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Kidney International, ISSN 0085-2538, 11/2015, Volume 88, Issue 5, pp. 1030 - 1046
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 06/2012, Volume 188, Issue 11, pp. 5438 - 5447
Journal Article
Cancer Prevention Research, ISSN 1940-6207, 07/2011, Volume 4, Issue 7, pp. 1021 - 1029
Journal Article
Nature Cell Biology, ISSN 1465-7392, 03/2018, Volume 20, Issue 3, pp. 307 - 319
Reactive oxygen species (ROS) contribute to tissue damage and remodelling mediated by the inflammatory response after injury. Here we show that ROS, which... 
STEM-CELLS | PHAGOCYTOSIS | ACTIVATION | SUPEROXIDE-PRODUCTION | ROS | KINASE | PTEN | NEURONS | MECHANISMS | OUTGROWTH | CELL BIOLOGY | Ganglia, Spinal - injuries | Sciatic Nerve - injuries | Axons - enzymology | Reactive Oxygen Species - metabolism | Exosomes - pathology | Sciatic Nerve - physiopathology | Spinal Cord Injuries - genetics | Spinal Cord Injuries - pathology | Endocytosis | CX3C Chemokine Receptor 1 - metabolism | Ganglia, Spinal - enzymology | Peripheral Nerve Injuries - physiopathology | Spinal Cord Injuries - enzymology | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Disease Models, Animal | Dyneins - metabolism | Cell Line | NADPH Oxidase 2 - deficiency | Macrophages - pathology | Signal Transduction | Sciatic Nerve - pathology | Mice, Inbred C57BL | Peripheral Nerve Injuries - pathology | PTEN Phosphohydrolase - metabolism | Nuclear Proteins - metabolism | Endosomes - pathology | Nerve Regeneration | Macrophages - enzymology | Mice, Knockout | Animals | Peripheral Nerve Injuries - enzymology | NADPH Oxidase 2 - metabolism | Axons - pathology | Ganglia, Spinal - pathology | Exosomes - enzymology | Endosomes - enzymology | Spinal Cord Injuries - physiopathology | NADPH Oxidase 2 - genetics | Nerve Degeneration | Peripheral Nerve Injuries - genetics | Sciatic Nerve - enzymology | Axons | Regeneration | Reactive oxygen species | NADP (Coenzyme) | Nervous system | Cellular signal transduction | Research | Gene expression | Oxidase | Recovery of function | Spinal injuries | CX3CR1 protein | Inflammatory response | AKT protein | Exosomes | Macrophages | Inactivation | NAD(P)H oxidase | Signal transduction | Dorsal root ganglia | Neurodegeneration | Dieback | CYBB protein | Degeneration | Cell body | Oxygen | Deactivation | Inflammation | Ganglia | 1-Phosphatidylinositol 3-kinase | Signaling | Dynein | PTEN protein | Sciatic nerve | Endosomes | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 2002, Volume 36, Issue 1, pp. 57 - 68
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 11/2016, Volume 37, Issue 2, pp. 328 - 340
OBJECTIVE—The calcium composition of atherosclerotic plaque is thought to be associated with increased risk for cardiovascular events, but whether plaque... 
atherosclerosis | coronary artery disease | macrophages | inflammation | biomarkers | C-REACTIVE PROTEIN | CORONARY-ARTERY CALCIUM | CARDIOVASCULAR EVENTS | VASCULAR CALCIFICATION | VULNERABLE PATIENT | RISK-ASSESSMENT STRATEGIES | COMPUTED-TOMOGRAPHY | IN-VIVO | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | NF-KAPPA-B | Up-Regulation | Prognosis | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Myocytes, Smooth Muscle - pathology | rac GTP-Binding Proteins - deficiency | rac GTP-Binding Proteins - metabolism | Male | Coronary Artery Disease - enzymology | Atherosclerosis - enzymology | Transfection | Interleukin-1beta - metabolism | rac GTP-Binding Proteins - genetics | Coronary Artery Disease - pathology | Inflammation Mediators - metabolism | Aortic Diseases - prevention & control | Vascular Calcification - mortality | Female | Vascular Calcification - enzymology | Aortic Diseases - enzymology | Interleukin 1 Receptor Antagonist Protein - pharmacology | Aorta - enzymology | Aortic Diseases - pathology | Atherosclerosis - pathology | Coronary Vessels - pathology | Genetic Predisposition to Disease | Coronary Vessels - enzymology | Macrophages - pathology | Signal Transduction | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Cells, Cultured | Neuropeptides - metabolism | Coronary Artery Disease - mortality | Plaque, Atherosclerotic | Vascular Calcification - pathology | Macrophages - enzymology | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Apolipoproteins E - genetics | Muscle, Smooth, Vascular - enzymology | Atherosclerosis - prevention & control | rac1 GTP-Binding Protein - metabolism | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 02/2013, Volume 112, Issue 4, pp. 675 - 688
RATIONALE:Matrix metalloproteinase (MMP)-28 regulates the inflammatory and extracellular matrix responses in cardiac aging, but the roles of MMP-28 after... 
fibroblast | MMP-28 | inflammation | macrophage phenotype | myocardial infarction | LYSYL OXIDASE | CARDIAC & CARDIOVASCULAR SYSTEMS | TGF-BETA | VENTRICULAR-FUNCTION | HEART-FAILURE | CORONARY MICROEMBOLIZATION | REPAIR | MATRIX-METALLOPROTEINASE-9 | PERIPHERAL VASCULAR DISEASE | EPILYSIN MMP-28 | TNF-ALPHA | TARGETED DELETION | HEMATOLOGY | Myocardial Infarction - blood | Cell Adhesion Molecules - genetics | Cicatrix - etiology | Extracellular Matrix Proteins - biosynthesis | Male | Matrix Metalloproteinases, Secreted - deficiency | Pulmonary Edema - etiology | Myocytes, Cardiac - enzymology | Myofibroblasts - metabolism | Ventricular Dysfunction, Left - enzymology | Ventricular Remodeling - genetics | Female | Myocardial Infarction - physiopathology | Transcription, Genetic | Cytokines - genetics | Heart Rupture - enzymology | Receptors, Cytokine - genetics | Matrix Metalloproteinase 9 - blood | Myocardial Infarction - enzymology | Macrophage Activation - physiology | Macrophages - classification | Cell Adhesion Molecules - biosynthesis | Receptors, Cytokine - biosynthesis | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Gene Expression Regulation | Ventricular Dysfunction, Left - etiology | Inflammation | Ventricular Remodeling - physiology | Macrophages - enzymology | Mice, Knockout | Collagen - metabolism | Myocardial Infarction - complications | Heart Rupture - etiology | Animals | Pulmonary Edema - enzymology | Matrix Metalloproteinases, Secreted - genetics | Matrix Metalloproteinases, Secreted - physiology | Protein-Lysine 6-Oxidase - metabolism | Cicatrix - enzymology | Mice | Cytokines - biosynthesis | Index Medicus
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 02/2016, Volume 5, Issue 2, p. n/a
Background-The choline-derived metabolite trimethylamine N-oxide (TMAO) has been demonstrated to contribute to atherosclerosis and is associated with coronary... 
atherosclerosis | cardiovascular disease | inflammation | endothelial cell | nuclear factor‐κB signaling | vascular smooth muscle cell | leukocyte adhesion | trimethylamine N‐oxide | Endothelial cell | Trimethylamine N-oxide | Nuclear factor-κB signaling | Atherosclerosis | Cardiovascular disease | Leukocyte adhesion | Vascular smooth muscle cell | Inflammation | MICROBIAL-METABOLISM | CARDIAC & CARDIOVASCULAR SYSTEMS | HYDROSTATIC-PRESSURE | HEART-FAILURE | ADHESION MOLECULES | trimethylamine N-oxide | HUMAN ENDOTHELIAL-CELLS | L-CARNITINE | LYSOPHOSPHATIDIC ACID | CARDIOVASCULAR-DISEASE | PHOSPHATIDYLCHOLINE | nuclear factor-kappa B signaling | Atherosclerosis - genetics | Coculture Techniques | Humans | Myocytes, Smooth Muscle - pathology | Aortitis - enzymology | NF-kappa B - metabolism | Aortitis - chemically induced | Leukocytes - enzymology | Atherosclerosis - enzymology | Receptors, LDL - deficiency | Female | Aortitis - pathology | Myocytes, Smooth Muscle - drug effects | Aorta - enzymology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Receptors, LDL - genetics | Atherosclerosis - pathology | Genetic Predisposition to Disease | Myocytes, Smooth Muscle - enzymology | Aorta - drug effects | Atherosclerosis - chemically induced | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Aortitis - genetics | Cell Adhesion - drug effects | Mice, Knockout | Aorta - pathology | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Signal Transduction - drug effects | Choline | Leukocytes - drug effects | Enzyme Activation | Methylamines - toxicity | Endothelial Cells - pathology | Endothelial Cells - enzymology | Muscle, Smooth, Vascular - enzymology | Endothelial Cells - drug effects | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2005, Volume 25, Issue 28, pp. 6594 - 6600
Journal Article
Science Translational Medicine, ISSN 1946-6234, 05/2010, Volume 2, Issue 32, pp. 32ra36 - ra36
Journal Article
Cancer Cell, ISSN 1535-6108, 07/2014, Volume 26, Issue 1, pp. 121 - 135
The tumor microenvironment plays a critical role in cancer progression, but the precise mechanisms by which stromal cells influence the epithelium are poorly... 
CELLS | ACTIVATION | INVASION | TGF-BETA | ONCOLOGY | REACTIVE STROMA | PATHWAY | PROTEIN P62 | PROSTATE-CANCER | AUTOPHAGY | TUMOR-GROWTH | CELL BIOLOGY | Inflammation - pathology | Fibroblasts - enzymology | Prostatic Intraepithelial Neoplasia - pathology | Oxidative Stress | TOR Serine-Threonine Kinases - metabolism | Sequestosome-1 Protein | Stromal Cells - pathology | Coculture Techniques | Humans | Tumor Microenvironment | Male | Multiprotein Complexes - genetics | Heat-Shock Proteins - deficiency | Heat-Shock Proteins - genetics | Multiprotein Complexes - metabolism | TOR Serine-Threonine Kinases - genetics | RNA Interference | Time Factors | Cell Transformation, Neoplastic - genetics | Interleukin-6 - metabolism | Fibroblasts - metabolism | PTEN Phosphohydrolase - genetics | Signal Transduction | Prostatic Hyperplasia - genetics | Mice, Knockout | Energy Metabolism | Prostatic Intraepithelial Neoplasia - enzymology | Adaptor Proteins, Signal Transducing - deficiency | Cell Line, Tumor | Glucose - metabolism | Mice | Proto-Oncogene Proteins c-myc - genetics | Prostatic Hyperplasia - enzymology | Prostatic Intraepithelial Neoplasia - genetics | Inflammation - enzymology | Prostatic Hyperplasia - pathology | Mechanistic Target of Rapamycin Complex 1 | Amino Acids - metabolism | Stromal Cells - enzymology | Prostatic Neoplasms - genetics | Transfection | HEK293 Cells | Inflammation Mediators - metabolism | Prostatic Neoplasms - pathology | Neoplasm Invasiveness | Heat-Shock Proteins - metabolism | Mice, Inbred C57BL | Cell Communication | PTEN Phosphohydrolase - metabolism | Cell Transformation, Neoplastic - metabolism | Proto-Oncogene Proteins c-myc - metabolism | Animals | Adaptor Proteins, Signal Transducing - genetics | Inflammation - genetics | Prostatic Neoplasms - enzymology | Adaptor Proteins, Signal Transducing - metabolism | Cell Transformation, Neoplastic - pathology | Glucose metabolism | Development and progression | Glucose | Prostate cancer | Dextrose | Tumors | Index Medicus
Journal Article