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by Jostins, Luke and Ripke, Stephan and Weersma, Rinse K and Duerr, Richard H and McGovern, Dermot P and Hui, Ken Y and Lee, James C and Schumm, L. Philip and Sharma, Yashoda and Anderson, Carl A and Essers, Jonah and Mitrovic, Mitja and Ning, Kaida and Cleynen, Isabelle and Theatre, Emilie and Spain, Sarah L and Raychaudhuri, Soumya and Goyette, Philippe and Wei, Zhi and Abraham, Clara and Achkar, Jean-Paul and Ahmad, Tariq and Amininejad, Leila and Ananthakrishnan, Ashwin N and Andersen, Vibeke and Anews, Jane M and Baidoo, Leonard and Balschun, Tobias and Bampton, Peter A and Bitton, Alain and Boucher, Gabrielle and Brand, Stephan and Büning, Carsten and Cohain, Ariella and Cichon, Sven and D'Amato, Mauro and de Jong, Dirk and Devaney, Kathy L and Dubinsky, Marla and Edwards, Cathryn and Ellinghaus, David and Ferguson, Lynnette R and Franchimont, Denis and Fransen, Karin and Gearry, Richard and Georges, Michel and Gieger, Christian and Glas, Jürgen and Haritunians, Talin and Hart, Ailsa and Hawkey, Chris and Hedl, Matija and Hu, Xinli and Karlsen, Tom H and Kupcinskas, Limas and Kugathasan, Subra and Latiano, Anna and Laukens, Debby and Lawrance, Ian C and Lees, Charlie W and Louis, Edouard and Mahy, Gillian and Mansfield, John and Morgan, Angharad R and Mowat, Craig and Newman, William and Palmieri, Orazio and Ponsioen, Cyriel Y and Potocnik, Uros and Prescott, Natalie J and Regueiro, Miguel and Rotter, Jerome I and Russell, Richard K and Sanderson, Jeremy D and Sans, Miquel and Satsangi, Jack and Schreiber, Stefan and Simms, Lisa A and Sventoraityte, Jurgita and Targan, Stephan R and Taylor, Kent D and Tremelling, Mark and Verspaget, Hein W and de Vos, Martine and Wijmenga, Cisca and Wilson, David C and Winkelmann, Juliane and Xavier, Ramnik J and Zeissig, Sebastian and Zhang, Bin and Zhang, Clarence K and Zhao, Hongyu and Silverberg, Mark S and Annese, Vito and Hakonarson, Hakon and Brant, Steven R and Radford-Smith, Graham and Mathew, Christopher G and Rioux, John D and Schadt, Eric E and ... and Int IBD Genetics Consortium IIBDGC and International IBD Genetics Consortium (IIBDGC) and The International IBD Genetics Consortium (IIBDGC) and Region Örebro län and Örebro universitet and Institutionen för medicinska vetenskaper
Nature, ISSN 0028-0836, 2012, Volume 491, Issue 7422, pp. 119 - 124
Crohn's disease and ulcerative colitis, the two common forms of inflammatory bowel disease (IBD), affect over 2.5 million people of European ancestry, with... 
HYPER-IGE SYNDROME | RISK LOCI | NETWORK | NUMBER | METAANALYSIS | TUBERCULOSIS | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | MUTATIONS | EXPRESSION | Crohn Disease - genetics | Humans | Inflammatory Bowel Diseases - immunology | Colitis, Ulcerative - genetics | Mycobacterium tuberculosis - immunology | Inflammatory Bowel Diseases - physiopathology | Colitis, Ulcerative - immunology | Host-Pathogen Interactions - immunology | Mycobacterium tuberculosis - pathogenicity | Haplotypes - genetics | Mycobacterium - pathogenicity | Mycobacterium Infections - genetics | Inflammatory Bowel Diseases - genetics | Crohn Disease - microbiology | Mycobacterium Infections - microbiology | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Reproducibility of Results | Mycobacterium - immunology | Crohn Disease - immunology | Genome, Human - genetics | Phenotype | Colitis, Ulcerative - microbiology | Host-Pathogen Interactions - genetics | Polymorphism, Single Nucleotide - genetics | Inflammatory Bowel Diseases - microbiology | Crohn Disease - physiopathology | Colitis, Ulcerative - physiopathology | Inflammatory bowel disease | Candidates | Tuberculosis | Architecture | Genetics | Genomes | Diabetes | Gene expression | Health risk assessment | Immune system | Index Medicus | Rheumatology and Autoimmunity | Basic Medicine | Medical Genetics | Medicinsk genetik | Clinical Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Reumatologi och inflammation | Medicinska och farmaceutiska grundvetenskaper | Klinisk medicin
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 05/2014, Volume 176, Issue 2, pp. 266 - 274
I nflammatory bowel diseases are characterized by a deregulated immune response targeting the gut bacterial flora. M ucosal‐associated invariant T ( MAIT )... 
immune system | MHC | class I‐like | cells | inflammatory bowel diseases | innate | Inflammatory bowel diseases | Innate T cells | MHC class I-like | Immune system | NATURAL-KILLER-CELLS | CYTOKINES | CROHNS-DISEASE | ULCERATIVE-COLITIS | IMMUNOLOGY | SUBSET | NKG2D | innate T cells | INFECTION | DEFINES | EXPRESSION | IMPROVES CYTOTOXICITY | T-Lymphocyte Subsets - immunology | Humans | Inflammatory Bowel Diseases - immunology | Interleukin-17 - immunology | NK Cell Lectin-Like Receptor Subfamily K - immunology | Ki-67 Antigen - metabolism | Male | Colitis, Ulcerative - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Ki-67 Antigen - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Intestinal Mucosa - immunology | Natural Killer T-Cells - metabolism | Inflammatory Bowel Diseases - pathology | Interleukins - immunology | Natural Killer T-Cells - pathology | Adult | Female | Receptors, Immunologic - immunology | Interleukin-17 - blood | Interleukins - blood | Colitis, Ulcerative - pathology | Inflammatory Bowel Diseases - blood | Colitis, Ulcerative - blood | Crohn Disease - immunology | Immunity, Innate - immunology | Microscopy, Confocal | T-Lymphocyte Subsets - pathology | Crohn Disease - pathology | T-Lymphocyte Subsets - metabolism | Crohn Disease - blood | Interferon-gamma - immunology | Interferon-gamma - blood | Receptors, Immunologic - metabolism | Intestinal Mucosa - pathology | Natural Killer T-Cells - immunology | Biological response modifiers | T cells | Analysis | Ulcerative colitis | Medical research | Bowel disease | Index Medicus | Original
Journal Article
Gut, ISSN 0017-5749, 04/2014, Volume 63, Issue 4, pp. 567 - 577
Background We analysed incidence, predictors, histological features and specific treatment options of anti-tumour necrosis factor α (TNF-α) antibody-induced... 
RHEUMATOID-ARTHRITIS | ULCERATIVE-COLITIS | DOUBLE-BLIND | PROINFLAMMATORY GENE-EXPRESSION | ACTIVE CROHNS-DISEASE | NECROSIS FACTOR THERAPY | GASTROENTEROLOGY & HEPATOLOGY | MAINTENANCE THERAPY | HUMAN KERATINOCYTES | INTERLEUKIN-12/23 MONOCLONAL-ANTIBODY | GENOME-WIDE ASSOCIATION | Th1 Cells - physiology | Prospective Studies | Humans | Inflammatory Bowel Diseases - immunology | Interleukin-12 - physiology | Interleukin-17 - immunology | Interleukin-17 - physiology | Psoriasis - etiology | Ustekinumab | Inflammatory Bowel Diseases - physiopathology | Male | Th1 Cells - immunology | Interleukins - physiology | Inflammatory Bowel Diseases - pathology | Interleukins - immunology | Tumor Necrosis Factor-alpha - immunology | Antibodies - immunology | Adult | Female | Inflammatory Bowel Diseases - complications | Interleukin-23 - physiology | Skin - pathology | Skin - immunology | Psoriasis - immunology | Antibodies - therapeutic use | Antibodies, Monoclonal, Humanized - therapeutic use | Interferon-gamma - physiology | Interleukin-23 - immunology | Psoriasis - physiopathology | Interferon-gamma - immunology | Interleukin-12 - immunology | Tumor Necrosis Factor-alpha - physiology | Skin - physiopathology | Antibodies, Monoclonal, Humanized - immunology | Complications and side effects | Inflammatory bowel diseases | Care and treatment | Psoriasis | Tumor necrosis factor | Influence | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Science, ISSN 0036-8075, 05/2016, Volume 352, Issue 6289, pp. 1116 - 1120
Journal Article
Science, ISSN 0036-8075, 2/2003, Volume 299, Issue 5609, pp. 1057 - 1061
Regulatory T cells engage in the maintenance of immunological self-tolerance by actively suppressing self-reactive lymphocytes. Little is known, however, about... 
T lymphocytes | Molecules | Cell growth | Inflammatory bowel diseases | B lymphocytes | Antigen presenting cells | Cultured cells | Reports | Autoimmune diseases | Cells | Gastritis | MUTANT | HOMEOSTASIS | LYMPHOCYTE-ASSOCIATED ANTIGEN-4 | ENTEROPATHY | CD25(+) | MULTIDISCIPLINARY SCIENCES | IMMUNOLOGICAL SELF-TOLERANCE | INDUCTION | AUTOIMMUNE-DISEASE | SCURFY MOUSE | T-Lymphocyte Subsets - immunology | T-Lymphocytes, Regulatory - metabolism | T-Lymphocyte Subsets - cytology | Inflammatory Bowel Diseases - immunology | Gastritis - prevention & control | Recombinant Fusion Proteins - metabolism | T-Lymphocytes, Regulatory - immunology | Antigens, CD - analysis | CD4-Positive T-Lymphocytes - immunology | DNA-Binding Proteins - metabolism | Receptors, Antigen, T-Cell - immunology | Thymus Gland - metabolism | Inflammatory Bowel Diseases - prevention & control | Transduction, Genetic | Lymphocyte Activation | Autoimmune Diseases - immunology | Self Tolerance | Thymus Gland - cytology | Immune Tolerance | Autoimmune Diseases - prevention & control | Mice, Transgenic | DNA-Binding Proteins - genetics | Mice, SCID | Animals | Gastritis - immunology | T-Lymphocytes - immunology | Mice | Mice, Inbred BALB C | Mutation | Forkhead Transcription Factors | Cytokines - biosynthesis | Receptors, Interleukin-2 - analysis | Genetics | Control | Genetic aspects | Research | T cells | Cellular control mechanisms | Immunology | Index Medicus
Journal Article
Immunity, ISSN 1074-7613, 2008, Volume 29, Issue 6, pp. 947 - 957
Journal Article