metabolism | Gastric Inhibitory Polypeptide - metabolism | Age Factors | Intestine, Small - pathology | Transcription Factors - deficiency | Male | Malabsorption Syndromes - metabolism | Intestinal Absorption | Cholecystokinin - metabolism | LIM-Homeodomain Proteins - deficiency | Glucose Metabolism Disorders - metabolism | Female | Chromogranin A - metabolism | Enteroendocrine Cells - metabolism | Weight Gain | Microfilament Proteins - genetics | Somatostatin - metabolism | Animals, Newborn | Enteroendocrine Cells - pathology | Glucose Tolerance Test | Glucagon-Like Peptide 1 - metabolism | Mice, Inbred C57BL | Genotype | Biomarkers - blood | Transcription Factors - genetics | Gastrins - metabolism | Malabsorption Syndromes - genetics | Mice, Knockout | Ghrelin - metabolism | LIM-Homeodomain Proteins - genetics | Phenotype | Animals | Diarrhea - genetics | Glucose Metabolism Disorders - genetics | Serotonin - metabolism | Glucose Metabolism Disorders - blood | Diarrhea - metabolism | Intestine, Small - metabolism | Blood Glucose - metabolism | Integrases - genetics | Intestinal Mucosa - pathology | Glucose metabolism | Pancreatic beta cells | Cell research | Gene mutations | Homeostasis | Genetic research | Genetic aspects | Research | Health aspects | Hormones and Signaling
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3. Full Text A targeted RNAi screen identifies endocytic trafficking factors that control GLP-1 receptor signaling in pancreatic β-cells
by Buenaventura, Teresa and Kanda, Nisha and Douzenis, Phoebe C and Jones, Ben and Bloom, Stephen R and Chabosseau, Pauline and Corrêa, Ivan R and Bosco, Domenico and Piemonti, Lorenzo and Marchetti, Piero and Johnson, Paul R and Shapiro, A.M. James and Rutter, Guy A and Tomas, Alejandra
Diabetes, ISSN 0012-1797, 03/2018, Volume 67, Issue 3, pp. 385 - 399
The glucagon-like peptide 1 (GLP-1) receptor (GLP-1R) is a key target for type 2 diabetes (T2D) treatment. Because endocytic trafficking of agonist-bound... 
CLATHRIN-MEDIATED ENDOCYTOSIS | RETROMER | TRANSPORT | GLUCAGON-LIKE PEPTIDE-1 | GLUCOSE | MEMBRANE | INSULIN-SECRETION | ENDOCRINOLOGY & METABOLISM | PROTEIN-COUPLED RECEPTORS | EXPRESSION | BINDING | Sorting Nexins - genetics | Humans | Lysosomes - enzymology | Exenatide | Recombinant Fusion Proteins - metabolism | DNA-Binding Proteins - metabolism | Glucagon-Like Peptide-1 Receptor - genetics | Glucagon-Like Peptide-1 Receptor - metabolism | Insulin-Secreting Cells - metabolism | Lysosomes - metabolism | RNA Interference | Incretins - pharmacology | Sorting Nexins - metabolism | Cyclic AMP - metabolism | Insulin Secretion | Lysosomes - drug effects | Cell Line | Microscopy, Electron, Transmission | Second Messenger Systems - drug effects | DNA-Binding Proteins - antagonists & inhibitors | Tissue Culture Techniques | Endocytosis - drug effects | Recombinant Fusion Proteins - chemistry | DNA-Binding Proteins - genetics | Peptides - pharmacology | Lysosomes - ultrastructure | Insulin-Secreting Cells - ultrastructure | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Calcium Signaling - drug effects | Mice | Sorting Nexins - antagonists & inhibitors | Venoms - pharmacology | Glucagon-Like Peptide-1 Receptor - agonists | Type 2 diabetes | Usage | Care and treatment | B cells | Research | Glucagon
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4. Full Text Incretin action in the pancreas: Potential promise, possible perils, and pathological pitfalls
by Drucker, Daniel J
Diabetes, ISSN 0012-1797, 10/2013, Volume 62, Issue 10, pp. 3316 - 3323
Glucagon-like peptide-1 (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP) are incretin hormones that control the secretion of insulin, glucagon,... 
DIPEPTIDYL PEPTIDASE-4 INHIBITOR | DRUG-INDUCED PANCREATITIS | GLUCAGON-LIKE PEPTIDE-1 | ALPHA-CELL HYPERPLASIA | BETA-CELLS | ENDOCRINOLOGY & METABOLISM | POSTPRANDIAL HYPERGLYCEMIA | NO EVIDENCE | GLP-1 RECEPTOR ACTIVATION | DEPENDENT INSULINOTROPIC POLYPEPTIDE | EXOCRINE PANCREAS | Cell Proliferation | Gastric Inhibitory Polypeptide - metabolism | Pancreas - physiopathology | Dipeptidyl-Peptidase IV Inhibitors - therapeutic use | Humans | Incretins - metabolism | Male | Receptors, Gastrointestinal Hormone - metabolism | Inflammation - metabolism | Insulin-Secreting Cells - metabolism | Female | Real-Time Polymerase Chain Reaction | Receptors, Glucagon - agonists | Acinar Cells - metabolism | Cricetinae | Atherosclerosis - drug therapy | Glucagon-Like Peptide 1 - metabolism | Atherosclerosis - physiopathology | Diabetes Mellitus - drug therapy | Diabetes Mellitus - metabolism | Pancreas - drug effects | Rats | Pancreas - metabolism | Atherosclerosis - metabolism | Blotting, Western | Animals | Glucagon-Like Peptide-1 Receptor | Diabetes Mellitus - physiopathology | Mice | Glucose metabolism | G proteins | Health aspects | Diabetes therapy | s in Diabetes
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5. Full Text Glucagon-like peptide-1 reduces hepatic lipogenesis via activation of AMP-activated protein kinase
by Ben-Shlomo, Shani and Zvibel, Isabel and Shnell, Mati and Shlomai, Amir and Chepurko, Elena and Halpern, Zamir and Barzilai, Nir and Oren, Ran and Fishman, Sigal
Journal of Hepatology, ISSN 0168-8278, 2010, Volume 54, Issue 6, pp. 1214 - 1223
Background & Aims Glucagon-like peptide-1 (GLP-1), a gut-derived peptide degraded by dipeptidyl peptidase-4 (DPP4), stimulates insulin secretion in response to... 
Gastroenterology and Hepatology | Incretins | Inflammatory cytokines | Liver | AMPK | Dipeptidylpetidase 4 | Lipogenesis | Glucagon-like peptide 1 | Gluconeogenesis | RAT-LIVER | METFORMIN | CELLS | HEPATOCYTES | INDUCED INSULIN-RESISTANCE | TISSUE GROWTH-FACTOR | GLUCOSE-METABOLISM | STEATOSIS | MICE | GASTROENTEROLOGY & HEPATOLOGY | AMP-Activated Protein Kinases - metabolism | Dipeptidyl Peptidase 4 - metabolism | Fatty Liver - pathology | Rats, Inbred F344 | Male | DNA Primers - genetics | Hepatocytes - metabolism | RNA, Messenger - metabolism | AMP-Activated Protein Kinases - deficiency | Liver - drug effects | Base Sequence | Lipids - blood | Inflammation Mediators - metabolism | Cyclic AMP - metabolism | Lipogenesis - physiology | Hepatocytes - drug effects | Fatty Liver - metabolism | Cytokines - metabolism | Glucagon-Like Peptide 1 - metabolism | Rats, Transgenic | Liver - metabolism | RNA, Messenger - genetics | Cells, Cultured | Glucagon-Like Peptide 1 - pharmacology | Rats | Dipeptidyl Peptidase 4 - genetics | Enzyme Activation - drug effects | Animals | Dipeptidyl Peptidase 4 - deficiency | Lipogenesis - drug effects | Metabolic Networks and Pathways - drug effects | AMP-Activated Protein Kinases - genetics | Fatty Liver - etiology | Phosphatases | Glucagon | Triglycerides | Glucose | Transforming growth factors | Fatty acids | Dextrose | Carnitine | Glucose metabolism | Angiotensin | Physiological aspects | Protein kinases | Protein binding
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6. Full Text Reduced β-cell secretory capacity in pancreatic-insufficient, but not pancreatic-sufficient, cystic fibrosis despite normal glucose tolerance
by Sheikh, Saba and Gudipaty, Lalitha and De Leon, Diva D and Hadjiliadis, Denis and Kubrak, Christina and Rosenfeld, Nora K and Nyirjesy, Sarah C and Peleckis, Amy J and Malik, Saloni and Stefanovski, Darko and Cuchel, Marina and Rubenstein, Ronald C and Kelly, Andrea and Rickels, Michael R
Diabetes, ISSN 0012-1797, 01/2017, Volume 66, Issue 1, pp. 134 - 144
Patients with pancreatic-insufficient cystic fibrosis (PI-CF) are at increased risk for developing diabetes. We determined beta-cell secretory capacity and... 
OBESE SUBJECTS | ADOLESCENTS | INSULIN-SECRETION | ENDOCRINOLOGY & METABOLISM | HOUR PLASMA-GLUCOSE | ADULTS | SENSITIVITY | INTOLERANCE | ORAL GLUCOSE | EPIDEMIOLOGY | CHILDREN | Exocrine Pancreatic Insufficiency - metabolism | Glucose Tolerance Test | Gastric Inhibitory Polypeptide - metabolism | Glucagon-Like Peptide 1 - metabolism | Proinsulin - metabolism | Cystic Fibrosis - metabolism | Humans | Middle Aged | Cystic Fibrosis - pathology | Incretins - metabolism | Pancreas - pathology | Male | Pancreas - metabolism | Young Adult | Insulin - metabolism | Insulin-Secreting Cells - metabolism | Adolescent | Glucose - metabolism | Adult | Female | Glucagon - metabolism | Insulin-Secreting Cells - pathology | C-Peptide - metabolism | Cystic fibrosis | Research | Glucose | Dextrose | Pathophysiology
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7. Full Text The metabolic profile of intrahepatic cholestasis of pregnancy is associated with impaired glucose tolerance, dyslipidemia, and increased fetal growth
by Martineau, Marcus G and Raker, Christina and Dixon, Peter H and Chambers, Jenny and Machirori, Mavis and King, Nicole M and Hooks, Melissa L and Manoharan, Ramya and Chen, Kenneth and Powrie, Raymond and Williamson, Catherine
Diabetes Care, ISSN 0149-5992, 02/2015, Volume 38, Issue 2, pp. 243 - 248
OBJECTIVEQuantification of changes in glucose and lipid concentrations in women with intrahepatic cholestasis of pregnancy (ICP) and uncomplicated pregnancy... 
FXR | ACTIVATION | ACID | HYPERGLYCEMIA | ENDOCRINOLOGY & METABOLISM | FARNESOID-X-RECEPTOR | SECRETION | OUTCOMES | ADVERSE PREGNANCY | INCRETIN | EXPRESSION | Glucose Intolerance - metabolism | Fetal Macrosomia - metabolism | Glucose Tolerance Test | Prospective Studies | Diabetes, Gestational - metabolism | Glucagon-Like Peptide 1 - metabolism | Humans | Metabolome - physiology | Birth Weight - physiology | Male | Metabolic Syndrome - metabolism | Cholesterol - metabolism | Gestational Age | Triglycerides - metabolism | Pregnancy | Cholestasis, Intrahepatic - metabolism | Fetal Development - physiology | Lipids - blood | Dyslipidemias - metabolism | Adult | Female | Pregnancy Complications - metabolism | Blood Glucose - metabolism | Pregnancy Outcome | Jaundice, Obstructive | Pregnant women | Growth | Analysis | Fetus | Glucose | Research | Metabolism | Health aspects | Cholestasis | Risk factors | Dextrose
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8. Full Text The role of SGLT1 and GLUT2 in intestinal glucose transport and sensing
by Röder, Pia V and Geillinger, Kerstin E and Zietek, Tamara S and Thorens, Bernard and Koepsell, Hermann and Daniel, Hannelore
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, p. e89977
Intestinal glucose absorption is mediated by SGLT1 whereas GLUT2 is considered to provide basolateral exit. Recently, it was proposed that GLUT2 can be... 
CALCIUM-ABSORPTION | DIFFUSIVE COMPONENT | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | SUGAR ABSORPTION | APICAL GLUT2 | COTRANSPORTER | RAT SMALL-INTESTINE | MICE | BRUSH-BORDER MEMBRANE | INCRETIN SECRETION | Intestines - drug effects | Intestinal Mucosa - metabolism | Incretins - blood | Intestinal Absorption - drug effects | Incretins - metabolism | Glucose - pharmacology | Insulin - blood | Glucose Transporter Type 2 - metabolism | Glucose Transporter Type 2 - deficiency | Sodium-Glucose Transporter 1 - deficiency | Insulin - metabolism | Animals | Glucose - metabolism | Mice | Sodium-Glucose Transporter 1 - metabolism | Blood Glucose - metabolism | Insulin Secretion | Tracers (Biology) | Glucose metabolism | Blood sugar | Glucose transporter | Membranes | Secretion | Chemoreception | Glucose | Kinases | Glucose transport | Insulin | Small intestine | Contamination | Absorption | Clonal deletion | Rodents | Deletion | Transporter | Bulk transporters
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9. Full Text Central GLP-1 receptor signalling accelerates plasma clearance of triacylglycerol and glucose by activating brown adipose tissue in mice
by Kooijman, Sander and Wang, Yanan and Parlevliet, Edwin T and Boon, Mariëtte R and Edelschaap, David and Snaterse, Gido and Pijl, Hanno and Romijn, Johannes A and Rensen, Patrick C. N
Diabetologia, ISSN 0012-186X, 2015, Volume 58, Issue 11, pp. 2637 - 2646
Glucagon-like peptide 1 (GLP-1) receptor (GLP-1R) agonism, used in the treatment of type 2 diabetes, has recently been shown to increase thermogenesis via the... 
GLP-1 | Medicine & Public Health | Human Physiology | Triacylglycerol | Metabolic Diseases | Internal Medicine | Brown adipose tissue | Mice | Exendin-4 | TRIGLYCERIDE CLEARANCE | THERMOGENESIS | ENERGY-EXPENDITURE | GLUCAGON-LIKE PEPTIDE-1 | DIET | METABOLISM | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | CENTRAL-NERVOUS-SYSTEM | BRAIN | Glucagon-Like Peptide 1 - metabolism | Mice, Inbred C57BL | Male | Peptides - pharmacology | Triglycerides - metabolism | Glucagon-Like Peptide-1 Receptor - metabolism | Insulin - metabolism | Animals | Ion Channels - metabolism | Signal Transduction - drug effects | Body Composition - drug effects | Mitochondrial Proteins - metabolism | Incretins - pharmacology | Glucose - metabolism | Signal Transduction - physiology | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Venoms - pharmacology | Uncoupling Protein 1 | Glucagon-Like Peptide-1 Receptor - agonists | Body Composition - physiology | Adipose tissues | Type 2 diabetes | Glucose metabolism | Peptides | Triglycerides | Glucose | Fatty acids | Dextrose
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10. Full Text GLP-1R Agonists Promote Normal and Neoplastic Intestinal Growth through Mechanisms Requiring Fgf7
by Koehler, Jacqueline A and Baggio, Laurie L and Yusta, Bernardo and Longuet, Christine and Rowland, Katherine J and Cao, Xiemin and Holland, Dianne and Brubaker, Patricia L and Drucker, Daniel J
Cell Metabolism, ISSN 1550-4131, 03/2015, Volume 21, Issue 3, pp. 379 - 391
Glucagon-like peptide-1 (GLP-1) secreted from enteroendocrine L cells promotes nutrient disposal via the incretin effect. However, the majority of L cells are... 
STEM-CELLS | INHIBITION | TRUNCALLY VAGOTOMIZED SUBJECTS | COLORECTAL-CANCER | GLUCOSE | FACTOR RECEPTOR 2 | IN-VIVO | ENDOCRINOLOGY & METABOLISM | PROLIFERATION | EXPRESSION | GLUCAGON | CELL BIOLOGY | Receptor, IGF Type 1 - metabolism | Intestinal Mucosa - metabolism | Intestinal Mucosa - physiopathology | Colon - physiology | Intestine, Small - pathology | Incretins - metabolism | Male | Carcinogenesis - metabolism | Intestine, Small - physiology | Fibroblast Growth Factor 7 - metabolism | Glucagon-Like Peptide-1 Receptor - metabolism | Peptides - metabolism | Colon - physiopathology | Female | Mice, Inbred C57BL | Cell Proliferation - physiology | Epidermal Growth Factor - metabolism | Colon - metabolism | Intestinal Mucosa - physiology | Animals | Signal Transduction - physiology | Mice | Intestine, Small - metabolism | Venoms - metabolism | Glucagon-Like Peptide-1 Receptor - agonists | Antimitotic agents | Physiological aspects | Fibroblast growth factors | Glucagon | Antineoplastic agents | Growth
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11. Incretins and lipid metabolism
by Tsimihodimos, Vasilis and Elisaf, Moses
Current Medicinal Chemistry, ISSN 0929-8673, 04/2017, Volume 25, Issue 18, pp. 2133 - 2139
Recent findings indicate that incretin hormones and incretin-based therapies may affect the metabolism of lipoproteins, although the corresponding mechanisms... 
Incretins | HDL | GLP-1 | Dyslipidemia | Lipids | Triglycerides | GLP-2 | GIP | Glucagon-Like Peptide 1 - metabolism | Dipeptidyl-Peptidase IV Inhibitors - therapeutic use | Humans | Lipoproteins, HDL - metabolism | Incretins - metabolism | Lipoproteins, LDL - metabolism | Liver Diseases - drug therapy | Diabetes Mellitus, Type 2 - drug therapy | Glucagon-Like Peptide-1 Receptor - agonists | Triglycerides - metabolism | English language | Liver diseases | Liver | Central nervous system | Hormones | Metabolism | Apolipoproteins | Insulin | Low density lipoprotein | Lipoproteins | Fatty liver | Diet | Intestine | Arteriosclerosis | Atherosclerosis | Lipid metabolism
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12. Full Text The role of bile acids in reducing the metabolic complications of obesity after bariatric surgery: A systematic review
by Penney, N.C and Kinross, J and Newton, R.C and Purkayastha, S
International Journal of Obesity, ISSN 0307-0565, 11/2015, Volume 39, Issue 11, pp. 1565 - 1574
BACKGROUND: Bariatric surgery is currently the most efficacious treatment for obesity and its associated metabolic co-morbidities, such as diabetes. The... 
RESTING ENERGY-EXPENDITURE | NUTRITION & DIETETICS | GLUCAGON-LIKE PEPTIDE-1 | SLEEVE GASTRECTOMY | HUMAN GUT MICROBIOTA | Y GASTRIC BYPASS | GROWTH-FACTOR 19 | INSULIN-SECRETION | ENDOCRINOLOGY & METABOLISM | WEIGHT-LOSS | ILEAL INTERPOSITION SURGERY | FARNESOID-X RECEPTOR | Bariatric Surgery | Metabolic Diseases - surgery | Obesity, Morbid - surgery | Receptors, G-Protein-Coupled - metabolism | Humans | Gastrointestinal Microbiome | Endoplasmic Reticulum - metabolism | Homeostasis | Incretins - metabolism | Bile Acids and Salts - metabolism | Lipid Metabolism | Treatment Outcome | Obesity, Morbid - complications | Energy Metabolism | Metabolic Diseases - etiology | Weight Loss | Glucose - metabolism | Metabolic Diseases - prevention & control | Obesity, Morbid - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Obesity | Complications and side effects | Care and treatment | Usage | Bile acids | Surgery | Influence | Research | Health aspects | Index Medicus
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13. Full Text Incretin hormones regulate microglia oxidative stress, survival and expression of trophic factors
by Spielman, Lindsay Joy and Gibson, Deanna Lynn and Klegeris, Andis
European Journal of Cell Biology, ISSN 0171-9335, 05/2017, Volume 96, Issue 3, pp. 240 - 253
The incretin hormones glucagon-like peptide (GLP)-1 and glucose-dependent insulinotropic polypeptide (GIP) are primarily known for their metabolic function in... 
Incretins | Alzheimer’s disease | Cell death | Microglia degeneration | Parkinson’s disease | Apoptosis |