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Journal Article
Molecular and Cellular Biochemistry, ISSN 0300-8177, 8/2012, Volume 367, Issue 1, pp. 73 - 84
The pathophysiological mechanisms leading to neuronal injury in middle cerebral artery occlusion (MCAO) model of cerebral stroke are complex and multifactorial... 
Life Sciences | Biochemistry, general | MCAO | NF-κB | Piperine | COX-2 | NOS-2 | Medical Biochemistry | Oncology | Behavioral deficit | Cardiology | ACTIVATION | CYCLOOXYGENASE-2 | NITRIC-OXIDE SYNTHASE-2 | STROKE PATIENTS | CYTOKINE EXPRESSION | DAMAGE | CELL BIOLOGY | INFARCT VOLUME | NF-kappa B | MICE | STRESS | PROSTAGLANDIN E-2 | Reperfusion Injury - etiology | Rats, Wistar | Motor Activity - drug effects | Male | NF-kappa B - metabolism | Neuroprotective Agents - pharmacology | Piperidines - pharmacology | Cyclooxygenase 2 - genetics | Infarction, Middle Cerebral Artery - complications | Infarction, Middle Cerebral Artery - drug therapy | Cytokines - blood | Alkaloids - pharmacology | Reperfusion Injury - pathology | Anti-Inflammatory Agents - pharmacology | Cytokines - secretion | Inflammation Mediators - blood | Rats | Infarction, Middle Cerebral Artery - pathology | Down-Regulation - drug effects | Animals | Reperfusion Injury - prevention & control | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Cyclooxygenase 2 - metabolism | Polyunsaturated Alkamides - pharmacology | Muscle Strength - drug effects | Benzodioxoles - pharmacology | Nitric Oxide Synthase Type II - metabolism | Index Medicus | Brain | Stroke | Antiinflammatory agents | Neurons | Inflammation | Interleukin 6 | NF- Kappa B protein | Reperfusion | Ischemia | Cell death | Cerebral blood flow | Interleukin 1 | Cyclooxygenase-2 | Injuries | Tumor necrosis factor- alpha
Journal Article
Journal Article
Brain Research, ISSN 0006-8993, 2014, Volume 1559, pp. 46 - 54
Abstract Nobiletin, a citrus polymethoxylated flavone, is reported to possess various pharmacological activities such as anticancer, anti-inflammation, and... 
Neurology | Citrus polymethoxylated flavone | Ischemic stroke | Neutrophil infiltration | Nobiletin | Neuroprotectant | Motor function deficit | TARGET | IMPROVES MEMORY IMPAIRMENT | FOCAL ISCHEMIA | NEUROSCIENCES | STROKE | INFLAMMATION | MICE | EXPRESSION | BRAIN | Neurons - pathology | Rats, Wistar | Apoptosis - drug effects | Reperfusion Injury - drug therapy | Motor Activity - drug effects | Male | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Ischemic Attack, Transient - complications | Neuroprotective Agents - pharmacology | Neuroprotective Agents - pharmacokinetics | Infarction, Middle Cerebral Artery - complications | Infarction, Middle Cerebral Artery - drug therapy | Reperfusion Injury - complications | Neurons - drug effects | Flavones - pharmacology | Disease Models, Animal | Ischemic Attack, Transient - pathology | Neutrophils - pathology | Ischemic Attack, Transient - drug therapy | Brain Edema - etiology | Flavones - pharmacokinetics | Reperfusion Injury - pathology | Brain Edema - pathology | Neutrophils - drug effects | Rats | Treatment Outcome | Infarction, Middle Cerebral Artery - pathology | Brain - drug effects | Animals | Anti-Inflammatory Agents, Non-Steroidal - pharmacokinetics | Brain - pathology | Brain Edema - drug therapy | Antioxidants | Brain | Brain damage | Cerebral ischemia | Alzheimer's disease | Protein binding | Index Medicus
Journal Article
Stroke, ISSN 0039-2499, 05/2011, Volume 42, Issue 5, pp. 1237 - 1243
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e89450 - e89450
Aim: Glycyrrhizin (GL) has been reported to protect against ischemia and reperfusion (I/R)-induced injury by inhibiting the cytokine activity of high mobility... 
ISCHEMIA-REPERFUSION INJURY | STROKE | CELLS | LIVER-INJURY | CHROMATIN PROTEIN HMGB1 | POSTISCHEMIC BRAIN | MEDIATOR | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MOBILITY GROUP BOX-1 | HMG-1 | Neuroprotective Agents - therapeutic use | Cerebral Infarction - drug therapy | Inflammation - pathology | Apoptosis - drug effects | Reperfusion Injury - drug therapy | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Cerebral Infarction - complications | Reperfusion Injury - blood | Inflammation - complications | Brain Ischemia - blood | Glycyrrhizic Acid - pharmacology | Neuroprotective Agents - pharmacology | Infarction, Middle Cerebral Artery - complications | Inflammation - drug therapy | Cerebral Infarction - blood | HMGB1 Protein - metabolism | Infarction, Middle Cerebral Artery - drug therapy | Reperfusion Injury - complications | p38 Mitogen-Activated Protein Kinases - metabolism | HMGB1 Protein - antagonists & inhibitors | Cerebral Infarction - pathology | Brain Ischemia - complications | Reperfusion Injury - pathology | Antioxidants - pharmacology | Infarction, Middle Cerebral Artery - pathology | Rats, Sprague-Dawley | Infarction, Middle Cerebral Artery - blood | Glycyrrhizic Acid - therapeutic use | Animals | MAP Kinase Signaling System - drug effects | Brain Ischemia - drug therapy | Brain Ischemia - pathology | Oxidative Stress - drug effects | Oxidative stress | Brain | Chromosomal proteins | Cytokines | Ischemia | Inflammation | Enzyme-linked immunosorbent assay | Apoptosis | Occlusion | Cytochrome | Cerebral cortex | Kinases | Caspase-3 | HMGB1 protein | Western blotting | Interleukin 6 | Reperfusion | Cerebral blood flow | Rodents | Inhibition | Pretreatment | Stresses | Glycyrrhizin | Cortex | Rats | Caspase | JNK protein | Tumor necrosis factor-α | Nitric-oxide synthase | Stress | Neurological diseases | Polymerase chain reaction | Cytochrome c | Serum levels | Signaling | Injury prevention | Molecular modelling | Nitric oxide | Immunofluorescence | Index Medicus
Journal Article