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Clinical and experimental immunology, ISSN 0009-9104, 2014, Volume 176, Issue 2, pp. 266 - 274
Summary Inflammatory bowel diseases are characterized by a deregulated immune response targeting the gut bacterial flora. Mucosal... 
immune system | MHC class I‐like | innate T cells | inflammatory bowel diseases | Inflammatory bowel diseases | Innate T cells | MHC class I-like | Immune system | NATURAL-KILLER-CELLS | CYTOKINES | CROHNS-DISEASE | ULCERATIVE-COLITIS | IMMUNOLOGY | SUBSET | NKG2D | INFECTION | DEFINES | EXPRESSION | IMPROVES CYTOTOXICITY | T-Lymphocyte Subsets - immunology | Humans | Inflammatory Bowel Diseases - immunology | Interleukin-17 - immunology | NK Cell Lectin-Like Receptor Subfamily K - immunology | Ki-67 Antigen - metabolism | Male | Colitis, Ulcerative - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Ki-67 Antigen - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Intestinal Mucosa - immunology | Natural Killer T-Cells - metabolism | Inflammatory Bowel Diseases - pathology | Interleukins - immunology | Natural Killer T-Cells - pathology | Adult | Female | Receptors, Immunologic - immunology | Interleukin-17 - blood | Interleukins - blood | Colitis, Ulcerative - pathology | Inflammatory Bowel Diseases - blood | Colitis, Ulcerative - blood | Crohn Disease - immunology | Immunity, Innate - immunology | Microscopy, Confocal | T-Lymphocyte Subsets - pathology | Crohn Disease - pathology | T-Lymphocyte Subsets - metabolism | Crohn Disease - blood | Interferon-gamma - immunology | Interferon-gamma - blood | Receptors, Immunologic - metabolism | Intestinal Mucosa - pathology | Natural Killer T-Cells - immunology | Biological response modifiers | T cells | Analysis | Ulcerative colitis | Medical research | Bowel disease | Original
Journal Article
世界胃肠病学杂志:英文版, ISSN 1007-9327, 2016, Volume 22, Issue 11, pp. 3117 - 3126
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2013, Volume 144, Issue 1, pp. 36 - 49
Psycho-neuro-endocrine-immune modulation through the brain-gut axis likely has a key role in the pathogenesis of inflammatory bowel disease (IBD... 
Gastroenterology and Hepatology | Autonomic Nervous System | Brain-Gut Axis | Corticotropin-Releasing Factor | Stress | SYMPATHETIC-NERVOUS-SYSTEM | CROHNS-DISEASE | ULCERATIVE-COLITIS | CHOLINERGIC ANTIINFLAMMATORY PATHWAY | STRESS-RELATED ALTERATIONS | CORTICOTROPIN-RELEASING-FACTOR | HEART-RATE-VARIABILITY | RECTAL MUCOSAL MEASURES | QUALITY-OF-LIFE | GASTROENTEROLOGY & HEPATOLOGY | PITUITARY-ADRENAL AXIS | Pituitary-Adrenal System - physiopathology | Brain - physiopathology | Humans | Autonomic Nervous System - physiology | Gastrointestinal Tract - physiopathology | Stress, Physiological | Inflammatory Bowel Diseases - physiopathology | Autonomic Nervous System - physiopathology | Brain - physiology | Corticotropin-Releasing Hormone - metabolism | Animals | Anxiety - complications | Depression - complications | Hypothalamo-Hypophyseal System - physiology | Inflammatory Bowel Diseases - etiology | Inflammatory Bowel Diseases - psychology | Gastrointestinal Tract - innervation | Hypothalamo-Hypophyseal System - physiopathology | Inflammatory Bowel Diseases - therapy | Pituitary-Adrenal System - physiology | Stress, Psychological - complications | Corticotropin releasing hormone | Neurosciences | C-reactive protein | Heart beat | Corticosteroids | Gastrointestinal diseases | Alternative medicine | Stress (Psychology) | Mitogens | Tumors | Brain | Hépatology and Gastroenterology | Pituitary-Adrenal System | Gastrointestinal Tract | Inflammatory Bowel Diseases | Neurons and Cognition | Depression | Corticotropin-Releasing Hormone | Life Sciences | Human health and pathology | Immunology | Stress, Psychological | Anxiety | Hypothalamo-Hypophyseal System
Journal Article
世界胃肠病学杂志:英文版, ISSN 1007-9327, 2016, Volume 22, Issue 25, pp. 5655 - 5667
The incidence of inflammatory bowel diseases(IBD)-Crohn’s disease(CD) and ulcerative colitis(UC... 
side | effects | colitis;Drugrelated | and | bowel | Crohn’s | disease;Ulcerative | adverse | reactions;Inflammatory | Inflammatory bowel disease | Crohn's disease | Extraintestinal manifestations | Malnutrition | Drugrelated side effects and adverse reactions | Ulcerative colitis | RHEUMATOID-ARTHRITIS | LICHEN-PLANUS | LOW-DOSE METHOTREXATE | CROHNS-DISEASE | ULCERATIVE-COLITIS | OPPORTUNISTIC INFECTIONS | PYOSTOMATITIS-VEGETANS | Drug-related side effects and adverse reactions | EVIDENCE-BASED CONSENSUS | LYMPHOPROLIFERATIVE DISORDER | GASTROENTEROLOGY & HEPATOLOGY | Humans | Malnutrition - etiology | Oral Ulcer - etiology | Crohn Disease - complications | Cheilitis - physiopathology | Drug-Related Side Effects and Adverse Reactions | Gingivitis - physiopathology | Cheilitis - etiology | Colitis, Ulcerative - drug therapy | Stomatitis, Aphthous - etiology | Mouth Diseases - physiopathology | Colitis, Ulcerative - complications | Stomatitis - physiopathology | Lip Diseases - physiopathology | Oral Ulcer - physiopathology | Malnutrition - physiopathology | Stomatitis - etiology | Mouth Diseases - etiology | Crohn Disease - drug therapy | Stomatitis, Aphthous - physiopathology | Gingivitis - etiology | Immunosuppressive Agents - adverse effects | Crohn Disease - physiopathology | Lip Diseases - etiology | Colitis, Ulcerative - physiopathology | Topic Highlight | Crohn’s disease
Journal Article
Gut, ISSN 0017-5749, 01/2018, Volume 67, Issue 1, pp. 108 - 119
... with a decrease in the genus Roseburia in the gut microbiota of healthy controls without gut complaints. Disease localisation is a major determinant of the IBD-associated gut... 
BACTERIA | CROHNS-DISEASE | VARIANTS | RARE | GENOTYPE | ULCERATIVE-COLITIS | RISK | LOCI | ASSOCIATION | BACTERIAL INTERACTIONS | INFLAMMATORY BOWEL DISEASE | GENETICS | INTESTINAL BACTERIA | CROHNS | GASTROENTEROLOGY & HEPATOLOGY | Severity of Illness Index | Crohn Disease - genetics | Risk Assessment - methods | Genetic Predisposition to Disease | Humans | Middle Aged | Colitis, Ulcerative - genetics | Male | Colitis, Ulcerative - pathology | Dysbiosis - complications | Dysbiosis - microbiology | Gastrointestinal Microbiome - genetics | Case-Control Studies | Feces - microbiology | Colitis, Ulcerative - microbiology | Dysbiosis - genetics | Host-Pathogen Interactions - genetics | Crohn Disease - pathology | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Adult | Crohn Disease - microbiology | Female | Inflammatory Bowel Diseases - microbiology | Inflammatory bowel diseases | Care and treatment | Genetic aspects | Research | Microbiota (Symbiotic organisms) | Genetic variation | Phenotypes | Bacterial infections | Breastfeeding & lactation | rRNA 16S | Pathogenesis | Genomes | Inflammation | Crohn's disease | Family medical history | Crohns disease | Inflammatory bowel disease | Genetic variance | Microbiota | Antibiotics | Intestine | Gastroenterology | Peripheral blood | Genetics | Gene loci | Colon | Endoscopy | Acetic acid | NOD2 protein | Gut microbiota | Healthy controls | Host genetics
Journal Article
Journal of pediatric gastroenterology and nutrition, ISSN 0277-2116, 2013, Volume 58, Issue 6, pp. 1 - 806
Journal Article
Immunology and Allergy Clinics of North America, ISSN 0889-8561, 08/2015, Volume 35, Issue 3, pp. 413 - 437
Journal Article
Nature (London), ISSN 1476-4687, 2019, Volume 567, Issue 7746, pp. 49 - 55
.... Breakdown of the epithelial barrier underpins inflammatory bowel disease (IBD). However, the specific contributions of each epithelial-cell subtype to this process are unknown... 
STEM-CELLS | DENDRITIC CELLS | MUCIN | RNA-SEQ | MULTIDISCIPLINARY SCIENCES | GENES | TISSUES | EXPRESSION | GENOME-WIDE ASSOCIATION | MUCUS LAYERS | RESOURCE | Colon - cytology | Intestinal Mucosa - metabolism | WAP Four-Disulfide Core Domain Protein 2 | Humans | Colitis, Ulcerative - genetics | Male | Stem Cells - cytology | Stem Cells - metabolism | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Transcription, Genetic | Epithelial Cells - cytology | Single-Cell Analysis | Tight Junctions - metabolism | Genetic Predisposition to Disease - genetics | Natriuretic Peptides - metabolism | Colon - pathology | Health | Biomarkers - analysis | Epithelial Cells - classification | Epithelial Cells - pathology | Colitis, Ulcerative - pathology | Intestinal Mucosa - microbiology | Goblet Cells - metabolism | Animals | Proteins - metabolism | Colitis, Ulcerative - microbiology | Ion Channels - metabolism | Epithelial Cells - microbiology | Stem Cells - pathology | Inflammatory Bowel Diseases - microbiology | Colon - microbiology | Mice | Goblet Cells - pathology | Goblet Cells - cytology | Hydrogen-Ion Concentration | Intestinal Mucosa - pathology | Physiological aspects | Epithelial cells | Identification and classification | Gastrointestinal diseases | Inflammatory bowel diseases | Mucosal immunity | Goblet cells | Transcription | Tight junctions | Mucus | Absorptivity | Uroguanylin | Epithelium | Disease control | Immunity | Progenitor cells | Mucous membrane | Inflammatory bowel disease | Intestine | Stem cells | Cells (biology) | Satiety | Breakdown | Colon | Cancer
Journal Article