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PloS one, ISSN 1932-6203, 03/2012, Volume 7, Issue 3, p. e33814
.... Although evidence points to consider exposure to BPA as a risk factor for insulin resistance, its actions on whole body metabolism and on insulin-sensitive tissues are still unclear... 
LEPTIN | SKELETAL-MUSCLE | CELLS | ENDOCRINE DISRUPTORS | MULTIDISCIPLINARY SCIENCES | INSULIN-SECRETION | SENSITIVITY | MECHANISMS | EXPRESSION | ESTROGEN-RECEPTOR | EXPOSURE | Phosphorylation | Benzhydryl Compounds | Phenols - toxicity | Liver - metabolism | Male | Muscle, Skeletal - metabolism | Insulin Receptor Substrate Proteins - metabolism | Insulin - metabolism | Animals | Liver - drug effects | Signal Transduction - drug effects | Estrogens, Non-Steroidal - toxicity | Injections, Subcutaneous | Muscle, Skeletal - drug effects | Glucose - metabolism | Receptor, Insulin - metabolism | Mice | Proto-Oncogene Proteins c-akt - metabolism | Energy Metabolism - drug effects | Mitogen-Activated Protein Kinases - metabolism | Tyrosine | Type 2 diabetes | Bisphenol-A | Liver | Muscles | Glucose | Insulin | Risk factors | Dextrose | Epoxy resins | Analysis | Polycarbonates | Physiological aspects | Insulin resistance | Mitogens | Protein kinases | Endocrine disruptors | Energy metabolism | Homeostasis | AKT protein | Signal transduction | Rodents | Animal tissues | Polymers | Polycarbonate | Locomotor activity | Body temperature | Abnormalities | Diabetes mellitus | MAP kinase | Metabolism | Resistance factors | Skeletal muscle | Energy balance | Bisphenol A | Signaling | Insulin receptor substrate 1 | Protein kinase | Food intake | Phenols | Calorimetry
Journal Article
Cell Cycle, ISSN 1538-4101, 06/2011, Volume 10, Issue 11, pp. 1750 - 1756
Increasing evidence supports a connection between cancer and metabolism and emphasizes the need to understand how tumors respond to the metabolic microenvironment and how tumor cell metabolism is regulated... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | IRS proteins | Metformin | Insulin receptor | Metabolism | IGF-1 receptor | TYROSINE PHOSPHORYLATION | KINASE | metformin | AKT | FACTOR-I | CELL-SURVIVAL | INSULIN-RECEPTOR-SUBSTRATE-1 IRS-1 | CELL BIOLOGY | BREAST-CANCER | GLUCOSE-HOMEOSTASIS | GROWTH | metabolism | cancer | insulin receptor | GLUT1 EXPRESSION | Neoplasms - metabolism | Receptor, Insulin - metabolism | Neoplasms - etiology | Insulin Receptor Substrate Proteins - metabolism | Humans | Extra View
Journal Article
Journal Article
Journal Article
Journal Article
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2003, Volume 278, Issue 46, pp. 45777 - 45784
...). IL-6, in contrast to TNF-alpha, did not increase pS-307 of insulin-receptor substrate (IRS... 
IRS 1 | PROTEIN | LIPOLYSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | SENSITIVITY | RECEPTOR | GLUCOSE-TRANSPORT | TNF-ALPHA | RELEASE | EXPRESSION | ADIPOSE-TISSUE | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Phosphorylation | Humans | Middle Aged | Repressor Proteins | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Suppressor of Cytokine Signaling Proteins | Dose-Response Relationship, Drug | Adipose Tissue - metabolism | Mitogen-Activated Protein Kinase Kinases - metabolism | Muscle Proteins | MAP Kinase Kinase 4 | Biological Transport | Time Factors | Insulin Receptor Substrate Proteins | JNK Mitogen-Activated Protein Kinases | Adult | Transcription, Genetic | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Monosaccharide Transport Proteins - metabolism | Tyrosine - chemistry | RNA - metabolism | Cytokines - metabolism | Glucose Transporter Type 4 | Insulin Resistance | 3T3-L1 Cells | Reverse Transcriptase Polymerase Chain Reaction | Suppressor of Cytokine Signaling 3 Protein | Blotting, Northern | Tyrosine - metabolism | Animals | Proteins - metabolism | Adipocytes - metabolism | Glucose - metabolism | Mice | Transcription Factors | insulin receptors | pS-307 protein | peroxisome proliferator-activated receptors | IRS-1 protein | GLUT-4 protein | JNK protein | insulin-receptor substrate 2 | insulin resistance | glucose transporter GLUT4 | insulin-receptor substrate 1 protein
Journal Article
Nature Cell Biology, ISSN 1465-7392, 06/2005, Volume 7, Issue 6, pp. 601 - 611
.... Insulin receptor substrate proteins regulate a necdin-E2F4 interaction... 
MATRIX METALLOPROTEINASES | ACTIVATION | INHIBITION | PROTEIN | INTERACTS | RESISTANCE | ADIPOCYTE DIFFERENTIATION | ADIPOGENESIS | PRADER-WILLI-SYNDROME | ADIPOSE-TISSUE | CELL BIOLOGY | Oligonucleotide Array Sequence Analysis | Gene Expression Regulation, Developmental - genetics | Adipocytes - cytology | Gene Expression Profiling | Stem Cells - cytology | Wnt Proteins | PPAR gamma - metabolism | Stem Cells - metabolism | DNA-Binding Proteins - metabolism | Cell Differentiation - genetics | Intercellular Signaling Peptides and Proteins - metabolism | E2F4 Transcription Factor | Membrane Proteins - metabolism | Nuclear Proteins - genetics | Repressor Proteins - metabolism | PPAR gamma - genetics | Membrane Proteins - genetics | Cells, Cultured | Intercellular Signaling Peptides and Proteins - genetics | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Signal Transduction - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Cyclic AMP Response Element-Binding Protein - genetics | Adipose Tissue, Brown - cytology | Animals | Adipocytes - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Adipose Tissue, Brown - growth & development | Receptor, Insulin - metabolism | Adipose Tissue, Brown - metabolism | Mice | Physiological aspects | Receptors | Research | Cell differentiation | Gene expression | Insulin
Journal Article
Nature cell biology, ISSN 1476-4679, 2004, Volume 6, Issue 4, pp. 351 - 357
Journal Article