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by Shungin, Dmitry and Winkler, Thomas W and Croteau-Chonka, Damien C and Ferreira, Teresa and Locke, Adam E and Mägi, Reedik and Strawbridge, Rona J and Pers, Tune H and Fischer, Krista and Justice, Anne E and Workalemahu, Tsegaselassie and Wu, Joseph M. W and Buchkovich, Martin L and Heard-Costa, Nancy L and Roman, Tamara S and Drong, Alexander W and Song, Ci and Gustafsson, Stefan and Day, Felix R and Esko, Tonu and Fall, Tove and Kutalik, Zoltán and Luan, Jian’an and Randall, Joshua C and Scherag, André and Vedantam, Sailaja and Wood, Andrew R and Chen, Jin and Fehrmann, Rudolf and Karjalainen, Juha and Kahali, Bratati and Liu, Ching-Ti and Schmidt, Ellen M and Absher, Devin and Amin, Najaf and Anderson, Denise and Beekman, Marian and Bragg-Gresham, Jennifer L and Buyske, Steven and Demirkan, Ayse and Ehret, Georg B and Feitosa, Mary F and Goel, Anuj and Jackson, Anne U and Johnson, Toby and Kleber, Marcus E and Kristiansson, Kati and Mangino, Massimo and Mateo Leach, Irene and Medina-Gomez, Carolina and Palmer, Cameron D and Pasko, Dorota and Pechlivanis, Sonali and Peters, Marjolein J and Prokopenko, Inga and Stančáková, Alena and Ju Sung, Yun and Tanaka, Toshiko and Teumer, Alexander and Van Vliet-Ostaptchouk, Jana V and Yengo, Loïc and Zhang, Weihua and Albrecht, Eva and Ärnlöv, Johan and Arscott, Gillian M and Bandinelli, Stefania and Barrett, Amy and Bellis, Claire and Bennett, Amanda J and Berne, Christian and Blüher, Matthias and Böhringer, Stefan and Bonnet, Fabrice and Böttcher, Yvonne and Bruinenberg, Marcel and Carba, Delia B and Caspersen, Ida H and Clarke, Robert and Warwick Daw, E and Deelen, Joris and Deelman, Ewa and Delgado, Graciela and Doney, Alex S. F and Eklund, Niina and Erdos, Michael R and Estrada, Karol and Eury, Elodie and Friedrich, Nele and Garcia, Melissa E and Giedraitis, Vilmantas and Gigante, Bruna and Go, Alan S and Golay, Alain and Grallert, Harald and Grammer, Tanja B and Gräßler, Jürgen and Grewal, Jagvir and Groves, Christopher J and Haller, Toomas and Hallmans, Goran and ... and The PAGE Consortium and The International Endogene Consortium and The MAGIC Investigators and The ReproGen Consortium and The CKDGen Consortium and The ADIPOGen Consortium and The ICBP and The CARDIOGRAMplusC4D Consortium and The GENIE Consortium and The GLGC and The GEFOS Consortium and The MuTHER Consortium and The LifeLines Cohort Study and PAGE Consortium and LifeLines Cohort Study and CARDIOGRAMplusC4D Consortium and MuTHER Consortium and ICBP and ADIPOGEN Consortium and CKDGen Consortium and GEFOS Consortium and Int Endogene Consortium and GENIE Consortium and MAGIC Investigators and ReproGen Consortium and GLGC and ADIPOGen Consortium and International Endogene Consortium and Göteborgs universitet and Gothenburg University and Institutionen för medicin, avdelningen för invärtesmedicin och klinisk nutrition and Sahlgrenska Academy and Centre for Bone and Arthritis Research and Sahlgrenska akademin and Institute of Medicine, Department of Internal Medicine and Clinical Nutrition
Nature (London), ISSN 1476-4687, 2015, Volume 518, Issue 7538, pp. 187 - 196
.... To increase our understanding of the genetic basis of body fat distribution and its molecular links to cardiometabolic traits, here we conduct genome-wide association meta-analyses of traits related... 
ABDOMINAL ADIPOSITY | METAANALYSIS | VARIANTS | MULTIDISCIPLINARY SCIENCES | COMMON SNPS | GLYCEMIC TRAITS | RISK | FALSE DISCOVERY | GENOME-WIDE ASSOCIATION | ADIPOGENIC DIFFERENTIATION | SEXUAL-DIMORPHISM | Body Mass Index | Genome-Wide Association Study | Age Factors | Neovascularization, Physiologic - genetics | Epigenesis, Genetic | Humans | Male | Continental Population Groups - genetics | Sex Characteristics | Obesity - genetics | Europe - ethnology | Genome, Human - genetics | Adipose Tissue - metabolism | Insulin - metabolism | Models, Biological | Adipocytes - metabolism | Insulin Resistance - genetics | Polymorphism, Single Nucleotide - genetics | Female | Body Fat Distribution | Transcription, Genetic - genetics | Adipogenesis - genetics | Waist-Hip Ratio | Quantitative Trait Loci - genetics | Adipose tissues | Quantitative trait loci | Genetic research | Genetic aspects | Research | Metabolism | Health aspects | Studies | Body mass index | Genealogy | Body fat | Insulin resistance | Genetics | Genomes | Abdomen | Meta-analysis | Life Sciences | Adipocytes/metabolism Adipogenesis/genetics Adipose Tissue/metabolism Age Factors Body Fat Distribution Body Mass Index Continental Population Groups/genetics Epigenesis, Genetic Europe/ethnology Female Genome, Human/genetics Genome-Wide Association Study Humans Insulin/metabolism Insulin Resistance/genetics Male Models, Biological Neovascularization, Physiologic/genetics Obesity/genetics Polymorphism, Single Nucleotide/genetics Quantitative Trait Loci/genetics Sex Characteristics Transcription, Genetic/genetics Waist-Hip Ratio | Folkhälsovetenskap, global hälsa, socialmedicin och epidemiologi | Endokrinologi och diabetes | Public Health, Global Health, Social Medicine and Epidemiology | Endocrinology and Diabetes
Journal Article
Journal Article
Nature genetics, ISSN 1061-4036, 2017, Volume 49, Issue 9, pp. 1392 - 1397
.... Phenome-wide association scanning showed that CCDC92 likely affects coronary artery disease through insulin resistance pathways, whereas experimental analysis suggests that ARHGEF26 influences... 
PROVIDES INSIGHTS | METAANALYSIS | GENETICS & HEREDITY | NUCLEOTIDE EXCHANGE FACTOR | SUSCEPTIBILITY LOCI | ARCHITECTURE | RISK | TRAITS | VARIANT | GENOME-WIDE ASSOCIATION | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Leukocyte Rolling - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Middle Aged | Cells, Cultured | Genotype | Logistic Models | Male | United Kingdom | Rho Guanine Nucleotide Exchange Factors - genetics | Health Information Systems | Carrier Proteins - genetics | Phenotype | Coronary Artery Disease - genetics | Coronary Artery Disease - pathology | HEK293 Cells | Insulin Resistance - genetics | Adult | Female | Aged | Polymorphism, Single Nucleotide | Transendothelial and Transepithelial Migration - genetics | Complications and side effects | Development and progression | Insulin resistance | Genetic aspects | Disease susceptibility | Research | Coronary heart disease | Repositories | Leukocyte migration | Nucleotide sequence | Coronary artery | Genomes | Leukocytes | Insulin | Coronary artery disease | Disease control | Loci | Disease resistance | Pathways | Genetic analysis | DNA methylation | Gene loci | Mutation | Heart diseases | Deoxyribonucleic acid--DNA | coronary artery disease | population genetics | gene-expression | genome-wide association studies
Journal Article
Diabetes care, ISSN 0149-5992, 2009, Volume 32, Issue 11, pp. S362 - 367
Journal Article
The American Journal of Human Genetics, ISSN 0002-9297, 07/2013, Volume 93, Issue 1, pp. 141 - 149
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2009, Volume 106, Issue 34, pp. 14564 - 14569
A conserved insulin-like pathway modulates both aging and pathogen resistance in Caenorhabditis elegans... 
Salmonella | Pathogens | Phagocytes | Epithelial cells | Salmonella infections | Genes | Nematodes | Bacteria | Infections | Genetic mutation | BACTERIAL PATHOGENS | LIFE-SPAN | IMMUNITY | LEGIONELLA-PNEUMOPHILA | CAENORHABDITIS-ELEGANS | CROHNS-DISEASE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | C-ELEGANS | DICTYOSTELIUM-DISCOIDEUM | CELL-DEATH | Caenorhabditis elegans - microbiology | Dictyostelium - microbiology | Immunity, Innate - genetics | Autophagy - physiology | Green Fluorescent Proteins - genetics | Intestinal Mucosa - cytology | RNA Interference | Receptor, Insulin - genetics | Phagosomes - ultrastructure | Autophagy - genetics | Vesicular Transport Proteins | Green Fluorescent Proteins - metabolism | Microscopy, Electron, Transmission | Salmonella typhimurium - physiology | Transcription Factors - physiology | Intestinal Mucosa - ultrastructure | Salmonella typhimurium - genetics | Animals, Genetically Modified | Caenorhabditis elegans - genetics | Epithelial Cells - ultrastructure | Intestinal Mucosa - microbiology | Longevity - genetics | Receptor, Insulin - physiology | Transcription Factors - genetics | Host-Pathogen Interactions | Caenorhabditis elegans - ultrastructure | Animals | Epithelial Cells - microbiology | Survival Analysis | Dictyostelium - genetics | Caenorhabditis elegans Proteins - physiology | Mutation | Caenorhabditis elegans Proteins - genetics | Forkhead Transcription Factors | Microscopy, Fluorescence | Prevention | Pathogenic microorganisms | Genetic aspects | Salmonellosis | Research | Bacterial genetics | Salmonella typhimurium | Infection | Overexpression | Life span | Aging | Lysosomes | Forkhead protein | Protein-tyrosine kinase receptors | Replication | Insulin | Phagocytosis | Biological Sciences
Journal Article
PLoS Genetics, ISSN 1553-7390, 10/2016, Volume 12, Issue 10, p. e1006396
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 2013, Volume 62, Issue 6, pp. 1876 - 1887
Journal Article