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World journal of gastroenterology : WJG, ISSN 1007-9327, 2016, Volume 22, Issue 11, pp. 3117 - 3126
Journal Article
Clinical and experimental immunology, ISSN 0009-9104, 2014, Volume 176, Issue 2, pp. 266 - 274
Summary Inflammatory bowel diseases are characterized by a deregulated immune response targeting the gut bacterial flora. Mucosal... 
immune system | MHC class I‐like | innate T cells | inflammatory bowel diseases | Inflammatory bowel diseases | Innate T cells | MHC class I-like | Immune system | NATURAL-KILLER-CELLS | CYTOKINES | CROHNS-DISEASE | ULCERATIVE-COLITIS | IMMUNOLOGY | SUBSET | NKG2D | INFECTION | DEFINES | EXPRESSION | IMPROVES CYTOTOXICITY | T-Lymphocyte Subsets - immunology | Humans | Inflammatory Bowel Diseases - immunology | Interleukin-17 - immunology | NK Cell Lectin-Like Receptor Subfamily K - immunology | Ki-67 Antigen - metabolism | Male | Colitis, Ulcerative - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Ki-67 Antigen - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Intestinal Mucosa - immunology | Natural Killer T-Cells - metabolism | Inflammatory Bowel Diseases - pathology | Interleukins - immunology | Natural Killer T-Cells - pathology | Adult | Female | Receptors, Immunologic - immunology | Interleukin-17 - blood | Interleukins - blood | Colitis, Ulcerative - pathology | Inflammatory Bowel Diseases - blood | Colitis, Ulcerative - blood | Crohn Disease - immunology | Immunity, Innate - immunology | Microscopy, Confocal | T-Lymphocyte Subsets - pathology | Crohn Disease - pathology | T-Lymphocyte Subsets - metabolism | Crohn Disease - blood | Interferon-gamma - immunology | Interferon-gamma - blood | Receptors, Immunologic - metabolism | Intestinal Mucosa - pathology | Natural Killer T-Cells - immunology | Biological response modifiers | T cells | Analysis | Ulcerative colitis | Medical research | Bowel disease | Original
Journal Article
Clinical and experimental immunology, ISSN 0009-9104, 2013, Volume 172, Issue 3, pp. 410 - 416
Summary Interleukin (IL)‐37 is a member of the IL‐1 cytokine family. We investigated IL‐37b expression in the inflamed mucosa of inflammatory bowel disease... 
innate immunity | TNF‐α | anti‐inflammatory cytokine | TNF-α | Innate immunity | Anti-inflammatory cytokine | CROHNS-DISEASE | TNF-alpha | IL-1 FAMILY | KAPPA-B | IMMUNOLOGY | PROTEIN-KINASE KINASE | IDENTIFICATION | COLONIC SUBEPITHELIAL MYOFIBROBLASTS | PATHOGENESIS | OVEREXPRESSION | INHIBITOR | anti-inflammatory cytokine | CELL | Crohn Disease - genetics | Interleukin-1 - genetics | Intestinal Mucosa - metabolism | Gene Expression - drug effects | Myofibroblasts - immunology | Humans | Inflammatory Bowel Diseases - immunology | Colitis, Ulcerative - genetics | Crohn Disease - metabolism | NF-kappa B - metabolism | Phosphatidylinositol 3-Kinases - metabolism | RNA, Messenger - metabolism | Colitis, Ulcerative - immunology | Inflammatory Bowel Diseases - metabolism | Transcription Factor AP-1 - metabolism | Case-Control Studies | Intestinal Mucosa - drug effects | MAP Kinase Signaling System | Myofibroblasts - metabolism | Intestinal Mucosa - immunology | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Adaptive Immunity | Caco-2 Cells | Interleukin-1 - metabolism | Myofibroblasts - pathology | Colitis, Ulcerative - metabolism | RNA, Messenger - genetics | Cells, Cultured | Colitis, Ulcerative - pathology | Immunity, Innate | Myofibroblasts - drug effects | Crohn Disease - immunology | Tumor Necrosis Factor-alpha - pharmacology | Chemokine CXCL10 - genetics | Crohn Disease - pathology | Chemokine CXCL10 - metabolism | Intestinal Mucosa - pathology | Interleukins | Biological response modifiers | Gastrointestinal diseases | Inflammatory bowel disease | Proteins | Colon | Cytokines | Original
Journal Article
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2017, Volume 152, Issue 2, pp. 327 - 339.e4
Journal Article
Journal Article
Nature communications, ISSN 2041-1723, 2018, Volume 9, Issue 1, pp. 3728 - 11
...ARTICLE NLRP1 restricts butyrate producing commensals to exacerbate inflammatory bowel disease Hazel Tye1,2, Chien-Hsiung Yu1,2, Lisa A. Simms3, Marcel R. de... 
Inflammasomes | Signal Transduction | Humans | Mice, Inbred C57BL | Gastrointestinal Microbiome | Clostridiales | Interferon-gamma/metabolism | Male | Mice, Transgenic | Adaptor Proteins, Signal Transducing/genetics | Colon/pathology | Animals | Gene Deletion | Intestinal Mucosa/metabolism | T-Lymphocytes/cytology | Vancomycin/pharmacology | Rectum/metabolism | Apoptosis Regulatory Proteins/genetics | Female | Interleukin-18/metabolism | Mice | Colitis/metabolism | Inflammatory Bowel Diseases/drug therapy | Butyrates/metabolism | ACTIVATION | INTERLEUKIN-18 | CROHNS-DISEASE | COLITIS | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | MICROBIOTA | POLYMORPHISM | PYROPTOSIS | SEVERITY | Butyrates - metabolism | Intestinal Mucosa - metabolism | Interferon-gamma - metabolism | Inflammatory Bowel Diseases - metabolism | Vancomycin - pharmacology | Apoptosis Regulatory Proteins - genetics | Inflammatory Bowel Diseases - drug therapy | Colon - pathology | Rectum - metabolism | Apoptosis Regulatory Proteins - metabolism | T-Lymphocytes - cytology | Adaptor Proteins, Signal Transducing - genetics | Colitis - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Interleukin-18 - metabolism | Inflammatory bowel diseases | Commensals | Mucosa | Vancomycin | Immunity | Antiinfectives and antibacterials | Interleukin 18 | Inflammatory bowel disease | Receptors | Microorganisms | Intestine | γ-Interferon | Rectum | Colon | Supplementation | Ulcerative colitis
Journal Article
Nature (London), ISSN 1476-4687, 2019, Volume 567, Issue 7746, pp. 49 - 55
.... Breakdown of the epithelial barrier underpins inflammatory bowel disease (IBD). However, the specific contributions of each epithelial-cell subtype to this process are unknown... 
STEM-CELLS | DENDRITIC CELLS | MUCIN | RNA-SEQ | MULTIDISCIPLINARY SCIENCES | GENES | TISSUES | EXPRESSION | GENOME-WIDE ASSOCIATION | MUCUS LAYERS | RESOURCE | Colon - cytology | Intestinal Mucosa - metabolism | WAP Four-Disulfide Core Domain Protein 2 | Humans | Colitis, Ulcerative - genetics | Male | Stem Cells - cytology | Stem Cells - metabolism | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Transcription, Genetic | Epithelial Cells - cytology | Single-Cell Analysis | Tight Junctions - metabolism | Genetic Predisposition to Disease - genetics | Natriuretic Peptides - metabolism | Colon - pathology | Health | Biomarkers - analysis | Epithelial Cells - classification | Epithelial Cells - pathology | Colitis, Ulcerative - pathology | Intestinal Mucosa - microbiology | Goblet Cells - metabolism | Animals | Proteins - metabolism | Colitis, Ulcerative - microbiology | Ion Channels - metabolism | Epithelial Cells - microbiology | Stem Cells - pathology | Inflammatory Bowel Diseases - microbiology | Colon - microbiology | Mice | Goblet Cells - pathology | Goblet Cells - cytology | Hydrogen-Ion Concentration | Intestinal Mucosa - pathology | Physiological aspects | Epithelial cells | Identification and classification | Gastrointestinal diseases | Inflammatory bowel diseases | Mucosal immunity | Goblet cells | Transcription | Tight junctions | Mucus | Absorptivity | Uroguanylin | Epithelium | Disease control | Immunity | Progenitor cells | Mucous membrane | Inflammatory bowel disease | Intestine | Stem cells | Cells (biology) | Satiety | Breakdown | Colon | Cancer
Journal Article