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The Journal of immunology (1950), ISSN 0022-1767, 01/2014, Volume 192, Issue 2, pp. 623 - 629
Chronic inflammation is known to promote metabolic dysregulation in obesity and type 2 diabetes. Although the precise origin of the unchecked inflammatory... 
IMMUNE-SYSTEM | INFLAMMATION | DISEASE | RESISTANCE | FAT | AKT | IMMUNOLOGY | DIET-INDUCED OBESITY | KEY PLAYERS | ADIPOSE-TISSUE | CUTTING EDGE | Tumor Necrosis Factor-alpha - metabolism | Antigens, CD - immunology | T-Lymphocytes, Regulatory - metabolism | Epithelial Cells - metabolism | TOR Serine-Threonine Kinases - metabolism | Obesity - immunology | Insulin - immunology | Interferon-gamma - metabolism | Antigens, CD - metabolism | T-Lymphocytes, Regulatory - immunology | Epithelium - immunology | Signal Transduction - immunology | Inflammation - metabolism | Tumor Necrosis Factor-alpha - immunology | Interleukin-10 - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Receptor, Insulin - immunology | Macrophages - immunology | Hyperinsulinism - metabolism | Intra-Abdominal Fat - immunology | Transforming Growth Factor beta - immunology | Epithelium - metabolism | Mice, Inbred C57BL | Apyrase - metabolism | Cells, Cultured | Apyrase - immunology | CTLA-4 Antigen - metabolism | Inflammation - immunology | CTLA-4 Antigen - immunology | Intra-Abdominal Fat - metabolism | Obesity - metabolism | TOR Serine-Threonine Kinases - immunology | Insulin - metabolism | Macrophages - metabolism | Animals | Hyperinsulinism - immunology | Proto-Oncogene Proteins c-akt - immunology | Epithelial Cells - immunology | Interferon-gamma - immunology | Interleukin-10 - antagonists & inhibitors | Receptor, Insulin - metabolism | Mice | Interleukin-10 - immunology | Transforming Growth Factor beta - metabolism
Journal Article
Journal Article
The journal of clinical endocrinology and metabolism, ISSN 1945-7197, 2017, Volume 102, Issue 5, pp. 1468 - 1477
Context: Increasing evidences suggest a correlation between gut and type 1 diabetes (T1D). Objective: The objective of this study is to evaluate the gut... 
INCREASED INTESTINAL PERMEABILITY | HEALTHY-CHILDREN | DIFFERS | FECAL MICROBIOTA | DISEASE | ENDOCRINOLOGY & METABOLISM | CELL AUTOIMMUNITY | GUT MICROBIOTA | MELLITUS | ONSET | Receptors, CCR2 - genetics | Serum Amyloid P-Component - immunology | Receptors, CCR2 - immunology | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Transcriptome | Child, Preschool | Male | Monocyte Chemoattractant Proteins - immunology | Vascular Endothelial Growth Factor A - genetics | Antigens, CD - genetics | Interleukin-4 Receptor alpha Subunit - genetics | Young Adult | Celiac Disease - microbiology | Cyclooxygenase 2 - genetics | Intestinal Mucosa - immunology | Diabetes Mellitus, Type 1 - microbiology | Tumor Necrosis Factor-alpha - immunology | Child | Real-Time Polymerase Chain Reaction | Duodenum - immunology | Vascular Endothelial Growth Factor A - immunology | C-Reactive Protein - genetics | Intestinal Mucosa - microbiology | Reverse Transcriptase Polymerase Chain Reaction | Monocyte Chemoattractant Proteins - genetics | Chemokine CCL22 - genetics | Chemokine CCL19 - genetics | Adolescent | RNA, Ribosomal, 16S - genetics | C-Reactive Protein - immunology | Antigens, CD - immunology | Infant | Case-Control Studies | Duodenum - microbiology | Chemokine CCL22 - immunology | Celiac Disease - immunology | Adult | Female | Diabetes Mellitus, Type 1 - immunology | Chemokine CCL19 - immunology | Cyclooxygenase 2 - immunology | Antigens, Differentiation, Myelomonocytic - immunology | Diabetes Mellitus, Type 1 - genetics | Gastrointestinal Microbiome - genetics | Interleukin-4 Receptor alpha Subunit - immunology | Antigens, Differentiation, Myelomonocytic - genetics | Serum Amyloid P-Component - genetics | Aged | Immunohistochemistry | CC chemokine receptors | rRNA 16S | CCL19 protein | Pathogenesis | Mucosa | Microbiomes | Diabetes mellitus (insulin dependent) | Macrophages | Small intestine | Gene sequencing | CCL22 protein | Microbiota | CCR2 protein | Intestine | Interleukin 4 receptors | Digestive tract | Duodenum | Digestive system | Abnormalities | Diabetes mellitus | Inflammation | Tumor necrosis factor-α | Gene expression | Ribonucleic acid--RNA | Disease control | Patients | Celiac disease | Biopsy | Infiltration | Diabetes | Cyclooxygenase-2 | Autoimmune diseases | Monocyte chemoattractant protein 1
Journal Article
Journal Article
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 05/2012, Volume 109, Issue 19, pp. E1143 - E1152
Obesity triggers a low-grade systemic inflammation, which plays an important role in the development of obesity-associated metabolic diseases. In searching for... 
Cluster of differentiation 1d | Alpha-galactosylceramide | Glycolipid-reactive T cells | Obesity-induced inflammation | glycolipid-reactive T cells | NKT CELLS | INKT CELLS | GLUCOSE-INTOLERANCE | MULTIDISCIPLINARY SCIENCES | GLYCOLIPID ANTIGENS | INNATE IMMUNE-SYSTEM | FATTY-ACIDS | NONALCOHOLIC STEATOHEPATITIS | DEFICIENT MICE | DANGER SIGNALS | alpha-galactosylceramide | cluster of differentiation 1d | obesity-induced inflammation | ADIPOSE-TISSUE | Adipose Tissue, White - immunology | Antigens, CD1d - immunology | Obesity - immunology | Adipose Tissue, White - metabolism | Lipids - immunology | Male | Dietary Fats - immunology | Galactosylceramides - administration & dosage | Obesity - genetics | Lymphocyte Activation - immunology | Flow Cytometry | Natural Killer T-Cells - metabolism | Dietary Fats - administration & dosage | Inflammation Mediators - metabolism | Antigens, CD1d - genetics | CD8-Positive T-Lymphocytes - metabolism | Female | Macrophages - immunology | Cytokines - immunology | Fatty Liver - genetics | Inflammation Mediators - immunology | Adipose Tissue, White - pathology | Cytokines - metabolism | Mice, Inbred C57BL | Cells, Cultured | Inflammation - immunology | Lipids - administration & dosage | Galactosylceramides - physiology | Antigens, CD1d - metabolism | Mice, Knockout | Macrophages - metabolism | Animals | Galactosylceramides - immunology | Fatty Liver - immunology | Insulin Resistance - genetics | Inflammation - genetics | Insulin Resistance - immunology | Mice | CD8-Positive T-Lymphocytes - immunology | Natural Killer T-Cells - immunology | Biological Sciences | PNAS Plus
Journal Article
by Mouraux, Stéphane and Bernasconi, Eric and Pattaroni, Céline and Koutsokera, Angela and Aubert, John-David and Claustre, Johanna and Pison, Christophe and Royer, Pierre-Joseph and Magnan, Antoine and Kessler, Romain and Benden, Christian and Soccal, Paola M and Marsland, Benjamin J and Nicod, Laurent P and Jougon, J and Velly, J.-F and Rozé, H and Blanchard, E and Dromer, C and Antoine, M and Cappello, M and Ruiz, M and Sokolow, Y and Vanden Eynden, F and Van Nooten, G and Barvais, L and Berré, J and Brimioulle, S and De Backer, D and Créteur, J and Engelman, E and Huybrechts, I and Ickx, B and Preiser, T.J.C and Tuna, T and Van Obberghe, L and Vancutsem, N and Vincent, J.-L and De Vuyst, P and Etienne, I and Féry, F and Jacobs, F and Knoop, C and Vachiéry, J.L and Van den Borne, P and Wellemans, I and Amand, G and Collignon, L and Giroux, M and Angelescu, D and Chavanon, O and Hacini, R and Pirvu, A and Porcu, P and Albaladejo, P and Allègre, C and Bataillard, A and Bedague, D and Briot, E and Casez-Brasseur, M and Colas, D and Dessertaine, G and Durand, M and Francony, G and Hebrard, A and Marino, M.R and Oummahan, B and Protar, D and Rehm, D and Robin, S and Rossi-Blancher, M and Augier, C and Bedouch, P and Boignard, A and Bouvaist, H and Briault, A and Camara, B and Claustre, J and Chanoine, S and Dubuc, M and Quétant, S and Maurizi, J and Pavèse, P and Pison, C and Saint-Raymond, C and Wion, N and Chérion, C and Grima, R and Jegaden, O and Maury, J.-M and Tronc, F and Flamens, C and Paulus, S and Mornex, J.-F and Philit, F and Senechal, A and Glérant, J.-C and Turquier, S and Gamondes, D and Chalabresse, L and ... and SysCLAD Consortium
Journal of allergy and clinical immunology, ISSN 0091-6749, 02/2018, Volume 141, Issue 2, pp. 718 - 729.e7
Homeostatic turnover of the extracellular matrix conditions the structure and function of the healthy lung. In lung transplantation, long-term management... 
microbiota | fibroblasts | macrophages | Airway remodeling | matrix | ALLOGRAFT DYSFUNCTION | PDGF-D | IMMUNOLOGY | ALLERGY | ACUTE EXACERBATIONS | INFLAMMATION | SERUM-LEVELS | DISEASE | IDIOPATHIC PULMONARY-FIBROSIS | EXPRESSION | COPD | Lung - microbiology | Lung - pathology | Microbiota - immunology | Macrophages - pathology | Humans | Middle Aged | Extracellular Matrix - immunology | Male | Fibroblasts - pathology | Airway Remodeling - immunology | Signal Transduction - immunology | Bacteria - immunology | Lung Transplantation | Bacteria - classification | Adult | Female | Fibroblasts - immunology | Extracellular Matrix - pathology | Lung - immunology | Macrophages - immunology | Chitinase | Cell culture | rRNA 16S | Platelet-derived growth factor | Transplants & implants | Ontology | Crosstalk | Lung transplantation | Parenchyma | Transplantation | Remodeling | Macrophages | Gene sequencing | Streptococcus infections | Respiratory tract | Cell activation | Microbiota | Xenografts | Fibroblasts | Bacteria | Extracellular matrix | Metalloproteinase | Alveoli | Growth factors | Deoxyribonucleic acid--DNA | Thrombospondin | Biodegradation | Heparan sulfate | Kidneys | Bronchus | Principal components analysis | Inflammation | Gene expression | Ribonucleic acid--RNA | Gene amplification | Pulmonary fibrosis | Collagen | Biomarkers | Structure-function relationships | Life Sciences | IGF, Insulin-like growth factor | KO, KEGG ortholog | GO, Gene Ontology | PDGFD, Platelet-derived growth factor D | SPP1, Secreted phosphoprotein 1 | CFU, Colony-forming unit | COPD, Chronic obstructive pulmonary disease | rRNA, Ribosomal RNA | CLAD, Chronic lung allograft dysfunction | KEGG, Kyoto Encyclopedia of Genes and Genomes | SysCLAD, System prediction of Chronic Lung Allograft Dysfunction | BAL, Bronchoalveolar lavage | CHI3L1, Chitinase 3-like 1 | THP-DM, THP1-derived macrophages | MMP, Matrix metallopeptidase | IQR, Interquartile range | THBS1, Thrombospondin 1
Journal Article
PloS one, ISSN 1932-6203, 01/2014, Volume 9, Issue 1, p. e86289
Infections with helminth parasites prevent/attenuate auto-inflammatory disease. Here we show that molecules secreted by a helminth parasite could prevent Type... 
B-CELLS | AUTOIMMUNE | TGF-BETA | SCHISTOSOMA-MANSONI | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | INFECTION | ANTIGENS | MICE | TRICHURIS-SUIS THERAPY | IDENTIFICATION | T-Lymphocytes, Regulatory - immunology | Helminths - immunology | Autoimmunity - immunology | Female | Diabetes Mellitus, Type 1 - immunology | T-Lymphocytes, Regulatory - parasitology | Transforming Growth Factor beta - immunology | Diabetes Mellitus, Type 1 - parasitology | Lectins - immunology | Macrophages, Peritoneal - immunology | Lymph Nodes - immunology | beta-N-Acetylhexosaminidases - immunology | Pancreas - immunology | B7-H1 Antigen - immunology | Macrophages, Peritoneal - parasitology | Diabetes Mellitus, Experimental - immunology | Diabetes Mellitus, Experimental - parasitology | Cell Differentiation - immunology | Fasciola hepatica - immunology | Autoantibodies - immunology | Animals | Lymph Nodes - parasitology | Interferon-gamma - immunology | Mice, Inbred NOD | Mice | Autoimmunity | Genetic markers | Type 1 diabetes | Environmental law | Immunoglobulin G | B cells | T cells | Health aspects | Insulitis | Disease | Infections | Lymphocytes T | Parasites | Macrophages | Inflammatory diseases | Lymph nodes | Proteins | Prevention | Lymphocytes | Splenocytes | Rodents | Pancreas | Antigens | Autoantibodies | Cytokines | Immunomodulation | Diabetes mellitus | Mammals | Gene expression | Insulin | Studies | γ-Interferon | PD-L1 protein | Interferon | Diabetes | Autoimmune diseases | Peritoneum
Journal Article
Blood, ISSN 1528-0020, 2009, Volume 114, Issue 15, pp. 3244 - 3254
Journal Article