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The Journal of Immunology, ISSN 0022-1767, 01/2004, Volume 172, Issue 1, pp. 567 - 576
The signaling mechanism by which the anti-inflammatory cytokine IL-10 mediates suppression of proinflammatory cytokine synthesis remains largely unknown.... 
RHEUMATOID-ARTHRITIS | TUMOR-NECROSIS-FACTOR | DNA-BINDING | HUMAN NEUTROPHILS | TYROSINE PHOSPHORYLATION | INTERLEUKIN-10 RECEPTOR | GENE-EXPRESSION | KAPPA-B-ALPHA | IMMUNOLOGY | HUMAN MONOCYTES | MONONUCLEAR PHAGOCYTES | Protein Binding - genetics | Protein Biosynthesis | Interleukin-6 - antagonists & inhibitors | Humans | Tumor Necrosis Factor-alpha - genetics | Immunoglobulins - genetics | Lipopolysaccharides - antagonists & inhibitors | RNA, Messenger - metabolism | Suppressor of Cytokine Signaling Proteins | Repressor Proteins - antagonists & inhibitors | Antigens, CD - metabolism | Trans-Activators - physiology | Protein Tyrosine Phosphatases - antagonists & inhibitors | RNA, Messenger - biosynthesis | Protein Tyrosine Phosphatases - genetics | Inflammation Mediators - physiology | Glycoproteins - genetics | DNA-Binding Proteins - physiology | Protein Tyrosine Phosphatases - biosynthesis | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction - genetics | DNA - metabolism | Down-Regulation - genetics | Macrophages - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 2 | Repressor Proteins - biosynthesis | Up-Regulation - immunology | Interleukin-10 - antagonists & inhibitors | Lipopolysaccharides - pharmacology | Adenoviruses, Human - genetics | Interleukin-10 - immunology | Tumor Necrosis Factor-alpha - biosynthesis | Phosphorylation | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Antigens, CD - biosynthesis | Receptors, Cell Surface | Receptors, IgG - biosynthesis | Receptors, IgG - antagonists & inhibitors | Interleukin-10 - physiology | Signal Transduction - immunology | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Signaling Lymphocytic Activation Molecule Family Member 1 | Receptors, Tumor Necrosis Factor - antagonists & inhibitors | RNA, Messenger - antagonists & inhibitors | Receptors, Tumor Necrosis Factor, Type II | Trans-Activators - genetics | Inflammation Mediators - antagonists & inhibitors | Trans-Activators - biosynthesis | Immunoglobulins - biosynthesis | Macrophages - immunology | Inflammation Mediators - immunology | Receptors, Tumor Necrosis Factor - metabolism | Immune Sera - pharmacology | Proteins - physiology | Cells, Cultured | Glycoproteins - antagonists & inhibitors | Histocompatibility Antigens Class II - biosynthesis | Tissue Inhibitor of Metalloproteinase-1 - antagonists & inhibitors | Transcription Factors - antagonists & inhibitors | Transcription Factors - biosynthesis | Up-Regulation - genetics | DNA-Binding Proteins - genetics | DNA - antagonists & inhibitors | Glycoproteins - biosynthesis | Suppressor of Cytokine Signaling 3 Protein | Down-Regulation - immunology | Interleukin-6 - biosynthesis | Receptors, Tumor Necrosis Factor - biosynthesis | STAT3 Transcription Factor | Trans-Activators - antagonists & inhibitors | Genetic Vectors | DNA-Binding Proteins - biosynthesis | Tumor Necrosis Factor-alpha - antagonists & inhibitors | SOCS-3 protein | Index Medicus | Abridged Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2012, Volume 188, Issue 5, pp. 2127 - 2135
It is estimated that 1 billion people around the world are vitamin D deficient. Vitamin D deficiency has been linked to various inflammatory diseases. However,... 
ACTIVATION | GENE | X RECEPTOR | MACROPHAGES | INFLAMMATION | PROSTATE-CANCER | DISEASE | NUCLEAR RECEPTOR SUPERFAMILY | TNF-ALPHA | IMMUNOLOGY | DIFFERENTIAL REGULATION | Dual Specificity Phosphatase 1 - deficiency | Bone Marrow Cells - enzymology | Humans | L Cells (Cell Line) | Male | Monocytes - immunology | Lipopolysaccharides - antagonists & inhibitors | Mice, 129 Strain | MAP Kinase Signaling System - immunology | Phosphorylation - genetics | MAP Kinase Signaling System - genetics | Bone Marrow Cells - immunology | Inflammation Mediators - metabolism | Monocytes - pathology | Adult | Bone Marrow Cells - drug effects | Inflammation Mediators - antagonists & inhibitors | Phosphorylation - immunology | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Macrophages - immunology | Dual Specificity Phosphatase 1 - genetics | Macrophages - pathology | Mice, Inbred C57BL | Cells, Cultured | Vitamin D - pharmacology | Macrophages - enzymology | Mice, Knockout | Monocytes - drug effects | Monocytes - enzymology | Animals | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Lipopolysaccharides - pharmacology | Dual Specificity Phosphatase 1 - metabolism | Macrophages - drug effects | Mice | Cytokines - antagonists & inhibitors | Drug Delivery Systems - methods | Cytokines - biosynthesis | Index Medicus | Abridged Index Medicus
Journal Article
British Journal of Nutrition, ISSN 0007-1145, 2014, Volume 111, Issue 7, pp. 1221 - 1230
Several reports have indicated that dietary intake of DHA is associated with lower prevalence of periodontitis. In the present study, we investigated the... 
Lipopolysaccharide | Proinflammatory mediators | Prevotella intermedia | DHA | INFLAMMATORY DISEASES | ACTIVE RHEUMATOID-ARTHRITIS | PORPHYROMONAS-GINGIVALIS | GINGIVAL CREVICULAR FLUID | ACTINOBACILLUS-ACTINOMYCETEMCOMITANS | NUTRITION & DIETETICS | NITRIC-OXIDE PRODUCTION | POLYUNSATURATED FATTY-ACIDS | STIMULATES RELEASE | DESTRUCTIVE PERIODONTAL-DISEASE | NF-KAPPA-B | Dietary Fats - metabolism | Heme Oxygenase-1 - metabolism | Interleukin-6 - antagonists & inhibitors | Periodontitis - microbiology | Protoporphyrins - pharmacology | NF-kappa B - metabolism | Interleukin-1beta - genetics | Lipopolysaccharides - antagonists & inhibitors | Metalloporphyrins - pharmacology | Periodontitis - metabolism | Heme Oxygenase-1 - genetics | Anti-Inflammatory Agents, Non-Steroidal - antagonists & inhibitors | Docosahexaenoic Acids - antagonists & inhibitors | Interleukin-1beta - metabolism | Nitric Oxide Synthase Type II - antagonists & inhibitors | Inflammation Mediators - metabolism | Inflammation Mediators - antagonists & inhibitors | Membrane Proteins - metabolism | Anti-Inflammatory Agents, Non-Steroidal - metabolism | Interleukin-6 - metabolism | Macrophages - immunology | Interleukin-1beta - antagonists & inhibitors | NF-kappa B - antagonists & inhibitors | Lipopolysaccharides - toxicity | Periodontitis - immunology | Interleukin-6 - genetics | Membrane Proteins - genetics | Nitric Oxide - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Dietary Fats - antagonists & inhibitors | Heme Oxygenase-1 - antagonists & inhibitors | Membrane Proteins - agonists | Periodontitis - prevention & control | Gene Expression Regulation - drug effects | Macrophages - metabolism | Animals | MAP Kinase Signaling System - drug effects | Membrane Proteins - antagonists & inhibitors | Nitric Oxide Synthase Type II - genetics | Prevotella intermedia - metabolism | Macrophages - drug effects | Mice | Lipopolysaccharides - isolation & purification | Macrophage Activation - drug effects | Nitric Oxide - metabolism | Cell Line, Transformed | Docosahexaenoic Acids - metabolism | Nitric Oxide Synthase Type II - metabolism | Pathogens | Immunology | Nutrition | Gum disease | Fatty acids | Rodents | Index Medicus
Journal Article
Journal of Ethnopharmacology, ISSN 0378-8741, 07/2014, Volume 154, Issue 3, pp. 711 - 718
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2006, Volume 281, Issue 33, pp. 23658 - 23667
MAPK-activated protein kinase 2 (MAPKAP K2 or MK2) is one of several kinases directly regulated by p38 MAPK. A role for p38 MAPK in the pathology of Alzheimer... 
TUMOR-NECROSIS-FACTOR | IN-VITRO | BIOCHEMISTRY & MOLECULAR BIOLOGY | MAPKAP KINASE-2 | CENTRAL-NERVOUS-SYSTEM | MICE | AMYLOID PRECURSOR PROTEIN | TNF-ALPHA | P38 KINASE | BRAIN | TAU-HYPERPHOSPHORYLATION | Protein Kinases - metabolism | Neurons - pathology | Phosphorylation | Protein Kinases - genetics | Coculture Techniques | Humans | Protein-Serine-Threonine Kinases | Astrocytes - pathology | Cerebral Cortex - pathology | Astrocytes - enzymology | Lipopolysaccharides - antagonists & inhibitors | Cerebral Cortex - cytology | Alzheimer Disease - pathology | Chemokines - antagonists & inhibitors | Inflammation Mediators - metabolism | Interferon-gamma - antagonists & inhibitors | Inflammation Mediators - antagonists & inhibitors | Protein Kinases - deficiency | p38 Mitogen-Activated Protein Kinases - metabolism | Protein Kinases - biosynthesis | Disease Models, Animal | Astrocytes - cytology | Cell Line | Cerebral Cortex - enzymology | Chemokines - secretion | Microglia - secretion | Mice, Inbred C57BL | Cytokines - secretion | Intracellular Signaling Peptides and Proteins | Microglia - enzymology | Rats | Mice, Transgenic | Alzheimer Disease - enzymology | Down-Regulation - genetics | Mice, Knockout | Amyloid beta-Peptides - antagonists & inhibitors | Animals | Peptide Fragments - antagonists & inhibitors | Neurons - enzymology | Mice | Cytokines - antagonists & inhibitors | Alzheimer Disease - genetics | Index Medicus
Journal Article
The Journal of Immunology, ISSN 0022-1767, 08/2006, Volume 177, Issue 3, pp. 1967 - 1974
Journal Article
Journal Article
The Journal of Immunology, ISSN 0022-1767, 12/2008, Volume 181, Issue 12, pp. 8633 - 8641
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2011, Volume 187, Issue 3, pp. 1157 - 1165
Monocytes attracted by tumor-induced chronic inflammation differentiate to APCs, the type of which depends on cues in the local tumor milieu. In this work, we... 
CANCER PATIENTS | IN-VIVO EXPRESSION | IMMUNE-RESPONSE | SQUAMOUS INTRAEPITHELIAL LESIONS | HUMAN DENDRITIC CELLS | REGULATORY T-CELLS | GENE-EXPRESSION | ANTIGEN-PRESENTING CELLS | TUMOR-CELLS | TNF-ALPHA | IMMUNOLOGY | Dinoprostone - physiology | Human papillomavirus 18 - immunology | Interleukin-6 - antagonists & inhibitors | Coculture Techniques | Humans | Neoplasm Proteins - physiology | Neoplasm Proteins - immunology | Neoplasm Proteins - antagonists & inhibitors | Uterine Cervical Neoplasms - pathology | Antibodies, Blocking - pharmacology | Monocytes - immunology | Human papillomavirus 16 - immunology | Th1 Cells - immunology | Cell Movement - immunology | Papillomavirus Infections - pathology | Th1 Cells - metabolism | Th1 Cells - virology | Interleukin-6 - physiology | Monocytes - pathology | Papillomavirus Infections - immunology | Female | Inflammation Mediators - antagonists & inhibitors | Inflammation Mediators - physiology | Macrophages - immunology | Interleukin-12 - biosynthesis | Inflammation Mediators - immunology | Antigen-Presenting Cells - metabolism | Macrophages - classification | Macrophages - pathology | Macrophage Activation - immunology | Cells, Cultured | Uterine Cervical Neoplasms - prevention & control | Uterine Cervical Neoplasms - immunology | Antigen-Presenting Cells - immunology | Papillomavirus Infections - metabolism | Interleukin-6 - immunology | Cell Line, Tumor | HeLa Cells | Index Medicus | Abridged Index Medicus
Journal Article
British Journal of Nutrition, ISSN 0007-1145, 02/2014, Volume 111, Issue 3, pp. 415 - 423
Dietary redox-active/antioxidant phytochemicals may help control or mitigate the inflammatory response in chronic inflammatory bowel disease (IBD). In the... 
Indicaxanthin | Inflammatory bowel disease | Redox-active phytochemicals | Inflammation | In vitro models | NADPH Oxidases - chemistry | Betaxanthins - isolation & purification | Interleukin-6 - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Enterocytes - metabolism | Antioxidants - metabolism | Humans | Inflammatory Bowel Diseases - immunology | NADPH Oxidases - metabolism | Cell Membrane Permeability | NF-kappa B - metabolism | Inflammatory Bowel Diseases - metabolism | Intestinal Absorption | Enterocytes - secretion | Interleukin-1beta - metabolism | Nitric Oxide Synthase Type II - antagonists & inhibitors | Inflammation Mediators - metabolism | Interleukin-8 - secretion | Opuntia - chemistry | Inflammation Mediators - antagonists & inhibitors | Interleukin-1beta - antagonists & inhibitors | NF-kappa B - agonists | Pyridines - isolation & purification | Pyridines - therapeutic use | Caco-2 Cells | NF-kappa B - antagonists & inhibitors | Fruit - chemistry | Cyclooxygenase 2 - chemistry | NADPH Oxidases - antagonists & inhibitors | Betaxanthins - metabolism | Interleukin-8 - antagonists & inhibitors | Inflammatory Bowel Diseases - diet therapy | Betaxanthins - therapeutic use | Interleukin-6 - secretion | NADPH Oxidase 1 | Enterocytes - immunology | Antioxidants - therapeutic use | Reactive Oxygen Species - antagonists & inhibitors | Pyridines - metabolism | Antioxidants - isolation & purification | Cyclooxygenase 2 - metabolism | Enzyme Activation | Nitric Oxide Synthase Type II - metabolism | Index Medicus
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