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Nature Medicine, ISSN 1078-8956, 02/2017, Volume 23, Issue 2, pp. 174 - 184
Low-grade, chronic inflammation has been associated with many diseases of aging, but the mechanisms responsible for producing this inflammation remain unclear.... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | INTERLEUKIN-18 LEVELS | PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | CARDIOVASCULAR-DISEASE | RECEPTOR | ESSENTIAL-HYPERTENSION | PROTEINS | T-CELLS | AGE | CELL BIOLOGY | Toll-Like Receptor 5 - genetics | Humans | Middle Aged | Oxidative Stress - immunology | Transcriptome | Immunoblotting | Male | Purinergic P1 Receptor Antagonists - pharmacology | Young Adult | Toll-Like Receptor 8 - immunology | Aging - immunology | Toll-Like Receptor 6 - immunology | Aging - genetics | Calcium-Binding Proteins - immunology | Interleukin-1beta - metabolism | Aged, 80 and over | Caffeine - pharmacology | Hypertension - genetics | Intracellular Signaling Peptides and Proteins - genetics | Interleukin 1 Receptor Antagonist Protein - immunology | Carotid Intima-Media Thickness | Mortality | Neutrophils - drug effects | Hypertension - immunology | Nucleotides - metabolism | Toll-Like Receptor 5 - immunology | Monocytes - drug effects | Regression Analysis | Phenotype | Mice | Calcium-Binding Proteins - genetics | Metabolomics | Pulse Wave Analysis | Neutrophil Activation - drug effects | Cytidine - pharmacology | Interleukin 1 Receptor Antagonist Protein - genetics | Intracellular Signaling Peptides and Proteins - immunology | Cytidine - analogs & derivatives | CARD Signaling Adaptor Proteins - genetics | Toll-Like Receptor 6 - genetics | Adult | Female | Blood Pressure - drug effects | Blood Platelets - drug effects | Macrophages - immunology | Cytokines - immunology | CARD Signaling Adaptor Proteins - immunology | Cell Line | Vascular Stiffness - genetics | Cytokines - metabolism | Oxidative Stress - genetics | Interleukin-1beta - immunology | Adenine - pharmacology | Inflammation - immunology | Inflammasomes - genetics | Vascular Stiffness - immunology | Animals | Inflammasomes - immunology | Toll-Like Receptor 8 - genetics | Inflammation - genetics | Aged | Platelet Activation - drug effects | Genetic aspects | Immune response | Gene expression | Health aspects | Aging | Inflammation | Immune system | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, pp. e0175153 - e0175153
IL-1 beta is a potent player in cutaneous inflammation and central for the development of a Th17 micro-milieu in autoinflammatory diseases including psoriasis.... 
VITAMIN-D | SYSTEMIC-LUPUS-ERYTHEMATOSUS | INTERFERON-GAMMA | MULTIDISCIPLINARY SCIENCES | PSORIASIN S100A7 | INFLAMMATORY CASPASES | KERATINOCYTES | 1,25-DIHYDROXYVITAMIN D-3 | MOLECULAR-CLONING | EXPRESSION | T-CELLS | Caspases - immunology | Humans | Interleukin-17 - immunology | Interleukin-17 - pharmacology | Interleukin-1beta - genetics | S100 Proteins - immunology | Psoriasis - genetics | S100 Proteins - genetics | Th17 Cells - drug effects | Transfection | Inflammasomes - drug effects | Psoriasis - pathology | Adaptor Proteins, Signal Transducing - immunology | Apoptosis Regulatory Proteins - genetics | Skin - pathology | Skin - immunology | Th17 Cells - pathology | Interleukin-1beta - secretion | Psoriasis - immunology | S100 Calcium Binding Protein A7 | Apoptosis Regulatory Proteins - immunology | DNA - immunology | Signal Transduction | Caspases - genetics | Cells, Cultured | Gene Expression Regulation | Interleukin-17 - genetics | Interleukin-1beta - immunology | Vitamin D - pharmacology | Keratinocytes - cytology | Keratinocytes - immunology | DNA - genetics | Interleukin-17 - antagonists & inhibitors | Keratinocytes - drug effects | Inflammasomes - immunology | Adaptor Proteins, Signal Transducing - genetics | Th17 Cells - immunology | Interferon-gamma - pharmacology | Interleukins | Physiological aspects | Development and progression | Skin diseases | Genetic aspects | Inflammation | Research | Cell culture | Transcription | Interleukin | Helper cells | Lymphocytes T | Inflammatory diseases | Vitamin D | Lymphocytes | Interleukin 1 | Lesions | Deoxyribonucleic acid--DNA | Psoriasis | Cytokines | Dermatology | Keratinocytes | Caspase | Epidermis | Hazards | Gene expression | Caspase-5 | Hospitals | Biopsy | Interferon | Autoimmune diseases | Apoptosis | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2014, Volume 111, Issue 30, pp. 11181 - 11186
Acute glaucoma is a sight-threatening condition characterized by a sudden and substantial rise in intraocular pressure (IOP) and consequent retinal ganglion... 
Glaucoma | Reperfusion | Messenger RNA | Ischemia | Toll like receptors | Central nervous system | Retina | Brain damage | Intraocular pressure | Microglia | Retinal ischemia/reperfusion injury | Cell apoptosis | BRAIN-INJURY | MICROGLIA ACTIVATION | INTERLEUKIN-1-BETA | MULTIDISCIPLINARY SCIENCES | REPERFUSION INJURY | POPULATIONS | retinal ischemia/reperfusion injury | ANGLE-CLOSURE GLAUCOMA | cell apoptosis | TOLL-LIKE RECEPTORS | CENTRAL-NERVOUS-SYSTEM | MICE | CEREBRAL-ISCHEMIA | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Caspase 8 - metabolism | Caspase 1 - metabolism | Interleukin-1beta - genetics | Caspase 8 - genetics | Interleukin-1beta - metabolism | Apoptosis Regulatory Proteins - genetics | Eye Proteins - genetics | Glaucoma - metabolism | Disease Models, Animal | Acute Disease | Rats | Toll-Like Receptor 4 - genetics | Receptors, Cytoplasmic and Nuclear - genetics | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Toll-Like Receptor 4 - metabolism | Inflammasomes - genetics | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Eye Proteins - metabolism | Caspase 1 - genetics | Adaptor Proteins, Signal Transducing - genetics | Mice | Adaptor Proteins, Signal Transducing - metabolism | Enzyme Activation - genetics | Glaucoma - genetics | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus | Biological Sciences | reperfusion injury | retinal ischemia
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 665 - 680
Cellular lipid metabolism has been linked to immune responses; however, the precise mechanisms by which de novo fatty acid synthesis can regulate inflammatory... 
MEDICINE, RESEARCH & EXPERIMENTAL | OXIDATIVE STRESS | GLUCOSE-METABOLISM | ATP-CITRATE LYASE | UCP2 | SEPTIC SHOCK | CELL-GROWTH | CRITICALLY-ILL PATIENTS | INTENSIVE INSULIN THERAPY | CANCER | MITOCHONDRIAL UNCOUPLING PROTEINS | Fatty Acid Synthase, Type I - genetics | Fatty Acid Synthase, Type I - biosynthesis | Lipids - genetics | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Ion Channels - genetics | Caspase 1 - metabolism | Mitochondrial Proteins - genetics | Interleukin-1beta - genetics | Lipids - biosynthesis | Sepsis - pathology | Interleukin-1beta - metabolism | Mitochondrial Proteins - metabolism | Sepsis - metabolism | Macrophages - pathology | Down-Regulation - genetics | Inflammasomes - genetics | Sepsis - genetics | Mice, Knockout | Carrier Proteins - genetics | Enzyme Induction - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Sepsis - chemically induced | Ion Channels - metabolism | Caspase 1 - genetics | Sepsis - therapy | Mice | Uncoupling Protein 2 | Interleukin-18 - genetics | Interleukin-18 - metabolism | Cellular proteins | Immune response | Synthesis | Sepsis | Development and progression | Lipids | Genetic aspects | Properties | Enzymes | Cytokines | Pathogenesis | Mortality | Glucose | Metabolism | Gene expression | Fatty acids | Proteins | Studies | Metabolites | Consent | Rodents | Index Medicus | Abridged Index Medicus
Journal Article
Nature Immunology, ISSN 1529-2908, 07/2015, Volume 16, Issue 8, pp. 859 - 870
The receptor NLRP3 is involved in the formation of the NLRP3 inflammasome that activates caspase-1 and mediates the release of interleukin 1β (IL-1β) and... 
NIH 3T3 Cells | Asthma - metabolism | Inflammasomes - metabolism | Oligonucleotide Array Sequence Analysis | NLR Family, Pyrin Domain-Containing 3 Protein | Interferon Regulatory Factors - metabolism | Gene Expression Profiling | Th2 Cells - immunology | Promoter Regions, Genetic - genetics | CD4-Positive T-Lymphocytes - immunology | Cell Differentiation - genetics | Signal Transduction - immunology | Neoplasms, Experimental - immunology | Asthma - immunology | Neoplasms, Experimental - genetics | Trans-Activators - genetics | Female | Interleukin-4 - genetics | Carrier Proteins - immunology | Trans-Activators - immunology | Interleukin-4 - metabolism | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Interferon Regulatory Factors - genetics | Gene Expression Regulation, Neoplastic - immunology | Signal Transduction - genetics | Protein Binding - immunology | Reverse Transcriptase Polymerase Chain Reaction | Th2 Cells - metabolism | Asthma - genetics | Blotting, Western | Inflammasomes - genetics | Mice, Knockout | Carrier Proteins - genetics | Cell Differentiation - immunology | Interleukin-4 - immunology | Interferon Regulatory Factors - immunology | Animals | Carrier Proteins - metabolism | Inflammasomes - immunology | Cell Line, Tumor | Trans-Activators - metabolism | Mice | Neoplasms, Experimental - metabolism | Promoter Regions, Genetic - immunology | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 01/2013, Volume 110, Issue 5, pp. 1851 - 1856
A flagellin-independent caspase-1 activation pathway that does not require NAIP5 or NRLC4 is induced by the intracellular pathogen Legionella pneumophila .... 
Cell death | Innate immunity | Inflammasome | PATTERN-RECOGNITION | innate immunity | cell death | inflammasome | MULTIDISCIPLINARY SCIENCES | NLRP3 INFLAMMASOME ACTIVATION | INFECTION | RECEPTORS | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Adaptor Proteins, Vesicular Transport - genetics | Caspase 1 - metabolism | Immunoblotting | Legionella pneumophila - genetics | Adaptor Proteins, Vesicular Transport - metabolism | Necrosis | Caspases - metabolism | Flagellin - genetics | Flagellin - metabolism | Apoptosis Regulatory Proteins - genetics | Cytoskeletal Proteins - metabolism | Receptor, Interferon alpha-beta - genetics | Macrophages - microbiology | Receptor, Interferon alpha-beta - metabolism | Calcium-Binding Proteins - metabolism | Cytokines - metabolism | Caspases - genetics | Mice, Inbred C57BL | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Macrophages - cytology | Legionella pneumophila - metabolism | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Host-Pathogen Interactions | Carrier Proteins - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Caspase 1 - genetics | CARD Signaling Adaptor Proteins | Bone Marrow Cells - microbiology | Legionella pneumophila - physiology | Mice | Enzyme Activation | Mutation | Myeloid Differentiation Factor 88 - metabolism | Bone Marrow Cells - metabolism | Apoptosis | Calcium-Binding Proteins - genetics | Legionella pneumophila | Genetic aspects | Research | Macrophages | Properties | Immunological research | Proteins | Bacteria | Medical treatment | Cells | Index Medicus | Biological Sciences
Journal Article
PLoS genetics, ISSN 1553-7404, 04/2019, Volume 15, Issue 4, pp. e1007739 - e1007739
Sleep disordered breathing (SDB)-related overnight hypoxemia is associated with cardiometabolic disease and other comorbidities. Understanding the genetic... 
INTERMITTENT HYPOXIA | OXYGEN DESATURATION | HEART-FAILURE | GENETICS & HEREDITY | OSTEOPOROTIC FRACTURES | COGNITIVE IMPAIRMENT | APNEA-HYPOPNEA | NLRP3 INFLAMMASOME | SEX-DIFFERENCES | PULMONARY-FIBROSIS | GENOME-WIDE ASSOCIATION | Humans | Middle Aged | Male | Gene Regulatory Networks | Genetic Variation | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Young Adult | Interleukin-18 Receptor alpha Subunit - genetics | Serine Endopeptidases - genetics | Aged, 80 and over | Adult | Female | Hexokinase - genetics | Genome-Wide Association Study | Oxyhemoglobins - metabolism | Extracellular Matrix Proteins - genetics | Computational Biology | Sleep Apnea Syndromes - blood | Sleep Apnea Syndromes - genetics | Sleep - genetics | Nerve Tissue Proteins - genetics | Cell Adhesion Molecules, Neuronal - genetics | Oxygen - blood | Hypoxia - blood | Hypoxia - genetics | Adolescent | Aged | Polymorphism, Single Nucleotide | Quantitative Trait Loci | Physiological aspects | Sleep disorders | Genetic aspects | Oxyhemoglobin | Interleukins | Genomics | Mortality | Genomes | Health aspects | Funding | Critical care | Single-nucleotide polymorphism | Epidemiology | Population genetics | Reelin protein | Consortia | Hypoxemia | Interleukin 1 | Bioinformatics | Public health | Phenotypes | Inflammation | Genetic diversity | Gene expression | Hexokinase | Interleukin 18 | Medicine | Nutrition research | Genetic variance | Sleep | Hospitals | Womens health | Replication | Respiration | Oxygenation | Index Medicus
Journal Article
Blood, ISSN 0006-4971, 03/2018, Volume 131, Issue 13, pp. 1442 - 1455
Hemophagocytic lymphohistiocytosis (HLH) and macrophage activation syndrome (MAS) are life-threatening hyperferritinemic systemic inflammatory disorders.... 
CAUSES AUTOINFLAMMATION | INTERFERON-GAMMA | INFLAMMASOME ACTIVATION | JUVENILE IDIOPATHIC ARTHRITIS | FAMILIAL HEMOPHAGOCYTIC LYMPHOHISTIOCYTOSIS | GENE-EXPRESSION | MICE | ONSET STILLS-DISEASE | NLRC4 CAUSES | HEMATOLOGY | CD8(+) T-CELLS | Interleukin-18 - immunology | Lymphohistiocytosis, Hemophagocytic - pathology | Humans | Pyrin - genetics | Interleukin-1beta - genetics | Macrophage Activation Syndrome - immunology | Mutation, Missense | CARD Signaling Adaptor Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein - genetics | Signal Transduction - immunology | Calcium-Binding Proteins - immunology | Intestinal Mucosa - immunology | Pyrin - immunology | Apoptosis Regulatory Proteins - genetics | Macrophage Activation Syndrome - genetics | Lymphohistiocytosis, Hemophagocytic - genetics | Lymphohistiocytosis, Hemophagocytic - immunology | CARD Signaling Adaptor Proteins - immunology | Apoptosis Regulatory Proteins - immunology | Intercellular Signaling Peptides and Proteins - genetics | Interleukin-1beta - immunology | Signal Transduction - genetics | Inflammasomes - genetics | Mice, Knockout | Animals | NLR Family, Pyrin Domain-Containing 3 Protein - immunology | Macrophage Activation Syndrome - pathology | Inflammasomes - immunology | Mice | Interleukin-18 - genetics | Intercellular Signaling Peptides and Proteins - immunology | Amino Acid Substitution | Calcium-Binding Proteins - genetics | Intestinal Mucosa - pathology | Index Medicus | Abridged Index Medicus | 8 | 12 | 34 | Immunobiology and Immunotherapy
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2009, Volume 284, Issue 36, pp. 24035 - 24048
The role of endogenous inducers of inflammation is poorly understood. To produce the proinflammatory master cytokine interleukin (IL)-1β, macrophages need... 
GENE | LEUCINE-RICH PROTEOGLYCANS | CASPASE-1 | MACROPHAGES | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNATE IMMUNE-RESPONSE | HYALURONAN | IL-1-BETA | SECRETION | ATP | NALP3 INFLAMMASOME | Toll-Like Receptor 2 - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Immunity, Innate - genetics | Extracellular Matrix - metabolism | Caspase 1 - metabolism | Male | Ureteral Obstruction - metabolism | Interleukin-1beta - genetics | Inflammation - metabolism | Kidney - metabolism | Carrier Proteins - immunology | Signal Transduction | Proteoglycans - immunology | Extracellular Matrix Proteins - genetics | Proteoglycans - metabolism | Toll-Like Receptor 4 - genetics | Biglycan | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Ureteral Obstruction - immunology | Macrophages - metabolism | Receptors, Purinergic P2 - metabolism | Toll-Like Receptor 2 - immunology | Mice | Proteoglycans - genetics | Extracellular Matrix - immunology | Kidney - immunology | Caspase 1 - immunology | Extracellular Matrix - genetics | Receptors, Purinergic P2 - genetics | Ureteral Obstruction - genetics | Interleukin-1beta - biosynthesis | Lung - metabolism | Receptors, Purinergic P2 - immunology | Extracellular Matrix Proteins - metabolism | Macrophages - immunology | Carrier Proteins - biosynthesis | Interleukin-1beta - immunology | Inflammation - immunology | Protein Structure, Tertiary - genetics | Receptors, Purinergic P2X7 | Carrier Proteins - genetics | Immunity, Innate - immunology | Animals | Caspase 1 - genetics | Extracellular Matrix Proteins - immunology | Receptors, Purinergic P2X4 | Inflammation - genetics | Lung - immunology | Animal models | Reactive oxygen species | Oligomerization | Proteoglycans | Transcription | Lung | Inflammation | Macrophages | Kidney | Stress | Caspase-1 | Purine P2X receptors | Signal transduction | Cell activation | TLR2 protein | Interleukin 1 | Toll-like receptors | Extracellular matrix | Sepsis | Cooperativity | Injuries | Index Medicus | Glycobiology and Extracellular Matrices
Journal Article
Cell, ISSN 0092-8674, 09/2016, Volume 167, Issue 1, pp. 187 - 202.e17
Inflammasome complexes function as key innate immune effectors that trigger inflammation in response to pathogen- and danger-associated signals. Here, we... 
keratosis lichenoides chronica | germline | IL-1 | keratinocytes | multiple self-healing squamous cell carcinoma | ASC | gain-of-function | inflammasome | cancer | genodermatosis | NLRP1 | MSSE | MSPC | skin inflammation | APOPTOSIS | IMMUNITY | CELLS | DOMAIN | NALP1 | INTERLEUKIN-1-BETA | VITILIGO | BIOCHEMISTRY & MOLECULAR BIOLOGY | KERATINOCYTES | NLRC4 CAUSES | AUTOINFLAMMATORY DISEASE | CELL BIOLOGY | Adaptor Proteins, Signal Transducing - chemistry | Keratosis - genetics | Inflammasomes - metabolism | Pyrin - chemistry | Humans | Chromosomes, Human, Pair 17 - genetics | Hyperplasia - genetics | Apoptosis Regulatory Proteins - genetics | Germ-Line Mutation | Protein Domains | Keratosis - pathology | Carcinoma - pathology | Skin Neoplasms - pathology | Amino Acid Sequence | Interleukin-1 - metabolism | Epidermis - pathology | Genetic Predisposition to Disease | Paracrine Communication | Signal Transduction | Apoptosis Regulatory Proteins - chemistry | Inflammasomes - genetics | Syndrome | Hyperplasia - pathology | Pedigree | Skin Neoplasms - genetics | Adaptor Proteins, Signal Transducing - genetics | Carcinoma - genetics | Oligomers | Medical colleges | Gene mutations | Medical genetics | Disease susceptibility | Genetic aspects | Skin | Health aspects | Skin cancer | Proteins | Squamous cell carcinoma | Index Medicus
Journal Article