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1. Full Text Innate mucosal‐associated invariant T (MAIT) cells are activated in inflammatory bowel diseases
by Serriari, N.‐E and Eoche, M and Lamotte, L and Lion, J and Fumery, M and Marcelo, P and Chatelain, D and Barre, A and Nguyen‐Khac, E and Lantz, O and Dupas, J.‐L and Treiner, E
Clinical & Experimental Immunology, ISSN 0009-9104, 05/2014, Volume 176, Issue 2, pp. 266 - 274
I nflammatory bowel diseases are characterized by a deregulated immune response targeting the gut bacterial flora. M ucosal‐associated invariant T ( MAIT )... 
immune system | MHC | class I‐like | cells | inflammatory bowel diseases | innate | Inflammatory bowel diseases | Innate T cells | MHC class I-like | Immune system | NATURAL-KILLER-CELLS | CYTOKINES | CROHNS-DISEASE | ULCERATIVE-COLITIS | IMMUNOLOGY | SUBSET | NKG2D | innate T cells | INFECTION | DEFINES | EXPRESSION | IMPROVES CYTOTOXICITY | T-Lymphocyte Subsets - immunology | Humans | Inflammatory Bowel Diseases - immunology | Interleukin-17 - immunology | NK Cell Lectin-Like Receptor Subfamily K - immunology | Ki-67 Antigen - metabolism | Male | Colitis, Ulcerative - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Ki-67 Antigen - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Intestinal Mucosa - immunology | Natural Killer T-Cells - metabolism | Inflammatory Bowel Diseases - pathology | Interleukins - immunology | Natural Killer T-Cells - pathology | Adult | Female | Receptors, Immunologic - immunology | Interleukin-17 - blood | Interleukins - blood | Colitis, Ulcerative - pathology | Inflammatory Bowel Diseases - blood | Colitis, Ulcerative - blood | Crohn Disease - immunology | Immunity, Innate - immunology | Microscopy, Confocal | T-Lymphocyte Subsets - pathology | Crohn Disease - pathology | T-Lymphocyte Subsets - metabolism | Crohn Disease - blood | Interferon-gamma - immunology | Interferon-gamma - blood | Receptors, Immunologic - metabolism | Intestinal Mucosa - pathology | Natural Killer T-Cells - immunology | Biological response modifiers | T cells | Analysis | Ulcerative colitis | Medical research | Bowel disease | Original
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2. Full Text Impact of antibodies to infliximab on clinical outcomes and serum infliximab levels in patients with inflammatory bowel disease (IBD): A meta-analysis
by Nanda, Kavinderjit S and Cheifetz, Adam S and Moss, Alan C
American Journal of Gastroenterology, ISSN 0002-9270, 01/2013, Volume 108, Issue 1, pp. 40 - 47
OBJECTIVES : Antibodies to infliximab (ATIs) have been associated with loss of clinical response and lower serum infliximab (IFX) levels in some studies of... 
DOSE INTENSIFICATION | ANTI-TNF | IMMUNOGENICITY | THERAPY | CROHNS-DISEASE | ULCERATIVE-COLITIS | RANDOMIZED-TRIAL | MAINTENANCE INFLIXIMAB | BIOLOGICS | GASTROENTEROLOGY & HEPATOLOGY | ASSOCIATION | Inflammatory Bowel Diseases - drug therapy | Antibodies, Anti-Idiotypic - blood | Humans | Inflammatory Bowel Diseases - immunology | Antibodies, Monoclonal - pharmacokinetics | Anti-Inflammatory Agents, Non-Steroidal - immunology | Antibodies, Monoclonal - therapeutic use | Inflammatory Bowel Diseases - blood | Biomarkers - blood | Anti-Inflammatory Agents, Non-Steroidal - blood | Antibodies, Monoclonal - blood | Infliximab | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Treatment Failure | Anti-Inflammatory Agents, Non-Steroidal - pharmacokinetics | Maintenance Chemotherapy | Antibodies, Monoclonal - immunology
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3. Full Text Proactive therapeutic concentration monitoring of infliximab may improve outcomes for patients with inflammatory bowel disease: Results from a pilot observational study
by Vaughn, Byron P and Martinez-Vazquez, Manuel and Patwardhan, Vilas R and Moss, Alan C and Sandborn, William J and Cheifetz, Adam S
Inflammatory Bowel Diseases, ISSN 1078-0998, 11/2014, Volume 20, Issue 11, pp. 1996 - 2003
Background: Infliximab (IFX) is effective in the treatment of inflammatory bowel disease; however, the effect is often not durable. It is unknown if proactive... 
Inflammatory bowel disease | Therapeutic drug monitoring | Infliximab concentration | Infliximab | Optimized monotherapy | infliximab concentration | CROHNS-DISEASE | infliximab | ULCERATIVE-COLITIS | optimized monotherapy | BIOLOGICS | GASTROENTEROLOGY & HEPATOLOGY | inflammatory bowel disease | therapeutic drug monitoring | MAINTENANCE THERAPY | Inflammatory Bowel Diseases - drug therapy | Prognosis | Follow-Up Studies | Humans | Inflammatory Bowel Diseases - immunology | Middle Aged | Antibodies, Monoclonal - pharmacokinetics | Antibodies, Monoclonal - therapeutic use | Male | Inflammatory Bowel Diseases - blood | Case-Control Studies | Tissue Distribution | Pilot Projects | Gastrointestinal Agents - therapeutic use | Adult | Female | Retrospective Studies | Drug Monitoring | Gastrointestinal Agents - pharmacokinetics
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4. Full Text Circulating CD4+ and CD8+ T cells are activated in inflammatory bowel disease and are associated with plasma markers of inflammation
by Funderburg, Nicholas T and Stubblefield Park, Samantha R and Sung, Hannah C and Hardy, Gareth and Clagett, Brian and Ignatz‐Hoover, James and Harding, Clifford V and Fu, Pingfu and Katz, Jeffry A and Lederman, Michael M and Levine, Alan D
Immunology, ISSN 0019-2805, 09/2013, Volume 140, Issue 1, pp. 87 - 97
Inflammatory bowel disease ( IBD ) is characterized by damage to the gut mucosa and systemic inflammation. We sought to evaluate the role of chronic... 
microbial translocation | T‐cell activation | inflammatory bowel disease | Inflammatory bowel disease | Microbial translocation | T-cell activation | PROTEIN | CROHNS-DISEASE | PERMEABILITY | IMMUNE RECONSTITUTION | ULCERATIVE-COLITIS | IMMUNOLOGY | TNF | PATHOGENESIS | BIOMARKERS | PREDICTS PROGNOSIS | EXPRESSION | Humans | Inflammatory Bowel Diseases - immunology | Middle Aged | HLA-DR Antigens - blood | Male | Colitis, Ulcerative - immunology | ADP-ribosyl Cyclase 1 - blood | Case-Control Studies | CD4-Positive T-Lymphocytes - immunology | Lipopolysaccharides - blood | C-Reactive Protein - metabolism | Interleukin-6 - blood | Adult | Female | Lymphocyte Activation | Inflammation Mediators - blood | Membrane Glycoproteins - blood | Inflammatory Bowel Diseases - blood | Biomarkers - blood | Colitis, Ulcerative - blood | Crohn Disease - immunology | Crohn Disease - blood | Inflammatory Bowel Diseases - microbiology | CD8-Positive T-Lymphocytes - immunology | Interferon-gamma - blood | Cohort Studies | Interleukin-2 - blood | Gastrointestinal system | Inflammation | Biological response modifiers | Binding proteins | T cells | Mitogens | Ulcerative colitis | Protein binding | Proteins | Plasma | Lymphocytes | Rodents | Original
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5. Full Text The association of tissue anti-TNF drug levels with serological and endoscopic disease activity in inflammatory bowel disease: The ATLAS study
by Yarur, Andres J and Jain, Anjali and Sussman, Daniel A and Barkin, Jamie S and Quintero, Maria A and Princen, Fred and Kirkland, Richard and Deshpande, Amar R and Singh, Sharat and Abreu, Maria T
Gut, ISSN 0017-5749, 02/2016, Volume 65, Issue 2, pp. 249 - 255
Objective The aim of this study was to assess the correlation between serum and intestinal anti-tumour necrosis factor (TNF) levels, and their relationship to... 
MAINTENANCE | TUMOR-NECROSIS-FACTOR | CROHNS-DISEASE | INDUCTION THERAPY | ADALIMUMAB | ULCERATIVE-COLITIS | RANDOMIZED-TRIAL | INFLIXIMAB | GASTROENTEROLOGY & HEPATOLOGY | FACTOR-ALPHA | REMISSION | Inflammatory Bowel Diseases - drug therapy | Cross-Sectional Studies | Infliximab - analysis | Adalimumab - blood | Humans | Tumor Necrosis Factor-alpha - blood | Inflammatory Bowel Diseases - blood | Infliximab - blood | Intestinal Mucosa - chemistry | Adalimumab - analysis | Tumor Necrosis Factor-alpha - analysis | Inflammatory bowel diseases | Care and treatment | Dosage and administration | Adalimumab | Tumor necrosis factor | Inflammatory bowel disease | Studies | Immunoglobulins | Gangrene | Tumor necrosis factor-TNF | Inflammation | Clinical medicine | Endoscopy | Drug dosages | Patients | Crohns disease
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6. Full Text Inflammatory bowel disease and thromboembolism
by Petros Zezos Georgios Kouklakis Fred Saibil
世界胃肠病学杂志：英文版（电子版）, ISSN 1007-9327, 2014, Volume 20, Issue 38, pp. 13863 - 13878
Patients with inflammatory bowel disease(IBD) have an increased risk of vascular complications. Thromboembolic complications,both venous and arterial,are... 
Inflammatory | bowel | Crohn’s | disease | Ulcera | dis-ease | Hypercoagulability | Inflammatory bowel disease | Treatment | Endothelial dysfunction | Crohn's disease | Thromboembolism | Epidemiology | Thrombosis | Ulcerative colitis | RISK-FACTORS | CROHNS-DISEASE | CAUSE-SPECIFIC MORTALITY | ISCHEMIC-HEART-DISEASE | VON-WILLEBRAND-FACTOR | MUCOSAL MICROCIRCULATION | DEEP VENOUS THROMBOSIS | MOLECULAR-WEIGHT HEPARIN | ACTIVE ULCERATIVE-COLITIS | PROTEIN-S DEFICIENCY | GASTROENTEROLOGY & HEPATOLOGY | Prognosis | Risk Assessment | Humans | Risk Factors | Inflammation Mediators - blood | Inflammatory Bowel Diseases - physiopathology | Inflammatory Bowel Diseases - blood | Thromboembolism - epidemiology | Inflammatory Bowel Diseases - epidemiology | Thromboembolism - blood | Thromboembolism - prevention & control | Blood Coagulation | Thromboembolism - physiopathology | Inflammatory Bowel Diseases - therapy | Review | Crohn’s disease
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7. Full Text Concentrations of Adalimumab and Infliximab in Mothers and Newborns, and Effects on Infection
by Julsgaard, Mette and Christensen, Lisbet A and Gibson, Peter R and Gearry, Richard B and Fallingborg, Jan and Hvas, Christian L and Bibby, Bo M and Uldbjerg, Niels and Connell, William R and Rosella, Ourania and Grosen, Anne and Brown, Steven J and Kjeldsen, Jens and Wildt, Signe and Svenningsen, Lise and Sparrow, Miles P and Walsh, Alissa and Connor, Susan J and Radford-Smith, Graham and Lawrance, Ian C and Andrews, Jane M and Ellard, Kathrine and Bell, Sally J
Gastroenterology, ISSN 0016-5085, 2016, Volume 151, Issue 1, pp. 110 - 119
Background & Aims Little is known about in utero exposure to and postnatal clearance of anti–tumor necrosis factor (anti-TNF) agents in neonates. We... 
Gastroenterology and Hepatology | Safety | Inflammatory Bowel Diseases | Vaccination | ERA Study | ANTIBODIES | MANAGEMENT | IMMUNOGLOBULINS | CONSENSUS | PREGNANCY | WOMEN | TRANSPORT | INFLAMMATORY-BOWEL-DISEASE | GASTROENTEROLOGY & HEPATOLOGY | NURSING MOTHERS | Inflammatory Bowel Diseases - drug therapy | Pregnancy Complications - blood | Prospective Studies | Maternal-Fetal Exchange | Humans | Inflammatory Bowel Diseases - blood | Fetal Blood - metabolism | Mothers | Pregnancy Complications - drug therapy | New Zealand | Pregnancy | Adalimumab - blood | Denmark | Adult | Female | Gastrointestinal Agents - blood | Infliximab - blood | Australia | Infant, Newborn | Infants (Newborn) | Infliximab | Health aspects
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8. Full Text Chemokine and cytokine levels in inflammatory bowel disease patients
by Singh, Udai P and Singh, Narendra P and Murphy, E. Angela and Price, Robert L and Fayad, Raja and Nagarkatti, Mitzi and Nagarkatti, Prakash S
Cytokine, ISSN 1043-4666, 01/2016, Volume 77, pp. 44 - 49
Crohn’s disease (CD) and ulcerative colitis (UC), two forms of inflammatory bowel disease (IBD), are chronic, relapsing, and tissue destructive lesions that... 
Chemokine | Inflammation | Inflammatory bowel disease (IBD) | Crohn’s disease (CD) | Ulcerative colitis (UC) | Cytokine | Crohn's disease (CD) | DEXTRAN SULFATE SODIUM | IL-10(-/-) MICE | CROHNS-DISEASE | INCREASED EXPRESSION | BIOCHEMISTRY & MOLECULAR BIOLOGY | ULCERATIVE-COLITIS | IMMUNOLOGY | CELL BIOLOGY | INTESTINAL INFLAMMATION | EXPERIMENTAL COLITIS | MONOCYTE-CHEMOATTRACTANT PROTEIN-1 | RECEPTORS | T-CELLS | Chemokines - blood | T-Lymphocytes, Helper-Inducer - metabolism | Enzyme-Linked Immunosorbent Assay | Humans | Middle Aged | Inflammation Mediators - blood | Male | Inflammatory Bowel Diseases - blood | Colitis, Ulcerative - blood | Macrophages - metabolism | Crohn Disease - blood | Adult | Female | Aged | Neutrophils - metabolism | Cytokines - blood | Cytokines | Gastrointestinal diseases
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9. Full Text Peripheral and intestinal regulatory CD4+CD25(high) T cells in inflammatory bowel disease
by Maul, J and Loddenkemper, C and Mundt, P and Berg, E and Giese, T and Stallmach, A and Zeitz, M and Duchmann, R
GASTROENTEROLOGY, ISSN 0016-5085, 06/2005, Volume 128, Issue 7, pp. 1868 - 1878
Background & Aims: Regulatory CD25+ T cells (T-reg) are effective in the prevention and down-regulation of inflammatory bowel disease (IBD) in animal models.... 
IN-VITRO | ACTIVATION | CROHNS-DISEASE | CD4(+)CD25(+) | TOLERANCE | ULCERATIVE-COLITIS | FUNCTIONAL-CHARACTERIZATION | LAMINA PROPRIA | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | BLOOD | Down-Regulation | Humans | Inflammatory Bowel Diseases - immunology | Middle Aged | Male | Inflammatory Bowel Diseases - blood | Case-Control Studies | CD4-Positive T-Lymphocytes - immunology | Flow Cytometry | Biopsy | Intestinal Mucosa - immunology | Inflammatory Bowel Diseases - pathology | Adolescent | Adult | Female | Aged | Receptors, Interleukin-2 - analysis
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10. Full Text Expression of CCR6 and CXCR6 by Gut-Derived CD4+/CD8α+ T-Regulatory Cells, Which Are Decreased in Blood Samples From Patients With Inflammatory Bowel Diseases
by Godefroy, Emmanuelle and Alameddine, Joudy and Montassier, Emmanuel and Mathé, Justine and Desfrançois-Noël, Juliette and Marec, Nadège and Bossard, Céline and Jarry, Anne and Bridonneau, Chantal and Le Roy, Amandine and Sarrabayrouse, Guillaume and Kerdreux, Elise and Bourreille, Arnaud and Sokol, Harry and Jotereau, Francine and Altare, Frédéric
Gastroenterology, ISSN 0016-5085, 10/2018, Volume 155, Issue 4, pp. 1205 - 1217
BACKGROUND & AIMS: Faecalibacterium prausnitzii, a member of the Clostridium IV group of the Firmicutes phylum that is abundant in the intestinal microbiota,... 
Immune Regulation | Bacteria | Microbiota | T-Cell Response | IMMUNITY | INDUCE | MICROBIOME | FAECALIBACTERIUM-PRAUSNITZII | PATHOGENESIS | RESPONSES | COLITIS | MICE | HEALTH | GASTROENTEROLOGY & HEPATOLOGY | Receptors, CCR6 - immunology | T-Lymphocytes, Regulatory - metabolism | Intestinal Mucosa - metabolism | Cell Proliferation | Humans | Inflammatory Bowel Diseases - immunology | Colon - immunology | Case-Control Studies | T-Lymphocytes, Regulatory - immunology | Receptors, CXCR6 - immunology | Intestinal Mucosa - immunology | Inflammatory Bowel Diseases - pathology | CD8-Positive T-Lymphocytes - metabolism | Receptors, CCR6 - blood | T-Lymphocytes, Regulatory - microbiology | CD8-Positive T-Lymphocytes - microbiology | Colon - pathology | Lymphocyte Activation | Cells, Cultured | Inflammatory Bowel Diseases - blood | Intestinal Mucosa - microbiology | Colon - metabolism | Faecalibacterium prausnitzii - immunology | Phenotype | Receptors, CXCR6 - blood | Inflammatory Bowel Diseases - microbiology | Colon - microbiology | CD8-Positive T-Lymphocytes - immunology | Intestinal Mucosa - pathology | Life Sciences | Cancer
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11. Full Text FERGIcor, a Randomized Controlled Trial on Ferric Carboxymaltose for Iron Deficiency Anemia in Inflammatory Bowel Disease
by Evstatiev, Rayko and Marteau, Philippe and Iqbal, Tariq and Khalif, Igor L and Stein, Jürgen and Bokemeyer, Bernd and Chopey, Ivan V and Gutzwiller, Florian S and Riopel, Lise and Gasche, Christoph and FERGI Study Grp and FERGI Study Group
Gastroenterology, ISSN 0016-5085, 2011, Volume 141, Issue 3, pp. 846 - 853.e2
Background & Aims Iron deficiency anemia (IDA) is common in chronic diseases and intravenous iron is an effective and recommended treatment. However, dose... 
Gastroenterology and Hepatology | Crohn's Disease | Ulcerative Colitis | Intravenous Iron | ERYTHROPOIETIN | MANAGEMENT | CROHNS-DISEASE | EFFICACY | SUPPLEMENTATION | ORAL IRON | SUCROSE | QUALITY-OF-LIFE | THROMBOCYTOSIS | GASTROENTEROLOGY & HEPATOLOGY | Maltose - administration & dosage | Anemia, Iron-Deficiency - drug therapy | Outcome Assessment (Health Care) | Humans | Middle Aged | Ferric Compounds - administration & dosage | Male | Anemia, Iron-Deficiency - blood | Dose-Response Relationship, Drug | Young Adult | Inflammatory Bowel Diseases - epidemiology | Aged, 80 and over | Adult | Female | Maltose - therapeutic use | Inflammatory Bowel Diseases - drug therapy | Maltose - adverse effects | Comorbidity | Hemoglobins - metabolism | Treatment Outcome | Inflammatory Bowel Diseases - blood | Anemia, Iron-Deficiency - epidemiology | Maltose - analogs & derivatives | Glucaric Acid | Adolescent | Ferric Compounds - therapeutic use | Aged | Ferric Compounds - adverse effects | Infusions, Intravenous | Prevention | Medical colleges | Safety regulations | Transferrin | Analysis | Iron deficiency anemia | Glycosylated hemoglobin | Chronic diseases | Crohn's disease | Ulcerative colitis | Cancer
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12. Full Text Pretreatment 25-Hydroxyvitamin D Levels and Durability of Anti–Tumor Necrosis Factor–α Therapy in Inflammatory Bowel Diseases
by Zator, Zachary A and Cantu, Stephanie M and Konijeti, Gauree Gupta and Nguyen, Deanna D and Sauk, Jenny and Yajnik, Vijay and Ananthakrishnan, Ashwin N
Journal of Parenteral and Enteral Nutrition, ISSN 0148-6071, 3/2014, Volume 38, Issue 3, pp. 385 - 391
Introduction: Emerging evidence supports an immunologic role for 25-hydroxyvitamin D (25(OH)D) in inflammatory bowel disease (IBD). Here we examined if... 
Crohn's disease | ulcerative colitis | biologic therapy | vitamin D | SURGERY | CROHNS-DISEASE | REDUCED RISK | ANTI-TNF THERAPY | IBD | VITAMIN-D DEFICIENCY | NUTRITION & DIETETICS | COLITIS | D-RECEPTOR | ASSOCIATION | Inflammatory Bowel Diseases - drug therapy | Follow-Up Studies | Vitamin D Deficiency - complications | Humans | Proportional Hazards Models | Male | Inflammatory Bowel Diseases - blood | Vitamin D - blood | Young Adult | Vitamin D Deficiency - blood | Sensitivity and Specificity | Adolescent | Adult | Female | Vitamin D - analogs & derivatives | Retrospective Studies | Inflammatory Bowel Diseases - complications | Dietary Supplements | Tumor Necrosis Factor-alpha - antagonists & inhibitors
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