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Journal of Experimental Medicine, ISSN 0022-1007, 02/2010, Volume 207, Issue 2, pp. 273 - 280
Journal Article
Journal Article
Biochimie, ISSN 0300-9084, 05/2015, Volume 112, pp. 139 - 150
The four known ID proteins (ID1-4, Inhibitor of Differentiation) share a homologous helix loop helix (HLH) domain and act as dominant negative regulators of... 
ID4 | DNA-Protein interaction | Protein–protein interaction | Tumor suppressor gene | Cancer | Protein-protein interaction | DOXORUBICIN-INDUCED APOPTOSIS | UNFAVORABLE PROGNOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR-SUPPRESSOR GENES | POOR DIFFERENTIATION | CELLULAR SENESCENCE | NEGATIVE REGULATOR | BREAST-CANCER | EPIGENETIC INACTIVATION | PROSTATE-CANCER CELLS | EXPRESSION PATTERN | Prostatic Neoplasms - metabolism | Prostatic Neoplasms - pathology | Basic Helix-Loop-Helix Transcription Factors - genetics | Humans | Inhibitor of Differentiation Proteins - genetics | Inhibitor of Differentiation Protein 1 - antagonists & inhibitors | Male | Neoplasm Proteins - antagonists & inhibitors | Inhibitor of Differentiation Proteins - metabolism | Inhibitor of Differentiation Protein 1 - metabolism | Neoplasm Proteins - metabolism | Inhibitor of Differentiation Protein 2 - metabolism | Inhibitor of Differentiation Proteins - antagonists & inhibitors | Prostatic Neoplasms - genetics | Basic Helix-Loop-Helix Transcription Factors - metabolism | Inhibitor of Differentiation Protein 2 - genetics | Cell Line, Tumor | Inhibitor of Differentiation Protein 1 - genetics | Inhibitor of Differentiation Protein 2 - antagonists & inhibitors | Transcription, Genetic | Neoplasm Proteins - genetics | Proteins | DNA | Genetic research | Genetic aspects | Genetic transcription | Prostate cancer | DNA-ligand interactions | protein-protein interaction | cancer | DNA-protein interaction | tumor suppressor gene
Journal Article
Immunity, ISSN 1074-7613, 11/2012, Volume 37, Issue 5, pp. 905 - 916
Langerhans cells (LCs), the dendritic cells (DCs) in skin epidermis, possess an exceptional life cycle and developmental origin. Here we identified two types... 
PROGENITORS | HOMEOSTASIS | AUTOCRINE/PARACRINE TGF-BETA-1 | ID2 | BONE-MARROW | IN-VIVO | MICE | LANGERIN(+) DENDRITIC CELLS | IMMUNOLOGY | IDENTIFICATION | PRECURSORS | Inflammation - pathology | Skin - cytology | Bone Marrow - immunology | Langerhans Cells - pathology | Monocytes - cytology | Skin - metabolism | Monocytes - metabolism | Monocytes - immunology | Cell Differentiation - genetics | Inflammation - metabolism | Ultraviolet Rays | Bone Marrow - metabolism | Inhibitor of Differentiation Protein 2 - genetics | Langerhans Cells - immunology | Monocytes - pathology | Antigens, Surface - metabolism | Transcription Factors - immunology | Skin - immunology | Epidermis - metabolism | Epidermis - pathology | Gene Expression | Antigens, Surface - genetics | Inflammation - immunology | Inhibitor of Differentiation Protein 2 - immunology | Transcription Factors - genetics | Inhibitor of Differentiation Protein 2 - metabolism | Transcription Factors - metabolism | Cell Differentiation - immunology | Animals | Epidermis - immunology | Inflammation - genetics | Mice | Antigens, Surface - immunology | Langerhans Cells - metabolism | Epidermis - cytology | Molecular genetics | Dendritic cells | Analysis | Genes | Skin | Inflammation | Universities and colleges | Gene expression | Bone marrow | Flow cytometry | Software | Transplants & implants | Microscopy
Journal Article
Nature Genetics, ISSN 1061-4036, 2014, Volume 46, Issue 5, pp. 451 - 456
Journal Article
Molecular Cell, ISSN 1097-2765, 01/2017, Volume 65, Issue 2, pp. 247 - 259
Monoubiquitination and deubiquitination of FANCD2:FANCI heterodimer is central to DNA repair in a pathway that is defective in the cancer predisposition... 
core complex | RING E3 | Fanconi anemia | FANCD2 | deubiquitination | FANCB | enzyme mechanism | monoubiquitination | DNA repair | NUCLEAR ACCUMULATION | MONOUBIQUITINATED FANCD2 | TARGETED DISRUPTION | COMPLEX | DNA-REPAIR PATHWAY | CROSS-LINK REPAIR | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTEINS | DAMAGE | COMPLEMENTATION GROUP-B | LIGASE | CELL BIOLOGY | Fanconi Anemia Complementation Group D2 Protein - genetics | Fanconi Anemia - metabolism | Humans | Multiprotein Complexes | Protein Multimerization | Substrate Specificity | Fanconi Anemia Complementation Group A Protein - metabolism | DNA-Binding Proteins - metabolism | Fanconi Anemia Complementation Group Proteins - metabolism | Ubiquitination | Transfection | Time Factors | Fanconi Anemia - genetics | Ubiquitin-Specific Proteases - metabolism | Recombinant Proteins - metabolism | Cell Line | Fanconi Anemia Complementation Group Proteins - genetics | Nuclear Proteins - metabolism | Fanconi Anemia Complementation Group G Protein - metabolism | DNA - metabolism | Inhibitor of Differentiation Protein 2 - metabolism | DNA - genetics | Fanconi Anemia Complementation Group C Protein - metabolism | Fanconi Anemia Complementation Group D2 Protein - metabolism | Protein Binding | Fanconi Anemia Complementation Group L Protein - metabolism | Fanconi Anemia Complementation Group E Protein - metabolism | Ubiquitin | Chemotherapy | Ligases | Genomics | Research institutes | Cancer | Fanconi's anemia | Proteins | Medical research | Molecular genetics | Medicine, Experimental
Journal Article
Nature, ISSN 0028-0836, 2016, Volume 537, Issue 7620, pp. 412 - 416
Journal Article
Nature Immunology, ISSN 1529-2908, 2009, Volume 10, Issue 10, pp. 1118 - 1124
Journal Article