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Journal Article
Nature Medicine, ISSN 1078-8956, 05/2016, Volume 22, Issue 5, pp. 516 - 523
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 8/2014, Volume 34, Issue 8, pp. 1306 - 1314
Lipocalin-2 (LCN2) is a secreted protein of the lipocalin family, but little is known about the expression or the role of LCN2 in the central nervous system.... 
blood-brain barrier | ischemia/reperfusion | lipocain-2 | neuroinflammation | astrocyte | microglia | MEDIATOR | BARRIER | IRON | GELATINASE-ASSOCIATED LIPOCALIN | NEUROSCIENCES | DAMAGE | INFLAMMATORY MECHANISMS | ENDOCRINOLOGY & METABOLISM | HEMATOLOGY | ISCHEMIC-STROKE | UP-REGULATION | PROGRESSION | Neurons - pathology | Oncogene Proteins - genetics | Reperfusion Injury - etiology | Cell Proliferation | Neuroglia - pathology | Coculture Techniques | Astrocytes - pathology | Acute-Phase Proteins - genetics | Infarction, Middle Cerebral Artery - immunology | Infarction, Middle Cerebral Artery - metabolism | Ischemic Attack, Transient - complications | Astrocytes - immunology | Culture Media | Infarction, Middle Cerebral Artery - complications | Lipocalins - genetics | Neuroglia - immunology | Neurons - metabolism | Acute-Phase Proteins - deficiency | Ischemic Attack, Transient - metabolism | Reperfusion Injury - metabolism | Disease Models, Animal | Ischemic Attack, Transient - pathology | Neutrophil Infiltration - immunology | Reperfusion Injury - pathology | Cell Survival | Mice, Inbred C57BL | Neurons - immunology | Infarction, Middle Cerebral Artery - pathology | Mice, Knockout | Animals | Lipocalin-2 | Reperfusion Injury - immunology | Oncogene Proteins - deficiency | Mice | Ischemic Attack, Transient - immunology | Astrocytes - metabolism | Index Medicus | blood–brain barrier | Original | reperfusion | ischemia
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 11/2014, Volume 15, Issue 11, pp. 20913 - 20926
Inflammation and oxidative stress play an important part in the pathogenesis of focal cerebral ischemia/reperfusion (I/R) injury, resulting in neuronal death.... 
Oxidative stress | Neuroinflammation | Cerebral ischemia-reperfusion injury | Chrysin | CELLS | WISTAR RATS | cerebral ischemia-reperfusion injury | ARTERY OCCLUSION | BRAIN-INJURY | BIOCHEMISTRY & MOLECULAR BIOLOGY | REPERFUSION | chrysin | CHEMISTRY, MULTIDISCIPLINARY | DAMAGE | neuroinflammation | PATHWAY | AMELIORATION | NF-KAPPA-B | ISCHEMIC-STROKE | oxidative stress | Neuroprotective Agents - therapeutic use | Inflammation - pathology | Middle Cerebral Artery - immunology | Cytokines - analysis | Reperfusion Injury - drug therapy | Male | Brain Ischemia - immunology | Infarction, Middle Cerebral Artery - immunology | Infarction, Middle Cerebral Artery - metabolism | Flavonoids - therapeutic use | Inflammation - metabolism | Inflammation - drug therapy | Middle Cerebral Artery - metabolism | Anti-Inflammatory Agents - therapeutic use | Infarction, Middle Cerebral Artery - drug therapy | Reperfusion Injury - metabolism | Cytokines - immunology | Reperfusion Injury - pathology | Mice, Inbred C57BL | Middle Cerebral Artery - drug effects | Middle Cerebral Artery - pathology | Brain Ischemia - prevention & control | Inflammation - immunology | Infarction, Middle Cerebral Artery - pathology | Antioxidants - therapeutic use | Animals | Reperfusion Injury - immunology | Brain Ischemia - pathology | Oxidative Stress - drug effects
Journal Article
Stroke, ISSN 0039-2499, 10/2015, Volume 46, Issue 10, pp. 2916 - 2925
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2011, Volume 25, Issue 4, pp. 715 - 726
Abstract 17β-Estradiol (E) and progesterone (P) are neuroprotective factors in the brain preventing neuronal death under different injury paradigms. Our... 
Psychiatry | Allergy and Immunology | MCAO | Chemokine | Stroke | Progesterone | Estrogen | Microglia | FUNCTIONAL RECOVERY | ESTROGEN-MEDIATED NEUROPROTECTION | MALE-MICE | EXPERIMENTAL STROKE | IMMUNOLOGY | FOCAL ISCHEMIA | SEX STEROIDS | NEUROSCIENCES | RECEPTOR-ALPHA | TRAUMATIC BRAIN-INJURY | INFLAMMATORY RESPONSE | ISCHEMIC-STROKE | Rats, Wistar | Immune Tolerance - physiology | Male | Infarction, Middle Cerebral Artery - immunology | Recovery of Function | Brain - metabolism | Stroke - pathology | Lymphocytes - physiology | Lymphocytes - immunology | Microglia - immunology | Microglia - physiology | Neuroprotective Agents - pharmacology | Infarction, Middle Cerebral Artery - complications | Female | Progesterone - pharmacology | Estradiol - pharmacology | Macrophages - immunology | Stroke - immunology | Chemokines - immunology | Estradiol - physiology | Macrophages - physiology | Gonadal Steroid Hormones - pharmacology | Rats | Infarction, Middle Cerebral Artery - pathology | Stroke - etiology | Animals | Analysis of Variance | Sex Factors | Brain - pathology | Progesterone - physiology | Chemokines - metabolism | Brain - immunology | Drug Combinations | Immunologic Factors - pharmacology | Prevention | Neurons | Lymphocytes | Physiological aspects | Brain damage | Personal injuries | Gene expression | Vascular endothelial growth factor | Estradiol
Journal Article
STEM CELLS Translational Medicine, ISSN 2157-6564, 10/2015, Volume 4, Issue 10, pp. 1131 - 1143
The effects of mesenchymal stem cell (MSC)‐derived extracellular vesicles were compared with those of MSCs i.v. delivered 1, 3, and 5 days or 1 day after focal... 
Angiogenesis | Adult stem cells | Cellular therapy | Clinical translation | Nervous system | Tissue regeneration | Mesenchymal stem cells | FUNCTIONAL RECOVERY | BRAIN-INJURY | FOCAL CEREBRAL-ISCHEMIA | BONE-MARROW | VERSUS-HOST-DISEASE | MESENCHYMAL STEM-CELLS | CELL & TISSUE ENGINEERING | THERAPEUTIC BENEFIT | IMMUNE-RESPONSES | NEURAL PROGENITOR CELLS | MARROW STROMAL CELLS | Infarction, Middle Cerebral Artery - physiopathology | Ataxia - etiology | Male | Infarction, Middle Cerebral Artery - immunology | Infarction, Middle Cerebral Artery - therapy | Extracellular Vesicles - transplantation | Mesenchymal Stromal Cells - ultrastructure | Infarction, Middle Cerebral Artery - complications | Myeloid Cells - immunology | Disease Models, Animal | Lymphocyte Activation | Mice, Inbred C57BL | Nerve Regeneration | Random Allocation | Organ Specificity | Ataxia - prevention & control | Culture Media, Conditioned | Rotarod Performance Test | Animals | Brain - pathology | Lymphocyte Count | Mice | Myeloid Cells - pathology | Brain - immunology | Mesenchymal Stem Cell Transplantation | Cell Movement | Stroke | Data analysis | Traumatic brain injury | Immune response | Mesenchyme | Clinical trials | Administrative support | Patients | Neurogenesis | Carotid arteries | Studies | Prevention | Vesicles | Immunosuppression | Ischemia | Peripheral blood | Stem cells | Writing | Laboratory animals | Brain injury | Veins & arteries | Enabling Technologies for Cell-Based Clinical Translation
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 01/2015, Volume 35, Issue 2, pp. 583 - 598
Journal Article
Journal of Neuroimmunology, ISSN 0165-5728, 2007, Volume 190, Issue 1, pp. 28 - 33
Journal Article
The Journal of Immunology, ISSN 0022-1767, 06/2006, Volume 176, Issue 11, pp. 6523 - 6531
Journal Article